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1 sion with subsequent reperfusion to generate spinal cord ischemia.
2 arteries are effective for the treatment of spinal cord ischemia.
3 on systemic blood pressure in a rat model of spinal cord ischemia.
4 onstrated to be neuroprotective in brain and spinal cord ischemia.
5 microl/day for 7 days prior to induction of spinal cord ischemia.
6 otocol may decrease the rates of devastating spinal cord ischemia.
10 azoxide improves neurological function after spinal cord ischemia and reperfusion by diminishing leve
13 otection protocol could decrease the risk of spinal cord ischemia and/or paraplegia and consists of p
14 a critical determinant of outcome following spinal cord ischemia, and controlled peri-operative bloo
15 y matter involvement similar to that seen in spinal cord ischemia, and three (16%) had isolated white
17 y early detection and immediate treatment of spinal cord ischemia before it evolves to infarction.
19 es the potential catastrophic consequence of spinal cord ischemia from an unexpectedly prolonged aort
20 ctors and interventions to prevent and treat spinal cord ischemia has the potential to prevent spinal
21 ive events associated with increased IM were spinal cord ischemia (HR = 9.46; 95% CI: 3.98-22.47; P <
23 are consistent with the idea that transient spinal cord ischemia induces the formation of a death-in
28 Neurologic events included stroke (5.0%) and spinal cord ischemia (permanent 1.7%, temporary 7.9%).
33 ntials (MEPs) monitoring can promptly detect spinal cord ischemia (SCI) from aortic clamping during o
34 has demonstrated low risks of mortality and spinal cord ischemia (SCI), but few large series have be
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