ライフサイエンスコーパス: spontaneous recovery

1 Five untreated subjects had spontaneous recovery.

2 ermine the mechanisms mediating and limiting spontaneous recovery.

3 old hyperoxia-treated rats, suggesting slow, spontaneous recovery.

4 neither affect hepatic hemodynamics nor the spontaneous recovery.

5 that the findings could not be attributed to spontaneous recovery.

6 ay 10, the CR was again measured to evaluate spontaneous recovery.

7 n the circulation after treatment-induced or spontaneous recovery.

8 isms downstream of tRNA(Glu) may explain the spontaneous recovery.

9 ear often reappears after extinction through spontaneous recovery.

10 oning and the Immediate group displayed less spontaneous recovery.

11 ty receptor expression) at baseline predated spontaneous recovery.

12 a(2+) experienced sustained swelling without spontaneous recovery.

13 t of sertraline while reducing the chance of spontaneous recovery.

14 frequency and intensity of stimulation, and spontaneous recovery.

15 lockade of reconsolidation does not show any spontaneous recovery.

16 Higher rates of spontaneous recovery (56%) and transplantation (31%) wit

17 ndertaken where there was little evidence of spontaneous recovery, 7-41 months after the initial inju

18 e of extinction when tested 7 d later or its spontaneous recovery after 2 wk.

19 Motor cortical plasticity contributes to spontaneous recovery after incomplete spinal cord injury

20 To investigate mechanisms underlying spontaneous recovery after incomplete spinal cord injury

21 red adult mammalian spinal cord shows little spontaneous recovery after injury.

22 solateral corticospinal pathway in mediating spontaneous recovery after SCI and support a focus on sp

23 tamatergic NMDA receptors is associated with spontaneous recovery after SH and enhanced recovery afte

24 This spontaneous recovery and convergence demonstrated that B

25 More significantly, spontaneous recovery and dishabituation were not as clea

26 Furthermore, immediate extinction can yield spontaneous recovery and prolong fear renewal.

27 nd CD8(+) Treg play an important role in the spontaneous recovery and resistance from reinduction of

28 e inhibitory DREADD receptor hM4Di abrogated spontaneous recovery and resulted in a greater change in

29 This abrogated spontaneous recovery and resulted in a greater change in

30 s, the results remain inconclusive regarding spontaneous recovery and the timing of extinction and ar

31 d a decrement in the CR that did not undergo spontaneous recovery and underwent only modest reinstate

32 equency-dependent decline in responsiveness, spontaneous recovery, and dishabituation by a novel stim

33 Habituation, spontaneous recovery, and dishabituation of the electric

34 rase (TRMU) deficiency, stand out by showing spontaneous recovery, and provide the key to treatments

35 eases cocaine-priming-induced reinstatement, spontaneous recovery, and renewal of cocaine seeking in

36 forms of classical conditioning, we observed spontaneous recovery but not reinstatement of extinguish

37 f women and minorities, and shows infrequent spontaneous recovery, but transplantation affords excell

38 truncal alopecia totalis by 20-23 days, with spontaneous recovery by 28 days.

39 revealed early deficits followed by partial spontaneous recovery by 6 weeks after injury.

40 Animal studies suggest that spontaneous recovery can be abolished if extinction occu

41 Some evidence suggests, however, that spontaneous recovery can be enhanced by modulating neura

42 Spontaneous recovery can occur after incomplete spinal c

43 However, after weeks of spontaneous recovery, considerable reactivation of the h

44 Contextual renewal (Experiment 3) and spontaneous recovery (Experiment 4) of spatial preferenc

45 While myeloid DCs showed spontaneous recovery following HAART in both the groups,

46 /activation-induced cell death (AICD) in the spontaneous recovery from acute disease.

47 Patients with spontaneous recovery from ALF showed significantly highe

48 n liver regeneration and might contribute to spontaneous recovery from ALF.

49 nal APC capacity during our investigation on spontaneous recovery from autoimmune glomerulonephritis

50 Spontaneous recovery from chronic infection is rare and

51 correlates with less severity and more rapid spontaneous recovery from disease in parental B10.PL or

52 rols disease-causing effectors, resulting in spontaneous recovery from disease.

53 long-term antiviral therapy as compared with spontaneous recovery from HBV infection.

54 shold of the T cell response associated with spontaneous recovery from HCV infection, they have impor

55 the importance of nongenetic determinants in spontaneous recovery from HCV infection.

56 and 10 %, respectively and after 2 weeks of spontaneous recovery from immobilization these fibres we

57 amine (d-AMPH) 24 h after injury accelerates spontaneous recovery from this particular deficit.

58 surgically in severe neonatal injures; if no spontaneous recovery has occurred by 3 months, and if ne

59 y myopathy' is an exception because it shows spontaneous recovery if infants survive the first months

60 halomyelitis (EAE), B cells are required for spontaneous recovery in acute models.

61 ccessible might provide deeper insights into spontaneous recovery in humans.

62 The extent of spontaneous recovery in individual animals varied substa

63 ntation, there was a progressive increase in spontaneous recovery in sham-transplanted animals.

64 context-shift participants, and there was no spontaneous recovery in the same-context participants.

65 cur with time after injury, the magnitude of spontaneous recovery is limited.

66 -mediated autoimmune diseases, indicate that spontaneous recovery is mediated by regulatory T cells (

67 gh full recovery is rare, a modest degree of spontaneous recovery is observed consistently in a broad

68 e out of 100,000 children per year, and that spontaneous recovery is typical.

69 This suggests that reinstatement, but not spontaneous recovery, is a preparation- or substrate-dep

70 investigate potential mechanisms underlying spontaneous recovery, lesions of defined components of t

71 These data suggest that partial spontaneous recovery may occur in 6-month-old perinatal

72 al-network models indicate that this form of spontaneous recovery may result from the storage of dist

73 ediction of spontaneous recovery (SR) or non-spontaneous recovery (NSR; death or transplantation).

74 est that FGF2 and GGF2 may contribute to the spontaneous recovery observed after SCI by stimulating t

75 Testing for spontaneous recovery occurred 96 hr after acquisition.

76 otoneurons 7 days post-axotomy and a partial spontaneous recovery occurred by 21 days.

77 aim of enhancing and surpassing the limited spontaneous recovery occurring in animal models and ulti

78 Significant spontaneous recovery occurs after essentially all forms

79 Spontaneous recovery occurs in a minority of patients wi

80 nc., Deerfield, IL) until transplantation or spontaneous recovery occurs.

81 ighboring nodes (external failure) and (iii) spontaneous recovery of a component.

82 of hematin to the inhibited apoenzyme led to spontaneous recovery of about 50% of cyclooxygenase acti

83 We found that this led to spontaneous recovery of B.

84 r increase in the percentage of CD4 T cells, spontaneous recovery of cell-mediated immunity (CMI) was

85 spatially embedded and random networks where spontaneous recovery of components can mitigate spontane

86 opaminergic neurons in the midbrain, exhibit spontaneous recovery of dopaminergic nerve terminals.

87 Rats were subsequently extinguished and spontaneous recovery of drug-seeking behavior following

88 essary for the installation, maintenance, or spontaneous recovery of extinction of CFC.

89 yeblink CRs has never been demonstrated, and spontaneous recovery of extinguished eyeblink CRs has no

90 ol prior to immediate extinction showed less spontaneous recovery of fear during extinction retrieval

91 ited effectiveness and many subjects exhibit spontaneous recovery of fear, as also evident in animal

92 To define the mechanisms mediating spontaneous recovery of function after incomplete SCI, w

93 Such children have a capacity for spontaneous recovery of growth-hormone production on rem

94 to identify the mechanism(s) underlying this spontaneous recovery of hypoxic phrenic responses led us

95 Spontaneous recovery of ipsilateral diaphragm muscle act

96 Although there is an apparent rapid and spontaneous recovery of left ventricular ejection fracti

97 motor cortex, which may underlie some of the spontaneous recovery of motor function seen after injury

98 There is a subsequent spontaneous recovery of normal phloem morphology, which

99 Spontaneous recovery of renal function occurred after er

100 Spontaneous recovery of S1PR1 expression on normal B and

101 ter C(2) hemisection that coincided with the spontaneous recovery of some ipsilateral phrenic functio

102 Development and spontaneous recovery of symptoms in STIA was reflected i

103 In birds, spontaneous recovery of the hair cells of the inner ear

104 ction training, was successful in preventing spontaneous recovery of the memory 24-72 h following ext

105 expression, and matrix cross-linking during spontaneous recovery of up to 366 days.

106 dicted the successful reduction of overnight spontaneous recovery on a day-by-day basis.

107 her deterioration and buy time for potential spontaneous recovery or bridge to liver transplantation.

108 ed hepatic function in ALF allowing time for spontaneous recovery or transplantation.

109 riant was also significantly associated with spontaneous recovery [P = 0.04, OR = 3.51 (1.0-12.5)] in

110 te human post-SCI pathophysiology, including spontaneous recovery processes.

111 n the elevated plus maze test and had higher spontaneous recovery rates from helplessness behavior co

112 roke, the period with the greatest degree of spontaneous recovery, reduce the number of stroke patien

113 1 knockdown was not found to be sensitive to spontaneous recovery, reinstatement, or a shift in the t

114 The molecular mechanisms governing the spontaneous recovery seen following brain injury remain

115 ured by c-fos protein immunoreactivity) when spontaneous recovery (SR) of a conditioned taste aversio

116 Spontaneous recovery (SR) of a CTA (a revival of the tas

117 s were performed regarding the prediction of spontaneous recovery (SR) or non-spontaneous recovery (N

118 sure to the unconditioned stimulus (US)--and spontaneous recovery--the return of an extinguished CR w

119 loss of consciousness and postural tone with spontaneous recovery; the most common form is vasovagal

120 After 2 weeks of spontaneous recovery there still was an average strength

121 This significant, yet incomplete, spontaneous recovery was accompanied by extensive sprout

122 rolonged for context-shift participants, and spontaneous recovery was observed in the same-context pa

123 Spontaneous recovery was observed in three patients; the

124 Spontaneous recovery was remarkably common if support ca

125 Spontaneous recovery was robust in Experiments 3 and 4.

126 To investigate the neural basis of spontaneous recovery, we used kinematic, physiological a

127 s with acute HCV (n = 22), including 10 with spontaneous recovery, were prospectively studied.

128 he remaining three patients made a rapid and spontaneous recovery with no further symptoms suggestive

129 Fifteen participants showed spontaneous recovery within 1-6 months.

130 with aphasia in the acute period experience spontaneous recovery within the first six months after t