ライフサイエンスコーパス: spleen cell

1 cluding Con A, PMA/ionomycin, and allogeneic spleen cells.

2 leens, which was produced mainly by adherent spleen cells.

3 ed in vitro transforming activity in chicken spleen cells.

4 nsplanted with B10.D2 (H-2d) bone marrow and spleen cells.

5 liver metastasis resulted that contained GFP spleen cells.

6 omplete adjuvant with infusion of allogeneic spleen cells.

7 nhance REL's transforming ability in chicken spleen cells.

8 ne marrow-derived dendritic cells as well as spleen cells.

9 on by adoptive transfer of WT lymph node and spleen cells.

10 e times the number of osteoclasts as control spleen cells.

11 mined by ELISA after in vitro stimulation of spleen cells.

12 ound to be comparable to those of control B6 spleen cells.

13 ed primarily in the cytoplasm of transformed spleen cells.

14 1 alpha(+/+)) or knockout (MIP-1 alpha(-/-)) spleen cells.

15 IL-1beta, IL-6, and TNF-alpha production by spleen cells.

16 ntigen were substantially reduced in TP(-/-) spleen cells.

17 ed in rats immunized by i.p. injection of BN spleen cells.

18 .6 GnRH/lacZ construct is able to express in spleen cells.

19 4 T cells in peptide-stimulated, day 75 p.i. spleen cells.

20 y, stimulated IFN-gamma production in normal spleen cells.

21 tes induced in NOD.scid mice by diabetic NOD spleen cells.

22 f T helper-1 (T(H)1) profile of cytokines by spleen cells.

23 t of long-term IgG-producing bone marrow and spleen cells.

24 vation of MV-specific B cells from MV-immune spleen cells.

25 Th17 cytokine production by Der f-stimulated spleen cells.

26 ic molecule, Bcl-xL, was reduced in PKCtheta spleen cells.

27 ice that were transferred with NF-kappaB-Luc spleen cells.

28 ot affect REL's ability to transform chicken spleen cells.

29 anti-CD154 and transfusion of donor-specific spleen cells.

30 In DNMAML1-transduced spleen cells, a dramatic decrease in MZB cells was prese

31 observed with the OT-1 cells from VPLNs and spleen cells after anti-CD137 injections.

32 c-tolerant)CBA, versus (BALB/c-rejected)CBA, spleen cells after challenge with BALB/c antigen.

33 mitogen-activated protein kinase patterns in spleen cells after treatment, which showed reduced mitoc

34 distinguish between normal brain, liver and spleen cells and between colon cancer and normal colon c

35 rway hyperresponsiveness, cytokine levels in spleen cells and bronchoalveolar lavage fluid (BALF), an

36 LR7 and induced cytokine production in mouse spleen cells and human PBMCs and higher levels of IFN-al

37 f IFN-gamma secretion was observed in murine spleen cells and in human PBMC.

38 n vitro reduced IL-6 production by activated spleen cells and increased expression of TGF-beta in den

39 ts were subsequently exposed to diabetogenic spleen cells and monitored until graft failure.

40 ent and depletion of normal CD4(+) or CD8(+) spleen cells and RT-PCR analysis of selected cytokines i

41 When GFP spleen cells and the RFP cancer cells were coinjected in

42 riving a lacZ reporter gene in both cultured spleen cells and transiently expressing zebrafish.

43 ells (BMCs), bone marrow macrophages (BMMs), spleen cells, and RAW264.7 cells were evaluated by analy

44 In BMMs, spleen cells, and RAW264.7 cells, osteoclast differentia

45 lungs than did mice that received nonimmune spleen cells, and TNF receptor-1 KO mice were not fully

46 oplasm to the nucleus in transformed chicken spleen cells; and (4) that mutations in REL, in addition

47 At the peak of infection, the majority of spleen cells are immature CD71(-)Ter119(+) reticulocytes

48 Furthermore, REL-S525P-transformed chicken spleen cells are more resistant to TNFalpha-induced cell

49 expression in REL-S525P-transformed chicken spleen cells as compared to wild-type REL-transformed ce

50 en compared with autologous donor irradiated spleen cells as control modulators, DBMC significantly i

51 of disease caused by polyclonal diabetogenic spleen cells as well as the highly diabetogenic monoclon

52 ntation and not by immunization with porcine spleen cells, as xenografted mutant mice developed an ef

53 munized subcutaneously with injected C57BL/6 spleen cells at 2 or 8 weeks after grafting, after which

54 positive CD4 and CD8 cells in RSV-stimulated spleen cells at all times p.i., while the percentage of

55 activity and hsp70 synthesis are induced in spleen cells at significantly lower temperatures relativ

56 s restored by cotransfer of normal syngeneic spleen cells at twice the concentration of Skn-immune ce

57 ransferred with Skn-immune cells plus normal spleen cells, but also treated with anti-IL-7 Ab, no lon

58 cyte-derived fibrocytes from wild-type mouse spleen cells, but not from SIGN-R1(-/-) mouse spleen cel

59 ole in the initial transformation of primary spleen cells by v-Rel, although distinct requirements fo

60 es from endogenous cells but that introduced spleen cells can also contribute to islet recovery.

61 ice with an immune adjuvant and semisyngenic spleen cells can reverse the disease but found that sple

62 tudies, transferring Ly5.1(+) unfractionated spleen cell CD4(+) or CD8(+) T cells from dnTGFbetaRII m

63 Finally, Cradd-deficient spleen cells, CD4(+) T cells, and mice respond to T cell

64 a cDNA encoding guinea pig IFN-gamma from a spleen cell cDNA library.

65 as further confirmed by in vitro chondrocyte/spleen cell co-culture assay.

66 horylation of STAT5 in T regulatory cells in spleen cells compared with mice treated with anti-CTLA-4

67 rmine whether IL-7 expression without normal spleen cell cotransfer could modulate lesion development

68 ticoid receptor activation in LPS-stimulated spleen cell cultures in vitro.

69 g exogenous recombinant mouse IL-10 to ACAID spleen cell cultures lacking gammadelta T cells.

70 uanine modification showed activity in mouse spleen cell cultures, in vivo in mice, and in human cell

71 s towards a Th1-type in OVA-sensitized mouse spleen cell cultures.

72 single locus was sufficient to "repolarize" spleen cell cultures.

73 wards a Th1 type in antigen-sensitized mouse spleen cell cultures.

74 st-induced IL-6 in lupus-prone MRL/lpr mouse spleen cell cultures.

75 and concentration-dependent fashion in mouse spleen-cell cultures costimulated with IL-2.

76 pG motif induced cytokine secretion in mouse spleen-cell cultures.

77 re accompanied by similar periodic cycles of spleen cell cytokine secretions and nitric oxide and int

78 were immunized with donor (Dark Agouti, RT1) spleen cells (day -5).

79 e systemic response in allergen-rechallenged spleen cells demonstrated no significant alteration in a

80 Flow cytometry of spleen cells demonstrated recovery of CD4(+) cells to >6

81 cells can reverse the disease but found that spleen cells did not contribute to the observed recovery

82 ice to nonimmune B6 mice, but CD8+ -depleted spleen cells did not protect against infection.

83 noted in the peritoneal cavity tissue site; spleen cells displayed a predominant population of doubl

84 with syngeneic BALB/c (H-2d) bone marrow and spleen cells do not develop disease.

85 ay actively interfere with the function of a spleen cell DST.

86 However, activated CD8 spleen cells elicited rejection of Lewis rat pancreas xe

87 Furthermore, IFN-gamma secretion from mouse spleen cells exposed to a weak TCR signal was increased

88 ravenously with donor BALB/c bone marrow and spleen cells expressing TCRalpha and TCRbeta transgenes

89 ODG activity was detected in spleen cell extracts of wild type age-matched mice, but

90 ing then that B6-Mer(-/-) recipients of bm12 spleen cells failed to develop anti-dsDNA and anti-chrom

91 umns produced more IFN-gamma mRNA than whole spleen cells following stimulation with concanavalin A o

92 The serum specimens for antibodies and spleen cells for memory T cell responses to RSV antigens

93 We report that Itch(-/-) bone marrow and spleen cells formed more osteoclasts than cells from WT

94 Adoptive transfer of BM and spleen cells from allergic donors treated with anti-CD20

95 observed to respond in vivo and in vitro to spleen cells from allogeneic H-2(q) mice, and specificit

96 ct recognition of HLA-A2-derived peptides by spleen cells from allograft recipients was also higher o

97 CII-stimulated spleen cells from anti-C5-treated mice produced lower le

98 Spleen cells from AP-3-deficient mice have a reduced abi

99 rthritis was transferred to SCID mice, using spleen cells from arthritic WT and CCR5(-/-) mice.

100 ment for Ag presentation by B cells, because spleen cells from B cell-deficient mice processed and pr

101 Disease was induced by injection of spleen cells from B10.D2 mice into BALB/c mice deficient

102 SCID mice reconstituted with whole spleen cells from BALB/c donors rejected guinea pig corn

103 In contrast, spleen cells from burn-injured CD4(-/-) mice produced cy

104 Spleen cells from C3H/HeJ and C57BL/10ScNJ LPS-nonrespon

105 Adoptive transfer of spleen cells from CM-immunized mice into naive recipient

106 or to target ratios of 50:, 100:, and 200:1, spleen cells from control rats exhibited greater than 35

107 Spleen cells from CpG-treated neonates produce large amo

108 Spleen cells from day-3 infected TNFR1-deficient mice se

109 Lipopolysaccharide-stimulated spleen cells from EC-SOD-KO mice produced greater levels

110 Spleen cells from exposed animals partially recovered th

111 in PTH-treated co-cultures of osteoblast and spleen cells from Fgf2-/mice compared with Fgf2+/+ mice.

112 marrow cultures or co-cultures of osteoblast-spleen cells from Fgf2-/mice was significantly impaired.

113 pecific T cell proliferation was diminished, spleen cells from gamma-chain(-/-) mice successfully ado

114 Stimulation of ex vivo spleen cells from HIV-1 transgenic mice with TLR4, TLR2,

115 Spleen cells from HIV-1-Tg mice deficient in TLR2 (Tg/TL

116 e vaccine strain (LVS) in a mouse model, and spleen cells from immune mice are often used for immunol

117 Recipients of spleen cells from immunized CD4+ KO mice had 6 times mor

118 We adaptively transferred protection using spleen cells from immunized CD4+ KO mice to nonimmune B6

119 reased levels of IL-23p19 mRNA expression in spleen cells from immunized IL-12Rbeta2(-/-) mice compar

120 An ex vivo cytokine production assay using spleen cells from infected [PE x BR] F2 mice together wi

121 However, adherent spleen cells from infected animals produced more nitric

122 Spleen cells from infected C57BL/6 mice underwent signif

123 Spleen cells from infected MHC II-deficient mice produce

124 mor-induced suppressor cells, we transferred spleen cells from mice bearing progressive MCA205 sarcom

125 immunospot assay results indicated that the spleen cells from mice immunized with a sopB mutant show

126 tometry was used to evaluate bone marrow and spleen cells from mice infected with P. gingivalis and c

127 Spleen cells from mice receiving either form of compleme

128 Cytokine release by spleen cells from mice vaccinated with Hsp60 produced su

129 Spleen cells from mice with progressive EAE produced les

130 However, transfer of spleen cells from mildly arthritic KO donors to SCID hos

131 Thymus and spleen cells from MoMLV-inoculated mice were plated on M

132 not abrogate inhibition of proliferation of spleen cells from MV-infected cotton rats.

133 eta and RANTES mRNA could be detected in the spleen cells from naive animals stimulated with LPS or P

134 ity to B-cell-deficient immune environments, spleen cells from naive C57BL/6 [wild-type (WT)] and B-c

135 Transfer of CD4 but not CD8 spleen cells from naive C57BL/6 mice into Rag2 mice led

136 posed mice suppressed the immune function of spleen cells from normal mice, but not those from knocko

137 In contrast, spleen cells from Pax5(-/-) mice produce as much as five

138 Spleen cells from PG490-88-treated mice could not respon

139 Spleen cells from protein-depleted rats exhibited no gre

140 GFP-specific restimulation of spleen cells from R. typhi(GFPuv)-infected BALB/c mice e

141 Cytotoxic spleen cells from reconstituted mice lysed third-party H

142 mparison of LCV-infected B cells and CD20(+) spleen cells from rhesus monkeys shows increased express

143 Spleen cells from Salmonella-infected T-bet-deficient mi

144 t of IFN-gamma and IL-2 produced by isolated spleen cells from sensitized animals.

145 Spleen cells from SRW-sensitized mice contained a large

146 21 phosphorylation, we reconstituted primary spleen cells from STAT4-deficient mice with wild-type an

147 oliferated robustly in response to IC-pulsed spleen cells from the AM14 RF BCR transgenic mice.

148 tion of IgG spots in the bone marrow and the spleen cells from the antiYIL-6RYtreated mice.

149 Subsequently, the spleen cells from the immunized mice were used as the so

150 Spleen cells from the mice that underwent transplantatio

151 Spleen cells from the sST2-Fc-treated mice produced sign

152 Replication-competent virus was detected in spleen cells from these nonviremic Hsp90 inhibitor-treat

153 TIEG(-/-) mice were cultured with marrow and spleen cells from TIEG(+/+) mice.

154 Spleen cells from TMEV-infected mice were stimulated wit

155 Spleen cells from TMEV-infected SJL/J mice stimulated wi

156 Lymph node and spleen cells from tolerized or control donors were harve

157 Spleen cells from transgenic mice were also transferred

158 immunotherapy revealed that the transfer of spleen cells from tumor-bearing mice severely inhibited

159 rom a WEHI-3B cell challenge after receiving spleen cells from vaccinated mice 1 day before challenge

160 Antigen-stimulated spleen cells from VDR KO mice showed an impaired Th1 cel

161 In contrast, spleen cells from W/W(v) mice infected both in vitro and

162 Adoptive transfer of fractionated spleen cells from wild-type mice to MHC-II KO mice indic

163 Injection of naive mice with apoptotic spleen cells generated by irradiation led to DC changes

164 nt assays, but REL-S471N-transformed chicken spleen cells had increased levels of MnSOD protein as co

165 virus-specific precursor B cells than normal spleen cells, had no significant impact on the course of

166 whether tumor cells escaping J558-B7 immune spleen cells harbor mutations in the P1A epitope.

167 However, the frequencies of spleen cells harboring latent MHV-68 genomes were the sa

168 hibited reactivation of peritoneal cells and spleen cells harvested from mice lacking B lymphocytes,

169 fected peripheral blood mononuclear cells or spleen cells has been shown to be an efficient means of

170 treatment of NOD-scid recipients of diabetic spleen cells, however, hastened the onset of disease, sh

171 Culturing spleen cells in BMP4, SCF under hypoxic conditions resul

172 s significantly lower on CD4(+) and F4/80(+) spleen cells in CD28 peptide-treated mice.

173 to sustain the transformed state of chicken spleen cells in culture, suggesting that direct and spec

174 cultures, were much more effective than bulk spleen cells in expanding the numbers of Tregs.

175 on myeloid cells 1) was upregulated in swine spleen cells in response to S. suis infection.

176 interleukin 4 and interferon gamma by mouse spleen cells in response to the immunizing peptides were

177 polarized the cytokine release of VPLNs and spleen cells in response to tumor antigen toward a type

178 es suffices to malignantly transform primary spleen cells in stringent soft agar assays and produce f

179 rmore, RELDelta424-490-ER transforms chicken spleen cells in the absence of estrogen, but the additio

180 d that antibody production from FVIII-primed spleen cells in vitro were profoundly inhibited in the p

181 e REL more efficient at transforming chicken spleen cells in vitro.

182 ion factor can malignantly transform chicken spleen cells in vitro.

183 shes the ability of REL to transform chicken spleen cells in vitro.

184 ral vector can malignantly transform chicken spleen cells in vitro.

185 t activated p53 accumulated in the nuclei of spleen cells in WT and AhR-null mice after DMBA treatmen

186 ith beta1AR(-/-) bone marrow progenitors and spleen cells, indicating that beta1AR signaling on immun

187 f donor bone marrow and CD4+ T cell-depleted spleen cells induced chimerism without causing GVHD in o

188 tivation of Jurkat T-cells and mouse primary spleen cells induces binding of Ku80 and NF90 to the IL-

189 negative or B-cell receptor (BCR)-transgenic spleen cells into B-cell-deficient and CD8(+) T-cell-dep

190 The injection of spleen cells into immune-compromised mice resulted in fi

191 e responses induced by transfer of B6 CD4(+) spleen cells into irradiated MHC class II disparate B6.C

192 ecific (P25 CD4(+) TCR transgenic) wild-type spleen cells into sanroqueRag1(-/-) mice actually led to

193 AID) Tregs were induced by injecting C57BL/6 spleen cells into the AC of BALB/c mice.

194 port that the transplantation of DBA/2 donor spleen cells into thymectomized MHC-matched allogeneic B

195 muMT(-8) mice that received no spleen cells invariably succumbed to the infection withi

196 eloped by fusion of mouse myeloma cells with spleen cells isolated from an H5N1-virus-immunized mouse

197 Spleen cells isolated from the transgenic mice displayed

198 ansferred with tetAg7/p79(+) T cells and NOD spleen cells, like mice transferred with NOD spleen cell

199 ODN-treated immature DC pulsed with B10 (H2) spleen cell lysate elicited markedly lower proliferative

200 When given as DLI together with wild-type spleen cells, marked expansion of GVH-reactive 2C CD8+ T

201 VCAM-1-dependent spleen cell migration under 2 dynes/cm(2) laminar flow w

202 ther inhibition of concanavalin A-stimulated spleen cells nor keyhole limpet hemocyanin-specific prol

203 A significant increase in spleen cell numbers associated with a striking increase

204 Spleen cells obtained from B. burgdorferi-infected, "art

205 In the autoimmune diabetes model, whole spleen cells obtained from diabetic NOD mice induced aut

206 In contrast, spleen cells obtained from infected, IL-10-deficient C57

207 nerated a combinatorial library derived from spleen cells of a patient with SLE who had just previous

208 eeks of sensitization of recipient mice with spleen cells of donor origin, healthy composite grafts i

209 cells, and recall assays were performed with spleen cells of immunized mast cell-deficient and wild-t

210 Cytokine secretion from spleen cells of mice vaccinated with the encapsulated vj

211 ced using genetic material obtained from the spleen cells of rabbits immunized with UO(2)(2+)-DCP con

212 lated epitope-specific CTLs in both PBLs and spleen cells of transgenic rabbits.

213 while it induced the generation of IL-10 in spleen cells of treated EAE animals.

214 We show not only specific capture of primary spleen cells on protein-DNA microarray spots but also th

215 spleen cells, like mice transferred with NOD spleen cells only, developed diabetes.

216 T cells could be stimulated by either donor spleen cells or recipient APCs.

217 luble Langerin did not bind to lymph node or spleen cells, or keratinocytes as assessed via flow cyto

218 facilitate IFN signaling, as demonstrated in spleen cells overexpressing sIFNAR2a, which displayed qu

219 B6.Sle1 and B6.Nba2 mice were evaluated for spleen cell phenotype, numbers of splenic Ig-secreting c

220 naling events can be detected within a whole spleen cell population and identify a role for LIF in th

221 We monitored spleen cell populations during Salmonella infection and

222 TNF-alpha, IL-1beta, and IL-6 production by spleen cell populations prepared from wild-type (WT), Ra

223 By analyzing unmanipulated whole spleen cell populations we have aimed to mimic this in v

224 environment can be restored with transferred spleen cell preparations containing CD8+ dendritic cells

225 a novel cell-transfer protocol that enables spleen cell priming in the absence of disease.

226 ized mast cell-deficient and wild-type mouse spleen cells produced IFN-gamma and IL-17 in response to

227 timulation, the Matk/CHK(-/-) lymph node and spleen cells produced significantly lower IFN-gamma leve

228 Spleen cell production of interferon-gamma, RANTES, and

229 bs was reduced in the Crry-Tg mice, although spleen cell proliferation in response to collagen type I

230 ter immunization, four self peptides induced spleen cell proliferation of T cells from naive MRL/lpr,

231 ble KO mice demonstrated reduced PG-specific spleen cell proliferation, but the production of Th cyto

232 ry activity, as determined by in vitro mouse spleen cell proliferation, cytokine secretion, and in vi

233 of IgG2a antibody and suppressed CII-induced spleen cell proliferation.

234 ) plus a donor-specific transfusion (DST) of spleen cells prolongs skin allograft survival in mice th

235 MHC-I-deficient male spleen cells provided a source of male Ag that could not

236 mice reconstituted with CD4-depleted BALB/c spleen cells rejected guinea pig corneas in a delayed fa

237 Collagen-specific spleen cell responses in IL-21R.Fc-treated mice were obs

238 L-5 and IL-4]-, or ThIL-17 [IL-17]-producing spleen cells) responses.

239 FACS analysis of mouse spleen cells revealed proinflammatory macrophages expres

240 NK cells isolated from WAS patient spleen cells showed increased expression of DNAM-1 and h

241 elevated in VL plasma, and at pretreatment, spleen cells showed significantly elevated mRNA levels o

242 tration of tolerogenic amylase-coupled fixed spleen cells significantly ameliorated disease severity,

243 to abrogate suppression mediated by tolerant spleen cells so excluding any of these molecules as crit

244 B6 or B6.129S1-IL12rb2(tm1Jm) (B6.IL12R-/-) spleen cells (SpC) in a MHC class II-disparate MLC.

245 ulture supernatant of human PBMCs and murine spleen cells stimulated with anti-CD3 antibody.

246 by naive TCR75 T cells and by nontransgenic spleen cells stimulated with anti-CD3.

247 okine (RANTES) mRNA expression in guinea pig spleen cells stimulated with concanavalin A, lipopolysac

248 pleen cells, but not from SIGN-R1(-/-) mouse spleen cells, suggesting that CD209/SIGN-R1 is required

249 ice lacking B lymphocytes, but not wild-type spleen cells, suggesting that IFN-gamma may inhibit reac

250 t, 80% of muMT(-8) mice injected with normal spleen cells survived and resolved the infection.

251 6 such peptides elicited lymph node cell and spleen cell T cell proliferation in both MRL/lpr (stimul

252 ce showed much less death among their F4/80+ spleen cells than did infected wild-type mice, and they

253 derived from mouse lungs bound less well to spleen cells than wild-type virus.

254 so, infected B6 mice have increased CD11b(+) spleen cells that express interleukin-10 (IL-10).

255 sease in recipients of B6.IL-12R(-/-) CD4(+) spleen cells that received a TNF inhibitor-encoding aden

256 uced by transplanting B10.D2 bone marrow and spleen cells to lethally irradiated BALB/cJ mice, is a m

257 We used spleen cells to stimulate the mixed leukocyte reaction (

258 e ability of encephalitogenic lymph node and spleen cells to transfer EAE.

259 ition of male but not female MHC-I-deficient spleen cells to wild-type syngeneic female EC induced MH

260 treatment of the donor male MHC-I-deficient spleen cells, to inhibit proteasome function, markedly e

261 ecting a donor-specific transfusion (DST) of spleen cells together with blocking alphaCD154 antibody

262 s introduced into the mice by infusion or by spleen cell transfer from recombinant hBDDFVIII-immunize

263 adiation, followed by B10.D2 bone marrow and spleen cell transplantation.

264 nse of lymphocyte-deficient RAG(-/-) mice to spleen cells transplanted from either allogeneic or syng

265 se, whereas mutant mice immunized by porcine spleen cells via i.p. injection failed to do so.

266 6, and monocyte chemoattractant protein 1 by spleen cells was also decreased after diterpenoid therap

267 limpet hemocyanin-specific proliferation of spleen cells was altered after infection with the recomb

268 a, TNF-alpha, and IL-1beta by LPS-stimulated spleen cells was decreased, and IL-10 was increased, in

269 ither CD8 T cells alone or with CD4-depleted spleen cells was ineffective in preventing PAH, whereas

270 N-gamma, TNF-alpha, and IL-1beta in cultured spleen cells was reduced by in vivo treatment with low d

271 measured by gamma interferon production from spleen cells, was comparable in both wild-type and TLR4-

272 Alternatively, NF-kappaB-Luc spleen cells were adoptively transferred into Rag2 -/- m

273 The spleen cells were also GFP positive.

274 antigen in adjuvant and 10 days later, their spleen cells were cultured for 5 to 7 days and the CTL r

275 the expansion of stress BFU-E, but only when spleen cells were cultured in BMP4 + SCF at low-oxygen c

276 Likewise, DCs and not spleen cells were effective in sustaining expression of

277 responses of TFF2-expressing macrophages and spleen cells were examined by cytokine enzyme-linked imm

278 GFP spleen cells were found in the liver metastases that res

279 1-IL-12rb2(tm1Jm) (B6.IL-12R(-/-)) responder spleen cells were found to be comparable to those of con

280 C57BL/6 spleen cells were injected into the anterior chamber (AC

281 Saline (control) or allogeneic CB6F1 spleen cells were injected intravenously into 2CF1 and B

282 Spleen cells were phenotyped, and enzyme-linked immunosp

283 to provoke alloreactivity, because Mer(-/-) spleen cells were recognized by bm12 T cells in MLR.

284 Spleen cells were shown to move from the posterior mesen

285 Thymus and spleen cells were stained with CD1d tetramers that had b

286 en-specific cells were not anergic, although spleen cells were still strikingly anergic.

287 Spleen cells were stimulated in culture with overlapping

288 CD14(+) spleen cells were the main source of IL-27 mRNA, whereas

289 Nonadherent spleen cells were the major source of IL-10 in IOE-infec

290 Injection of MHC-II((-/-)) spleen cells, which can provide CD4(+) T-cell-independen

291 Transfer of BCR-transgenic spleen cells, which contained approximately 10 times few

292 s with BMP-2 supported osteoclastogenesis of spleen cells with a concomitant increase in expression o

293 )D(b-/-) mice reacted in vitro to allogeneic spleen cells with an apparent frequency comparable to co

294 The culture of NOD mouse spleen cells with aPC reduced the secretion of inflammat

295 Nevertheless, treatment of transformed spleen cells with leptomycin B causes wild-type REL and

296 Costimulation of HIV-1 transgenic mice spleen cells with LPS and STF or CpG DNA induced TNF-alp

297 er stimulation of mediastinal lymph node and spleen cells with UV-inactivated measles virus at variou

298 y, displayed strong expression in thymus and spleen cells within lymphocytic cells, moderate expressi

299 fically eliminate viral peptide-pulsed donor spleen cells within minutes after adoptive transfer and

300 Culture of Pax5(-/-) spleen cells yields a population of adherent cells that