ライフサイエンスコーパス: spinal injury

1 out S1 CST involvement in recovery following spinal injury.

2 etour in restoration of motor function after spinal injury.

3 orelimb movements, and can be regained after spinal injury.

4 r function is important in rehabilitation of spinal injury.

5 sponsible for a lack of neurogenesis after a spinal injury.

6 uitable as a therapeutic approach to chronic spinal injury.

7 h volunteers and in patients with incomplete spinal injury.

8 tion in multiple motor systems after chronic spinal injury.

9 es influencing functional recovery following spinal injury.

10 ency in patients with, for example, cervical spinal injury.

11 plasticity, which may foster recovery after spinal injury.

12 ependent response of the spinal cord after a spinal injury.

13 tical representation of tactile events after spinal injury.

14 transmit neural signals to bypass a chronic spinal injury.

15 ing performance, particularly after neonatal spinal injury.

16 ving transgenic mice over several days after spinal injury.

17 ders such as respiratory insufficiency after spinal injury.

18 osed as a strategy to improve movement after spinal injury.

19 to improve walking ability in patients with spinal injury.

20 equired preconditions established by chronic spinal injury.

21 h by corticospinal tract (CST) neurons after spinal injury.

22 and improved breathing in rats with cervical spinal injuries.

23 of cAMP, however, is useless as therapy for spinal injuries.

24 lation will be useful for treatment of human spinal injuries.

25 rats (Rattus norvegicus) receiving thoracic spinal injury 3 d to 8 months earlier and recorded 1 d a

26 se noradrenergic receptors proliferate after spinal injury and descending noradrenergic pathways cont

27 ated with respiratory insufficiency, such as spinal injury and motor neuron disease.

28 ll number of investigations done on cervical spinal injury and tetraplegia, and the differences in le

29 eurocan, which increase in the vicinity of a spinal injury, and aggrecan, which decreases, into the l

30 inal sympathetic reflexes is increased after spinal injury, and these reflexes may result in life-thr

31 Most spinal injuries are incomplete, leaving spared neural pa

32 apeutics to treat traumatic brain injury and spinal injury as well as neuronal and cardiac ischemic e

33 ects was positively correlated with American Spinal Injuries Association (ASIA) grade: the smallest a

34 In two of two patients with American Spinal Injuries Association grade C traumatic spinal cor

35 evere traumatic spinal cord injury (American Spinal Injuries Association grades A-C).

36 NfL levels correlated with American Spinal Injury Association (ASIA) motor score at baseline

37 ination with gross motor scoring by American Spinal Injury Association Impairment Scale and Inflammat

38 inal cord injury (cervical level 4, American Spinal Injury Association Impairment Scale category A).

39 children with cerebral AVF and the American Spinal Injury Association impairment scale in children w

40 nt had cervical injuries, 53.4% had American Spinal Injury Association injury severity A (complete in

41 cluding Brief Pain Inventory, ASIA (American Spinal Injury Association) impairment scale, SF-36 Short

42 Spinal injury disrupts connections between the brain and

43 , after the bladder has been disconnected by spinal injury from the pontine micturition centre, vanil

44 together, these results indicate that lumbar spinal injuries have more severe consequences on hindlim

45 is responsible for 6 to 19% and 9% to 14% of spinal injuries in children and adults, respectively.

46 Spinal injuries in children remain a challenge despite s

47 We suggest that after neonatal spinal injury in the rat, the transplant-mediated reesta

48 ations in non-regenerating species.Following spinal injury in zebrafish, non-neural cells establish a

49 e conditions, such as sleep apnoea, cervical spinal injury or amyotrophic lateral sclerosis.

50 ep apnea, or respiratory insufficiency after spinal injury or during neurodegenerative diseases.

51 plasticity and improves motor function after spinal injury or during neuromuscular disease.

52 ed (e.g., sleep apnea, apnea of prematurity, spinal injury, or motor neuron disease).

53 Brain and spinal injury reduce mobility and often impair sensorimo

54 These findings demonstrate that spinal injury results in increased taurine concentration

55 icant increase in phosphorylated ERK1 at the spinal injury site after in vivo ChABC treatment, indica

56 t axon processes were able to grow through a spinal injury site.

57 Seven weeks after spinal injury, the Basso-Beattie Bresnahan locomotor sco

58 etic nerve activity is maintained after high spinal injury through circuits that remain in question.

59 ed a general rehabilitation ward, a regional spinal injuries unit and stroke rehabilitation ward.

60 cruited from the patient lists of a tertiary spinal injury unit and a Specialist Continence Service.

61 y, whereas one patient with low suspicion of spinal injury was partially extended.