ライフサイエンスコーパス: spontaneously hypertensive

1 ry studies showed that Mcoln1(-/-) mice were spontaneously hypertensive.

2 the individual CYP4A genes in the kidneys of spontaneously hypertensive and Wistar-Kyoto rats.

3 ion had minimal effects on the baroreflex in spontaneously hypertensive BPH-2 control mice, which exh

4 prevents ischemic and hemorrhagic stroke in spontaneously hypertensive, genetically stroke-prone (SH

5 S development in obese diabetic Zucker fatty/Spontaneously hypertensive heart failure F1 hybrid (ZSF1

6 Spontaneously hypertensive heart failure myocytes were c

7 measured in 5 groups of rats: (1) obese male spontaneously hypertensive heart failure rats (SHHF) at

8 alazine (a vasodilator) were administered to spontaneously hypertensive heart failure rats between 6

9 action coupling and nitroso-redox balance in spontaneously hypertensive heart failure rats with dilat

10 l NO synthase-deficient (NOS1(-/-)) mice and spontaneously hypertensive heart failure rats, to test t

11 Male, Sprague-Dawley and Spontaneously-Hypertensive-Heart-Failure (SHHF) rats wer

12 remodeling in a model of established HF, the spontaneously hypertensive/HF (SHHF) rat.

13 min and rosiglitazone) in a genetic model of spontaneously hypertensive, insulin-resistant rats (SHHF

14 4A8 mRNA was also found in 3- and 4-week-old spontaneously hypertensive kidneys.

15 Elevated Chga occurs in spontaneously hypertensive rat (SHR) adrenal glands and

16 oad LVH using 2 standard rat models: (1) the spontaneously hypertensive rat (SHR) and (2) aortic band

17 The spontaneously hypertensive rat (SHR) and its normotensiv

18 xygen consumption have been described in the spontaneously hypertensive rat (SHR) and may contribute

19 binant inbred (RI) strains, derived from the spontaneously hypertensive rat (SHR) and normotensive Br

20 organism, mated it with the stroke-resistant spontaneously hypertensive rat (SHR) and performed a gen

21 quantify metabolic substrate shifts with the spontaneously hypertensive rat (SHR) as a model of left

22 for this heightened action of Ang II in the spontaneously hypertensive rat (SHR) brain neurons.

23 regulatory brain areas, was increased in the spontaneously hypertensive rat (SHR) compared to the Wis

24 in was significantly enhanced in the RVLM of spontaneously hypertensive rat (SHR) compared with normo

25 rent at physiological membrane potentials in spontaneously hypertensive rat (SHR) compared with Wista

26 The adult spontaneously hypertensive rat (SHR) has been shown to e

27 The spontaneously hypertensive rat (SHR) has been suggested

28 The spontaneously hypertensive rat (SHR) has been widely use

29 IL-17 family in genetic hypertension in the spontaneously hypertensive rat (SHR) has not been invest

30 factor that stimulates myocytes growth, from spontaneously hypertensive rat (SHR) heart and patients

31 The spontaneously hypertensive rat (SHR) is a model of these

32 In contrast, the spontaneously hypertensive rat (SHR) is highly resistant

33 The spontaneously hypertensive rat (SHR) is insulin resistan

34 The spontaneously hypertensive rat (SHR) is often used as a

35 P-catalyzed EET formation was altered in the spontaneously hypertensive rat (SHR) kidney.

36 Using the spontaneously hypertensive rat (SHR) model of ADHD, we r

37 In the spontaneously hypertensive rat (SHR) model of genetic hy

38 system to safely ablate the CB in vivo in a spontaneously hypertensive rat (SHR) model of hypertensi

39 tatively the cTnI phosphorylation changes in spontaneously hypertensive rat (SHR) model of hypertensi

40 n pre- and early-onset hypertension from the spontaneously hypertensive rat (SHR) model, which repres

41 chronic cardiac injury was evaluated in the spontaneously hypertensive rat (SHR) model.

42 robust in vivo blood pressure lowering in a spontaneously hypertensive rat (SHR) model.

43 We tested the hypothesis that in spontaneously hypertensive rat (SHR) NO produced central

44 fold in renal cortical S9 fractions from the spontaneously hypertensive rat (SHR) relative to the nor

45 middle cerebral artery occlusion (tMCAO) in spontaneously hypertensive rat (SHR) resulted in signifi

46 ng clinically relevant cyclic stretch and in spontaneously hypertensive rat (SHR) retina.

47 The spontaneously hypertensive rat (SHR) strain exists in li

48 Previous studies in the spontaneously hypertensive rat (SHR) support a role for

49 Previous studies in the spontaneously hypertensive rat (SHR) support a role for

50 For this study, we hypothesized that spontaneously hypertensive rat (SHR) vessels should have

51 P expression is significantly reduced in the spontaneously hypertensive rat (SHR) which could contrib

52 and SHR.WKY-Sa) derived from a cross of the spontaneously hypertensive rat (SHR) with the Wistar-Kyo

53 aintenance of elevated blood pressure in the spontaneously hypertensive rat (SHR), a genetic model fo

54 nd maintenance of high blood pressure in the spontaneously hypertensive rat (SHR), a model of primary

55 n pre- and early-onset hypertension from the spontaneously hypertensive rat (SHR), a model that resem

56 The spontaneously hypertensive rat (SHR), a putative animal

57 The spontaneously hypertensive rat (SHR), a widely used anim

58 vents the development of hypertension in the spontaneously hypertensive rat (SHR), an animal model fo

59 In the spontaneously hypertensive rat (SHR), in spite of normal

60 olic heart failure (HF) were examined in the spontaneously hypertensive rat (SHR).

61 rtriglyceridemia and hyperinsulinemia of the spontaneously hypertensive rat (SHR).

62 d to be increased in proximal tubules of the spontaneously hypertensive rat (SHR).

63 when administered orally at 10 mg/kg to the spontaneously hypertensive rat (SHR).

64 ADHD as well as an animal model of ADHD, the spontaneously hypertensive rat (SHR).

65 ain-specific translational regulation in the spontaneously hypertensive rat (SHR/Ola).

66 eart rate (HR), observed in the stroke-prone spontaneously hypertensive rat (SHRSP(HD)), is a primary

67 We used the stroke-prone spontaneously hypertensive rat (SHRSP) as a model organi

68 The stroke-prone spontaneously hypertensive rat (SHRSP) is a genetically

69 ntegrity and wall pathology were examined in spontaneously hypertensive rat and chronic angiotensin I

70 ntegrity and wall pathology were examined in spontaneously hypertensive rat and chronic angiotensin I

71 lodalton protein isolated from hypertrophied spontaneously hypertensive rat and dilated cardiomyopath

72 1a(+) immune cells are dominant in the (SHR) spontaneously hypertensive rat and expand in response to

73 This study explored the spontaneously hypertensive rat as an animal model of pul

74 himeric Wistar-Kyoto rats reconstituted with spontaneously hypertensive rat BM.

75 The spontaneously hypertensive rat can be a model of pulmona

76 in, a hypertrophic activator identified from spontaneously hypertensive rat heart and cardiomyopathic

77 and myocardial microstructure of healthy and spontaneously hypertensive rat hearts at the ages of 12

78 the cortex and outer medulla of 1-4-week-old spontaneously hypertensive rat kidneys relative to the c

79 ins long-term blood pressure regulation in a spontaneously hypertensive rat model over 4 weeks.

80 We have used a spontaneously hypertensive rat model to investigate the

81 either the overload of heart failure (or the spontaneously hypertensive rat model) or the profound un

82 nder Dox-induced cardiotoxic conditions in a spontaneously hypertensive rat model.

83 e rats and transplanted into recipient adult spontaneously hypertensive rat spleens.

84 The increase in blood pressure in spontaneously hypertensive rat was associated with gut p

85 od pressure in the aorta-ligated rat and the spontaneously hypertensive rat when administered orally.

86 reflex-function curves is exaggerated in the spontaneously hypertensive rat where the cardiac compone

87 rom normal (Wistar Kyoto) and hypertrophied (spontaneously hypertensive rat) rats was investigated by

88 hesized that in hypertensive diabetic state (spontaneously hypertensive rat), the CB is sensitized by

89 dependent on the carotid body (CB) input in spontaneously hypertensive rat, a model that also exhibi

90 ion-induced damage than is the kidney of the spontaneously hypertensive rat, and (b) establish the fe

91 an animal model of stroke, the stroke-prone spontaneously hypertensive rat, implicated the gene enco

92 s chronic hypertension develops in the young spontaneously hypertensive rat, medullary Na,K-ATPase ac

93 izes the authors' recent experience with the spontaneously hypertensive rat, the best experimental mo

94 In the MCAO model, conducted in the spontaneously hypertensive rat, the more polar 3-hydroxy

95 y approximately 15 mm Hg, exclusively in the spontaneously hypertensive rat.

96 term reduction in high blood pressure in the spontaneously hypertensive rat.

97 id formation and renal function in the young spontaneously hypertensive rat.

98 Furthermore, antihypertensive effects in spontaneously hypertensive rats (SHR) also revealed that

99 the susceptibility of 5- to 6-month-old male spontaneously hypertensive rats (SHR) and age/sex-matche

100 The development of hypertension in spontaneously hypertensive rats (SHR) and hyperactive vo

101 edback (TGF) activity in 7-wk-old, euvolemic spontaneously hypertensive rats (SHR) and in Wistar-Kyot

102 ne rats (SHR-SP) have more brain injury than spontaneously hypertensive rats (SHR) and normotensive c

103 Spontaneously hypertensive rats (SHR) and normotensive W

104 smutase (ECSOD) reduces arterial pressure in spontaneously hypertensive rats (SHR) and whether its he

105 ght neurobehavioral tests were performed for Spontaneously Hypertensive rats (SHR) and Wistar Kyoto r

106 Cell counts were also made in spontaneously hypertensive rats (SHR) and Wistar-Kyoto (

107 Spontaneously hypertensive rats (SHR) are the most widel

108 myosin light chain kinase (smMLCK) by using spontaneously hypertensive rats (SHR) as an experimental

109 solated carotid body glomus cells from young spontaneously hypertensive rats (SHR) before the onset o

110 mined in DHI, Losartan, and placebo- treated Spontaneously Hypertensive Rats (SHR) by both noninvasiv

111 Ischemic tolerance was induced in spontaneously hypertensive rats (SHR) by injection of a

112 d renal sympathetic nerve activity (RSNA) in spontaneously hypertensive rats (SHR) compared to normot

113 red in distal mesenteric arteries from adult spontaneously hypertensive rats (SHR) compared with norm

114 ion showed greater levels of PRR mRNA in the spontaneously hypertensive rats (SHR) compared with norm

115 gher in the paraventricular nucleus (PVN) of spontaneously hypertensive rats (SHR) compared with thei

116 uscle cells (ASMCs) obtained from adult male spontaneously hypertensive rats (SHR) compared with thos

117 After a period of stable hypertrophy, male spontaneously hypertensive rats (SHR) develop heart fail

118 wever, AMPAR-EPSCs of labeled PVN neurons in spontaneously hypertensive rats (SHR) displayed inward r

119 Experiments used spontaneously hypertensive rats (SHR) during the early d

120 Recent evidence in vivo indicates that spontaneously hypertensive rats (SHR) exhibit an increas

121 We used Spontaneously Hypertensive Rats (SHR) exhibiting many fe

122 Spontaneously hypertensive rats (SHR) had a 90 min middl

123 Spontaneously hypertensive rats (SHR) have an activated

124 nsive strain rats having the highest and the spontaneously hypertensive rats (SHR) having the lowest

125 emitted at a higher pitch by male Wistar and spontaneously hypertensive rats (SHR) in an intensified

126 Spontaneously hypertensive rats (SHR) respond with exagg

127 ied the metabolic effects of pioglitazone in spontaneously hypertensive rats (SHR) that harbor a dele

128 d Ang II-induced inhibition of baroreflex in spontaneously hypertensive rats (SHR) versus WKY rats.

129 Spontaneously hypertensive rats (SHR) were compared to n

130 Male 20-week-old spontaneously hypertensive rats (SHR) were divided into

131 Groups of spontaneously hypertensive rats (SHR) were given 1 mg/kg

132 Spontaneously hypertensive rats (SHR) were treated with

133 al behavior were examined in male and female spontaneously hypertensive rats (SHR), a commonly used a

134 evation of peripheral vascular resistance in spontaneously hypertensive rats (SHR), a glucocorticoid-

135 rted that subjects with ADHD as well as male spontaneously hypertensive rats (SHR), a strain that is

136 KY) rats with borderline hypertension and in spontaneously hypertensive rats (SHR), a widely used gen

137 ng in the PVN in sympathetic overactivity in spontaneously hypertensive rats (SHR), a widely used gen

138 responses are impaired in arterioles of male spontaneously hypertensive rats (SHR), but they are stil

139 In spontaneously hypertensive rats (SHR), cannabinoid-1 rec

140 In spontaneously hypertensive rats (SHR), high NaCl diets i

141 us injection of AdECSOD(R213G) or AdECSOD in spontaneously hypertensive rats (SHR), immunostaining de

142 t regressing left ventricular hypertrophy in spontaneously hypertensive rats (SHR), possibly because

143 intain resting sympathetic vasomotor tone in spontaneously hypertensive rats (SHR).

144 sartan on the progression of renal injury in spontaneously hypertensive rats (SHR).

145 ts and increases smMLCK promoter activity in spontaneously hypertensive rats (SHR).

146 II (Ang II) and associated tissue injury in spontaneously hypertensive rats (SHR).

147 tail artery from normotensive rats (NTR) or spontaneously hypertensive rats (SHR).

148 lar smooth muscle cells (VSMC) isolated from spontaneously hypertensive rats (SHR).

149 Sprague-Dawley (SD), Wistar-Kyoto (WKY), and spontaneously hypertensive rats (SHR).

150 in normotensive (Wistar Kyoto rat, WKY) and spontaneously hypertensive rats (SHR).

151 s been shown to induce ischemic tolerance in spontaneously hypertensive rats (SHR).

152 nteracted the development of hypertension in spontaneously hypertensive rats (SHR).

153 of normotensive Wistar Kyoto rats (WKY) and spontaneously hypertensive rats (SHR).

154 follow the development of chronic changes in spontaneously hypertensive rats (SHR).

155 st the ability to suppress blood pressure in spontaneously hypertensive rats (SHR).

156 12a2 results from altered DNA methylation in spontaneously hypertensive rats (SHR).

157 ment linked to the hypertensive phenotype in spontaneously hypertensive rats (SHR).

158 nsive effect of amaranth protein/peptides on spontaneously hypertensive rats (SHR).

159 inea pigs; and (3) vasoregulatory effects in spontaneously hypertensive rats (SHR).

160 inflammation (SI; at a septic-like model) in spontaneously hypertensive rats (SHR).

161 in a rat model of chronic hypertension, the spontaneously hypertensive rats (SHR).

162 y on BP, cardiac function, and remodeling in spontaneously hypertensive rats (SHR).

163 ransplanted into unilaterally nephrectomized spontaneously hypertensive rats (SHR-RT1.N strain) that

164 in response to increased flow, stroke-prone spontaneously hypertensive rats (SHR-SP) exhibited a sma

165 Female spontaneously hypertensive rats (SHR; age, 4 months) wer

166 onducted on 4- to 5-week-old prehypertensive spontaneously hypertensive rats (SHRs) and age-matched n

167 N-mediated actions of Ang II are enhanced in spontaneously hypertensive rats (SHRs) and contribute to

168 aimed to observe effects of BSJYD on LVH in spontaneously hypertensive rats (SHRs) and explore its p

169 sparks in ventricular cells from 6-month-old spontaneously hypertensive rats (SHRs) and from age- and

170 eurons isolated from the stellate ganglia of spontaneously hypertensive rats (SHRs) and their normote

171 corded in spinally projecting PVN neurons in spontaneously hypertensive rats (SHRs) and Wistar-Kyoto

172 bers from normotensive Wistar-Kyoto rats and spontaneously hypertensive rats (SHRs) by cytofluorimetr

173 In pre-hypertensive (PH) and adult spontaneously hypertensive rats (SHRs) carotid body type

174 ed hypoxia-related proteins in the testes of spontaneously hypertensive rats (SHRs) compared to Wista

175 n increased level in the PVN tissue in adult spontaneously hypertensive rats (SHRs) compared with age

176 ker, candesartan, was orally administered to spontaneously hypertensive rats (SHRs) for 40 days, foll

177 a larger amplitude and shorter decay time in spontaneously hypertensive rats (SHRs) than in Wistar-Ky

178 Two cohorts of Spontaneously Hypertensive Rats (SHRs) were exposed to 1

179 Immune-competent spontaneously hypertensive rats (SHRs) were implanted wi

180 vior and social interaction were examined in spontaneously hypertensive rats (SHRs), a commonly used

181 tivity of spinally projecting PVN neurons in spontaneously hypertensive rats (SHRs), but not in normo

182 In spontaneously hypertensive rats (SHRs), high ABP is asso

183 not different between Wistar-Kyoto (WKY) and spontaneously hypertensive rats (SHRs), renal ETBR phosp

184 These effects are exaggerated in spontaneously hypertensive rats (SHRs), resulting in an

185 study, we examined conditioned inhibition in spontaneously hypertensive rats (SHRs), the most well-va

186 on NTS baroreceptor second-order neurons in spontaneously hypertensive rats (SHRs).

187 dritic spine density are increased in NTS of spontaneously hypertensive rats (SHRs).

188 labelled spinally projecting PVN neurons in spontaneously hypertensive rats (SHRs).

189 ex function in Wistar-Kyoto (WKY) but not in spontaneously hypertensive rats (SHRs).

190 ta(1)-ARs as well as lower blood pressure in spontaneously hypertensive rats (SHRs).

191 ist improves the baroreceptor reflex gain in spontaneously hypertensive rats (SHRs).

192 tivity of PVN presympathetic neurons in male spontaneously hypertensive rats (SHRs).

193 kDa (LFH <3 kDa) were orally administered to spontaneously hypertensive rats (SHRs).

194 ing shift that diminishes GABA inhibition in spontaneously hypertensive rats (SHRs).

195 VN neurons and sympathetic vasomotor tone in spontaneously hypertensive rats (SHRs).

196 icroangiopathy in salt-loaded, stroke-prone, spontaneously hypertensive rats (SHRSP) without controll

197 in male and female 16 week old stroke prone spontaneously hypertensive rats (SHRSP).

198 blood pressure lasting for four-six hours in spontaneously hypertensive rats after a single dose of T

199 al tumor incidence and origin in a cohort of spontaneously hypertensive rats aged 20-24 months (SHRs;

200 duced a long-term antihypertensive effect in spontaneously hypertensive rats and antifibrotic effects

201 DDR2-integrin-beta1 link was also evident in spontaneously hypertensive rats and DDR2-knockout mice.

202 , purified, and sequenced from the hearts of spontaneously hypertensive rats and dilated cardiomyopat

203 ertensive rats (BHR), the first offspring of spontaneously hypertensive rats and normotensive Wistar-

204 ivation, attenuated hypertension in both the spontaneously hypertensive rats and the chronic angioten

205 Fetal hepatocytes were harvested from spontaneously hypertensive rats and transplanted into re

206 manner, with similar levels of inhibition in spontaneously hypertensive rats and Wistar-Kyoto rats; m

207 antly larger increases in MAP and HR in male spontaneously hypertensive rats as compared to normotens

208 ide hydrolase (sEH) reduce blood pressure in spontaneously hypertensive rats as well as the findings

209 ide antihypertensive effect was evaluated in spontaneously hypertensive rats at an oral dose of 10 mg

210 with blood pressure returning to baseline in spontaneously hypertensive rats by three days after trea

211 upregulated or downregulated in 12-week-old spontaneously hypertensive rats compared to normotensive

212 mmunohistochemical staining of the aortae of spontaneously hypertensive rats demonstrated strong corr

213 Wistar Kyoto rat BM, the resultant chimeric spontaneously hypertensive rats displayed significant re

214 l ester was given by daily s.c. injection to spontaneously hypertensive rats exposed to filtered air

215 The right MCA was occluded in spontaneously hypertensive rats for 0, 60 and 120 min.

216 (AT(1)R-AS) in a retroviral vector prevents spontaneously hypertensive rats from developing hyperten

217 (ii) Spontaneously hypertensive rats have more orexin neurons

218 Spontaneously hypertensive rats received either bilatera

219 f the cationic peptides (200mg/kgbodywt.) to spontaneously hypertensive rats resulted in a more rapid

220 m channel blocker, SNX-111, was evaluated in spontaneously hypertensive rats subjected to 60 min of f

221 greater renal vasoconstriction in 7-wk-old, spontaneously hypertensive rats than in Wistar-Kyoto rat

222 Here, in ventricular myocytes from spontaneously hypertensive rats that develop heart failu

223 e used an animal model of ischemic stroke in spontaneously hypertensive rats to determine whether or

224 Male spontaneously hypertensive rats underwent occlusion of t

225 blood pressure reduction in both normal and spontaneously hypertensive rats via interactions with th

226 ithelial cells derived from Wistar-Kyoto and spontaneously hypertensive rats were grown to confluency

227 RF-RDN (n=14) or sham-RDN (n=14) treatment, spontaneously hypertensive rats were subjected to 30 min

228 Halothane-anesthetized spontaneously hypertensive rats were subjected to middle

229 Spontaneously hypertensive rats were treated with 2, 3-d

230 Five-day twice daily treatment of spontaneously hypertensive rats with TMinh-23 produced s

231 MT- 3 infusion was also investigated in SHR (spontaneously hypertensive rats).

232 lly, AM404 reduced hyperactivity in juvenile spontaneously hypertensive rats, a putative model of att

233 n distal middle cerebral artery occlusion in spontaneously hypertensive rats, a strain representative

234 In contrast, in spontaneously hypertensive rats, administration of C-21

235 nd we have described disordered breathing in spontaneously hypertensive rats, an animal model of gene

236 enase and the development of hypertension in spontaneously hypertensive rats, and 2) a relationship b

237 sympathetic stellate ganglion of Wistar and spontaneously hypertensive rats, and human-induced pluri

238 n myotrophin-induced cardiac hypertrophy, in spontaneously hypertensive rats, and in dilated cardiomy

239 After BM ablation in spontaneously hypertensive rats, and reconstitution with

240 asting (~6 hours) antihypertensive effect in spontaneously hypertensive rats, associated with a signi

241 recording in freely moving normotensive and spontaneously hypertensive rats, high-resolution echocar

242 tolic blood pressure (Delta=48+/-5 mm Hg) in spontaneously hypertensive rats, indicating that SNX5 de

243 d 20-HETE formation in renal microsomes from spontaneously hypertensive rats, it has been proposed th

244 In older spontaneously hypertensive rats, it reduced left ventric

245 In spontaneously hypertensive rats, MGR and HDCF effectivel

246 efective in prehypertensive and hypertensive spontaneously hypertensive rats, presenting a potential

247 In anesthetized spontaneously hypertensive rats, the middle cerebral art

248 After oral administration to spontaneously hypertensive rats, the S1P1 selective comp

249 enteric resistance arteries of wild-type and spontaneously hypertensive rats, with a greater inhibiti

250 antagonist SC51322 reduced blood pressure in spontaneously hypertensive rats.

251 al cerebral ischemia and 24 h reperfusion in spontaneously hypertensive rats.

252 e similar in the kidneys of Wistar-Kyoto and spontaneously hypertensive rats.

253 cle in vitro and decreases blood pressure in spontaneously hypertensive rats.

254 ng mice, mice with diet-induced obesity, and spontaneously hypertensive rats.

255 ertensive rats, with a greater inhibition in spontaneously hypertensive rats.

256 mean arterial pressure (MAP) in anesthetized spontaneously hypertensive rats.

257 voke a required restoration of MAP levels in spontaneously hypertensive rats.

258 prehypertensive and 10-week-old hypertensive spontaneously hypertensive rats.

259 ificantly upregulated compared with sham-RDN spontaneously hypertensive rats.

260 pressure in response to vasoconstrictors in spontaneously hypertensive rats.

261 eported for insulin resistance and BP in the spontaneously hypertensive rats.

262 Obese ZSF1 (Zucker fatty and spontaneously hypertensive) rats were randomized to stan

263 ratory reflex conditions and elevated in the spontaneously hypertensive (SH) rat - and yet the exact

264 attenuates the development of high BP in the spontaneously hypertensive (SH) rat model of human essen

265 normotensive Wistar-Kyoto (WKY) rat and the spontaneously hypertensive (SH) rat to inhalation and in

266 tablishment of the hypertensive state in the spontaneously hypertensive (SH) rat.

267 Wistar-Kyoto normotensive (WKY) and spontaneously hypertensive (SH) rats implanted with radi

268 a indices were also noted in prehypertensive spontaneously hypertensive (SH) rats, which are known to

269 amus and brainstem of Wistar Kyoto (WKY) and spontaneously hypertensive (SH) rats.

270 in normotensive Wistar-Kyoto (WKY) rats and spontaneously hypertensive (SH) rats.

271 Male healthy Wistar Kyoto (WKY), spontaneously hypertensive (SH), and SH heart failure (S

272 flex is known to be hyper-responsive in both spontaneously hypertensive (SHR) and Goldblatt hypertens

273 re isolated from 18- to 24-month-old failing spontaneously hypertensive (SHR) or age-matched Wistar-K

274 c transport kinetics in male young and adult spontaneously hypertensive (SHR) rats compared with age-

275 ECS refractoriness in Wistar-Kyoto (WKY) and spontaneously hypertensive (SHR) rats which vary in aden

276 in Wistar-Kyoto (WKY), Sprague-Dawley (SD), spontaneously hypertensive (SHR), and deoxycorticosteron

277 of the cerebellar fastigial nucleus (FN) in spontaneously hypertensive (SHR), Wistar-Kyoto (WKY) and

278 development was assessed in male and female spontaneously hypertensive (SHR), Wistar-Kyoto (WKY), an

279 The stroke-prone spontaneously hypertensive (SHRSP) rat is a model of hum

280 tion of the blood-brain barrier (BBB) in the spontaneously hypertensive stroke prone rats (SHRSP), an

281 and blood-brain barrier (BBB) disruption in spontaneously hypertensive stroke prone rats (SHRSP).

282 a, and risk factors of metabolic syndrome in spontaneously hypertensive stroke-prone (SHRSP) and Wist

283 antan-1-yl-ureido) dodecanoic acid (AUDA) in spontaneously hypertensive stroke-prone (SHRSP) rats pro

284 In spontaneously hypertensive stroke-prone (SP) rats receiv

285 The spontaneously hypertensive stroke-prone rat (SHRSP) is a

286 We tested the following hypotheses: a) spontaneously hypertensive stroke-prone rats (SHR-SP) ha

287 and predisposition to metabolic syndrome in spontaneously hypertensive stroke-prone rats.

288 9/9) and 36/14 (+/- 4/3), respectively, for spontaneously hypertensive Wistar Kyoto rats (SHR), and

289 red among 4 strains of rats: Sprague-Dawley, Spontaneously Hypertensive, Wistar Kyoto (WKY), and Brow