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1 tal nerve (GON) that is a branch of the C(2) spinal root.
2 ell loss and axonal degeneration in anterior spinal roots.
3 were also detected in motor axons of ventral spinal roots analyzed after 7 d in vivo after a peripher
4 ed the extensive loss of large fibers in the spinal roots and nerves, and the paucity of demyelinatio
6 i; spheroids and loss of myelinated axons in spinal roots and peripheral nerves; increased myocyte ap
8 -ATPase in lumbar dorsal root ganglia (DRG), spinal roots, and skeletal muscle samples of amphibian (
9 e probe binds spinal sensory tracts, ventral spinal roots, and the sympathetic chain but does not bin
10 diculitis; neuritis and demyelination in the spinal roots; and inflammation with neurodegeneration in
11 pain; in adults, chronic pain is usual after spinal root avulsion injuries, and this is often intract
14 ocalization of restored sensation in avulsed spinal root dermatomes, now presumably routed via nerves
15 lead to Schwann cell-specific expression in spinal roots extending to multiple peripheral nerves.
21 channels on axons in the dorsal and ventral spinal roots of the dystrophic mouse 129/ReJ-Lama2dy was
22 al root ganglia on day 3 p.i., to the dorsal spinal roots on days 4 and 5 p.i., and to second- and th
24 ractice, surgical re-implantation of avulsed spinal roots provides a degree of motor recovery, but th
26 ts exhibited reduced (p<0.05) SP-LI in their spinal roots, while basal levels in spinal CSF were not
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