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1 sions depend on the precise placement of the spindle apparatus.
2 y contributing to the formation of a bipolar spindle apparatus.
3 the formation of a microtubule-based bipolar spindle apparatus.
4 icrotubules emanating from both poles of the spindle apparatus.
5 l all sister chromosomes bi-orient along the spindle apparatus.
6 ormation of a microtubule (MT)-based bipolar spindle apparatus.
7 ndensation, nuclear kinesis, and assembly of spindle apparatus.
8 g mitosis, when clathrin concentrates at the spindle apparatus.
9 replicated sister chromatids to the mitotic spindle apparatus.
10 whereas a pool of Nup96 colocalized with the spindle apparatus.
11 factors required for assembly of the mitotic spindle apparatus.
12 TACC4 proteins could no longer target to the spindle apparatus.
13 nce showed that PP5 localizes to the mitotic spindle apparatus.
14 attern but no direct colocalization with the spindle apparatus.
15 hromosome segregation depends on the mitotic spindle apparatus, a bipolar array of microtubules nucle
18 the attachment of chromosomes to the mitotic spindle apparatus and is required for faithful chromosom
19 critical component of the centrosome/mitotic spindle apparatus and its absence triggers p53-mediated
21 s (MSCs), which monitor the integrity of the spindle apparatus and prevent cells from progressing int
22 he separation of duplicate chromatids by the spindle apparatus and the delivery of one set of chromos
23 (4%) had anticytoplasmic (mitochondrial and spindle apparatus) antibodies with a titer of 80 and two
31 riatal cells, HAP-1 localized to the mitotic spindle apparatus, especially at spindle poles and on ve
32 , are the only structural constituent of the spindle apparatus essential for cleavage furrow inductio
34 ine/threonine phosphorylation as the mitotic spindle apparatus forms, and this phosphorylation persis
39 chromosomes did not align correctly onto the spindle apparatus in the majority of Mlh1(-/-) spermatoc
40 ation that was most prominent in the mitotic spindle apparatus in variant HT1080 human fibrosarcoma c
42 and abrogates EG5 recruitment to the mitotic spindle apparatus, leading to spindle disorganization.
44 PRL-1 relocalizes to the centrosomes and the spindle apparatus, proximal to the centrosomes, in a far
46 show that LIN-5 is a novel component of the spindle apparatus required for chromosome and spindle mo
48 of communication between chromosomes and the spindle apparatus that may be widely conserved among euk
51 teraction of an apparently symmetric mitotic spindle apparatus with a clearly polarized cell cortex.
53 oint proteins monitor the interaction of the spindle apparatus with the kinetochores, halting anaphas
54 location of TACC4 from the centrosome to the spindle apparatus with the majority of TACC4 at the spin
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