ライフサイエンスコーパス: spinal fluid

1 to the canalostomy due to influx of cerebral spinal fluid.

2 f normal appearing white matter and cerebral spinal fluid.

3 where they block circulation of the cerebral spinal fluid.

4 hamsters using 2-microl aliquots of cerebral spinal fluid.

5 omography and glucose monitoring in cerebral spinal fluid.

6 y culture or detection of Bb DNA in blood or spinal fluid.

7 of peptides dissolved in artificial cerebral spinal fluid.

8 e where they are transported to the cerebral spinal fluid.

9 secondary sites, principally from blood and spinal fluid.

10 tum and lung tissue samples; and pleural and spinal fluids.

11 h 16 additional NTHi isolates from blood and spinal fluid, 17 nasopharyngeal or throat isolates from

12 CK-8 (0 or 1.6 microg) in artificial cerebro-spinal fluid (5 microl).

13 Surprisingly, cerebral spinal fluid Abeta levels transiently rise, and when Ab:

14 interneurons (21/25) in artificial cerebral spinal fluid (aCSF) exhibited spontaneous tonic spiking

15 Artificial cerebral spinal fluid (aCSF) or 100 microM bumetanide in aCSF wer

16 xt, BLA was infused with artificial cerebral spinal fluid (ACSF) or ACSF containing an N-methyl-D-asp

17 stimulation with high-K+ artificial cerebral spinal fluid (aCSF) were observed.

18 osure to low bicarbonate artificial cerebral spinal fluid (aCSF), pH 6.70 for 30 min at 32 degrees C,

19 ed by microdialysis with artificial cerebral spinal fluid allowed isolation of 14C-glutamine (63%), 1

20 were absence of correlation between cerebral spinal fluid amyloid-beta1-42 and Boston naming and Trai

21 t-tau, brain amyloid-beta load via cerebral spinal fluid amyloid-beta1-42 and vascular disease via w

22 inal fluid t-tau) and amyloid load (cerebral spinal fluid amyloid-beta1-42) all independently explain

23 Cerebral spinal fluid analysis revealed elevated levels of proinf

24 Immunoglobulin in cerebral spinal fluid and antibody secreting cells (ASC) within t

25 ations of allopregnanolone in human cerebral spinal fluid and in rat brain sections.

26 Reports of rotavirus RNA in the cerebral spinal fluid and serum of children infected with rotavir

27 ryptococcal polysaccharides in the serum and spinal fluid and the lack of cellular infiltrates in the

28 s in human neocortex, white matter, cerebral spinal fluid, and serum by immunostaining and measuremen

29 itial reduction in the serum antibodies, the spinal fluid antibodies remained persistently elevated.

30 Serum or cerebral spinal fluid antigen titers did not correlate with morta

31 Cerebral spinal fluid antigen titers were higher with meningeal v

32 nuclear ASPP2 in vivo at the blood-cerebral spinal fluid barrier (the brain's barrier to inflammatio

33 between pulse pressure and distinct cerebral spinal fluid biomarker profiles.

34 primary viruses from autopsy brain, cerebral spinal fluid, blood, spleen, and lymph node samples from

35 to correlate with viral RNA in the cerebral spinal fluid but not with plasma viral load, consistent

36 ection, but this test may not be positive in spinal fluid collected less than 8 days after the onset

37 Analysis of imatinib mesylate cerebral spinal fluid concentrations revealed levels 155- fold lo

38 ese include V2b interneurons and the central spinal fluid-contacting Kolmer-Agduhr cells in the verte

39 Solutions of artificial cerebral spinal fluid containing 50 muM DA, 50 muM NE, and a DA-N

40 Severe cerebral spinal fluid (CSF) abnormalities were present in 1 patie

41 ith a similar half-life in both the cerebral spinal fluid (CSF) and the CNS.

42 A cerebral spinal fluid (CSF) cavity within the conus medullaris ha

43 eviewed to identify the presence of cerebral spinal fluid (CSF) clefts around the tumors.

44 Cerebral spinal fluid (CSF) examination showed marked abnormaliti

45 the first time in the regulation of cerebral spinal fluid (CSF) homeostasis.

46 pathophysiologic changes within the cerebral spinal fluid (CSF) of these patients.

47 drocephalus is a common disorder of cerebral spinal fluid (CSF) physiology resulting in abnormal expa

48 Plasma and cerebral spinal fluid (CSF) samples were collected periodically f

49 Biotin is likely transported into cerebral spinal fluid (CSF) via one or more specific transporters

50 mentalization between the blood and cerebral spinal fluid (CSF) was apparent with all neurological ca

51 nd lymph node tissues, bone marrow, cerebral spinal fluid (CSF), plasma, and very large numbers of pe

52 n the production and circulation of cerebral spinal fluid (CSF), suggesting that loss of mFu disrupts

53 es and spinal cord to circulate the cerebral spinal fluid (CSF).

54 the concentrations of Abeta1-42 in cerebral spinal fluid (CSF).

55 (n = 152), pleural fluid (n = 76), cerebral spinal fluid (CSF; n = 152), pericardial fluid (n = 131)

56 th cloned peripheral blood and cerebrospinal spinal fluid-derived CTL and bulk PBMC from HAM/TSP pati

57 hage-tropic virus isolated from the cerebral spinal fluid, did not use CCR5 or CXCR4.

58 , blood pressure mean arterial pressure, and spinal fluid drainage with naloxone (SFDN).

59 risk using hypothermia, naloxone, steroids, spinal fluid drainage, intercostal ligation, and optimiz

60 Clinical history, neurological exam, spinal fluid examination, and electrophysiological evalu

61 his study, BJAB0715, which was isolated from spinal fluid, exhibit much more divergence compared with

62 nvade the spinal canal and obstruct cerebral spinal fluid flow.

63 oteolytic fragment of cystatin C in cerebral spinal fluids from patients diagnosed with multiple scle

64 erized in vitro using an artificial cerebral spinal fluid gelatin.

65 ited robust in vivo activity in the cerebral spinal fluid glycine biomarker model in both rodents and

66 PCR identification of HSV in spinal fluid has become the diagnostic gold standard due

67 The mean cerebral spinal fluid HIV load was 5949 copies/mL.

68 of retraction: the article "Role of Cerebral Spinal Fluid in Space Flight Induced Ocular Changes and

69 tissue from either the blood or the cerebro-spinal fluid is limited.

70 Detection of Epstein-Barr virus in spinal fluid is useful in the diagnosis of primary centr

71 as the predominant sequence type of cerebral spinal fluid isolates from cases of meningitis.

72 pendent marker of Alzheimer's disease, i.e., spinal fluid levels of tau, provided corresponding evide

73 ing of Epstein-Barr virus DNA copy number in spinal fluid may be useful in assessing response.

74 While cerebral spinal fluid neuregulin levels were unaltered in patient

75 levated tau levels are found in the cerebral spinal fluid of AD patients.

76 FN-gamma), which is elevated in the cerebral spinal fluid of HAD patients.

77 ated in humans and elevates cGMP in cerebral spinal fluid of healthy volunteers, confirming that it i

78 The presence of EBV DNA in the cerebral spinal fluid of HIV patients with focal brain lesions st

79 e detected in seminal plasma and/or cerebral spinal fluid of six individuals.

80 nd a significantly high level of H2 S in the spinal fluid of the ALS patients.

81 Hypothalamic tanycytes contact the cerebral spinal fluid of the third ventricle and send processes i

82 y unreported autoantibodies contained in the spinal fluid of three individuals with paraneoplastic ne

83 ains the only approved procedure for testing spinal fluids of patients with possible neurosyphilis.

84 umbar puncture for determination of cerebral spinal fluid phosphorylated tau (P-tau) and beta-amyloid

85 specimens: bronchoalveolar lavage, cerebral spinal fluid, plasma, and whole blood.

86 malize unknown peak mobilities in a cerebral spinal fluid sample.

87 s was used to genotype parasites in cerebral spinal fluid samples from 8 of 10 human immunodeficiency

88 parasites/ml in amniotic fluid and cerebral spinal fluid samples.

89 rade 3) cerebellar herniation and absence of spinal fluid spaces around the cerebellum.

90 relate with mortality at 90 days or cerebral spinal fluid sterilization at 2 weeks.

91 ural magnetic resonance imaging and cerebral spinal fluid t-tau) and amyloid load (cerebral spinal fl

92 sonance images, neuronal injury via cerebral spinal fluid t-tau, brain amyloid-beta load via cerebral

93 in as well as GM, white matter, and cerebral spinal fluid tissue probability maps to facilitate tissu

94 e in CXCL1 and CXCL10 levels in the cerebral spinal fluid, TNF-alpha expression in the ependymal regi

95 At the same time that cerebral spinal fluid viral load increased in macaques that devel

96 Therefore, cerebral spinal fluid volume was decreased (Delta-40ml, 95% CI =

97 Spinal fluid was inflammatory in 86%, and CRMP-5-IgG in

98 For sampling, artificial cerebral spinal fluid was infused in through one channel at 50 nL

99 ts in steady-state equilibrium with cerebral spinal fluid where it has the potential to serve as a bi

100 ctivity is also detectable in human cerebral spinal fluid where its expression appears to be altered