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1 of Tgfbr2-deficient invasive transition zone squamous cell carcinoma.
2 ctinic keratosis is a precursor to cutaneous squamous cell carcinoma.
3 ochemistry in 96 patients with head and neck squamous cell carcinoma.
4 poor prognosis in melanoma and head and neck squamous cell carcinoma.
5 r smokers and in participants diagnosed with squamous cell carcinoma.
6 rom the HF junctional zone and predispose to squamous cell carcinoma.
7 vival of five cancers, such as head and neck squamous cell carcinoma.
8 keratinocytes, the cell-type susceptible to squamous cell carcinoma.
9 prostate cancer, urothelial cancer, and skin squamous cell carcinoma.
10 associated with skin keratosis and cutaneous squamous cell carcinoma.
11 ll proportion of the disease can progress to squamous cell carcinoma.
12 was not observed in patients with esophageal squamous cell carcinoma.
13 ity, photosensitivity, and increased risk of squamous cell carcinoma.
14 h T1/T2 clinically node-negative oral cavity squamous cell carcinoma.
15 of miR-203-induced transcriptomic changes in squamous cell carcinoma.
16 harbor high-grade dysplasia or even invasive squamous cell carcinoma.
17 l features with their malignant counterpart, squamous cell carcinoma.
18 marker and therapeutic target for cutaneous squamous cell carcinoma.
19 candidiasis, hypothyroidism, and esophageal squamous cell carcinoma.
20 up-regulated in human actinic keratosis and squamous cell carcinoma.
21 t to CHCT/MRI in patients with head and neck squamous cell carcinoma.
22 ed in national registries, such as basal and squamous cell carcinomas.
23 metimes with keratin pearls, consistent with squamous cell carcinomas.
24 ting toxicity in patients with head and neck squamous cell carcinomas.
25 ntigen for cancer diagnosis specifically for squamous cell carcinomas.
26 e (PI3Kalpha), are frequent in head and neck squamous cell carcinomas.
28 ophageal cancer (84.7% adenocarcinoma, 15.2% squamous cell carcinoma), 335 (9.3%) were treated with A
29 ted, 287 (62.8%) were BCCs, 106 (23.2%) were squamous cell carcinoma, 39 (8.5%) were lichen planus-li
30 eal adenocarcinomas, 323 cases of esophageal squamous cell carcinoma, 698 cases of gastric cardia ade
31 In other types of squamous epithelia and squamous cell carcinomas, a similar control mechanism is
33 ma, esophageal adenocarcinoma, or esophageal squamous cell carcinoma.Among older American adults, bot
34 zard ratios across subgroups, including both squamous cell carcinoma and adenocarcinoma, both cN0 and
36 sive phenotypes in lung adenocarcinoma, lung squamous cell carcinoma and breast carcinoma cancer cell
38 epidermal development, psoriasis, cutaneous squamous cell carcinoma and re-epithelisation are highli
40 as a predictive marker for HDACi response in squamous cell carcinomas and lymphomas, and unveiled let
41 increase was seen in Bowen disease, invasive squamous cell carcinoma, and a superficial type of basal
43 in lung adenocarcinoma as compared with lung squamous cell carcinoma, and that neutrophils are the mo
44 fty-six percent had adenocarcinomas, 34% had squamous cell carcinomas, and 10% had another histology.
46 d its capability in quantitative analysis of squamous cell carcinoma antigen in human serum with reco
47 ak current variation to the concentration of squamous cell carcinoma antigen in the range of 0.001-0.
48 borrheic keratosis) or mutant p53 (cutaneous squamous cell carcinoma) appears to dictate whether a le
49 volved in the progression of transition zone squamous cell carcinoma are poorly understood, hence rep
52 The aim of this article is to describe oral squamous cell carcinoma as it appears in different imagi
53 ith histologically verified primary head and squamous cell carcinoma at Odense University Hospital fr
54 in the pharyngeal mucosal space (n=16) with squamous cell carcinoma being the most common pathology.
56 tients treated by chemoradiotherapy for anal squamous cell carcinoma between October 2007 and October
58 analyses of patients with EGFR FISH-positive squamous-cell carcinoma cancers are encouraging and supp
60 re being treated for basal cell carcinoma or squamous cell carcinoma (cases) and 100 individuals who
63 against naturally HPV-infected head and neck squamous cell carcinoma cells using IFN-gamma ELISPOT an
65 GFR with GFP by genome-editing of human oral squamous cell carcinoma cells, which were used to examin
68 nsformation of normal epidermis to cutaneous squamous cell carcinoma (cSCC) is causally linked to alt
72 ified genetic loci associated with cutaneous squamous cell carcinoma (cSCC) risk, but single-nucleoti
75 was recently proposed for advanced cutaneous squamous cell carcinomas (cSCC); however, its efficacy i
76 dvantage on normal and neoplastic (cutaneous squamous cell carcinoma, cSCC) human epidermal cells.
78 ted with CD44, HAS3, and ABCC1 expression in squamous cell carcinoma datasets and p63-HA pathway is a
79 tudy, we determined that human head and neck squamous cell carcinoma-derived HSC-3 cells contain a su
80 ons on the cellular and molecular origins of squamous cell carcinoma due to oncogenic mutations in Ra
81 -wide association study (GWAS) on esophageal squamous cell carcinoma (ESCC) in Han Chinese, we conduc
84 issues removed from patients with esophageal squamous cell carcinoma (ESCC) with poor prognosis, and
85 ophageal adenocarcinoma (EAC) and esophageal squamous cell carcinoma (ESCC), although evidence is lim
86 h enhanced malignant potential in esophageal squamous cell carcinoma (ESCC), among the deadliest of a
87 have efficacy in EGFR-amplified oesophageal squamous cell carcinoma (ESCC), but may become quickly i
90 half of the world's 500,000 new oesophageal squamous-cell carcinoma (ESCC) cases each year occur in
94 tions in which, although common in cutaneous squamous cell carcinoma, have not been identified in SKs
95 lanoma skin cancers, in particular cutaneous squamous cell carcinoma, have the highest standardized i
96 which were frequently dysregulated in human squamous cell carcinomas, highlighting their potential a
97 define an intrinsic subtype of head and neck squamous cell carcinoma (HNSC) that features pronounced
99 cetuximab in locally advanced head and neck squamous cell carcinoma (HNSCC) and biomarkers that pred
100 potential relationship between head and neck squamous cell carcinoma (HNSCC) and microbial dysbiosis,
102 to examine whether staging of head and neck squamous cell carcinoma (HNSCC) by upfront (18)F-FDG PET
103 panel of 49 established human head and neck squamous cell carcinoma (HNSCC) cell lines and report th
104 l Cell Lung Cancer (NSCLC) and Head and Neck Squamous Cell Carcinoma (HNSCC) cell lines and was due t
105 phenotypes in patient-derived head and neck squamous cell carcinoma (HNSCC) cells in vitro and in vi
106 papillomavirus (HPV)-positive head and neck squamous cell carcinoma (HNSCC) have better responses to
111 ely 6,000 single cells from 18 head and neck squamous cell carcinoma (HNSCC) patients, including five
113 e cell growth of p53-deficient head and neck squamous cell carcinoma (HNSCC) UM-SCC-1 cells both in i
114 ne and metabolic phenotypes in head and neck squamous cell carcinoma (HNSCC) were assessed and change
115 nt-resistant head and neck tumors, including squamous cell carcinoma (HNSCC), adenoid cystic carcinom
116 nt-resistant head and neck tumors, including squamous cell carcinoma (HNSCC), adenoid cystic carcinom
117 clinically validated target in head and neck squamous cell carcinoma (HNSCC), where EGFR-blocking ant
126 nome Atlas (TCGA) reveals that head and neck squamous cell carcinomas (HNSCC) harbor the most frequen
129 papillomavirus (HPV)-negative head and neck squamous cell carcinomas (HNSCCs) are deadly and common
131 owever, more recent studies of head and neck squamous cell carcinomas (HNSCCs) suggest that integrati
132 he absence of p53, Nanog induces spontaneous squamous cell carcinoma, identifying a novel role of Nan
135 ential therapeutic targets for prevention of squamous cell carcinoma in patients with Kindler syndrom
141 y genomic studies in other cancer types.Oral squamous cell carcinoma is a prevalent malignancy in Tai
144 gastric adenocarcinoma (AGS) and esophageal squamous cell carcinoma (KYSE30) cancer cells were studi
145 eck in locoregionally advanced head-and-neck squamous cell carcinoma (LAHNSCC) after concurrent chemo
146 iling to analyze TLS formation in human lung squamous cell carcinoma (LSCC) and in an experimental mo
147 specific copy number gain (CNG) in both lung squamous cell carcinoma (LSCC) and ovarian serous carcin
149 ibe a similar hypomethylated subtype of lung squamous cell carcinoma (LUSC) that is enriched for both
150 between lung adenocarcinoma (LUAD) and lung squamous cell carcinoma (LUSC), two main lung cancer sub
152 a, classified as adenocarcinoma (n = 4,036), squamous cell carcinoma (n = 1,998), small cell carcinom
155 randomized trials on secondary prevention of squamous cell carcinoma observed a reduction in cumulati
159 have recurrent copy-number variants in three squamous-cell carcinomas (oesophageal, head and neck and
165 se after chemoradiotherapy for patients with squamous cell carcinoma of the anus is 26 weeks from sta
166 the quality of outcome reporting in RCTs of squamous cell carcinoma of the anus is inconsistent.
167 h newly diagnosed, histologically confirmed, squamous cell carcinoma of the anus without metastatic d
171 chemoradiotherapy in locoregionally advanced squamous cell carcinoma of the head and neck (LA-SCCHN).
172 ucleus enhanced its interaction with PCNA in squamous cell carcinoma of the head and neck (SCCHN) cel
173 logically confirmed recurrent and metastatic squamous cell carcinoma of the head and neck after disea
175 ositol 3-kinase (PI3K) pathway activation in squamous cell carcinoma of the head and neck contributes
176 platinum-refractory recurrent or metastatic squamous cell carcinoma of the head and neck have few tr
178 ial in patients with recurrent or metastatic squamous cell carcinoma of the head and neck who progres
179 nfirmed diagnosis of recurrent or metastatic squamous cell carcinoma of the head and neck, and had an
180 titumour activity in recurrent or metastatic squamous cell carcinoma of the head and neck, supporting
181 fe for patients with recurrent or metastatic squamous cell carcinoma of the head and neck, these data
185 d in 2004 to 2012 with AJCC stage III to IVB squamous cell carcinoma of the oral cavity, oropharynx,
186 had to include patients with non-metastatic squamous cell carcinoma of the oral cavity, oropharynx,
192 dissection in the treatment of patients with squamous-cell carcinoma of the head and neck who have ad
193 wly diagnosed, biopsy-proven stage III or IV squamous-cell carcinoma of the oropharynx, positive for
194 n papillomavirus (HPV)-related oropharyngeal squamous cell carcinoma (OPSCC) has been reported predom
195 The incidence of HPV-related oropharyngeal squamous cell carcinoma (OPSCC) has increased more than
196 papillomavirus (HPV)-negative oropharyngeal squamous cell carcinoma (OPSCC) has shown resistance to
198 papillomavirus (HPV)-positive oropharyngeal squamous cell carcinoma (OPSCC) is disproportionately hi
200 apillomavirus (HPV)-associated oropharyngeal squamous cell carcinoma (OPSCC) is treatment-responsive.
201 and a separate dataset of HPV+ oropharyngeal squamous cell carcinoma (OPSCC) samples to identify diff
202 deline on radiation therapy in oropharyngeal squamous cell carcinoma (OPSCC) that was determined to b
203 ), the increasing incidence of oropharyngeal squamous cell carcinomas (OPSCCs) is attributable to hum
204 es an increasing proportion of oropharyngeal squamous cell carcinomas (OPSCCs), particularly in white
205 es an increasing proportion of oropharyngeal squamous cell carcinomas (OPSCCs), particularly in white
207 d "cutaneous squamous cell carcinoma or skin squamous cell carcinoma or cSCC or nonmelanoma skin neop
208 used: "human papillomavirus" and "cutaneous squamous cell carcinoma or skin squamous cell carcinoma
209 g first-line treatment for patients with non-squamous cell carcinoma or squamous cell carcinoma (with
217 ssible association between bacteria and oral squamous cell carcinoma (OSCC) remain inconclusive, larg
219 ors with stage I adenocarcinoma (P<0.003) or squamous cell carcinoma (P=0.023) in the TCGA data set.
220 nic mouse model of breast cancer and in oral squamous cell carcinoma patients, tumor development was
222 rofile in recurrent/metastatic head and neck squamous cell carcinoma previously treated with platinum
223 ments for recurrent/metastatic head and neck squamous cell carcinoma refractory to platinum and cetux
229 thway; SNPs at these loci appear to modulate squamous cell carcinoma risk independently of the pigmen
230 from the 1000 Genomes Project and esophageal squamous cell carcinoma samples show that seeksv has hig
231 Malignant tumors included melanoma (12%), squamous cell carcinoma (SCC) (9%), lymphoma (7%), and o
232 uded 53 patients with a history of cutaneous squamous cell carcinoma (SCC) after a first kidney trans
233 ic immunosuppression promotes nonmelanocytic squamous cell carcinoma (SCC) after kidney transplantati
234 ng keratinocyte cancers, including cutaneous squamous cell carcinoma (SCC) and basal cell carcinoma (
235 Here we document TCF7L1 upregulation in skin squamous cell carcinoma (SCC) and demonstrate that TCF7L
236 alignant skin lesions that can progress into squamous cell carcinoma (SCC) and invasive SCC if left u
237 It is postulated to increase the risk of squamous cell carcinoma (SCC) and other skin cancers in
238 ends in the risk of posttransplant cutaneous squamous cell carcinoma (SCC) are few and appear contrad
240 Here we demonstrate for the first time that squamous cell carcinoma (SCC) EVs were enriched with the
242 bution of voriconazole to the development of squamous cell carcinoma (SCC) in lung transplant recipie
243 ing to detect basal cell carcinoma (BCC) and squamous cell carcinoma (SCC) in Mohs micrographic surge
244 d incidence of basal cell carcinoma (BCC) or squamous cell carcinoma (SCC) in QSkin, a prospective st
247 ients (4.4%) had a partial response; one had squamous cell carcinoma (SCC) of the anus and one had SC
249 to urinary arsenic concentration and risk of squamous cell carcinoma (SCC) of the skin in a U.S. popu
250 18)F-FDG phosphorylation rate were higher in squamous cell carcinoma (SCC) than in adenocarcinoma (AC
251 Here, we report in a murine model of skin squamous cell carcinoma (SCC) that nuclear FAK regulates
252 ching of KRAS+ lung adenocarcinomas (ADC) to squamous cell carcinoma (SCC) through deletion of Lkb1 (
254 ophagus (BE), columnar cell metaplasia (CM), squamous cell carcinoma (SCC), and squamous epithelium (
255 s), including basal cell carcinoma (BCC) and squamous cell carcinoma (SCC), are the most common cance
256 s), including basal cell carcinoma (BCC) and squamous cell carcinoma (SCC), are the most common cance
257 rs for posttransplant skin cancer, including squamous cell carcinoma (SCC), melanoma (MM), and Merkel
259 of tumor-infiltrating T lymphocytes (TIL) in squamous cell carcinoma (SCC), using a systems biologic
265 The associations were somewhat stronger in squamous cell carcinomas (SCC, OR = 4.90) than in urothe
266 Keratinocyte carcinomas (KCs), consisting of squamous cell carcinomas (SCCs) and basal cell carcinoma
272 lanomas, 4 hemangiomas, 4 dermatofibromas, 2 squamous cell carcinomas [SCCs]) was selected and shown
273 abilized DeltaNp63alpha to enhance epidermal squamous cell carcinoma spheroid formation, invasion, an
275 denocarcinoma (AD) project and 504 from TCGA squamous cell carcinoma (SQCC) project-were classified u
276 illary urothelial carcinoma, small cell, and squamous cell carcinoma subtypes of invasive bladder can
277 surgical treatment modalities for subungual squamous cell carcinoma (SUSCC) without bone invasion ne
280 letion alleles is 50% in 143 genotyped oral squamous cell carcinoma -Taiwan samples (27A3B (-/-):89A
281 was associated with a nonlinear reduction in squamous cell carcinoma that became attenuated as consum
282 tion germline polymorphism in Taiwanese oral squamous cell carcinoma that impacts expression of APOBE
283 epidermal cancer stem cells (ECS cells), in squamous cell carcinoma, that form rapidly growing, inva
285 e establishes a chromatin state predisposing squamous cell carcinomas to undergo EMT and metastasis,
287 Methods Adult patients with oral cavity squamous cell carcinoma undergoing upfront surgical rese
288 with subgroup analyses by histological type (squamous cell carcinoma vs adenocarcinoma) and type of n
289 chanism leading to the development of vulvar squamous cell carcinoma (VSCC) from vulvar lichen sclero
290 sophageal adenocarcinoma, but not esophageal squamous cell carcinoma, was associated with reduced eso
291 prised a mixed papilloma/well-differentiated squamous cell carcinoma (wdSCC), exhibiting p53 loss, in
292 man papilloma virus-associated oropharyngeal squamous cell carcinoma, we hypothesized that adding cet
293 and in the prestyloid parapharyngeal space, squamous cell carcinoma were the most common lesions, re
294 r tumors specimens (319 adenocarcinomas; 142 squamous cell carcinomas) were profiled from 245 black p
295 e aspiration from the mass was suggestive of squamous cell carcinoma, which was confirmed on immunohi
296 al sources are lacking, such as in basal and squamous cell carcinomas, which are not included in nati
297 t in non-Hispanic whites, whereas esophageal squamous cell carcinoma with risk factors of tobacco and
299 patients with non-squamous cell carcinoma or squamous cell carcinoma (without positive markers, eg, E
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