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1 ortex (possibly as a consequence of cortical spreading depression).
2 round 30-fold smaller than that of a wave of spreading depression).
3 thermia may be due to a reduced incidence of spreading depression.
4 e feedback in the generation of seizures and spreading depression.
5  time scale of 3 min may have been caused by spreading depression.
6 rea of the brain surface is known to trigger spreading depression.
7 l as during pathological conditions, such as spreading depression.
8 f neurons that showed activation by cortical spreading depression.
9 er loss of function, and higher incidence of spreading depression.
10 h as an increased susceptibility to cortical spreading depression.
11 op severe and prolonged motor deficits after spreading depression.
12 he trigemino-vascular system and in cortical spreading depression.
13 neuronal excitability that leads to cortical spreading depression.
14 ding hearing, vision, epilepsy, and cortical spreading depression.
15 centration of glutamine (2-5 mM) resulted in spreading-depression.
16 Minocycline had no effect on astrogliosis or spreading depression, a wave of ionic transients thought
17 rst time it has been shown that the cortical spreading depression activated the trigeminovascular sys
18                                              Spreading depression activates neurophysiological pathwa
19 neurovascular dysfunction and evoke cortical spreading depression, an event that is widely thought to
20 cance to migraine of events such as cortical spreading depression and activation of the trigeminovasc
21 ical hypoperfusion responses during cortical spreading depression and alpha-chloralose anaesthesia.
22 tic glutamate receptors, which occurs during spreading depression and ischemic depolarization.
23 e for the occurrence of spontaneous cortical spreading depression and peri-infarct depolarizations in
24 ited increased susceptibility to subcortical spreading depression and reverberating spreading depress
25 flurane anesthesia showed that both cortical spreading depression and terminal anoxic depolarization
26 te MCA occlusion is caused by either or both spreading depression and transient ischemic depolarizati
27      We argue that the dynamics of seizures, spreading depression, and anoxic depolarization lie alon
28 le of glial K buffering against seizures and spreading depression, and provides novel insights into a
29 n other slices, APC was emulated by inducing spreading depression (as determined by a negative DC shi
30 ological ceiling that separates seizure from spreading depression, as well as predicted a second ceil
31 se gain-of-function effects lead to cortical spreading depression, aura, and potentially migraine.
32 ntral) trigeminovascular neurons by cortical spreading depression, but not their activation from the
33 s are the first to demonstrate that cortical spreading depression can be blocked in vivo using single
34                                       Leao's spreading depression can be observed in clinically stand
35                                     Cortical spreading depression caused long-lasting blood-flow enha
36        Migrainous aura is caused by cortical spreading depression (CSD) - a slowly advancing wave of
37   However, the relationship between cortical spreading depression (CSD) and headache has not been ful
38 ow (rCBF) changes that occur during cortical spreading depression (CSD) are considered to be an exper
39 , although, animal models highlight cortical spreading depression (CSD) as a potential candidate.
40 ing has confirmed the importance of cortical spreading depression (CSD) as the pathophysiological mec
41 t and characterize complex waves of cortical spreading depression (CSD) evoked with KCL placed upon t
42                                     Cortical spreading depression (CSD) has been documented to confer
43                                     Cortical spreading depression (CSD) has been implicated in migrai
44                                     Cortical spreading depression (CSD) has been suggested to underli
45                                     Cortical spreading depression (CSD) has long been implicated in m
46                   Animal studies of cortical spreading depression (CSD) have benefited greatly from t
47 tensive care and were classified as cortical spreading depression (CSD) if they took place in spontan
48                                     Cortical spreading depression (CSD) is a key pathogenetic step in
49                                     Cortical spreading depression (CSD) is a propagating event of neu
50                                     Cortical spreading depression (CSD) is a propagating wave of neur
51                                     Cortical spreading depression (CSD) is a self-propagating wave of
52                                     Cortical spreading depression (CSD) is a transient propagating ex
53                                     Cortical spreading depression (CSD) is a wave of neuronal depolar
54                                     Cortical spreading depression (CSD) is associated with release of
55                                     Cortical spreading depression (CSD) is characterized by slowly pr
56                                     Cortical spreading depression (CSD) is likely the underlying phen
57                                     Cortical spreading depression (CSD) is thought to be a fundamenta
58                                     Cortical spreading depression (CSD) may be the underlying mechani
59                                     Cortical spreading depression (CSD) was induced by transient (10
60 d, if so, the threshold for evoking cortical spreading depression (CSD), a process sharing characteri
61 icient to cause infarcts, triggered cortical spreading depression (CSD), a propagating slow depolariz
62 on exhibit increased propensity for cortical spreading depression (CSD), a propagating wave of neurog
63                                     Cortical spreading depression (CSD), a wave of neuronal depolariz
64 mice, we show that a single wave of cortical spreading depression (CSD), an animal model of migraine
65 nnels and are highly susceptible to cortical spreading depression (CSD), the electrophysiologic event
66                Here, we report that cortical spreading depression (CSD), the electrophysiological sub
67         We evaluated the ability of cortical spreading depression (CSD), the proposed mechanism of th
68 ring somatosensory stimulations and cortical spreading depression (CSD), the putative mechanism of th
69 n damage and ischemia often trigger cortical spreading depression (CSD), which aggravates brain damag
70 this study was to determine whether cortical spreading depression (CSD)--an event believed to underli
71 us and evoked activity in naive and cortical spreading depression (CSD)-sensitized trigeminovascular
72  range trigeminovascular neurons by cortical spreading depression (CSD).
73 s (with [n=7] or without [n=7] HH), cortical spreading depressions (CSD) were elicited to ascertain t
74                                    Recurrent spreading depression episodes were induced by applicatio
75 ecovery rate for [K+]o, and interval between spreading depression episodes were measured before and a
76 ection analysis) and in vivo (i.e., during a spreading depression event in an anesthetized rat).
77  factor in the clinical response to cortical spreading depression events.
78 d the effect of fremanezumab on the cortical spreading depression-evoked activation of mechanosensiti
79 s predicted a second ceiling that demarcates spreading depression from anoxic depolarization.
80                                     Although spreading depression has been known for over 50 years, r
81 velength optical intrinsic imaging, cortical spreading depression has been shown to have a triphasic
82             Upstream events such as cortical spreading depression have also been modeled recently and
83   The pathological phenomena of seizures and spreading depression have long been considered separate
84             Pathological conditions, such as spreading depression, have been linked to abnormal range
85                                 Hyperthermic spreading depression (HSD) in immature rat hippocampal s
86  and headache by demonstrating that cortical spreading depression, implicated in migraine visual aura
87 cerebral ischaemia, and blocking of cortical spreading depression improved stroke outcome in these mi
88 provides evidence for an integrative role of spreading depression in brain pathophysiology.
89 ound, global cerebral ischemia, and cortical spreading depression in C57BL6 mice; 1 day after probe d
90 ge, but it is unknown whether TNF influences spreading depression in cerebral cortex in vivo.
91  signals evoked by stimulation, seizures and spreading depression in intact brain differ from those o
92 ole in the return of [K+]o to baseline after spreading depression in the cortex in vivo.
93 urovascular changes associated with cortical spreading depression in the mouse.
94 mulation blocked the propagation of cortical spreading depression in two of eight animals.
95          Enhanced susceptibility to cortical spreading depression increased sensitivity to focal cere
96 ity in reducing the total number of cortical spreading depressions induced by potassium chloride.
97 mechanism for the susceptibility of cortical spreading depression initiation in migraine disorders.
98                                     Cortical spreading depression is a propagating wave of depolariza
99          The findings indicate that cortical spreading depression is accompanied by transient neurobe
100                Our understanding of cortical spreading depression is also advancing.
101        These findings indicate that cortical spreading depression is associated with a multiphasic al
102                                     Cortical spreading depression is associated with profound vascula
103                              Hypoxia-induced spreading depression-like depolarization (hypoxic SD, or
104 larizations (PIDs) are seemingly spontaneous spreading depression-like waves that negatively impact t
105                            Results support a spreading, depression-like neuroelectric event occurring
106  Vascular/metabolic uncoupling with cortical spreading depression may have important clinical consequ
107 strated significant activity in the cortical spreading depression model of migraine as we reported pr
108 atine for 3 h failed to prevent hyperthermic spreading depression occurrence; and (2) intracellular A
109 utic targets in the disorders where cortical spreading depression occurs.
110 oelectric and metabolic event likened to the spreading depression of Leao.
111 tibility to ischemic depolarizations akin to spreading depression predisposes migraineurs to infarcti
112             Altogether, these data show that spreading depression propagates between cortex, basal ga
113 ing, and c-fos immunohistochemistry to trace spreading depression propagation into subcortical struct
114 e showed that unlike the wild type, cortical spreading depression readily propagated into subcortical
115 t astrocytes normally extrude calcium during spreading depression, resulting in rapid recovery of the
116   Although intercellular Ca2+ waves resemble spreading depression (SD) and occur in hippocampal organ
117  spontaneous, recurring episodes of cortical spreading depression (SD) as early as 20 min post-inject
118                                              Spreading depression (SD) has been demonstrated followin
119 occlusion, as well as the effects of induced spreading depression (SD) in intact animals.
120       We have developed an in vitro model of spreading depression (SD) in rat neocortex.
121               We examined characteristics of spreading depression (SD) induced on the rat cortex 1 da
122                                              Spreading depression (SD) involves current flow through
123                                              Spreading depression (SD) is a propagating wave of neuro
124                                              Spreading depression (SD) is a wave of cellular depolari
125                                              Spreading depression (SD) is wave of profound depolariza
126                         The possibility that spreading depression (SD) of cortical activity, a phenom
127 stigated the role of spontaneous and induced spreading depression (SD) on the evolution of focal isch
128                      Spontaneous episodes of spreading depression (SD) originating in multiple source
129           Triggered by focal depolarisation, spreading depression (SD) represents a suitable mechanis
130 ant, on fluid shifts, cerebral perfusion and spreading depression (SD) using diffusion- (DWI) and per
131                                              Spreading depression (SD), a slow diffusion-mediated sel
132                                              Spreading depression (SD), a slowly propagating neuronal
133                              This study used spreading depression (SD), which is characterized by red
134                   In the ischemic periphery, spreading depression (SD)-like waves of depolarization w
135 , I(Na,P), in the generation of seizures and spreading depression (SD).
136  expression following unilateral neocortical spreading depression (SD).
137 receded by an aura likely caused by cortical spreading depression (SD).
138 ous transition between epileptic seizure and spreading depression states as the cell swells and contr
139 ssium or hypoxia induced), mixed seizure and spreading depression states, and the terminal anoxic "wa
140 mone levels in these mice modulated cortical spreading depression susceptibility in much the same way
141  smooth muscle Notch 3 mutations to enhanced spreading depression susceptibility, implicating the neu
142  mutation are highly susceptible to cortical spreading depression, the electrophysiological surrogate
143                                       During spreading depression, the evoked increase in cytosolic C
144        Amiloride was shown to block cortical spreading depression, the experimental correlate of aura
145     We tested the susceptibility of cortical spreading depression, the experimental correlate of migr
146               This paper shows that cortical spreading depression, the neural correlate of migraine a
147 at cortices, when administered post-cortical spreading depression, transcranial magnetic stimulation
148 unction, one during the propagating cortical spreading depression wave and a second much longer phase
149 t current shift associated with the cortical spreading depression wave was accompanied by marked arte
150  concentration increased during the cortical spreading depression wave, but recovered and remained at
151     After recovery from the initial cortical spreading depression wave, we observed a second phase of
152 istinct from the initial propagated cortical spreading depression wave.
153 tical spreading depression and reverberating spreading depression waves.
154 h mechanical and chemically-induced cortical spreading depression when administered immediately post-
155 euronal activities, from spikes to seizures, spreading depression (whether high potassium or hypoxia
156 ion and c-fos expression are associated with spreading depression, which is believed to contribute to
157 ity, and enhanced susceptibility to cortical spreading depression, which is the electrophysiological
158  TAC-Red-stained brain cortex in mice during spreading depression, with velocity 4.4 +/- 0.5 mm/min,
159 and transient ischaemia can trigger cortical spreading depression without an enduring tissue signatur

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