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1 ensitivity of 80 to 90% for the diagnosis of sporadic Creutzfeldt-Jakob disease.
2 enerally symmetric pattern of involvement in sporadic Creutzfeldt-Jakob disease.
3 ) diffusivities in the total white matter in sporadic Creutzfeldt-Jakob disease.
4 wed by the correct histological diagnosis of sporadic Creutzfeldt-Jakob disease.
5 xist in the most common human prion disease, sporadic Creutzfeldt-Jakob disease.
6 erentiating autoimmune encephalopathies from sporadic Creutzfeldt-Jakob disease.
7 tients had either inherited prion disease or sporadic Creutzfeldt-Jakob disease.
8 incorporated into the diagnostic criteria of sporadic Creutzfeldt-Jakob disease.
9 rebellar abnormalities that closely resemble sporadic Creutzfeldt-Jakob disease.
10 es and some have met diagnostic criteria for sporadic Creutzfeldt-Jakob disease.
11 ted individuals resembled those of classical sporadic Creutzfeldt-Jakob disease.
12 (Sc) exists both between and within cases of sporadic Creutzfeldt-Jakob disease.
13 olecular basis for a novel classification of sporadic Creutzfeldt-Jakob disease.
14 blot and immunohistochemistry in 19 cases of sporadic Creutzfeldt-Jakob disease.
15 ting observed in experimental, acquired, and sporadic Creutzfeldt-Jakob diseases.
16 zfeldt-Jakob disease (15 of 15 with definite sporadic Creutzfeldt-Jakob disease, 13 of 14 with probab
17 is detectable at low levels in some cases of sporadic Creutzfeldt-Jakob disease and conversely, that
18 ble spongiform encephalopathies (variant and sporadic Creutzfeldt-Jakob disease and genetic forms of
19 forms found in all of the major subtypes of sporadic Creutzfeldt-Jakob disease and in a case of iatr
21 ples from patients with and patients without sporadic Creutzfeldt-Jakob disease and tested them using
22 zfeldt-Jakob disease, 13 of 14 with probable sporadic Creutzfeldt-Jakob disease, and 2 of 2 with inhe
23 al PrP aggregates derived from patients with sporadic Creutzfeldt-Jakob disease are taken up and degr
24 the prion, in brain samples from humans with sporadic Creutzfeldt-Jakob disease, as well as in rodent
25 rogeneous and may be clinically mistaken for sporadic Creutzfeldt-Jakob disease because of a negative
26 of abnormal prion protein that characterizes sporadic Creutzfeldt-Jakob disease can be found in certa
27 ting on PrP(Sc) aggregates extracted from 60 sporadic Creutzfeldt-Jakob disease (CJD) and 6 variant C
28 Fourth, protease-resistant PrPSc cores in sporadic Creutzfeldt-Jakob disease (CJD) and Gerstmann-S
29 that prions from the brains of patients with sporadic Creutzfeldt-Jakob disease (CJD) bind to very lo
32 ted elk and deer with those in subjects with sporadic Creutzfeldt-Jakob disease (CJD), as well as CJD
33 els of the most common human prion disorder, sporadic Creutzfeldt-Jakob disease (CJD), in which prion
35 these were classified as either familial or sporadic Creutzfeldt-Jakob disease (CJD); there was no c
36 tensor imaging to study the white matter in sporadic Creutzfeldt-Jakob disease compared to healthy c
38 EEG has long been used in the diagnosis of sporadic Creutzfeldt-Jakob disease; however, the charact
39 eases cohorts (FTLD, ALS, Alzheimer disease, sporadic Creutzfeldt-Jakob disease, Huntington disease-l
41 ings from olfactory epithelium in diagnosing sporadic Creutzfeldt-Jakob disease in living patients.
44 o distinct disease phenotypes, identified as sporadic Creutzfeldt-Jakob disease (M/M2 sCJD) and spora
45 ity within the white matter of patients with sporadic Creutzfeldt-Jakob disease, mainly in the left h
46 ase (n = 59) and compare these with cases of sporadic Creutzfeldt-Jakob disease (n = 170) in the Unit
47 iseases can occur as an idiopathic disorder (sporadic Creutzfeldt-Jakob disease) or can be acquired,
48 eas extracts from the brains of familial and sporadic Creutzfeldt-Jakob disease patients produced the
50 of PRNP thought to confer susceptibility to sporadic Creutzfeldt-Jakob disease (rs1029273), all pati
51 list prion clinic who were suspected to have sporadic Creutzfeldt-Jakob disease (sCJD) and yet were f
52 Current cerebrospinal fluid (CSF) tests for sporadic Creutzfeldt-Jakob disease (sCJD) are based on t
53 oradic fatal insomnia (sFI) and a subtype of sporadic Creutzfeldt-Jakob disease (sCJD) identified as
56 e CSF RT-QuIC differentiated 94% of cases of sporadic Creutzfeldt-Jakob disease (sCJD) MM1 from the s
57 hological studies confirmed the diagnosis of sporadic Creutzfeldt-Jakob disease (sCJD) MM1 subtype.
59 in misfolding, the most common subtype being sporadic Creutzfeldt-Jakob disease (sCJD), a rapidly pro
60 sfolding, with the most common subtype being sporadic Creutzfeldt-Jakob disease (sCJD), a rapidly pro
61 ly distinct subtypes have been identified in sporadic Creutzfeldt-Jakob disease (sCJD), based on the
62 cts from brains of two patients, who died of sporadic Creutzfeldt-Jakob disease (sCJD), contained eit
64 ans on bound PrP species present in control, sporadic Creutzfeldt-Jakob disease (sCJD), or variant CJ
65 served in the heart tissue of a patient with sporadic Creutzfeldt-Jakob Disease (sCJD), the most comm
70 mean diffusivity within the white matter in sporadic Creutzfeldt-Jakob disease, suggesting possible
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