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1 lates between MZ and follicular areas of the splenic white pulp.
2 response to chemokines and cannot enter the splenic white pulp.
3 ical location in the peripheral areas of the splenic white pulp.
4 ructures and the maintenance of an organized splenic white pulp.
5 ing epidermal Langerhans cells do not access splenic white pulp.
6 anergic B cells at the T/B interface of the splenic white pulp.
7 s which mononuclear cells cross to enter the splenic white pulp.
8 ning fibers throughout diseased nodes and in splenic white pulp.
9 te S1P(1) and S1P(3) and to migrate into the splenic white pulp.
10 We report a decline in NA nerve density in splenic white pulp (45%) at 15 months of age compared wi
11 was also expressed as a meshlike network in splenic white pulp and in the medullary region of the ly
13 ficient to reduce B cell accumulation in the splenic white pulp and to promote egress of activated T
15 maller accumulations of lymphocytes in their splenic white pulp areas, with no evidence of focal aggr
20 profiles were in marginal zones surrounding splenic white pulp nodules, and only smaller numbers wer
21 o 50% of cells being positive focally in the splenic white pulp of six septic but in no nonseptic pat
22 and p40-expressing DC were both observed in splenic white pulp, p40(+) DC rarely colocalized with ba
25 -dependent induction of Puma occurred in the splenic white pulp, whereas Noxa and Bid were induced in
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