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1 functional connectome of the striatal medium spiny neuron.
2 ABAergic inhibition onto direct and indirect spiny neurons.
3 nduced degeneration of striatal medium-sized spiny neurons.
4 t not D2-dopamine receptor-expressing medium spiny neurons.
5 region, an effect mediated by D2-type medium spiny neurons.
6 in indirect pathway nucleus accumbens medium spiny neurons.
7 and Drd2-expressing (striatopallidal) medium spiny neurons.
8 f excitatory synaptic transmission in medium spiny neurons.
9 eficits in dendritic spine density of medium spiny neurons.
10 costriatal activity in direct pathway medium spiny neurons.
11 shable D1- and D2-receptor-expressing medium spiny neurons.
12 induction of dendritic spines on NAc medium spiny neurons.
13 duced by the D1 receptor agonist SKF38393 in spiny neurons.
14 ogical outcome in dendritic spines of medium spiny neurons.
15 ine and other first messengers in the medium spiny neurons.
16 e of GRK2 and less of arrestin-2 than medium spiny neurons.
17 ts of DRD2 in cultured mouse striatal medium spiny neurons.
18 ibitory interactions between striatal medium spiny neurons.
19 subcellular localization in striatal medium spiny neurons.
20 at least some of which arose from identified spiny neurons.
21 rons from progenitors fated to become medium spiny neurons.
22 axon terminals at dendritic spines of medium spiny neurons.
23 hat preferentially occurs in striatal medium spiny neurons.
24 preferentially occurs in the striatal medium spiny neurons.
25 d expression of both genes in D2-type medium spiny neurons.
26 transient window of disinhibition for medium spiny neurons.
27 ion in dopamine receptor 1-expressing medium spiny neurons.
28 nd potentiated excitatory synapses on medium spiny neurons.
29 spiking interneurons (FSIs) than onto medium spiny neurons.
30 entiation protocol providing striatal medium spiny neurons.
31 D1 dopamine receptor (Drd1)-enriched medium spiny neurons, accelerates the development of morphine t
32 which is highly expressed in striatal medium spiny neurons, acts as a selective inhibitor of certain
33 label about 1% striatal D1-expressing medium spiny neurons and allow visualization of their dendrites
34 lrhodopsin-2 expression restricted to medium spiny neurons and assessed the specificity and strength
36 ession of identity genes for striatal medium spiny neurons and in dysregulation of cyclic AMP signali
37 however, ERK activation diminishes in medium spiny neurons and increases in striatal cholinergic inte
40 by dendritic spine enlargement in accumbens spiny neurons and required up-regulated surface expressi
41 MNI-659 may identify early changes in medium spiny neurons and serve as a marker to predict conversio
42 egulates the excitability of striatal medium spiny neurons and that reduction of Foxp1 correlates wit
43 model system incorporating GABAergic medium spiny neurons and the HEK293 cells, stably expressing di
44 ine neurons as well as D1R-expressing medium spiny neurons and their glutamatergic inputs via NMDARs
47 ing two distinct populations of medium-sized spiny neurons, and little is known concerning the cell-t
48 ts to both D1 and D2 receptor-bearing medium spiny neurons are inhibited by adenosine, released upon
49 uced plasticity, and define NAc shell medium spiny neurons as a primary site of persistent AMPA-type
50 tified dopamine receptor-1 expressing medium spiny neurons as key mediators of the effects of p11 on
51 unchanged pPKA substrate levels in D1 medium spiny neurons as well as in cholinergic interneurons.
53 nalyses of HEK293 cell innervation by medium spiny neuron axons using immunocytochemistry, activity-d
54 Our data show that whether core NAc medium spiny neurons belong to the direct or indirect pathways
56 in striatonigral and striatopallidal medium spiny neurons but not in several interneuron populations
57 s to basally control PP2A in striatal medium spiny neurons but that dopamine acting via PKA inactivat
58 ic synapses on nucleus accumbens core medium spiny neurons, but it is unknown how achieving drug use
59 ratio of AMPA to NMDA currents in accumbens spiny neurons compared with yoked saline animals at 2 wk
63 e neurons and nucleus accumbens (NAc) medium spiny neurons, consistent with a presynaptic effect.
65 on in D2 dopamine receptor-expressing medium spiny neurons corrected the hyperlocomotor phenotype, wh
66 - and D2-dopamine receptor-expressing medium spiny neurons (D1-/D2-MSNs) comprise the nucleus accumbe
67 e receptor type 2-expressing striatal medium spiny neurons (D2-MSNs) are particularly affected, we hy
68 es of dopamine D2-receptor-expressing medium spiny neurons (D2-MSNs), one of the major cell types in
69 pression of Auts2 or Caln1 in D2-type medium spiny neurons demonstrated that both genes promote cocai
70 tia nigra pars compacta, yet striatal medium spiny neuron dendritic spine density was largely maintai
71 y, cortical thinning, degeneration of medium spiny neurons, dense mutant Htt inclusion formation, dec
72 ls and mice expressing mutant Htt, in medium spiny neurons derived from human HD iPSCs and in brain s
74 striatal direct- and indirect-pathway medium spiny neurons (dMSNs and iMSNs) and optically stimulated
75 ed in the direct and indirect pathway medium spiny neurons (dMSNs and iMSNs, respectively), 5-HT6 rec
76 dorsomedial relative to dorsolateral medium spiny neurons early in training and population responses
77 role of phosphodiesterase 2 (PDE2), a medium spiny neuron-enriched and cGMP-activated PDE, in AMPAR t
78 erexpression of FosB in striatonigral medium spiny neurons exacerbated dyskinetic behavior, whereas o
81 idum (VP) as a site of convergence of medium spiny neurons expressing dopamine (DA) receptor type 1 (
82 ssociated with synaptic plasticity in medium spiny neurons expressing dopamine D2 receptors (D2-MSNs)
83 leation, a significant proportion of layer 4 spiny neurons failed to remodel their apical dendrites,
84 ial prefrontal cortex and in striatal medium spiny neurons forming the direct or indirect pathways.
85 ional synaptic maturation in striatal medium spiny neurons from neonatal rats exposed to antiepilepti
86 of inhibitory, indirect pathway medium-sized spiny neuron (iMSN) NMDA-Rs has been used to address the
87 eceptors (D2Rs) from indirect-pathway medium spiny neurons (iMSNs) is sufficient to impair locomotor
88 estin signaling in 'indirect pathway' medium spiny neurons (iMSNs), because of their central role in
89 c synaptic transmission onto striatal medium spiny neurons in a manner reminiscent of endogenous mesD
90 d expression of DARPP-32 in accumbens medium spiny neurons in a pattern indicative of reduced transmi
93 delta specifically in the striatum of medium spiny neurons in mice yielded HD-like motor phenotypes,
94 eases the density of stubby spines on medium spiny neurons in NAc, and that maintaining this increase
96 ne-induced changes in excitability of medium spiny neurons in nucleus accumbens and gating the compos
98 e-cell recordings were collected from medium spiny neurons in striatal slices prepared from adult mic
100 ses a specific loss of HFS-LTP in the medium spiny neurons in the direct pathway without affecting LT
101 cortex, terminates on direct pathway medium spiny neurons in the dorsolateral striatum, and is stren
102 ty in dopamine D2 receptor-expressing medium-spiny neurons in the indirect, striatopallidal pathway i
103 nhanced the intrinsic excitability of medium spiny neurons in the NAc core and reduced the function a
106 dministration specifically in D2-type medium spiny neurons in the nucleus accumbens, an effect seen i
107 ed htt induces a preferential loss of medium spiny neurons in the striatum and, to a lesser extent, a
110 ations have noted changes in striatal medium spiny neurons, including abnormal activation of extracel
111 exhibit several abnormalities in NAc medium spiny neurons, including reduced presynaptic function an
112 xpression of dopamine D2 receptors in medium spiny neurons increases their membrane excitability and
113 that activation of Group I mGluRs in medium spiny neurons induces trafficking of GluA2 from the endo
114 ed in the knock-in mice by changes in medium spiny neuron intrinsic excitability and nucleus accumben
115 ss calbindin, a marker of neostriatal-matrix spiny neurons, known to establish long-range projections
116 well documented in nucleus accumbens medium spiny neurons, little is known regarding the underlying
118 rly in dopamine receptor-1 expressing medium spiny neurons, may underlie pathophysiological mechanism
119 ase (HD) is characterized by striatal medium spiny neuron (MSN) dysfunction, but the underlying mecha
120 t, in the absence of SAPAP3, striatal medium spiny neuron (MSN) excitatory synapses exhibit eCB-media
121 quitously expressed, but the striatal medium spiny neuron (MSN) is most susceptible to dysfunction an
122 e show increases in the expression of medium spiny neuron (MSN) markers with age, suggesting that the
123 ent studies were conducted to examine medium spiny neuron (MSN) morphology by extending these earlier
125 europlasticity adaptations in the two medium spiny neuron (MSN) subtypes, those enriched in dopamine
126 regulation in nucleus accumbens (NAc) medium spiny neuron (MSN) subtypes, those enriched in dopamine
127 ine action in nucleus accumbens (NAc) medium spiny neuron (MSN) subtypes, those enriched in dopamine
130 ut to both major classes of principal medium spiny neuron (MSN) that gave way to a prolonged enhancem
131 type within the NAc is the GABAergic medium spiny neuron (MSN), with two major subpopulations define
132 elopment were confirmed by studies of medium spiny neurons (MSN), which do not express Met but receiv
136 ignals converging onto striatal medium-sized spiny neurons (MSNs) and activate intracellular events i
137 knocked out CK2, in either D1- or D2-medium spiny neurons (MSNs) and characterized the mice behavior
139 GABAARs) mediate phasic inhibition of medium spiny neurons (MSNs) and influence behavioral responses
140 nd striatopallidal (indirect pathway) medium spiny neurons (MSNs) and its relevance to repetitive gro
141 striatonigral and striatopallidal) of medium spiny neurons (MSNs) and synapses belonging to two neura
144 ne subtype 2 (D2) receptor-expressing medium spiny neurons (MSNs) are selectively vulnerable to Tat e
148 g postsynaptic M1 mAChRs expressed on medium spiny neurons (MSNs) at the origin of the indirect stria
149 e 2 (D2) receptor-expressing striatal medium spiny neurons (MSNs) by breeding transgenic Tat-expressi
150 mice, striatal D1 receptor-expressing medium spiny neurons (MSNs) directly projecting to the substant
151 rdinated activation of two classes of medium spiny neurons (MSNs) expressing D1 or D2 dopamine recept
152 cally involved in addiction, contains medium spiny neurons (MSNs) expressing dopamine D1 or D2 recept
153 on of DARPP-32 occurs specifically in medium spiny neurons (MSNs) expressing dopamine D1 receptors (D
154 -regulation occurs selectively in NAc medium spiny neurons (MSNs) expressing dopamine D2 receptors (D
156 from an imbalance in the activity of medium spiny neurons (MSNs) from the direct (dMSNs) and indirec
157 dent dendritic spine loss in striatal medium spiny neurons (MSNs) from YAC128 transgenic HD mice.
160 Electrophysiological analysis of medium spiny neurons (MSNs) in the AcbSh revealed decreased amp
162 Recordings were performed from mouse medium spiny neurons (MSNs) in the NAc at the same time after t
163 synaptic strength or connectivity of medium spiny neurons (MSNs) in the NAc to induce social avoidan
164 e, dendritic length, and branching of medium spiny neurons (MSNs) in the NAc, and quantitative immuno
165 n dopamine D1 or D2 subpopulations of medium spiny neurons (MSNs) in the NAc, we found that SIRT1 pro
166 and morphology of dendritic spines on medium spiny neurons (MSNs) in the nucleus accumbens (NAc), a c
167 ructural and functional properties of medium spiny neurons (MSNs) in the nucleus accumbens (NAc).
168 n (t-SP) at glutamatergic synapses on medium spiny neurons (MSNs) in the nucleus accumbens core (NAco
169 normal function of striatal GABAergic medium spiny neurons (MSNs) is believed to contribute to the de
170 c synapses on ventral striatum (vSTR) medium spiny neurons (MSNs) is critical for shaping stress resp
171 riatum, colocalized with the striatal medium spiny neurons (MSNs) marker, D1-receptor associated sign
173 f GlyRs in accumbal dopamine receptor medium spiny neurons (MSNs) of C57BL/6J mice, analysing mRNA ex
174 naling is significantly suppressed in medium spiny neurons (MSNs) of DKO mice relative to WT mice.
176 timing-dependent plasticity (STDP) in medium spiny neurons (MSNs) of the core nucleus accumbens (NAc)
177 imbalance in the activity of striatal medium spiny neurons (MSNs) of the direct (dMSNs) and indirect
178 induces mushroom spinogenesis in the medium spiny neurons (MSNs) of the dorsal striatum in rats, whi
180 1 and D2 dopamine receptor-expressing medium spiny neurons (MSNs) of the NAc of PV+ interneuron silen
182 plasticity at excitatory synapses on medium spiny neurons (MSNs) of the nucleus accumbens (NAc) driv
184 ("incubated rats") exhibit changes in medium spiny neurons (MSNs) of the nucleus accumbens (NAc) that
185 erations in the synaptic structure of medium spiny neurons (MSNs) of the nucleus accumbens (NAc), a p
187 kephalin-expressing (Penk-expressing) medium spiny neurons (MSNs) of the nucleus accumbens shell (NAc
188 highlights relevant effects of ACh on medium spiny neurons (MSNs) of the striatum and suggests how it
189 nal-regulated kinase (ERK) pathway in medium spiny neurons (MSNs) of the striatum controls psychostim
191 ifications in nucleus accumbens (NAc) medium spiny neurons (MSNs) play a key role in adaptive and pat
193 activity of two subpopulations of NAc medium spiny neurons (MSNs) that are defined by their predomina
196 ted synaptic transmission in striatal medium spiny neurons (MSNs) through postsynaptic endocytosis of
197 NAc core of rats modulates GABAergic medium spiny neurons (MSNs) through presynaptic-glutamatergic a
198 or adenosine A2a receptor-expressing medium spiny neurons (MSNs) to determine the role of 2-AG signa
199 daptations in nucleus accumbens (NAc) medium spiny neurons (MSNs) underlie stress-induced depression-
200 s and paired recordings from CHIs and medium spiny neurons (MSNs) virally overexpressing G-protein-ac
201 y, the membrane excitability of NAcSh medium spiny neurons (MSNs) was decreased at 2 d after withdraw
203 ighly expressed PDE in striatal medium-sized spiny neurons (MSNs) with low micromolar affinity for bo
206 ighly enriched population of striatal medium spiny neurons (MSNs), a neuronal subpopulation that has
207 t majority of projection neurons, the medium spiny neurons (MSNs), and a small yet diverse population
208 NAc, dendritic spine formation on NAc medium spiny neurons (MSNs), and locomotor sensitization to coc
209 tions of projection neurons [striatal medium spiny neurons (MSNs), and neurons in the external segmen
210 rrents in a subpopulation of striatal medium spiny neurons (MSNs), and spontaneous rhythmic bursts of
212 p between direct and indirect pathway medium spiny neurons (MSNs), in addition to characterizing the
213 al projection neurons, known as medium-sized spiny neurons (MSNs), in both the direct and indirect pa
214 te gyrus granule cells or in striatal medium spiny neurons (MSNs), indicating predominant expression
215 ctivity of phasically active neurons [medium spiny neurons (MSNs), presumed projection neurons] and t
216 o form GABAergic synapses on adjacent medium spiny neurons (MSNs), providing feed-forward inhibition
217 ed cell type-specific distinctions in medium spiny neurons (MSNs), the main projection neurons in the
218 a large population of human striatal medium spiny neurons (MSNs), the main target of neurodegenerati
220 eases disynaptic GABAergic input onto medium spiny neurons (MSNs), the major output neurons of the st
221 c integration with cortical inputs to medium spiny neurons (MSNs), thus playing a crucial role in mot
222 gh the functional output of principle medium spiny neurons (MSNs), whereas dysfunctional output of NA
223 dopamine-receptor-expressing striatal medium spiny neurons (MSNs), which are interspersed and electri
224 enhanced in a subpopulation of medium-sized spiny neurons (MSNs), which could dampen striatal output
225 esulting from dysfunction of striatal medium spiny neurons (MSNs), which form the main output project
226 for these neuromodulators in striatal medium spiny neurons (MSNs), which play important roles in plas
227 dopamine receptor (D(1)R)-expressing medium spiny neurons (MSNs), which receive input from dopamine
228 neurons of the striatum are GABAergic medium spiny neurons (MSNs), whose collateral synapses onto nei
229 on and schizophrenia involve striatal medium spiny neurons (MSNs), yet their functional connectivity
230 use a computational network model of medium spiny neurons (MSNs)-fast-spiking interneurons (FSIs), b
251 e principal population of medium-sized dense spiny neurons (MSNs; 97-98% of all striatal neurons in r
252 ce that alterations in striatal medium-sized spiny neurons (MSSNs) giving rise to the direct (D1 rece
255 -soluble proteins highly expressed in medium spiny neurons of striatum that are phosphorylated in res
256 sm of signal transduction enriched in medium spiny neurons of striatum that likely mediates effects o
257 te differently synaptic plasticity in medium spiny neurons of the accumbens direct and indirect pathw
260 ne-induced dendritic spine changes on medium spiny neurons of the NAc and the rewarding effects of co
261 display a loss of dendritic spines in medium spiny neurons of the nucleus accumbens (Nacc) shell, acc
265 terneurons, pyramidal neurons and the medium spiny neurons of the striatum, implicating cortical and
266 hate and is highly expressed in medium-sized spiny neurons of the striatum, making it an attractive t
267 , when expressed in mice in GABAergic medium spiny neurons of the striatum, the beta-arrestin-biased
270 trate that altering cAMP signaling in medium spiny neurons of the ventral striatum can effectively mo
271 receptor-expressing nucleus accumbens medium spiny neurons owing to activation of the melanocortin 4
272 eteroreceptors located on non-DAergic medium spiny neurons participate in the control of DA levels.
274 amically modulated by uncertainty across the spiny neuron population, allowing the system to self-tun
275 terneurons elicited GABAergic IPSPs/IPSCs in spiny neurons powerful enough to significantly delay evo
276 Finally, direct optical activation of medium spiny neurons proved to be capable of supporting self-st
277 ns of striatal projection neurons-the medium spiny neurons-reliably encode locomotion and environment
279 nnected pairs of EGFP-TH(+) interneurons and spiny neurons showed that the interneurons elicited GABA
280 ceptor 2a in the striatum, markers of medium spiny neurons signaling via the indirect pathway, associ
283 in was sufficient to induce pMeCP2 in medium-spiny neurons, suggesting that the combinatorial regulat
285 ride channel into rat dorsal striatal medium spiny neurons, targets of strong dopamine innervation, t
287 10A) is an enzyme present in striatal medium spiny neurons that degrades the intracellular second mes
288 ral model, TANs modulate the excitability of spiny neurons, their population response to reinforcemen
289 recordings were performed in NAc core medium spiny neurons to assess the contribution of CP-AMPAR tra
290 al excitability, and the inability of medium spiny neurons to regulate activity-induced gene expressi
292 of gene expression in dorsal striatum medium spiny neurons-unlike most other modules, which showed no
293 e thalamus and D2-receptor-expressing medium spiny neurons via synaptic insertion of GluA2-lacking AM
295 receptors on nucleus accumbens (NAc) medium spiny neurons where they antagonize D(2) receptor activi
296 tant huntingtin primarily in striatal medium spiny neurons, which highly express phosphodiesterase 10
297 to both D1 and D2 receptor-expressing medium spiny neurons while D2 receptors specifically inhibit ex
298 RIIbeta reexpression in all striatal medium spiny neurons with Darpp32-Cre corrected the hyperlocomo
299 vel observation is that the dendrites of the spiny neurons within all three regions remain confined t
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