ライフサイエンスコーパス: spirometric

1 9-year cumulative incidence for spirometric abnormalities was 41.8% (number at risk: 576

2 alence estimates of respiratory symptoms and spirometric abnormalities were computed, and bivariate a

3 igh in LAM (55%), even in patients with mild spirometric abnormalities, and was correlated with airfl

4 change abnormality in smokers with only mild spirometric abnormalities.

5 frequent in LAM, even in patients with mild spirometric abnormalities.

6 ss disorder [PTSD], and panic disorder), and spirometric abnormalities.

7 improved with cyclosporine, as determined by spirometric analysis (10 events in the cyclosporine grou

8 Results were combined with spirometric and anthropometric measurements.

9 Spirometric and breath-by-breath gas exchange measuremen

10 and poorly controlled HIV infection worsens spirometric and diffusing capacity measurements, and acc

11 Spirometric and IOS (resistance of the respiratory syste

12 Spirometric and IOS indices of airway function were obta

13 All daily spirometric and ventilatory changes declined in magnitud

14 Cross-sectional regression analyses using spirometric, anthropometric, and socioeconomic data were

15 Spirometric assessment included forced expiratory volume

16 A single post-bronchodilator spirometric assessment may not be reliable for diagnosin

17 Anthropometric and spirometric assessments were undertaken.

18 or GLI-defined normal spirometry across GOLD spirometric categories.

19 for Chronic Obstructive Lung Disease (GOLD) spirometric category (1-4) on the basis of post-bronchod

20 are prevented from taking a deep breath, the spirometric changes occurring with aerosol MCh challenge

21 ommon treatment options and how clinical and spirometric characteristics affect outcomes is not well

22 It is also unclear which early spirometric characteristics identify individuals at risk

23 For screening participants with spirometric COPD (n = 6,436), there was a twofold increa

24 tiative for Chronic Obstructive Lung Disease spirometric criteria for lung-function impairment that w

25 et into discrete physiologic groups based on spirometric criteria.

26 severity were defined according to standard spirometric criteria.

27 (20%, 60%, and 100% VC) on the basis of the spirometric data collected from each subject.

28 We retrospectively analyzed spirometric data for 197 single-lung recipients.

29 ue of stage 0-p by retrospectively analyzing spirometric data for 203 adult bilateral lung transplant

30 We have reported normative spirometric data for 3- to 6-year-old children.

31 Spirometric data revealed a mildly diminished FEV(1) and

32 Spirometric data were consistent with higher constrictio

33 Prebronchodilation and postbronchodilation spirometric data were obtained from 560 children in the

34 Acceptable spirometric data were obtained from 728 (58% boys) child

35 cerbations, adverse events, health care use, spirometric data, and high-resolution computed tomograph

36 e for 296 subjects, 188 of whom had complete spirometric data.

37 lung transplant recipients with irreversible spirometric decline and control subjects matched by time

38 th PRM(fSAD) greater than or equal to 30% at spirometric decline lived on average 2.6 years less than

39 y the risk of death in patients with diverse spirometric decline patterns.

40 PRM(fSAD) at spirometric decline was evaluated as a prognostic marker

41 r lung transplant recipients presenting with spirometric decline.

42 and provide prognostic information following spirometric decline.

43 ion-based studies demonstrating incidence of spirometric-defined chronic obstructive pulmonary diseas

44 often patients initially met criteria for a spirometric diagnosis of COPD but then crossed the diagn

45 To determine whether a spirometric diagnosis of mild or moderate COPD is subjec

46 We studied the spirometric effects of albuterol nebulized with heliox d

47 rs or older when examined and who received a spirometric examination.

48 In the treatment group (n = 50) spirometric, Feno, residual volume (RV)/total lung capac

49 imitation was defined as post-bronchodilator spirometric (FEV1 /FVC) ratio <lower limit of normal.

50 Questionnaire responses and spirometric findings in participating workers were compa

51 Spirometric findings, health status, and dyspnea were al

52 studies were performed both without and with spirometric gating by using a spirometer to trigger scan

53 scanning at 90% of vital capacity (group 2, spirometric gating study).

54 tes is high and unlikely to improve by using spirometric gating.

55 ly reported COPD and a significant number of spirometric GWAS loci were at least nominally (P < 0.05)

56 GLI-defined spirometric impairment establishes clinically meaningful

57 e indicator of emphysema in subjects without spirometric impairment.

58 ween subject groups and were correlated with spirometric indexes.

59 llergic rhinitis (AR) and eczema, as well as spirometric indices and sensitization, were examined usi

60 In contrast, most spirometric indices from either the maximal or the parti

61 revealed evidence of gas trapping but normal spirometric indices in the cyst-positive group.

62 tion, measured by reductions in quantitative spirometric indices including forced expiratory volume a

63 ies of childhood respiratory disease whereas spirometric indices such as the FEF(25-75)/FVC ratio are

64 lly determined by reductions in quantitative spirometric indices, including forced expiratory volume

65 P = .03) but not wheezing symptoms, baseline spirometric indices, or response to bronchodilator.

66 When compared with pre-bronchodilator spirometric indices, the post-bronchodilator values demo

67 Raw were correlated with changes in several spirometric indices.

68 y described genome-wide significant COPD and spirometric loci were associated with emphysema or airwa

69 and having a dog or cat) on five measures of spirometric lung function among 8- to 16-year-old subjec

70 The purpose of this study was to evaluate spirometric lung function in normal children ages 3 to 6

71 validated food frequency questionnaire, and spirometric lung function testing.

72 Forced vital capacity (FVC), a spirometric measure of pulmonary function, reflects lung

73 At the last spirometric measurement (mean [+/-SD] age, 26.0+/-1.8 ye

74 lymphangioleiomyomatosis had improvement in spirometric measurements and gas trapping that persisted

75 Of the 14 patients who had full flow-volume spirometric measurements during infancy, 10 had FEF(25-7

76 The spirometric measurements FEV1, FVC, and the ratio FEV1/F

77 tory volume in 1 second (FEV1), representing spirometric measurements performed from childhood into a

78 A mixed-model analysis of longitudinal spirometric measurements that considered multiple risk f

79 We sought to use spirometric measurements to identify patterns of airway

80 Spirometric measurements were obtained at nursery and da

81 understanding the genetics underlying these spirometric measurements will increase our knowledge of

82 on, fraction of exhaled nitric oxide values, spirometric measurements, asthma control, and treatment

83 of obstruction, as defined by using routine spirometric measurements, can identify obstruction pheno

84 uterol, control subjects showed no change in spirometric measurements, lung attenuation, or bronchial

85 iance revealed no significant differences in spirometric measurements, maximal inspiratory pressure,

86 Secondary endpoints included other spirometric measurements, pulmonary exacerbations, and h

87 study, contributing 61,746 quality-screened spirometric measurements.

88 Two loci previously associated with spirometric measures (GSTCD and PTCH1) were related to F

89 mericans [AA]) to identify associations with spirometric measures (post-bronchodilator FEV1 and FEV1/

90 Associations between spirometric measures and FEV(1) decline and mortality we

91 and further suggest that post-bronchodilator spirometric measures are optimal phenotypes for COPD gen

92 Pulmonary function was characterized by the spirometric measures forced vital capacity (FVC) and for

93 Spirometric measures from a subset of 6,425 never-smokin

94 Spirometric measures from two time points were used to c

95 ammatory response to grain dust, we compared spirometric measures of airflow and bronchoalveolar lava

96 Spirometric measures of lung function are heritable trai

97 edications, and lack of reference values for spirometric measures of lung function in many subgroups

98 Spirometric measures of pulmonary function exhibited hig

99 Spirometric measures of pulmonary function have been sho

100 se studies, and its sustained improvement of spirometric measures over the 1 mo of testing in the stu

101 ight, body mass index, and smoking status on spirometric measures were adjusted through linear regres

102 The residual spirometric measures were analyzed for linkage to the ge

103 any patients have substantial improvement in spirometric measures with inhaled bronchodilator medicat

104 tory volume in one second (FEV(1)) and other spirometric measures.

105 tracheal irritation, coughing, or changes in spirometric measures.

106 Associations were also observed for other spirometric measures.

107 Lower thresholds of each spirometric metric were associated with increasing adjus

108 aged by using a 64-detector row scanner with spirometric monitoring at total lung capacity and during

109 imaged with a 64-detector row scanner, with spirometric monitoring at total lung capacity and during

110 th American insulators for whom chest X-ray, spirometric, occupational, and smoking data were collect

111 Spirometric outcomes (FEV(1), forced vital capacity, and

112 of central importance in asthma and proposes spirometric outcomes as core outcomes for all future NIH

113 rea as measured by HRCT and the mean partial spirometric outcomes were highly correlated: FEV(1)p (r(

114 flows, Asthma Control Questionnaire scores, spirometric parameters, peak expiratory flows, blood eos

115 dy, the impact and outcome of an obstructive spirometric pattern identified in transplant recipients

116 disease, and family studies have shown that spirometric phenotypes are heritable.

117 loci reported as genome-wide significant for spirometric phenotypes related to airflow limitation or

118 dentify genetic determinants of quantitative spirometric phenotypes, an autosomal 10-cM genomewide sc

119 We investigated whether differences exist in spirometric pulmonary function in healthy children acros

120 of chronic allograft dysfunction exhibiting spirometric, radiological, and histopathological charact

121 The patient had normal spirometric readings, lung volumes, diffusing capacity,

122 esidual volumes are not detected on standard spirometric readings.

123 Spirometric reference values for Caucasians, African-Ame

124 roid does not preclude a robust clinical and spirometric response to tapering oral prednisone.

125 The prevalence of spirometric restriction was 38.6% using National Health

126 We carried out similar analyses for spirometric restriction, chronic cough and chronic phleg

127 Overall, we found no association of spirometric restriction, chronic cough or chronic phlegm

128 rol Test score, >19/25 or 50% increase), (2) spirometric results (FEV1 >/=80% of predicted value or >

129 FEV1, FEF25-75, and FVC were taken from spirometric results and FEF25-75/FVC ratios were obtaine

130 re classified as having CLAD on the basis of spirometric results and were divided into three groups:

131 to assess asthma control in children because spirometric results are many times normal values.

132 There was no difference in baseline spirometric results between the C-C and C-UC groups, exc

133 Spirometric results worsened most often with LDI, and ma

134 Demographic information, spirometric results, ASUI scores, and other asthma quest

135 When interpreting spirometric results, consideration of the pretest probab

136 Spirometric results, dyspnea, and health-related quality

137 tween the two groups regarding age, sex, and spirometric results, whereas there was more profound hyp

138 re defined using questionnaire responses and spirometric results.

139 CI) 0.26-0.89), which is consistent with the spirometric results.

140 3% of the subjects, whereas 33.9% had normal spirometric results.

141 phan receptor gamma or alpha) increased with spirometric severity, stimulation of lung CD8(+) T cells

142 but neither correlated in concentration with spirometric severity.

143 s method is accurate, it was compared with a spirometric technique.

144 scan, and mixed ventilatory impairment in a spirometric test were revealed.

145 Spirometric, total body plethysmographic, and CT data (a

146 1.13 to 0.85 and improved health status and spirometric values (P<0.001 for all comparisons with pla

147 elation [r(m)] = 0.01, P = .64) or change in spirometric values (range of r(m) values: -0.56 to -0.31

148 ds, univariate analysis demonstrated similar spirometric values and bronchodilator responsiveness in

149 After lung transplantation, spirometric values are routinely followed to assess graf

150 re was a significant but small difference in spirometric values between sitting and standing position

151 n remission differed significantly for all 3 spirometric values compared with the trajectories in tho

152 Reduced spirometric values in first-degree relatives of early-on

153 dy in which we compared sitting and standing spirometric values in obese individuals.

154 Correlations between spirometric values or RA950 and number of pack-years wer

155 equency of exacerbations, health status, and spirometric values were also assessed.

156 or smokers and nonsmokers were compared with spirometric values, diffusing capacity of the lung for c

157 peak to trough) of mean circadian changes in spirometric variables were 2.0-3.2% of the mesor.

158 od chest illness and within-person change in spirometric volumes between age 35 and 45 yr, adjusting