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1 en associated with autoimmune conditions and sprue.
2 peutic potential for the treatment of celiac sprue.
3 oral peptidase supplement therapy for Celiac Sprue.
4 hly restricted diet for patients with celiac sprue.
5 ointestinal bleeding in patients with celiac sprue.
6 agents for the potential treatment of Celiac Sprue.
7           Seventy-eight patients with celiac sprue (59 women and 19 men) treated with a gluten-free d
8                                       Celiac Sprue, a widely prevalent autoimmune disease of the smal
9 agnostic investigation of diarrhea in celiac sprue after treatment seems warranted.
10 might become clonally expanded in refractory sprue and enteropathy-associated T-cell lymphoma.
11 f the gastrointestinal tract, such as celiac sprue and scleroderma, and their relationship with Barre
12 tected in about half of patients with celiac sprue and should be added to the list of factors that ca
13                     Patients with refractory sprue and/or ulcerative jejunitis are, therefore, suffer
14 f response to a gluten-free diet (refractory sprue) and ulcerative jejunitis are complications of cel
15  critical role in the pathogenesis of Celiac Sprue by binding to epitopes derived from dietary gluten
16  HLA-DQ2 predisposes an individual to celiac sprue by presenting peptides from dietary gluten to inte
17  of various human disorders including celiac sprue, certain neurological diseases, and some types of
18           An analysis of 15 untreated celiac sprue (CS) patients demonstrated that approximately 33%
19 syndrome (IBS-D) may have undiagnosed celiac sprue (CS).
20         The majority of patients with celiac sprue experience diarrhea before diagnosis.
21        Endomysial antibodies indicate celiac sprue in asymptomatic patients, and multiple endocrinopa
22                                       Celiac sprue is a chronic disease, which usually occurs in chil
23                                       Celiac sprue is a multifactorial disease characterized by an in
24                                       Celiac Sprue is an HLA DQ2 (or DQ8)-associated autoimmune disor
25                                       Celiac sprue is important to recognize and treat by gluten rest
26          Iron deficiency complicating celiac sprue is usually attributed to the malabsorption of diet
27         Celiac disease, also known as celiac sprue, is a gluten-induced autoimmune-like disorder of t
28 has been reported in association with celiac sprue, lymphoma, and hypogammaglobulinemia.
29                                   Refractory sprue (nonresponsive celiac disease) appears to be a man
30                                       Celiac Sprue, or gluten-sensitive enteropathy, is an inheritabl
31 r understanding the role of tTGase in Celiac Sprue, our results lay the groundwork for the design of
32  oral therapeutic enzyme for treating celiac sprue, our results provide a strong foundation for furth
33  in all food grains that are toxic to Celiac Sprue patients but are absent from all nontoxic food gra
34 ion in the small intestinal mucosa of Celiac Sprue patients therefore represents a potentially attrac
35 ten in the small intestinal mucosa of celiac sprue patients, our findings reveal a new strategy for i
36 zed by gluten-responsive T cells from celiac sprue patients.
37 lls from small intestinal biopsies of Celiac Sprue patients.
38 he inflammatory response to gluten in Celiac Sprue patients.
39 ived human T cell lines from 14 of 14 Celiac Sprue patients.
40                                  The classic sprue syndrome of steatorrhea and malnutrition coupled w
41 utenases (proposed oral therapies for celiac sprue) were proteolyzed in the absence or presence of a
42 atorrhea; and 7 patients with treated celiac sprue who had normal intestinal histologic features.
43                    After treatment of celiac sprue with a gluten-free diet, chronic diarrhea persists
44 glutaminase, the major autoantigen in Celiac Sprue, with substantially greater selectivity than known

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