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1 ress is also involved in the pathogenesis of spinal and bulbar muscular atrophy.
2 peutic potential of arimoclomol in mice with spinal and bulbar muscular atrophy.
3 ndrogen receptor, an Hsp90 client mutated in spinal and bulbar muscular atrophy.
4 ell culture and in a knock-in mouse model of spinal and bulbar muscular atrophy.
6 ve therapeutic potential in the treatment of spinal and bulbar muscular atrophy and may also be a pos
7 ould potentially be a therapeutic target for spinal and bulbar muscular atrophy and related polygluta
8 nclude dentatorubral pallidoluysian atrophy, spinal and bulbar muscular atrophy, and the spinocerebel
9 RA24 to the longer poly-Q AR in the X-linked spinal and bulbar muscular atrophied AR could contribute
11 nd FRAXE loci), myotonic dystrophy, X-linked spinal and bulbar muscular atrophy, Huntington's disease
12 n in the androgen receptor, causing X-linked spinal and bulbar muscular atrophy, impairs its function
18 sis in cultured embryonic motor neurons from spinal and bulbar muscular atrophy mice, which was accom
21 have a positive effect on muscle function in spinal and bulbar muscular atrophy patients, in addition
22 , we show that, in the polyglutamine disease spinal and bulbar muscular atrophy, proteolysis of the m
23 polyQ) has been linked to the development of spinal and bulbar muscular atrophy (SBMA or Kennedy dise
24 ion within the androgen receptor (AR) causes spinal and bulbar muscular atrophy (SBMA) and is associa
43 the androgen receptor (AR) protein leads to spinal and bulbar muscular atrophy (SBMA), a neurodegene
44 ested in creating a mouse model for X-linked spinal and bulbar muscular atrophy (SBMA), a neuromuscul
45 sterone levels is a therapeutic approach for spinal and bulbar muscular atrophy (SBMA), a polyglutami
47 ransgenic model of the polyglutamine disease spinal and bulbar muscular atrophy (SBMA), an adult-onse
49 pansion in the androgen receptor (AR) causes spinal and bulbar muscular atrophy (SBMA), an X-linked n
50 gen receptor (AR), a protein associated with spinal and bulbar muscular atrophy (SBMA), and the nucle
51 CAG repeat polyglutamine diseases, including Spinal and Bulbar Muscular Atrophy (SBMA), Huntington's
52 by a CAG/polyglutamine expansion, including spinal and bulbar muscular atrophy (SBMA), Huntington's
57 X syndrome (FRAX), myotonic dystrophy (DM), spinal and bulbar muscular atrophy (SBMA, also known as
61 receptor (AR) to Kennedy's disease (X-linked spinal and bulbar muscular atrophy) was a major step for
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