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1 he Ipl-null placentas, with expansion of the spongiotrophoblast.
2 rinth layer and significant reduction of the spongiotrophoblast.
3 nta to vascularize and form the labyrinthine spongiotrophoblast.
4 p.d7) die at midgestation and lack placental spongiotrophoblast.
5 sistent with metabolic stress throughout the spongiotrophoblast.
6 ginning at midgestation, specifically in the spongiotrophoblasts.
7 f apoptosis among uterine decidual cells and spongiotrophoblasts.
8 gen cell proliferation, resulting in reduced spongiotrophoblast and decidual layers in the placenta.
9 icate Sox21 specifically in the promotion of spongiotrophoblast and giant cell differentiation and es
10 oblast cells of the labyrinth, as well as in spongiotrophoblast and glycogen trophoblast cells.
11 nt placental morphology, most notably in the spongiotrophoblast and labyrinth layers.
12 sitive trophoblasts, which are indicative of spongiotrophoblast and syncytiotrophoblast differentiati
13 alysis demonstrated placental defects in the spongiotrophoblast and syncytiotrophoblast layers, resul
14 ell differentiation is largely restricted to spongiotrophoblasts and giant cells.
15 ngiotrophoblast layer in some strains, while spongiotrophoblasts and glycogen cells are almost comple
16                           Reduced numbers of spongiotrophoblasts and glycogen trophoblasts were also
17       SOCS3-deficient placentas have reduced spongiotrophoblasts and increased trophoblast secondary
18 grossly altered with the loss of the diploid spongiotrophoblasts and labyrinth and an expansion of th
19 , widespread penetration of the labyrinth by spongiotrophoblasts, and abnormal distribution of tropho
20 fferentiation of trophoblast stem cells into spongiotrophoblasts as opposed to giant cells.
21 d number of glycoproteins synthesized by the spongiotrophoblasts between mid gestation and birth refl
22 expression of IFN-stimulated genes (ISGs) in spongiotrophoblasts but not in other placental cells tha
23 tem is present in the ectoplacental cone and spongiotrophoblast cells.
24 lacentas due to a reduction in the number of spongiotrophoblast, glycogen trophoblast and sinusoidal
25 e gestation and was particularly abundant in spongiotrophoblasts In mouse embryo (day 10.5-18.5), TIM
26 n I variant, only which are expressed in the spongiotrophoblast, late in rat placental development be
27 r two, Plac8 expression is restricted to the spongiotrophoblast layer during development, whereas Pla
28 gfr(wa2) homozygous placentas have a reduced spongiotrophoblast layer in some strains, while spongiot
29 ngiotrophoblast tissue with extension of the spongiotrophoblast layer into the labyrinth.
30  and severely compromises the ability of the spongiotrophoblast layer to expand appropriately, second
31 ant cell differentiation, a reduction of the spongiotrophoblast layer, and an absence of labyrinth fo
32 g Blimp1(+) diploid cells present within the spongiotrophoblast layer.
33 esults in an expansion of the giant cell and spongiotrophoblast layers, and a decrease in labyrinthin
34 centas show greatly reduced labyrinthine and spongiotrophoblast layers, and increased numbers of gian
35 s also altered, including an increase in the spongiotrophoblast marker Mash2 and decreases in the lab
36  Second, a major target of BPA/BPS is either spongiotrophoblast or GCs within the junctional zone.
37                                 In addition, spongiotrophoblast progenitors are not maintained.
38 es led to a decrease in the area occupied by spongiotrophoblast relative to trophoblast giant cells (
39 hich in normal midgestation embryos exhibits spongiotrophoblast-specific maternally active/paternally
40 nlike that of the human placental CSPG, is a spongiotrophoblast-specific protein (SSP), which is expr
41 last giant cells and the underlying layer of spongiotrophoblasts that form the boundary between the m
42 ties included an expanded volume fraction of spongiotrophoblast tissue with extension of the spongiot
43 ed between dopamine(+) GCs and reductions in spongiotrophoblast to GC area ratio.
44 lation between 5-HT(+) GCs and reductions in spongiotrophoblast to GC area suggests that this neurotr