コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 prion-like propagation of oligomeric SOD1 in spinal cord.
2 ociceptors have distinct morphologies in the spinal cord.
3 gral part of neural circuits for itch in the spinal cord.
4 Fos immunoreactivity pattern in the cervical spinal cord.
5 that governs axonal wiring of the zebrafish spinal cord.
6 thin band of tissue at the caudal end of the spinal cord.
7 erotonin innervation of motor neurons in the spinal cord.
8 time window for development of the brain and spinal cord.
9 ained excitation of neurons in slices of rat spinal cord.
10 ophage activation in the white matter of the spinal cord.
11 circuits and large diameter afferents in the spinal cord.
12 appears to occur to a lesser extent than in spinal cord.
13 synaptic afferents in the dorsal horn of the spinal cord.
14 ement and/or acute blood products within the spinal cord.
15 that this is controlled at the level of the spinal cord.
16 ytes in the demyelinated lesion of nude mice spinal cord.
17 te receptor with opioids at the level of the spinal cord.
18 per and lower motor neurons in the brain and spinal cord.
19 pathways and large diameter afferents in the spinal cord.
20 ntials if they were timed to converge in the spinal cord.
21 iately after complete transection of the rat spinal cord.
22 e regeneration of their central axons in the spinal cord.
23 axons growing across lesion epicenter in the spinal cord.
24 ctant for commissural axons in the embryonic spinal cord.
25 maturation of supraspinal projections to the spinal cord.
26 ling robust penetration of DR axons into the spinal cord.
27 s from the peripheral nervous system and the spinal cord.
28 ectedly profound pathology in the human INCL spinal cord.
29 , with no evidence of re-entry back into the spinal cord.
30 tly affecting the brainstem, cerebellum, and spinal cord.
31 interactions between cortex, brainstem, and spinal cord.
32 g from the hypothalamus to the brainstem and spinal cord.
33 remove netrin from different regions of the spinal cord.
34 cross five lumbosacral segments in the human spinal cord.
35 neurons project mostly to the hindbrain and spinal cord.
36 ectable in skeletal muscle, heart, brain and spinal cord.
37 by mechanosensory feedback in the vertebrate spinal cord.
38 several hundred micrometers of the zebrafish spinal cord.
39 , and importantly, also in the injured human spinal cord.
40 t architectures within these lineages in the spinal cord.
41 h as autonomic regions of the brain stem and spinal cord.
42 s (NSPCs) that contribute to the regenerated spinal cord.
43 in those sections from mechanically injured spinal cords.
44 the latter was only observed in regenerated spinal cords.
45 nistered by stereotaxic injection into mouse spinal cords.
46 of axons growing into a lesion epicenter in spinal cord after a concomitant dorsal column transectio
48 hen activating muscles, motor neurons in the spinal cord also activate Renshaw cells, which provide r
50 l ganglionic eminence (MGE) into adult mouse spinal cord ameliorates mechanical and thermal hypersens
51 as an example of non-monotonic coding in the spinal cord and better explains observations in human ps
52 leptomeningeal disease and metastasis to the spinal cord and brain, resembling xenografts of human SH
54 ch as estradiol, that are synthesized in the spinal cord and brainstem and act locally to influence p
55 cine corelease is particularly common in the spinal cord and brainstem, but its presence in the midbr
56 found that AAVrh10 transduces neurons in the spinal cord and dorsal root ganglia of immunodeficient m
59 uleus (LC) projects throughout the brain and spinal cord and is the major source of central noradrena
62 ish mutants in which OPCs migrate out of the spinal cord and myelinate peripheral motor axons, we ass
63 ich there is widespread demyelination of the spinal cord and optic nerves, we also show that thinly r
64 t neural stem cells, derived from the murine spinal cord and organized as neurospheres, can be trigge
66 n of BzATP resulted in ROS production in the spinal cord and oxidative DNA damage in dorsal horn neur
67 rom adrenomyeloneuropathy affecting only the spinal cord and peripheral nerves (Expanded Disability S
68 represent the direct cortical outputs to the spinal cord and play important roles in motor control ac
70 Bar(CRH) neurons project to the lumbosacral spinal cord and ramify extensively in two regions: the d
73 (HD)-within the various compartments of the spinal cord and their potential impact on the local vasc
78 A-binding protein TDP-43 in their brains and spinal cords, and rare mutations in the gene encoding TD
80 Circuits in the sensorimotor system (e.g., spinal cord) are thought to be assembled sequentially [1
81 They support the concept of the state of the spinal cord as a negotiated equilibrium that reflects th
82 c stiffness of the injured rat neocortex and spinal cord at 1.5 and three weeks post-injury using ato
91 ymphocytes from the blood into the brain and spinal cord by blocking the adhesion molecule alpha4-int
93 +ChABC treatment of the chronically contused spinal cord can provide a permissive substrate for the r
94 ses excitability in DRG neurons and leads to spinal cord central sensitization and neuropathic pain s
95 tomic regions when comparing astrocytes from spinal cord, cerebellum, cerebral cortex, and hippocampu
96 t is hardwired; but it is now clear that the spinal cord changes continually as new behaviors are acq
97 fective at inducing hyperexcitability within spinal cord circuits compared with skin afferents, which
98 g genetic approaches in the mouse to map the spinal cord circuits that transmit and gate the cutaneou
99 Neurobiotin retrograde transport from the spinal cord combined with immunofluorescence revealed sp
100 d events (SREs) such as pathologic fracture, spinal cord compression, or the necessity for radiation
101 related event (defined as clinical fracture, spinal cord compression, radiation to bone, or surgery i
102 native codon-derived DPRs in chick embryonic spinal cord confirmed in vitro data, revealing that each
103 hanisms for epigenetic regulation within the spinal cord constitute the starting point for handedness
107 patients and correlated with representative spinal cord cross-sectional area regions at the C2 to C3
111 dipine treatment attenuated clinical EAE and spinal cord degeneration and promoted remyelination.
116 that many neurons in laminae I and V of the spinal cord dorsal horn and caudal spinal trigeminal nuc
117 cinergic inhibitory neurotransmission in the spinal cord dorsal horn gates nociceptive signaling, is
118 We found that ATP induces ROS production in spinal cord dorsal horn neurons, an effect eliminated by
119 athways that were changed in astrocytes from spinal cord during chronic EAE involved decreases in exp
120 e hypothesis that inducing plasticity in the spinal cord during chronic stroke could improve recovery
122 earch team has demonstrated that lumbosacral spinal cord epidural stimulation (scES) and activity-bas
123 POINTS: Pairing motor cortex stimulation and spinal cord epidural stimulation produced large augmenta
125 he prion-like seeding of SOD1 was assayed in spinal cord extracts of transgenic mice expressing a G85
126 gative, acylated SOD1 fibrils to organotypic spinal cord failed to produce the SOD1 inclusion patholo
127 ong transversal and longitudinal sections of spinal cord from control animals, but it strongly change
129 n and brainstem regions and project onto the spinal cord, have long been recognised as key links in t
131 d RGS10 protein levels are suppressed in the spinal cord in a nerve injury-induced neuropathic pain m
132 increase in sensory axon regeneration in the spinal cord in comparison to wildtype C3(+/+) mice.
133 hage-specific mRNA directly from the injured spinal cord in mice and performed RNA sequencing to inve
134 es over motor cortex and the dorsal cervical spinal cord in rats; motor evoked potentials (MEPs) were
135 ia from the central canal (CC) lining of the spinal cord in rodents should occur in the embryonic per
136 defects in axon projection of DRG toward the spinal cord in vivo Furthermore, live-cell imaging of en
138 s within the ependymal layer of the original spinal cord include populations of neural stem/progenito
139 nimals and was associated with reduced acute spinal cord infiltration of Ly6C(hi) effector monocytes.
141 udy participant was a 53-year-old man with a spinal cord injury (cervical level 4, American Spinal In
142 ied in 384 patients with clinically complete spinal cord injury (SCI) and consequent anejaculation.
148 STATEMENT Pain sensitization associated with spinal cord injury (SCI) involves poorly understood mala
149 NS trauma and disease.SIGNIFICANCE STATEMENT Spinal cord injury (SCI) leads to profound functional de
151 ays significance roles in recovery following spinal cord injury (SCI), and diabetes mellitus (DM) imp
154 Spasticity, a common complication after spinal cord injury (SCI), is frequently accompanied by c
158 influence many pathological processes after spinal cord injury (SCI), the intrinsic molecular mechan
160 esolution of inflammation is defective after spinal cord injury (SCI), which impairs tissue integrity
168 an reveal early inflammation associated with spinal cord injury after thoracic aortic ischemia-reperf
170 -cost portable BMI for survivors of cervical spinal cord injury and investigated it as a means to sup
171 at motor evoked potentials size increased in spinal cord injury and uninjured participants after the
172 c remodeling and involves netrin-1 signaling.Spinal cord injury can induce synaptic reorganization an
173 receptor, promotes recovery after traumatic spinal cord injury in mice, a benefit achieved in part b
175 of rehabilitation strategies in humans with spinal cord injury is to strengthen transmission in spar
178 f an individual with traumatic high-cervical spinal cord injury who coordinated reaching and grasping
179 th chronic tetraplegia, due to high-cervical spinal cord injury, can regain limb movements through co
180 veral neurological disorders such as stroke, spinal cord injury, multiple sclerosis, amyotrophic late
181 s neurological conditions, such as stroke or spinal cord injury, result in an impaired control of the
182 s system to restore motor function following spinal cord injury, the role of cortical targets remain
183 ipate in neuronal development, angiogenesis, spinal cord injury, viral invasion, and immune response.
190 ion in conditions such as cerebral palsy and spinal cord injury.SIGNIFICANCE STATEMENT Acquisition of
191 or stimulating axonal regeneration following spinal cord injury.SIGNIFICANCE STATEMENT Injury of peri
193 of myelinating Schwann cells in the injured spinal cord; invasion of peripheral myelinating (P0+) Sc
195 that develops following injuries to brain or spinal cord is a major obstacle for tissue repair in cen
198 lipid molecular order of myelin in the mouse spinal cord is significantly reduced throughout the prog
201 idges at the epicenter of traumatic cervical spinal cord lesions in 24 subacute tetraplegic patients.
203 d on highly specific molecular features from spinal cord level to the terminal nerves at wrist level.
204 ticospinal tract axons in the contralesional spinal cord makes a significant contribution to sensorim
205 e motor cortex is functionally linked to the spinal cord, making it more likely that spinal gene expr
206 blood-cerebrospinal fluid barrier and of the spinal cord meninges, but not by the endothelium of the
208 onstrate that endogenous RGS10 is present in spinal cord microglia, and RGS10 protein levels are supp
210 romuscular disorder characterized by loss of spinal cord motor neurons, muscle atrophy and infantile
212 urological examination, a baseline brain and spinal cord MRI scan obtained less than 3 months from cl
214 eolytic targets of calpain in Xenopus laevis spinal cord neurons both in vivo and in vitro Inhibition
222 her (64)Cu-rituximab uptake in the brain and spinal cord of huCD20tg EAE, and B220 immunostaining ver
223 n, particularly of minor U12 introns, in the spinal cord of mice 30 d after SMA induction, which was
227 cal changes also occur at every level of the spinal cord of PPT1-deficient (Ppt1(-/-) ) mice before t
228 eposition around demyelinated lesions in the spinal cord of Theiler's murine encephalomyelitis virus-
230 sex difference in the IMS-UPRmt, because the spinal cords of female, but not male, G93A-SOD1 mice sho
231 vent in patients with multifocal or isolated spinal cord or optic neuritis involvement at onset in co
233 ates the use of lgals-1(-/-) mice to develop spinal cord- or traumatic brain injury models for the ev
234 ted, matured, and integrated into the rodent spinal cords over a time frame that aligned with the nor
235 ncer, deep vein thrombosis, major procedure, spinal cord paralysis, venous injury, lower extremity fr
240 man reflex is associated with reduced dorsal spinal cord potassium chloride cotransporter expression
242 orsal horn and RVM neurons to uncover an RVM-spinal cord-primary afferent circuit controlling pain th
243 oncept introduces.SIGNIFICANCE STATEMENT The spinal cord provides a reliable final common pathway for
245 e deep dorsal horn is a poorly characterized spinal cord region implicated in processing low-threshol
248 stiffness properties that are well suited to spinal cord repair by supporting cell growth mechano-bio
254 Blockade (NMB) and another undergoing lumbar spinal cord (SC) transection, both serving as controls.
255 In sum, when a new behavior changes the spinal cord, sensory feedback to the brain guides furthe
258 king of Slack channels also occurs in intact spinal cord slices and that it is carried out by adaptor
259 INs using targeted patch-clamp recording in spinal cord slices from adult transgenic mice that expre
264 modulation of cortical evoked potentials by spinal cord stimulation was largest when the spinal elec
265 ology components for injured adult mammalian spinal cord that are different from those involved in no
266 dings suggest that there exists a pathway in spinal cord that ascends from periphery to mesolimbic re
267 re traditionally viewed as the output of the spinal cord that do not influence locomotor rhythmogenes
268 on the mechanisms of axonal guidance in the spinal cord that provide for a discussion of the current
269 ject directly to nuclei in the brainstem and spinal cord that regulate parasympathetic and sympatheti
270 the transcriptome of both the cerebellum and spinal cord that was consistent with glial activation an
272 physiological studies about reflexes in the spinal cord, the contribution of mechanosensory feedback
274 This new variant is undetectable in brain or spinal cord, the only and most abundant known sources of
278 iated viral vectors, serotype-9 (scAAV-9) in spinal cord tissues after intraspinal injection of mouse
280 he central canal of the brain ventricles and spinal cord to circulate the cerebral spinal fluid (CSF)
281 hundred fifty thousand axons emerge from the spinal cord to innervate the human upper limb, of which
282 nomic estrogen signaling within the cervical spinal cord to recover respiratory neuroplasticity in di
284 rvivors.SIGNIFICANCE STATEMENT The brain and spinal cord undergo adaptive rewiring ("plasticity") fol
285 By selectively ablating microglia in the spinal cord using a saporin-conjugated antibody to Mac1,
289 most completely absent in both the brain and spinal cord when intracranial and intrathecal injections
290 mutant SOD1 protein in the disease-affected spinal cord, where concomitant increases in copper and S
291 significantly increased in the injured mouse spinal cord, where it is predominantly found in astrocyt
292 ing between prefrontal areas, brainstem, and spinal cord, which might represent a flexible mechanism
293 The second derived feature is the very short spinal cord, which terminates midway along the thoracic
294 receptor and cytokine gene expression in the spinal cord, which were normalized by postnatal coloniza
295 that lacked CCR7 were retained in brain and spinal cord while wild type DC migrated to cervical lymp
296 The replacement tail includes a regenerated spinal cord with a simple morphology: an ependymal layer
297 Na](+) adducts was observed in samples from spinal cord with demyelination, while the intensity of t
298 direct and trans-synaptic infection from the spinal cord with rabies viruses that carry glycoproteins
299 form to the circumvolutions of the brain and spinal cord without damaging neural tissues or triggerin
300 ing stimulation of motor cortex and cervical spinal cord would strengthen motor responses through the
301 despite implantation into the injured rodent spinal cord, yet they support delayed functional recover
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。