ライフサイエンスコーパス: staining intensity

1 try (>/= 25% of tumor cells with at least 2+ staining intensity).

2 r positive tumor cell area and P = 0.005 for staining intensity).

3 scores (percentage of positive tumor cells x staining intensity).

4 rom 0 to 400 (percentage of positive cells x staining intensity).

5 l the direct role of transporter activity on staining intensity.

6 al acetylcholinesterase (AChE) histochemical staining intensity.

7 haracteristics were associated with relative staining intensity.

8 SP cell (NSP) subpopulations by differential staining intensity.

9 the eye can be partially predicted by LIN28A staining intensity.

10 processing led to a significant reduction in staining intensity.

11 sess the correlations between CT texture and staining intensity.

12 integrity, they suffered from irreproducible staining intensity.

13 ers of synaptic vesicles and reduced FM 1-43 staining intensity.

14 methylation events to each other and to DNA staining intensity.

15 dendritic arborization and reduced calbindin staining intensity.

16 eases in TH-ir neuron number, soma size, and staining intensity.

17 PERIOD nuclear translocation and its protein staining intensity.

18 ibited altered phase and amplitude of PERIOD staining intensity.

19 arent association between tumor response and staining intensity.

20 methyl paraoxon significantly decreased AChE staining intensity.

21 scattered and contained a considerably lower staining intensity.

22 estinal segments studied revealed comparable staining intensities.

23 chemistry showed the BPD group had decreased staining intensity and abnormal distribution of alveolar

24 We observed a change in CSE1L staining intensity and cellular localization by immunohi

25 of colon cancers, with a correlation between staining intensity and clinical stage; PTEN expression w

26 Increased staining intensity and frequency of NF-kappaB-positive c

27 However, sulprostone resulted in increased staining intensity and nuclear translocation of apoptosi

28 sis, and correlates with immunohistochemical staining intensity and numbers of thin cap atheroma.

29 d ratio, 1.78; P < 0.02) controlled for TP53 staining intensity and other survival-associated factors

30 t cancer patients revealed a decrease in the staining intensity and percentage of cells expressing HP

31 associated with a greater reduction in Sost staining intensity and sclerostin-positive osteocytes (r

32 tect a significant correlation between KIM-1 staining intensity and the occurrence of DGF.

33 The staining intensity and the percentage of immunoreactive

34 (f) immunostaining data are best reported as staining intensity and the percentage of positive cells;

35 s whether associations existed between COX-2 staining intensity and the RTOG 92-02 primary endpoints

36 ound to tissue sections correlated well with staining intensity and yielded phage that bound to neopl

37 tumour cells with membrane staining of >/=1+ staining intensity), and evaluable disease, who had not

38 an be detected, yields considerably enhanced staining intensities, and is compatible with using reduc

39 identified on the bases of their morphology, staining intensity, and projection pattern: 1) the preop

40 played differential HLA-DP2-peptide tetramer staining intensity, and sequence analysis of the distinc

41 d a strong signal, the regional variation in staining intensity appeared to be related to neuron size

42 ared to muscle meat as indicated by a higher staining intensity are a plausible explanation for the d

43 showed that Rap1GAP and MMP9 expression and staining intensity are correlated (P < 0.0001) and that,

44 Using moderate or strong staining intensity as positive (score = 3 or 4), evaluat

45 ermore, p63genomic amplification and protein staining intensity associated with better survival.

46 ous neuronal swelling in ON precluded raised staining intensity being an artifact of neuronal atrophy

47 lly above the ECM islands, with the greatest staining intensity being observed at adhesion sites alon

48 The difference in distribution and staining intensities between LCNEC and SCLC compared wit

49 sons demonstrated significant differences in staining intensity between clinically localized prostate

50 NR4A2, showed alterations in cell density or staining intensity between the groups, highlighting the

51 However, BDNF-LIR staining intensity but not NGF-LIR or PGP 9.5-LIR is inc

52 le amino acid substitution revealed that the staining intensity by anti-Valpha24 Abs depends on wheth

53 Although differences in protein staining intensity can often be visualized by difference

54 nalysis, positive NGAL expression and strong staining intensity correlated with decreased disease-fre

55 Staining intensity correlated with the extent of cell ly

56 The staining intensity declined subsequently.

57 r gamma-aminobutyric acid transporter (vGAT) staining intensity decrease, whereas the intrinsic excit

58 Consistent with our hypothesis, staining intensities demonstrated that increased amounts

59 vidin-biotin complex method and the relative staining intensity determined by image analysis.

60 PTEN staining, although the cytoplasmic PTEN-staining intensity did not differ significantly from tha

61 rent in early pachytene spermatocytes, while staining intensity diminished in diplotene and meiotical

62 ngs) or biochemical abnormalities (increased staining intensities for adhesive glycoproteins) along t

63 rodensitometric analyses of immunoperoxidase staining intensities for CYP1A1/1A2 revealed that intrah

64 Unexpectedly, distal synapses had the lowest staining intensities for vesicular proteins vGlut, vGAT,

65 Mean relative staining intensity for 4-aminobiphenyl-DNA was slightly

66 ffected the number of BrdU-labeled cells and staining intensity for a marker of neuronal differentiat

67 er, there was moderate to strong cytoplasmic staining intensity for all of the CEA(+) HNC lines by im

68 We also found a trend of increasing regional staining intensity for all positive GABAA receptor subun

69 In control specimens, the staining intensity for both G1 fragments increased with

70 Staining intensity for cNOS, but not neuron number was r

71 Mean staining intensity for CRP of macrophages and lipid core

72 ing to RIII for signal transduction, in that staining intensity for RIII was relatively low in limbal

73 or phospho-S6 and phospho-eIF4G with various staining intensities in FCDs and TSC tubers.

74 In contrast, staining intensity in 9 patients with PV (41%) was unifo

75 omerulosclerosis and C3d, IgG, IgG3, and IgA staining intensity in glomeruli in complement-inhibited

76 s heterogeneous and was associated with weak staining intensity in more than 20% of the megakaryocyte

77 The results showed that i) APP staining intensity in neurons was variable in the AD cor

78 tween the antisera were distinct patterns of staining intensity in subregions of glomeruli of the olf

79 Staining intensity in the ciliary muscle was assessed by

80 significant increases in microglial size and staining intensity in the cortex, corpus callosum and st

81 Strikingly, COX-2 staining intensity in the normal adjacent epithelium was

82 tern; heat shock greatly increased the HSP70 staining intensity in the undifferentiated cells and les

83 usting for age, sex, and BCC location, PD-L1 staining intensity in tumor cells increased with the num

84 To compare the staining intensity in various tissues, the mean optical

85 NC was sufficient to induce cGMP-IR, and the staining intensity increased as the SNC dose was increas

86 Staining intensity increased on treatment of cells with

87 The staining intensity is dependent on section thickness, su

88 morphology coincided with increased F-actin staining intensity, larger focal adhesion size, and elev

89 on SFRP methylation status and TP53 protein staining intensity may be a clinically powerful predicto

90 se euchromatin as a control for quantitative staining intensity measurements in other regions of the

91 After stratification of the relative staining intensities of 4-aminobiphenyl-DNA adduct level

92 Fourth, the relative immunofluorescence staining intensities of Na/K-ATPase, ankyrin-3, and fodr

93 ons revealed that 74.1% of the samples had a staining intensity of <6 in the PD samples as compared w

94 cer (n = 17) demonstrated mean AMACR protein staining intensity of 1.31 (95% confidence interval, 1.2

95 The PD-L1 immunohistochemical staining intensity of 78 treated BCCs compared with 60 t

96 The staining intensity of both enzyme and substrate in the n

97 t were parvalbumin positive and the relative staining intensity of both parvalbumin and a component o

98 n a 1.72 (+/- 0.26)-fold (n = 5) increase in staining intensity of Caco-2 cells.

99 association between patient survival and FAS staining intensity of carcinomas.

100 lesion-related differences in the number or staining intensity of cells containing estrogen receptor

101 The number, size, and staining intensity of choline acetyltransferase (ChAT)-i

102 The staining intensity of ecNOS was reduced in both diabetic

103 animals not given hormones, SP decreased the staining intensity of estrogen receptor immunoreactive (

104 The number and staining intensity of GluR1-positive cells increases dra

105 Qualitatively, the staining intensity of individual cells was higher in spr

106 osure had no effect on neuronal size or ChAT staining intensity of MS or DBv neurons when compared to

107 e in porin expression could be visualized by staining intensity of Omp F and Omp C on acrylamide gels

108 Mean staining intensity of oral cells of smokers was 1.6-fold

109 Three patterns of immunohistochemical staining intensity of p53 protein were seen: negative, w

110 Whereas the staining intensity of SG files was constantly high at di

111 The ChAT staining intensity of the axotomized RDLN declined in bo

112 al prostate tissue which correlated with the staining intensity of the corresponding cancer tissue.

113 T and PPT; nor were there differences in the staining intensity of the somata of neurons in the adult

114 ces were found among the three groups in the staining intensity of von Willebrand factor and GLUT1.

115 higher IHC scores (% immunopositive cells x staining intensity) of SCF and p-AKT were associated wit

116 er demonstrated no significant difference in staining intensity or area across the molecular layer in

117 Bands that differed in staining intensity or that were of low intensity were ob

118 tive scoring of anti-PSA immunohistochemical staining intensity paralleled the LCM quantitative immun

119 We have determined that these differences in staining intensity reflect differences in amounts of dyn

120 showed a 2-fold increase in NADPH-diaphorase staining intensity relative to sham-infected and Ad. bet

121 Staining distribution was noted and staining intensity scored on a semiquantitative scale of

122 tion, and fibrosis (stage), higher NF-kappaB staining intensity scores within bile ducts, proliferati

123 However, VE1 staining intensity varied among the primary tumors and c

124 TCRm mAb staining intensity varied for the five human tumor cell

125 /- 26.1% of tumor cell area was positive and staining intensity was 2.8 +/- 1.2).

126 Increasing PKC epsilon staining intensity was associated with high histologic g

127 Podoplanin staining intensity was associated with increasing levels

128 size of these plaques was smaller, and their staining intensity was diminished compared with the plaq

129 However, no difference in mean relative staining intensity was found between smokers and nonsmok

130 Staining intensity was graded 0 to 3+ (0 to 1+ was consi

131 urones was positive, and the median relative staining intensity was greater in nociceptive than LTM u

132 In PN sections (n = 31), median staining intensity was mild, median percentage of staine

133 The 3-nitrotyrosine staining intensity was not different.

134 roportional hazards model, whereas HER1/EGFR staining intensity was not.

135 A modest increase in hyaluronan staining intensity was observed in the epidermis of kelo

136 Staining intensity was quantified within each attachment

137 Staining intensity was reduced in HNPCCs compared with s

138 The staining intensity was similar between various layers of

139 sections (n = 31), median IRS was 3, median staining intensity was strong, and median percentage of

140 Megakaryocyte c-mpl staining intensity was uniformly moderate-to-strong in t

141 By differential tetramer staining intensities, we observed two distinct minor H a

142 Moderate staining intensities were found in most B-CLL and FL cas

143 Cell density and staining intensity were compared in the DLPFC, as well a

144 In particular, differences in staining intensity were consistently seen among the thre

145 A general waxing and waning of staining intensity were observed over this developmental

146 nohistochemistry, and the labeling index and staining intensity were quantified by the Image-Pro syst

147 ull population, however, NGAL expression and staining intensity were significantly associated with hi

148 e staining among five mAbs, including higher staining intensity when compared with that of MHG1152 (p

149 ocysts from heterozygous matings had reduced staining intensity with rhodamine 123 and NRF-1(-/-) bla

150 ingly, there was no detectable change in TIM staining intensity within the eight pacemaker s-LNvs.