ライフサイエンスコーパス: spontaneous metastasis

1 ) and from subcutaneous tumors in nude mice (spontaneous metastasis).

2 1 in vivo inhibited migration, invasion, and spontaneous metastasis.

3 ignificant reduction of stromal invasion and spontaneous metastasis.

4 han host Gal-1 in promoting tumor growth and spontaneous metastasis.

5 , enabling studies of early tumor growth and spontaneous metastasis.

6 cooperate to complete the entire process of spontaneous metastasis.

7 t primary tumor growth but strongly promotes spontaneous metastasis.

8 rolong animal survival in the AT6.1 model of spontaneous metastasis.

9 uced VP as well as a subsequent reduction in spontaneous metastasis.

10 tributes to tumor implantation, seeding, and spontaneous metastasis.

11 es was examined using an orthotopic model of spontaneous metastasis.

12 those xenografts exhibiting high degrees of spontaneous metastasis.

13 CLC)-derived angiogenesis, tumor growth, and spontaneous metastasis.

14 itor (A2BRi) decreases both experimental and spontaneous metastasis and combines with chemotherapy or

15 efficacious in inhibiting the development of spontaneous metastasis and increased overall tumor-free

16 intravenously form intravascular colonies to spontaneous metastasis and to a carcinoma model system.

17 o induce estrogen-independent growth, induce spontaneous metastasis, and decrease ER levels in breast

18 astatic suppression in vitro as well as in a spontaneous metastasis animal model, indicating that KA1

19 sms in metastasis and angiogenesis, we did a spontaneous metastasis assay using MDA-MB-435 human brea

20 hybrid clones was then tested in a standard spontaneous metastasis assay using SCID mice.

21 ene in the advancement of prostate cancer, a spontaneous metastasis assay was performed in a severe c

22 or including: suppression of metastasis in a spontaneous metastasis assay, promotion of apoptosis fol

23 In a spontaneous metastasis assay, the primary tumor size of

24 etastatic lesions to the lungs in an in vivo spontaneous metastasis assay.

25 Spontaneous metastasis assays using cells ectopically ex

26 truct, and the cells were tested in standard spontaneous metastasis assays.

27 -MB-468) or high (C8161, M24(met)) levels of spontaneous metastasis but no LVI.

28 the host compartment leads to an increase in spontaneous metastasis but not primary tumor growth or n

29 a marked decrease in liver colonization and spontaneous metastasis by LSLiM6 and HM7 cells, whereas

30 und a consistent and reliable improvement in spontaneous metastasis detection.

31 analysis that uses data from mouse models of spontaneous metastasis developing after surgical removal

32 ce after splenic-portal inoculation or after spontaneous metastasis during cecal growth.

33 gest fibrin(ogen) plays an important role in spontaneous metastasis, facilitating the stable adhesion

34 in neither prevented nor changed the rate of spontaneous metastasis formation after surgical removal

35 VEGFR1 activity does not affect the rate of spontaneous metastasis formation in a clinically relevan

36 or CD24 (Cd24a in mice) in tumorigenesis and spontaneous metastasis from the orthotopic site remains

37 ocal disseminated disease (11-12 weeks), and spontaneous metastasis (>20 weeks).

38 toic membrane model of metastasis as well as spontaneous metastasis in a murine model.

39 xamined in culture, in primary sites, and in spontaneous metastasis in chick embryos and nude mice.

40 In an in vivo model of spontaneous metastasis in immunocompromized mice, loss o

41 not captured with previous models, including spontaneous metastasis in particular, and provide a usef

42 Analysis of spontaneous metastasis in stably transfected antisense c

43 of five control cell-inoculated mice showed spontaneous metastasis in the lung, whereas none of the

44 cO4(-), we followed primary tumor growth and spontaneous metastasis in the presence or absence of eto

45 was sufficient to induce CSC properties and spontaneous metastasis in transformed human mammary epit

46 ioned media (TCM) was employed to accelerate spontaneous metastasis in tumor xenografts, and the anti

47 l cancer (CRC) mouse model system to develop spontaneous metastasis in vivo and compare its reproduci

48 d EMT in vitro, and the drugs also inhibited spontaneous metastasis in vivo When fluidity was unchang

49 CDK5 activity was important for spontaneous metastasis in vivo; xenografts of AT6.3 cell

50 ransduction that facilitates early stages of spontaneous metastasis leading to tumor cell intravasati

51 We conclude that spontaneous metastasis may be clonal because they are ra

52 an influential role of EDNRB in CNS melanoma spontaneous metastasis may provide both a target for the

53 In a spontaneous metastasis model originating from footpad in

54 sation, we used the human tumor-chick embryo spontaneous metastasis model to select in vivo high (PC-

55 Similarly, in a spontaneous metastasis model, PAI-1-overexpressing and P

56 at future studies incorporate orthotopic and spontaneous metastasis models (syngeneic and xenogenic)

57 of MB16F0 melanoma and the experimental and spontaneous metastasis models for the mouse pulmonary ca

58 By using orthotopic spontaneous metastasis models in nude mice, we show that

59 M polarization away from the M2 phenotype in spontaneous metastasis models of 4T1 breast cancer and B

60 lts from the experimental metastasis and the spontaneous metastasis models suggests that there are de

61 ty of the NK cell receptor NKp46/NCR1 in two spontaneous metastasis models, the B16F10.9 melanoma (B1

62 Using both experimental and spontaneous metastasis models, we show that genetic abla

63 mental, as well as more clinically relevant, spontaneous metastasis models, we visualize all stages o

64 in the absence of NK cells in both acute and spontaneous metastasis models.

65 on capacities in both chick embryo and mouse spontaneous metastasis models.

66 gnificantly reduced in both experimental and spontaneous metastasis models.

67 and can be applied in both experimental and spontaneous metastasis models.

68 experimental animal model for examining the spontaneous metastasis of bone-homing tumors and indicat

69 196b abrogated in vitro invasion and in vivo spontaneous metastasis of breast cancer cells, indicatin

70 The animal model exhibited spontaneous metastasis of H-RS cells to lymph nodes and

71 ntibody suppressed the orthotopic growth and spontaneous metastasis of highly metastatic, androgen-in

72 Here we report spontaneous metastasis of human prostate cancer xenograf

73 d its degradation products in the growth and spontaneous metastasis of Lewis lung carcinoma was direc

74 Spontaneous metastasis of TAg-positive tumor cells to re

75 tes robust myeloma tumor growth and supports spontaneous metastasis of tumor cells to bone.

76 The growth and spontaneous metastasis of VEGF-expressing tumor cells we

77 and non-EMT cells cooperate to complete the spontaneous metastasis process.

78 ncreatic and renal cell orthotopic models of spontaneous metastasis, targeted delivery of Dox produce

79 nthetic miRNAs, reduced the tumor growth and spontaneous metastasis to bone.

80 yeloma cells dramatically up-regulates their spontaneous metastasis to bone.

81 2 and clonal cell lines derived from it show spontaneous metastasis to lung and regional lymph nodes

82 vasion, intravasation into blood vessels and spontaneous metastasis to lungs.

83 nude mice and assessed for tumor growth and spontaneous metastasis to regional lymph nodes and lungs

84 accompanied by a trend toward a decrease in spontaneous metastasis to the lung.

85 C knockdown inhibits breast tumor growth and spontaneous metastasis to the lungs of mice following ma

86 to mammary fat pads of BALB/c mice exhibited spontaneous metastasis to the lungs, to the peritoneal c

87 nd other human melanoma lines to investigate spontaneous metastasis, we made the observation of marke