ライフサイエンスコーパス: spondyloarthritis

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1 e as targets for therapeutic intervention of spondyloarthritis.

2 anded in synovial tissues from patients with spondyloarthritis.

3 ) strongly predisposes to the development of spondyloarthritis.

4 causative in some cases of undifferentiated spondyloarthritis.

5 axis may be involved in the pathogenesis of spondyloarthritis.

6 , diagnosis and management of juvenile-onset spondyloarthritis.

7 s with chronic back pain and suspected axial spondyloarthritis.

8 tis (29% vs 80%, P < .001), undifferentiated spondyloarthritis (58% vs 93%, P < .001) and osteoarthri

9 state of ultrasound imaging with respect to spondyloarthritis and describes some of the limitations

10 With the use of HAART, the prevalence of spondyloarthritis and Diffuse Infiltrative Lymphocytosis

11 allele may contribute to the pathogenesis of spondyloarthritis and its unique phenotype through downs

12 e classification criteria for juvenile-onset spondyloarthritis and magnetic resonance imaging has all

13 ther forms of inflammatory arthritis such as spondyloarthritis and rheumatoid arthritis.

14 ical features and overall diagnoses of axial spondyloarthritis, and consequently significantly affect

15 HLA-B27 transgenic) rats, an animal model of spondyloarthritis, and correlated with disease susceptib

16 tes in the blood and joints of patients with spondyloarthritis, and increased numbers of IL-17A(+)GM-

17 ber of ankylosing spondylitis (AS) and axial spondyloarthritis (axSpA) Registries already in existenc

18 Analyses of undifferentiated spondyloarthritis continue to suggest that chlamydiae, a

19 ous rheumatic syndromes including arthritis, spondyloarthritis, DILS, vasculitides, connective tissue

20 Spondyloarthritis encompasses a group of common inflamma

21 of the interleukin-23/interleukin-17 axis in spondyloarthritis has important therapeutic implications

22 Juvenile-onset spondyloarthritis has variable clinical features that ma

23 rachomatis and C. pneumoniae in induction of spondyloarthritis have not been discussed.

24 5, a proton-sensing receptor associated with spondyloarthritis in genome-wide association studies and

25 Older children with psJIA have features of spondyloarthritis, including relative male preponderance

26 diagnosis of SpA according to Assessment of SpondyloArthritis International Society (ASAS) criteria

27 th at least 20% improvement in Assessment of Spondyloarthritis International Society (ASAS20) respons

28 The publication of the new Assessment of SpondyloArthritis International Society classification c

29 The new Assessment of SpondyloArthritis International Society classification c

30 In 2009, ASAS (Assessment in SpondyloArthritis international Society) association, pu

31 MHC class I allele, HLA-B27, contributes to spondyloarthritis pathogenesis is unknown.

32 with other HLA class I could play a role in spondyloarthritis pathogenesis.

33 terleukin-17 axis plays an important role in spondyloarthritis pathogenesis.

34 rmacokinetics was investigated in a group of spondyloarthritis patients.

35 and may contribute to the development of the spondyloarthritis phenotype.

36 HLA-A3, which is not associated with spondyloarthritis (SpA), is also a ligand for KIR3DL2.

37 t paradigms of psoriatic arthritis (PsA) and spondyloarthritis (SpA).

38 ence for their potential pathogenic roles in spondyloarthritis (SpA).

39 the classification of preradiographic axial spondyloarthritis (SpA).

40 al examination in detecting many features of spondyloarthritis, such as synovitis and enthesitis.

41 at are more characteristic of other forms of spondyloarthritis than rheumatoid arthritis.

42 ities, medication usage, and side-effects in spondyloarthritis underscores the need for combining dat

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