ライフサイエンスコーパス: spinal reflex

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1 to "normalize' the excitability of specific spinal reflexes.

2 bolism, learning and memory, nociception and spinal reflexes, and anxiety and related behaviors.

3 Contrary to evidence from adults that spinal reflexes are inhibited during twitching [9-11], t

4 hat the tail-flick test of pain depends on a spinal reflex because a similar response is observed in

5 r response to bending, suggesting that these spinal reflexes can be modified by supraspinal signals i

6 Spinal reflex circuit development requires the precise r

7 The organization of spinal reflex circuits relies on the specification of di

8 ch was likely to have been generated through spinal reflex circuits, as a template to learn a predict

9 like immunoreactivity; and (3) regulation of spinal reflex circuits, as shown by an increase in alpha

10 These spinal reflexes could contribute to feedback regulation

11 and survival of SMA mice, partially restores spinal reflexes, illustrating the reversibility of these

12 cessive mono- and polysynaptic low threshold spinal reflexes in rats.

13 of dopamine and D2-like receptor ligands on spinal reflexes in wild-type (WT) and D3-receptor knock-

14 is a dramatic reduction of extensor tone and spinal reflexes, including PLRs.

15 Understanding trunk roles in voluntary and spinal reflex integration after spinal cord injury and i

16 muscle as well as low threshold polysynaptic spinal reflexes involving afferents from other treated m

17 Impairment of the corresponding otolith-spinal reflexes may contribute substantially to falls.

18 ysfunction of lumbar motoneurons and altered spinal reflexes modulation.

19 y information from the urethra that controls spinal reflexes necessary to maintain continence and ach

20 that the nRO may influence the regulation of spinal reflexes of the pelvic floor.

21 Nerve ligations excluded contributions of spinal reflexes or distal axon reflexes to the distribut

22 assessed by their continued ability to block spinal reflex pathways during the reappearance of sponta

23 Transmission in spinal reflex pathways from the toes to the ankle flexor

24 al evidence, we propose the existence of two spinal reflex pathways involved in micturition: a pathwa

25 y has a potent effect on the excitability of spinal reflex pathways.

26 This priming of spinal reflex sensitivity was measured by both reduction

27 fferent antinociceptive systems to inhibit a spinal reflex, tail withdrawal from radiant heat.

28 idine) receptor-induced antinociception in a spinal reflex test.

29 s, is associated with gradual alterations in spinal reflexes that appear to contribute to skill acqui

30 o depress sensory input but may also amplify spinal reflexes; the mechanisms of this modulation withi

31 lso increased pain-related vocalizations and spinal reflexes through a mechanism that required mGluR5

32 immersion paradigm, which primarily assesses spinal reflexes to painful thermal stimuli.

33 leus and m. gastrocnemius is a short-latency spinal reflex triggered by ankle joint rotation.

34 nship between nociceptive brain activity and spinal reflex withdrawal activity in response to a clini

35 ise the nociceptive-specific brain activity, spinal reflex withdrawal and behavioural activity follow

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