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1 k transgenic mice following stimulation with staphylococcal enterotoxin A.
2 that during the response to the superantigen Staphylococcal enterotoxin A, activated CD8 cells, but n
3 onist anti-4-1BB Ab compared with mice given staphylococcal enterotoxin A and a control IgG.
4 ens of mice injected 21 days previously with staphylococcal enterotoxin A and an agonist anti-4-1BB A
5        Fluorophore-conjugated superantigens (staphylococcal enterotoxin A and B), presumed to enter c
6 A followed by a combination of superantigens staphylococcal enterotoxin A and staphylococcal enteroto
7                        Food poisoning due to staphylococcal enterotoxins A and B (SEA and SEB) affect
8 tigenic toxins toxic shock syndrome toxin-1, staphylococcal enterotoxins A and B, and exfoliative tox
9 uch as cow milk, the limits of detection for staphylococcal enterotoxins A and B, cholera toxin, botu
10 aneous identification of six protein toxins (staphylococcal enterotoxins A and B, cholera toxin, rici
11 on of three bacterial toxins: cholera toxin, staphylococcal enterotoxin A, and toxic shock syndrome t
12 s were sensitive to superantigen activity of staphylococcal enterotoxin A as cultured in the presence
13 pecific manner with the polyclonal activator staphylococcal enterotoxin A augmented the CTL response.
14 ction limits were found to be 0.01 ng/mL for staphylococcal enterotoxin A, cholera toxin, botulinum t
15                                              Staphylococcal enterotoxin A constructs containing targe
16 ce repeatedly injected with the superantigen staphylococcal enterotoxin A entered a tolerant state in
17 aureus Cowan strain 1-lipopolysaccharide- or staphylococcal enterotoxin A-induced signaling pathways
18  into thioglycollate-induced peritonitis and staphylococcal enterotoxin A-injected skin pouch.
19 ly, we demonstrated that the presentation of staphylococcal enterotoxin A, like TSST-1, is peptide de
20 gation times in response to the superantigen staphylococcal enterotoxin A on dendritic cells and a re
21 n T cell receptors were stimulated either by staphylococcal enterotoxin A or by major histocompatibil
22 hallenged with ordinarily sublethal doses of staphylococcal enterotoxin A or staphylococcal enterotox
23                                            A staphylococcal enterotoxin A SEA-SEE hybrid molecule con
24 wide range of ~3 decades, while detection of staphylococcal enterotoxin A (SEA) and toxic shock syndr
25 dy, we investigated the effect of intranasal staphylococcal enterotoxin A (SEA) exposure on murine lu
26 monstrate that bacterial isolates containing staphylococcal enterotoxin A (SEA) from the affected ski
27 ogress towards reference materials (RMs) for Staphylococcal enterotoxin A (SEA) in cheese.
28       We have previously shown that systemic staphylococcal enterotoxin A (SEA) injections cause CD4
29                                              Staphylococcal enterotoxin A (SEA) is a microbial supera
30 ither toxic shock syndrome toxin (TSST) 1 or staphylococcal enterotoxin A (SEA) presentation.
31                     Site-directed mutants of staphylococcal enterotoxin A (SEA) that have attenuated
32 g) mice and the Vbeta3-specific superantigen staphylococcal enterotoxin A (SEA) to further investigat
33 15 antimicrobial agents; and PCR analysis of staphylococcal enterotoxin A (SEA) to SEH, toxic shock s
34                                              Staphylococcal enterotoxin A (SEA), a superantigen produ
35 ls with human ASM cells pulsed with the SAg, staphylococcal enterotoxin A (SEA), elicited adherence a
36                       C-terminal residues of staphylococcal enterotoxin A (SEA), including H187, D225
37 mice injected intraperitoneally (i.p. ) with staphylococcal enterotoxin A (SEA), SEB, or SEC3 or toxi
38                    Ovalbumin (OVA)-specific, staphylococcal enterotoxin A (SEA)-nonreactive naive CD4
39 antibody 5T4 fused to a mutated superantigen staphylococcal enterotoxin A (SEA).
40 ine residues in the biological activities of staphylococcal enterotoxin A (SEA).
41 cus aureus produces a set of proteins (e.g., staphylococcal enterotoxin A [SEA], SEB, toxic shock syn
42 -dependent mouse model of SIRS that utilizes staphylococcal enterotoxin A specific for Vbeta3(+) T ce
43 rinsic defect lying downstream of the TCR in staphylococcal enterotoxin A-specific CD8(+) T cells.
44                      Nearly 10 times as many staphylococcal enterotoxin A-specific T cells were detec
45 activator LPS, the T cell mitogen Con A, and staphylococcal enterotoxin A superantigen.
46             We find that they also recognize staphylococcal enterotoxin A superantigens in a manner d
47 h vaccinia virus, allogeneic tumor cells, or staphylococcal enterotoxin A, the cytotoxic and IFN-gamm
48                                              Staphylococcal enterotoxin A was found by ELISA in mushr

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