ライフサイエンスコーパス: spongiosa

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1 ese genes in rat femoral metaphyseal primary spongiosa.

2 nd also abolishes development of the primary spongiosa.

3 in trabecular bone formation in the primary spongiosa and a delay in vascular invasion of the early

4 r both skeletal sites, regions of trabecular spongiosa and cortical bone were identified and segmente

5 hs was accompanied by a reduction in primary spongiosa and increased osteoclast surfaces on histomorp

6 on is dramatically diminished in the primary spongiosa and is eventually lost.

7 ric analysis was performed to study alveolar spongiosa and periodontal ligament (PDL) modeling dynami

8 ation induced increased turnover of alveolar spongiosa, and the activity was localized; dramatic esca

9 p had significantly (P <0.05) less calcified spongiosa bone surface, greater periodontal ligament sur

10 sts in either the bone collar or the primary spongiosa but generate ectopic chondrocytes.

11 ed presence of cartilaginous remnants in the spongiosa, confirming a decrease in resorption of the ca

12 ion rate) were three-fold greater, calcified spongiosa decreased by two-fold, and PDL surface increas

13 ue rather than to an increased volume of the spongiosa element of the leaflet itself.

14 All leaflets had variable increases in the spongiosa element within the leaflet itself with some di

15 asionally complete separation of it from the spongiosa element.

16 Primary bone spongiosa formation was also disturbed and was accompani

17 cells of the perichondrium, and the primary spongiosa in fetal growth plates of mice and chickens.

18 ing collagen synthesis) was increased in the spongiosa layer.

19 4 mm; P<0.0001) at 60 days with an increased spongiosa layer.

20 Here we show that MSPCs in primary spongiosa of long bone in mice at late puberty undergo n

21 Histomorphometric analyses at secondary spongiosa of the femur and at metaphysis of the L4 verte

22 Hairy attachment structures (fossula spongiosa), present in the ancestor of Reduvioidea, were

23 enchyme and correlated with expansion of the spongiosa (proteoglycan-rich) region in Adamts5(-/-) val

24 However, the primary spongiosa region of adult Col9a1 mutant mice showed an a

25 mulations and those estimated under infinite spongiosa transport techniques.

26 emonstrated, however, that a close surrogate-spongiosa volume (combined tissues of trabecular bone an

27 e obtained given knowledge of the trabecular spongiosa volume (SV) of the bone site.

28 ly, a predictive model of the total skeletal spongiosa volume (TSSV) would be a clinically useful too

29 Although the utility of spongiosa volume in estimating patient-specific active m

30 Physical sections of interior spongiosa were then taken and subjected to nuclear magne

31 had a defect in the formation of the primary spongiosa with reduced immature osteoid (new bone format

32 cked within an infinite extent of trabecular spongiosa, with no allowance for particle escape to cort

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