ライフサイエンスコーパス: spike-timing-dependent plasticity

1 odes emerge in this microcircuit motif under spike timing-dependent plasticity.

2 idirectional modification is consistent with spike timing-dependent plasticity.

3 firing patterns associated with induction of spike timing-dependent plasticity.

4 i-Hebbian timing rules that reflect in vitro spike timing-dependent plasticity.

5 a manner consistent with the predictions of spike timing-dependent plasticity.

6 tex excitability following the principles of spike-timing-dependent plasticity.

7 PAS is based on the principles of spike-timing-dependent plasticity.

8 similarity with experimental results seen in spike-timing-dependent plasticity.

9 rvous system based on the Hebbian concept of spike-timing-dependent plasticity.

10 consider to be a problem with Hebbian (i.e., spike-timing-dependent) plasticity.

11 onal synchronization has also been linked to spike timing-dependent plasticity, a cellular mechanism

12 nner not predicted by the well-known rule of spike timing-dependent plasticity and requiring activati

13 We review three of them-synaptic scaling, spike-timing dependent plasticity and synaptic redistrib

14 e reconciles apparent contradictions between spike-timing-dependent plasticity and previous work at C

15 show that in visual cortex the rules of this spike-timing-dependent plasticity are not rigid, but sha

16 luding long-term potentiation/depression and spike-timing-dependent plasticity, are mimicked.

17 indings implicate alterations in DCN bimodal spike timing-dependent plasticity as underlying mechanis

18 idence detection, grouping by synchrony, and spike-timing-dependent plasticity, as well as for the pr

19 nput can bidirectionally control the sign of spike timing-dependent plasticity at local synapses in r

20 ity in remote neurons and subsequently allow Spike-Timing-Dependent Plasticity based learning to occu

21 g as input, the model can be trained using a spike-timing-dependent plasticity-based learning rule to

22 In addition, a model with spike timing-dependent plasticity can overcome a natural

23 These results support the idea that spike timing-dependent plasticity can rapidly adjust the

24 on in vivo and in vitro, the degree to which spike timing-dependent plasticity can shape receptive fi

25 A cellular learning rule known as spike-timing-dependent plasticity can form, reshape and

26 integrated, open-source software (StdpC, for spike timing-dependent plasticity clamp).

27 ed in vitro, it is less clear to what degree spike timing-dependent plasticity contributes to shaping

28 his model gives rise to a large diversity of spike timing-dependent plasticity curves, most of which

29 ic changes induced at individual synapses by spike timing-dependent plasticity do not strictly follow

30 nized structure in a recurrent network, with spike timing-dependent plasticity driven by spontaneous

31 Hence, we propose that spike timing-dependent plasticity enables this microcirc

32 ogous to those governing the associative and spike timing-dependent plasticity exhibited by individua

33 Although spike timing-dependent plasticity has been well-characte

34 Spike-timing dependent plasticity has been associated wi

35 Cellular mechanisms underlying spike-timing-dependent plasticity have been studied exte

36 The plasticity rule is a form of spike timing dependent plasticity in which a presynaptic

37 participate in oscillatory phase coding and spike timing-dependent plasticity in coordination with o

38 at coincides with reinforcement, and Hebbian spike timing-dependent plasticity in Kenyon cells alone

39 We show that spike timing-dependent plasticity in L2/L3 pyramids from

40 ationship between binge alcohol drinking and spike timing-dependent plasticity in nucleus accumbens (

41 tine during adolescence alters the rules for spike timing-dependent plasticity in prefrontal networks

42 al pathways, our results are consistent with spike timing-dependent plasticity in reticulospinal circ

43 that enhances movement therapy by directing spike timing-dependent plasticity in spared motor pathwa

44 inal pathways, at timings predicted to cause spike timing-dependent plasticity in the brainstem.

45 The derived learning rule is consistent with spike-timing dependent plasticity in that a presynaptic

46 Here we studied metaplasticity affecting spike-timing-dependent plasticity, in which the polarity

47 Spike timing-dependent plasticity is accompanied by chan

48 meter of a gate bias voltage pulse, synaptic spike-timing-dependent plasticity learning behaviour is

49 ection, long-term potentiation/depression, a spike-timing-dependent plasticity learning rule, paired-

50 Here, we present a new class of spike-timing-dependent plasticity learning rules with lo

51 Correlated cholinergic input induces spike timing-dependent plasticity-like hippocampal synap

52 ch as temporally asymmetric LTP induction or spike timing-dependent plasticity) may be at work in bot

53 ation when synaptic strengths evolve through spike-timing dependent plasticity mechanisms.

54 We show that inhibitory spike-timing dependent plasticity (modelling non-associa

55 If memories are stored by spike timing-dependent plasticity, neuronal interactions

56 wo photon-guided focal stimulation, we found spike timing-dependent plasticity of proximal excitatory

57 se findings are the first demonstration that spike timing-dependent plasticity of residual corticospi

58 In addition, we determined that PSD and spike-timing-dependent plasticity operating in parallel

59 ponent analysis and bottom-up models such as spike-timing dependent plasticity or the Bienenstock-Coo

60 Furthermore, a spike timing-dependent plasticity protocol, in which two

61 A recent study shows that a spike-timing-dependent plasticity protocol can potentiat

62 cesses, including reinforcement learning and spike-timing-dependent plasticity, requires the use of a

63 can sculpt recurrent activity according to a spike timing-dependent plasticity rule, and that impairi

64 ronal cells, consistent with a bidirectional spike-timing-dependent plasticity rule previously derive

65 We describe a method to derive spike-timing-dependent plasticity rules for self-repair,

66 We are addressing here the question how spike timing-dependent plasticity shapes the computation

67 central to the acquisition of novel actions.Spike timing dependent plasticity (STDP) has been studie

68 kinetics, change the receptor's response to spike timing dependent plasticity (STDP) protocols, and

69 aptic weights are updated via brain-inspired spike timing dependent plasticity (STDP).

70 Spike timing-dependent plasticity (STDP) and other conve

71 Spike timing-dependent plasticity (STDP) as a Hebbian sy

72 Recent findings of spike timing-dependent plasticity (STDP) have stimulated

73 ge alcohol drinking to assess its effects on spike timing-dependent plasticity (STDP) in medium spiny

74 Many cortical synapses exhibit spike timing-dependent plasticity (STDP) in which the pr

75 In model studies, such spike timing-dependent plasticity (STDP) introduces the

76 Spike timing-dependent plasticity (STDP) is under neurom

77 cate that the signaling machinery underlying spike timing-dependent plasticity (STDP) may be separate

78 Spike timing-dependent plasticity (STDP) modifies synapt

79 activation of D1-type DA receptors regulates spike timing-dependent plasticity (STDP) of the medial p

80 nduces long-term depression (LTD) and shifts spike timing-dependent plasticity (STDP) toward LTD at G

81 Here, we report that spike timing-dependent plasticity (STDP) was absent in t

82 or indirect pathways determines the form of spike timing-dependent plasticity (STDP), the manner by

83 tion of developing retinotectal circuits via spike timing-dependent plasticity (STDP).

84 nd postsynaptic firing, in a paradigm termed spike timing-dependent plasticity (STDP).

85 iation (LTP), long-term depression (LTD) and spike-timing dependent plasticity (STDP) are demonstrate

86 on the mechanisms of eCB bidirectionality in spike-timing dependent plasticity (STDP) at corticostria

87 We demonstrate that spike-timing dependent plasticity (STDP) enhances synchr

88 Spike-timing dependent plasticity (STDP) is a widespread

89 However, its effect on the induction of spike-timing dependent plasticity (STDP) is not clear.

90 synapses are excitatory and undergo hebbian spike-timing dependent plasticity (STDP) on a +/-25 ms t

91 nnections are temporally opposed versions of spike-timing dependent plasticity (STDP), leading to a s

92 e mechanism for mediating such refinement is spike-timing dependent plasticity (STDP), which translat

93 etworks typically combine point neurons with spike-timing-dependent plasticity (STDP) as the learning

94 Classic in vitro studies have described spike-timing-dependent plasticity (STDP) at a synapse: t

95 at this can actually occur through a form of spike-timing-dependent plasticity (STDP) at the cerebell

96 However, investigations of spike-timing-dependent plasticity (STDP) at these synaps

97 Such spike-timing-dependent plasticity (STDP) follows rules t

98 how these memory traces could emerge through spike-timing-dependent plasticity (STDP) has been missin

99 This spike-timing-dependent plasticity (STDP) has been studie

100 nterest in the neuromorphic emulation of the spike-timing-dependent plasticity (STDP) Hebbian learnin

101 Spike-timing-dependent plasticity (STDP) is a form of lo

102 This phenomenon of spike-timing-dependent plasticity (STDP) is believed to

103 Spike-timing-dependent plasticity (STDP) is considered a

104 nt modification of synaptic strengths due to spike-timing-dependent plasticity (STDP) is sensitive to

105 Spike-timing-dependent plasticity (STDP) modifies synapt

106 Here we examine spike-timing-dependent plasticity (STDP) of inhibitory s

107 Here, we study the effects of ongoing spike-timing-dependent plasticity (STDP) on the stabilit

108 on synaptic plasticity, we trigger a single spike-timing-dependent plasticity (STDP) pairing once pe

109 S831D/S845D mice exhibited LTP induced with spike-timing-dependent plasticity (STDP) protocol at a l

110 Here, we show that in vivo a spike-timing-dependent plasticity (STDP) protocol-consis

111 As a result, the notion that a single spike-timing-dependent plasticity (STDP) rule alone can

112 nd their postsynaptic targets obey a Hebbian spike-timing-dependent plasticity (STDP) rule.

113 s are consistent with experimentally derived spike-timing-dependent plasticity (STDP) rules, suggesti

114 neurons are modified under the influence of spike-timing-dependent plasticity (STDP) rules.

115 The spike-timing-dependent plasticity (STDP), a synaptic lea

116 to underlie changes in neuronal responses is spike-timing-dependent plasticity (STDP), an up- or down

117 e-cell properties, such as spiking dynamics, spike-timing-dependent plasticity (STDP), and acetylchol

118 long-term synaptic plasticity, which we call spike-timing-dependent plasticity (STDP), depends on the

119 In spike-timing-dependent plasticity (STDP), the order and

120 cording to a biological learning rule called spike-timing-dependent plasticity (STDP).

121 e synaptic strength of cortical connections [spike-timing-dependent plasticity (STDP)], but how the p

122 stimulation (PAS) protocols induce forms of spike-timing-dependent-plasticity (STDP) when paired pul

123 Spike timing-dependent plasticity, STDP, has attracted c

124 an important computational function through spike timing-dependent plasticity: The capability to dis

125 tory synapses show long-term plasticity, but spike timing-dependent plasticity was seen only at excit

126 We show that spike-timing-dependent plasticity with both pre- and pos