ライフサイエンスコーパス: splenocyte

1 ation in 5-wk-old pups (ex vivo assay of pup splenocytes).

2 creased TNF production in antigen-stimulated splenocytes.

3 psis-adapted human blood monocytes and mouse splenocytes.

4 the proliferation of alloantigen-stimulated splenocytes.

5 by i.p. injection of class II-disparate bm12 splenocytes.

6 mised by immunosuppressive CD71(+) erythroid splenocytes.

7 d and separately cocultured with primary rat splenocytes.

8 red for efficient reactivation of MHV68 from splenocytes.

9 arable to those seen with stimulated UNG(-/-)splenocytes.

10 ), and interleukin-2 (IL-2) were detected in splenocytes.

11 ing and promoted T(H)2 cytokine responses in splenocytes.

12 er adoptive transfer of activated autologous splenocytes.

13 ocytes but did not show any effect on KCa3.1 splenocytes.

14 depletion was confirmed by flow cytometry of splenocytes.

15 NO) and the expression of iNOS in MBP-primed splenocytes.

16 xaggerated production of IFN-gamma from SIRS splenocytes.

17 e proinflammatory cytokines IL-6 and IL-8 in splenocytes.

18 natal mice or after co-culture with neonatal splenocytes.

19 munomodulatory potential using mouse primary splenocytes.

20 on-gamma levels in BALF and in OVA-incubated splenocytes.

21 ease of OmpA-enhanced IFN-gamma by Ag-primed splenocytes.

22 ng to increased interleukin-12 production in splenocytes.

23 d expression of phosphorylated Akt (PAkt) in splenocytes.

24 an sepsis blood leukocytes and murine sepsis splenocytes.

25 al for trypomastigotes but was not toxic for splenocytes.

26 e level of IFN-gamma production in Ag-primed splenocytes.

27 as greatly increased compared with wild type splenocytes.

28 eted population, but was also in sIgkappa(+) splenocytes.

29 d Th17, and Tc17 responses in immunized mice splenocytes.

30 human gamma interferon response in HIS mouse splenocytes.

31 normal as well as proteolipid protein-primed splenocytes.

32 mption of opsonized murine RBCs by double-KO splenocytes.

33 inflammation depends on alpha7nAChR-positive splenocytes.

34 ly reduced CD11b(+) monocyte counts in mouse splenocytes.

35 IL-10 secretion was enhanced by PcRH in splenocytes (235%), macrophages (150%) and in lymphocyte

36 ung leukocytes and in cryptococcal Ag-pulsed splenocytes, 3) diminished IgE production in sera, and 4

37 age, which was associated with increases in splenocyte accumulation and B-cell activation but not wi

38 juvant effect exerted by activated apoptotic splenocytes (ActApoSp) was reduced after immunization wi

39 Splenocytes adoptively transferred from mice with HF, bu

40 ient, B cells plus B-cell-depleted wild-type splenocytes adoptively transferred into B-cell-deficient

41 cing ability of i.v. administered Ag-coupled splenocytes (Ag-SP) in mice, which has been demonstrated

42 ymph nodes and spleen T-cell population, and splenocytes alloantigen responsiveness of graft recipien

43 Ablation of Cul3 in mature CD4(+) splenocytes also resulted in dramatically exaggerated Tf

44 polarization of wild type or CerS6-deficient splenocytes also revealed no defects in the development

45 Itfg2-deficient splenocytes also showed a defect in cell migration in vi

46 M-CSF expression in CD4+ and CD8+ T cells in splenocyte and purified T cell cultures.

47 d the cytokine secretion of CD4+ T cells and splenocytes and altered the cellular profile in the lymp

48 Splenocytes and bone marrow cells were exposed to nicoti

49 Ex vivo studies using splenocytes and bone marrow-derived macrophages revealed

50 evels in supernatants from cultures of mouse splenocytes and dendritic cells, as well as from human g

51 In vitro culture of splenocytes and flow-sorted dendritic cells and T cells

52 B cells from murine splenocytes and from blood samples of healthy donors wer

53 against Francisella tularensis using murine splenocytes and further demonstrated that the relative l

54 f IL-17 and its promoting cytokines in total splenocytes and in both CD4 and CD8 T cells following im

55 natures induced by each adjuvant in vitro in splenocytes and in vivo in muscle and lymph nodes using

56 mice enhanced bone resorption by co-cultured splenocytes and induced interleukin-6, a molecule that p

57 increased production of IL-5 and IL-13 from splenocytes and liver mononuclear cells (MNCs) of infect

58 Splenocytes and lung lymph node cells from sensitized EP

59 ovalbumin-specific IgE, a reduced number of splenocytes and lymph node cells with a decreased number

60 MHV68 established and maintained latency in splenocytes and peritoneal cells but did not reactivate

61 shed T cell activation induced by allogeneic splenocytes and protected allogeneic MIN6 beta cells and

62 Purified B220(+) cells from PA splenocytes and purified CD4(+) cells from naive (NA) sp

63 ation of ISGs in STING N153S fibroblasts and splenocytes and STING N154S SAVI patient fibroblasts.

64 HT2B receptors restored IL-17 secretion from splenocytes and Th17 cell differentiation in Tph1(-/-) m

65 re cocultured for 5 d with irradiated BALB/c splenocytes and then photodepleted (PD).

66 ast, all mice that received untreated primed splenocytes and third-party mice that received PD-treate

67 or no effect on the number of MoMLV-infected splenocytes and thymocytes.

68 ody levels, cytokine release by restimulated splenocytes) and local (infiltration of immune cells int

69 ergen-induced IL-13 and IL-4 production from splenocytes, and inflammatory cell infiltrations in the

70 Antibody levels, cytokine production by splenocytes, and splenic forkhead box P3 (FOXP3)(+) regu

71 ice, cecal ligation and puncture resulted in splenocyte apoptosis and significant lymphopenia after 3

72 d parasite burden and increased induction of splenocyte apoptosis.

73 cytotoxicity assays in which peptide-pulsed splenocytes are killed by CTL in the spleens of immunise

74 es and purified CD4(+) cells from naive (NA) splenocytes are the minimal requirements for the adoptiv

75 n was significantly increased in septic mice splenocytes as compared with shams.

76 uced IFN-gamma/TNF-alpha recall responses by splenocytes, as well as fewer IL-12p70-producing APCs.

77 Same-party mice that received PD-treated splenocytes at the time of transplant lived 100 d withou

78 uppressed proliferation of wild-type C57B/6J splenocytes but did not show any effect on KCa3.1 spleno

79 MPER restimulated, H-2(d)-restricted primed splenocytes by class II-blocking Abs), and failed to neu

80 In vitro restimulation of splenocytes by myelin oligodendrocyte glycoprotein 35-55

81 of the TGF-beta gene, activation of STAT6 in splenocytes by NaB, recruitment of STAT6 to the TGF-beta

82 ion of NaB-induced expression of TGF-beta in splenocytes by small interfering RNA knockdown of STAT6

83 he R144A mutant), flow cytometry analysis of splenocytes by tetramer and intracellular cytokine stain

84 ed a significant impairment in thymocyte and splenocyte CD1d gene and protein expression.

85 Mice were injected with allogeneic splenocytes, CD4(+) T cells, or CD8(+) T cells and treat

86 vo fluorescence in the spleen, and decreased splenocyte cell death.

87 Anti-tumor immune responses were measured in splenocytes collected from mice treated with IL PBS or P

88 une attack initiated by adoptive transfer of splenocytes compared with that of the same area before t

89 ed ITP, transfer of allogeneic MHC-immunized splenocytes completely prevented CD61-induced ITP develo

90 he spleen is a heterogeneous environment and splenocytes comprise multiple cell types.

91 ouse models were used to generate a diseased splenocyte conditioned media (D-SCM) containing immune c

92 tive transfer of VNS-conditioned alpha7nAChR splenocytes conferred protection to recipient mice subje

93 The SHR splenocytes constitutively expressed more RORgammat than

94 addition, treatment of human PBMCs or mouse splenocytes containing destabilizing STAT3 mutations wit

95 Peritransplant infusion of apoptotic donor splenocytes cross-linked with ethylene carbodiimide (ECD

96 In primary splenocyte culture, lipopolysaccharide increased TNF-alp

97 antileishmanial IL-17 cytokine production in splenocyte culture.

98 In addition, splenocytes cultured 24 days after RSD exhibited a prime

99 Splenocytes cultured from female mice exhibited fewer Tr

100 control animals, peanut recall responses in splenocyte cultures from nanoparticle-treated mice showe

101 lavage fluids as well as in supernatants of splenocyte cultures were assessed.

102 IL-2 and IFN-gamma cytokines in restimulated splenocyte cultures.

103 ystem tissues, and in activated T cells from splenocyte cultures.

104 ELISA, as were cytokine recall responses in splenocyte cultures.

105 allenge, despite OVA-specific IgE levels and splenocyte cytokine production in response to OVA stimul

106 of IFN-gamma, IL-6, and IL-17 despite normal splenocyte cytokine secretion.

107 to L. monocytogenes through the promotion of splenocyte death, we examined the effect of C5aR1 on typ

108 the culture supernatant of stimulated mouse splenocytes decreased IFN-gamma concentration.

109 hylepoxyquinomicin, or transferring p50(-/-) splenocytes, decreased CXCR4 expression on CD8(+) T cell

110 ge(-) (Lin(-)) CD117(+) (c-Kit(+)) CX3CR1(-) splenocytes depleted of known APCs were most proficient

111 Human cord blood (CB)-derived or mouse splenocyte-derived DCs were loaded with purified recombi

112 tive in Yop translocation into CHO cells and splenocyte-derived neutrophils and macrophages.

113 ated reduction in neonatal CD71(+) erythroid splenocytes did not alter murine neonatal survival to en

114 nd third-party mice that received PD-treated splenocytes died of lethal GVHD.

115 stimulated BDC-2.5.Ncf1(m1J) CD4 T cells and splenocytes displayed elevated synthesis of Th1 cytokine

116 mice received a whole splenocyte suspension, splenocytes ex vivo depleted of CD25+ cells, or MACS-iso

117 f cytokines from activated IELs but not from splenocytes ex vivo.

118 transfer experiments showed that transgenic splenocytes exhibited attenuated diabetogenic ability.

119 ulate the NKG2D receptor with tumor cells or splenocytes expressing Rae-1.

120 we performed a co-culture assay using mouse splenocytes (expressing PD-L1 receptor PD-1) and murine

121 nflammatory cytokine responses and activated splenocytes for enhanced leishmanicidal activity in asso

122 capsule and adoptively transferred 5 x 10(6) splenocytes from 6-week-old nonobese diabetic mice.

123 When treated with 2-(1-adamantyl)-ethanol, splenocytes from 8 wk-infected BALB/c mice showed signif

124 Treatment with VIPR2 agonist or splenocytes from agonist-treated mice resulted in increa

125 nflammation in mice receiving OVA-sensitized splenocytes from AQP3(-/-) mice compared with wild-type

126 LB/c recipients, with or without transfer of splenocytes from autologous primary recipients.

127 In splenocytes from B cell deficient muMT mice, induction o

128 hed alloHCT using bone marrow (BM) cells and splenocytes from B6 (H-2) donor mice transplanted into B

129 Splenocytes from BALB/c mice immunized with a recombinan

130 Splenocytes from C57BL/6 J (B6) AID(-/-) mice were used

131 ex vivo system for aneuploidy where primary splenocytes from Casp2(-/-) mice were exposed to anti-mi

132 FA-1 inhibition blocked IFN-gamma secretion, splenocytes from CD11a(-/-) mice did not respond to ISG1

133 press the recall antibody response of murine splenocytes from FVIII knockout mice to FVIII in vitro a

134 Studies that used splenocytes from GPR120-deficient mice have confirmed th

135 nked immunosorbent spot [ELISPOT] assays) by splenocytes from IKEPLUS-immunized C57BL/6J mice, we ide

136 portantly, results from adoptive transfer of splenocytes from immunized animals in a Parkinson's dise

137 Furthermore, splenocytes from immunized H(1-4)RKO mice, compared with

138 In addition, splenocytes from infected Cd14(-/-) mice exhibited incre

139 Flow cytometric analysis of splenocytes from infected mice revealed that cellular ex

140 tly lower level than that of nonfractionated splenocytes from LP-BM5-infected mice.

141 accumulation was significantly reduced when splenocytes from mice deficient in NK cells by genetic a

142 ens by coculturing the library proteins with splenocytes from mice immunized with a live-attenuated P

143 Splenocytes from mice vaccinated with chi10057(pYA5199)

144 uman promonocytes during inflammation and in splenocytes from mice with sepsis.

145 e, and IL-6 induction by 17beta-estradiol in splenocytes from naive female mice (p<0.05) suggested th

146 duced markedly lower IFN-gamma production in splenocytes from naive mice.

147 Furthermore, splenocytes from NOD-Alox15(null) mice are unable to tra

148 Moreover, polyclonal splenocytes from nonobese diabetic (NOD) mice transplace

149 or-specific IFN-gamma production compared to splenocytes from PBS-treated mice in both models.

150 y, a significantly decreased T1D transfer by splenocytes from prediabetic NOD donors was observed in

151 Splenocytes from primary and secondary recipients were a

152 IFN-gamma ELISpot analysis of splenocytes from rAAV-K-treated mice indicated reduced r

153 ed more IL-6 ex vivo in response to LPS than splenocytes from sham mice.

154 erferon gamma (IFN-gamma) recall response of splenocytes from T. cruzi-infected mice confirmed that 1

155 MOG-stimulated splenocytes from these mice showed elevated levels of Th

156 Adoptive transfer of splenocytes from ultrasound-treated (but not sham) mice

157 Splenocytes from VACV-WR-infected mice were assayed with

158 ere reconstituted with nonregulatory CD25(-) splenocytes from wild-type (WT) or Tlr9(-/-) mice, AKI w

159 Nonadherent splenocytes from wild-type mice expressed significantly

160 Splenocytes from wild-type mice infected i.v. produced s

161 Splenocytes from wild-type or LAIR-1(-/-) mice were stim

162 patitis, cytokines, antibodies, and Treg and splenocyte function.

163 blot analysis revealed that Notch2HCS mutant splenocytes had increased phospho-Akt and phospho-Jun N-

164 esence of collagen, whereas LAIR-1-deficient splenocytes had no attenuation.

165 d from peripheral blood mononuclear cells or splenocytes harvested from CSC-vaccinated hosts were cap

166 had been preincubated with noradrenaline or splenocytes harvested from stressed mice.

167 in the lungs at later disease stages as were splenocyte IL12/23p40 and IFN-gamma levels following ex

168 epletion of maternal Foxp3(+) cells from pup splenocytes illustrated a substantial role for lactation

169 allogeneic stimulation in vitro, allogeneic splenocyte immunization in vivo, and allogeneic heart tr

170 mediates development and immune function via splenocyte immunohistochemistry analysis in association

171 /-) splenocytes were comparable to wild type splenocytes in proliferation responses, alloreactivity,

172 IL-23 in DCs abolished IL-17 production from splenocytes in response to Ag challenge.

173 ncreased Th1 effector cytokine production by splenocytes in response to myelin oligodendrocyte gp35-5

174 ed expression of the CD161 surface marker on splenocytes in SHRs and normotensive control Wistar-Kyot

175 dramatically reduced cytokine production by splenocytes in vitro and markedly decreased HA-specific

176 potent immunosuppression of Con A-stimulated splenocytes in vitro, even at a 1:320 MSC/splenocyte rat

177 17) production by vaccine antigen-stimulated splenocytes in vitro.

178 Immunization with allogeneic splenocytes in vivo resulted in increased alloreactivity

179 we show that chromatin remodeling can render splenocytes incapable of transferring diabetes into immu

180 flammation induced by injection of activated splenocytes increased Deaf1-Var1 and Srsf10, but not Ptb

181 ng and killing of C jejuni were increased in splenocytes incubated with rapamycin compared with contr

182 and CCR2 expression were also determined in splenocytes incubated with serum obtained from CD36-expr

183 l blood mononuclear cells (PBMCs), and mouse splenocytes incubated without or with chaperone protein

184 ed an increase in nuclear 5-LO expression in splenocytes, indicating enzyme activation after GVHD.

185 ivo culture of cervical lymph node cells and splenocytes, indicating that in allergic mice SD favors

186 and posttransplant (donor) bone marrow, and splenocyte infusion followed by posttransplantation cycl

187 xed with IL-23 in primary cultures of murine splenocytes inhibits IL-23-mediated immune signaling.

188 iated ITP was initiated by transfer of their splenocytes into severe combined immunodeficiency (SCID)

189 experiments showed that MALT1 deficiency in splenocytes is sufficient for EAE resistance.

190 the proliferation and cytokine responses of splenocytes isolated from Met e 1-sensitized Balb/c mice

191 However, splenocytes isolated from mice 24 hours after ultrasound

192 of supernatants from polyclonally stimulated splenocytes isolated from mice experiencing secondary DE

193 Splenocytes isolated from tumor bearing mice treated wit

194 t CD11b(+) cells was not limited to neonatal splenocytes; it also occurred with adult and neonatal bo

195 To address these questions we revisited the splenocyte killing assay, using CTL specific for an epit

196 In primary mouse splenocytes, L-Hcy promoted rIFNgamma-induced inflammato

197 with esxL-expressing M. smegmatis and mouse splenocytes led to down-regulation of IL-2, a key cytoki

198 llowing adoptive transfer of CerS6-deficient splenocytes maybe related to their ability to migrate an

199 Here we show that F4/80(+)/CD11b(low) splenocytes mediate the resistance to DNA-damaging chemo

200 We observed that the splenocyte-mediated apoptosis of cancer cells during co-

201 try using peripheral blood, lymph nodes, and splenocytes obtained from dogs undergoing graft-versus-h

202 set of closely related inflammatory genes in splenocytes of acutely diabetic mice and their repressio

203 Splenocytes of control-diet-fed whey-sensitized donors t

204 By contrast, splenocytes of fish-oil-fed whey-sensitized - but not sh

205 Augmented PGE2 production was also found in splenocytes of infected mice, and administration of EP2

206 nced CD4(+) and CD8(+) T cell populations in splenocytes of mice.

207 Splenocytes of S. aureus-infected mice lost most of thei

208 presence of immunosuppressive mechanisms in splenocytes of S. aureus-infected mice that inhibited th

209 Splenocytes of sham- or whey-sensitized donor mice fed e

210 n in interleukin-17 by in vitro-restimulated splenocytes of TNFR1-deficient mice.

211 Thus, splenocytes of ultrasound-treated mice are capable of mo

212 We examined the impact of CD71(+) erythroid splenocytes on murine neonatal mortality to endotoxin ch

213 nhibiting CXCR4 in AA mice, using CXCR4(-/-) splenocytes or AMD3100, significantly reduced BM infiltr

214 ncubating brain-derived PrP(Sc) with primary splenocytes or cultured macrophage RAW 264.7 cells.

215 EMMPRIN expression in cultures of mouse splenocytes or human PBMCs was elevated upon polyclonal

216 to reactivate from either latently infected splenocytes or PECs.

217 Using splenocytes or purified CD4(+) cells that were sufficien

218 ular LVS growth, we found that IL-6 KO total splenocytes or purified T cells were slightly defective

219 In contrast to splenocytes, peripheral blood leukocytes (PBLs) represen

220 sponse and cytokine profile from reactivated splenocytes, plethysmography after aerosol challenge to

221 ased with age, reaching more than 30% of the splenocyte population at 38 weeks.

222 Splenocytes (presumably splenic monocytes and dendritic

223 s, but not rapid desensitization, suppressed splenocyte proliferation and production of IL-4, IL-5, a

224 The capacity of DC to stimulate allogenic splenocyte proliferation was also enhanced by GUWE treat

225 L-17 triggered a pro-inflammatory state; and splenocyte proliferation was elevated in response to ocu

226 s both chronic and acute exposure suppressed splenocyte proliferation, although viral replication rat

227 rsed the immunosuppressive effect of MSCs on splenocyte proliferation.

228 In anti-CD3-stimulated splenocytes, proliferation and secretion of Th1, Th2, an

229 d the complement receptor 3 (CR3), on murine splenocytes, purified B cells, and human neutrophils.

230 ed splenocytes in vitro, even at a 1:320 MSC/splenocyte ratio.

231 Primary splenocytes readily aggregated and formed osteoclast-lik

232 h the levels of cell death in thymocytes and splenocytes, respectively, as measured by flow cytometry

233 and numbers of IL-17-producing CD4(+) mouse splenocytes, respectively.

234 E formation, as indicated by splenectomy and splenocyte restoration experiments.

235 ECP-treated autologous splenocytes resulted in immune tolerance in the host, in

236 Increasing doses of splenocytes resulted in increased incidence of GVHD in R

237 Transcriptome analyses of Kap1-deleted B splenocytes revealed an up-regulation of PTEN, the enzym

238 brain-infiltrating T lymphocytes, and CD3(+)splenocytes (SCs) of EAE mic, and found that global RISC

239 Reactivated splenocytes secreted higher levels of Th2 cytokines with

240 Their splenocytes secreted more IL-10, had increased arginase

241 Isolated mouse CD90.2(+) splenocytes stimulated in vitro with anti-CD3/anti-CD28

242 n associated with miR-10a in embryonic chick splenocytes subjected to an in vitro miR-10a inhibitor t

243 eased IFN-gamma secretion levels using mouse splenocytes, suggesting that diosgenin may be useful in

244 coculture system, but cultures with T-bet-KO splenocyte supernatants contained less IFN-gamma and inc

245 d, or male mice; undetectable IL-6 levels in splenocyte supernatants from ovariectomized and male mic

246 tumor-infiltrating lymphocytes compared with splenocytes, supporting the localized nature of the intr

247 Recipient mice received a whole splenocyte suspension, splenocytes ex vivo depleted of C

248 onal work showed that cytokine production by splenocytes taken post mortem from patients who died of

249 Finally, by blocking RKIP in wild-type SIRS splenocytes, the IFN-gamma response by CD8(+) Vbeta3(+)

250 CD161a(+)/CD68(+) macrophages in SHR-derived splenocytes, their renal infiltration, and premature hyp

251 ptive transfer of enriched CD71(+) erythroid splenocytes to CD71(+)-reduced animals did not reduce ba

252 cytokine production, and abilities of primed splenocytes to control intracellular LVS growth, we foun

253 Transfer of BALB/c splenocytes to SCID mice conferred rapid disease followi

254 However, the response of splenocytes to sphingosine-1-phosphate was impaired in a

255 trains were also assessed for the ability of splenocytes to transfer diabetes in an adoptive transfer

256 s tolerance in a fully mismatched allogeneic splenocyte transfer model in mice.

257 ) cells in NOD mice or Foxp3(+) T cells from splenocytes transferred into NOD.scid mice did not decre

258 onoclonal antibody (mAb) plus donor-specific splenocyte transfusion (DST) induces alloantigen-specifi

259 4 costimulatory blockade plus donor-specific splenocyte transfusion (DST), although it failed to alte

260 de immune or tolerant (donor-specific BALB/c splenocyte transfusion -/+ anti-CD40L monoclonal antibod

261 BALB/c donor-specific splenocyte transfusion and anti-CD40L monoclonal antibod

262 CD40L mAb in conjunction with donor-specific splenocyte transfusion.

263 Murine splenocytes treated with TAT-Runx1.d190 showed an increa

264 that preemptive infusion of apoptotic donor splenocytes treated with the chemical cross-linker ethyl

265 transfer or diminution of CD71(+) erythroid splenocytes under these experimental conditions suggests

266 y AID and its removal by UNG2 glycosylase in splenocytes undergoing maturation and in B cell cancers.

267 erformed global gene expression profiling of splenocytes using high throughput microarray technology.

268 llergens: T(H)2-type cytokine secretion from splenocytes was decreased, whereas specific IgG(1) and I

269 nd sphingosine-1-phosphate lyase-1 (SPL1) in splenocytes was downregulated.

270 FSE staining, and the expression of GRAIL in splenocytes was measured by immunohistochemistry, real-t

271 MHV68 reactivation from latent splenocytes was not altered in the absence of the vUNG.

272 ction in immunosuppressive CD71(+) erythroid splenocytes, was likely responsible for the reported enh

273 Investigating primary mouse splenocytes, we could demonstrate that MALT1-induced MYC

274 Using antibody-mediated cell sorting of rat splenocytes, we demonstrated that r129 induces migration

275 s previously identified by the evaluation of splenocytes were also found to be differentially express

276 A were transferred into naive mice and their splenocytes were co-cultured with fresh OVA-loaded DCs.

277 etion of alloimmune responses, donor C57BL/6 splenocytes were cocultured for 5 d with irradiated BALB

278 The AID(-/-) splenocytes were comparable to wild type splenocytes in

279 reated BALB/c mice (tolerant Treg cell), and splenocytes were cotransferred into islet transplanted n

280 and NK (natural killer) cell populations in splenocytes were elevated in case of vaccinated mice.

281 PD-treated splenocytes were infused into lethally irradiated BALB/c

282 Splenocytes were isolated and subjected to flow cytometr

283 cytokines in supernatants of OVA-stimulated splenocytes were measured by means of ELISA.

284 L-5 and IL-10, secreted by terpenoid-treated splenocytes were measured using the ELISA method.

285 pheral cytokines from autoantigen-stimulated splenocytes were measured, and central nervous system in

286 tramers, C57BL/6 mice sensitized with BALB/c splenocytes were shown to harbor H-2K(d)-specific IgG(+)

287 L/6 CD61 KO (CD61(-)/H-2(b)) mice, and their splenocytes were transferred into severe combined immuno

288 When nondepleted splenocytes were transferred to induce antibody-mediated

289 In contrast, when B-cell-depleted splenocytes were transferred to induce T-cell-mediated I

290 he combination provided full protection when splenocytes were transferred.

291 and less Th2 cytokine production in exposed splenocyte, were evident in the glycated protein treated

292 eoclastogenesis when cocultured with primary splenocytes, whereas ABCs slightly but significantly pro

293 Adoptive transfer of CerS6-deficient splenocytes, which have significantly decreased levels o

294 ished migration toward S1P in the Pparg(C/-) splenocytes, which impeded lymphocyte egression.

295 a dramatic reduction in all major subsets of splenocytes, which was associated with elevated caspase-

296 Treatment of male splenocytes with an LMP2-selective inhibitor did not red

297 Stimulation of Fng tKO splenocytes with anti-CD3/CD28 beads or LPS gave reduced

298 Incubation of dendritic cells or splenocytes with BMP-4 did not affect lipopolysaccharide

299 Restimulation of splenocytes with the Abeta1-15:DT conjugate resulted in

300 Compared with SCID mice receiving naive splenocytes, within 2 weeks after transfer, the ITP SCID