ライフサイエンスコーパス: spinogenesis

1 portantly, foot-shock alone did not increase spinogenesis.

2 t E2-mediated activity in the DH drives mPFC spinogenesis.

3 eoglycan syndecan-2 (SDC2) induces dendritic spinogenesis.

4 tum to other brain regions through augmented spinogenesis.

5 ic spines during the developmental period of spinogenesis.

6 ted increase in cytosolic calcium levels and spinogenesis.

7 nA2-RasGAP activity is necessary to suppress spinogenesis.

8 not correlated with LTP, but with increased spinogenesis.

9 the NMDA receptor abolished activity-induced spinogenesis.

10 uggesting fasting induced synaptogenesis and spinogenesis.

11 any activity in the regulation of dendritic spinogenesis.

12 role of the proteasome in activity-dependent spinogenesis.

13 actin depolymerizing factor is required for spinogenesis.

14 tivation of Rac1, an established enhancer of spinogenesis.

15 rowth, branch formation, synaptogenesis, and spinogenesis.

16 xpression increases during a period of rapid spinogenesis.

17 and subsequent morphological changes led to spinogenesis.

18 tial loss of dendritic filopodia involved in spinogenesis.

19 g time-lapse imaging have reported increased spinogenesis.

20 rogenic regulation of the DH influences mPFC spinogenesis.

21 ombinant profilins rescued the impairment in spinogenesis, a hallmark in Fragile X syndrome, thereby

22 necessary and sufficient for fasting-induced spinogenesis and excitatory synaptic activity, and that

23 the elaborate responses of the spine during spinogenesis and experience-dependent plasticity.

24 n-B3 controls axon targeting and coordinates spinogenesis and neuronal activity within the amygdala.

25 DeltaFosB induction and prevented increased spinogenesis and sensitized amphetamine reward.

26 Although the molecular mechanisms underlying spinogenesis and spine synapse formation are being delin

27 synaptic adhesion GPCR that is required for spinogenesis and synaptogenesis in mice and rats.

28 receptor gamma co-activator 1alpha inhibits spinogenesis and synaptogenesis.

29 y supporting the involvement of drebrin A in spinogenesis and synaptogenesis.

30 of neurons, in vivo, in time to orchestrate spinogenesis and synaptogenesis.

31 exity and outgrowth toward TCA clusters; (3) spinogenesis; and (4) tuning of excitatory inputs.

32 Moreover, anxiogenesis and amygdalar spinogenesis are also triggered by chronic stress in con

33 induced enhanced drug reward, DeltaFosB, and spinogenesis are dependent on mating-induced dopamine D1

34 The activation of Rac1 initiates spinogenesis at an early stage and regulates the functio

35 rticostriatal synapse formation and striatal spinogenesis, but can itself be repressed by Foxp2 throu

36 ports, PTEN knockdown may not induce de novo spinogenesis, but instead may increase synaptic activity

37 Multiple proteins have roles in spinogenesis, but until now, a regulatory role for actin

38 The regulation of dendritic spinogenesis could explain the association of CTTNBP2 wi

39 se of Mef2c rescue vocalization and striatal spinogenesis defects of Foxp2-deletion mutants.

40 icating the importance of estradiol-mediated spinogenesis for female sexual receptivity (81.43 +/- 7.

41 the specific effect of CTTNBP2 on dendritic spinogenesis, here we investigate whether CTTNBP2NL has

42 all RhoGTPase, contributes to the absence of spinogenesis in immature neurons.

43 therapy (ET) enhances cognition and induces spinogenesis in neuronal circuits.

44 hese data demonstrate that estradiol-induced spinogenesis in the ARH is an important cellular mechani

45 a delayed onset of anxiety-like behavior and spinogenesis in the basolateral amygdala (BLA) of rats.

46 ty-like behavior, concomitant with increased spinogenesis in the basolateral amygdala.

47 mTOR activation is necessary for E2-induced spinogenesis in the DH and mPFC.

48 t inhibitory avoidance (IA) induces mushroom spinogenesis in the medium spiny neurons (MSNs) of the d

49 e molecular mechanisms regulating E2-induced spinogenesis in vivo are largely unknown.

50 Until now, it has been assumed that enhanced spinogenesis increased excitatory input received by the

51 Galphas activation was sufficient to enhance spinogenesis induced by glutamate photolysis in both dSP

52 the functional translation of BDNF-triggered spinogenesis into clearly defined morphological spine ty

53 We show that DOCK10 function in spinogenesis is mediated mainly by Cdc42 and its downstr

54 The initial stage of spinogenesis is thought to begin with the emergence of a

55 Excitatory spine synapse formation (spinogenesis) is a poorly understood yet pivotal period

56 Consistent with its normal role of slowing spinogenesis, loss of Icam5 induced precocious stubby sp

57 A profound dendritic spinogenesis occurs concurrent with the connectivity inc

58 E2 treatment elicits ERK- and mTOR-dependent spinogenesis on CA1 and mPFC pyramidal neurons, effects

59 We find that glutamate-induced spinogenesis requires opening of NMDARs (N-methyl-D-aspa

60 sion by recruiting AMPARs to synapses during spinogenesis, thus providing a mechanistic link between

61 through HIF-1alpha, mediates VEGF-dependent spinogenesis to underlie the EE-induced antidepressant-l

62 se data demonstrate that neurons undertaking spinogenesis upregulate actin expression, that actin ove

63 Surprisingly, estrogen-activated spinogenesis was associated with a decrease in CA1 hippo

64 ly, WRP function is critical at the onset of spinogenesis, when dendritic filopodia are prevalent.