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1 ogen activators or by endogenously expressed staphylokinase.
2 facilitate tissue invasion without producing staphylokinase.
3 produce the bacterial plasminogen activator, staphylokinase.
4 ith and match in size the staphylococcal PA, staphylokinase.
5 The majority of S. aureus strains produce staphylokinase, a plasminogen activator capable of inact
8 ion was established, the interaction between staphylokinase and plasminogen did not promote systemic
9 "fibrin-dependent mechanisms" (akin to both staphylokinase and tissue Pg activator) that include: 1)
11 erate sequence identity to streptokinase and staphylokinase, bacterial activators of human plasminoge
12 Homology modelling based upon structures of staphylokinase complexed with human microplasminogen sup
13 und directly to staphylokinase and augmented staphylokinase-dependent plasminogen activation and fibr
15 other two phages, phi 13 also introduces the staphylokinase gene (sak) and a second gene related to e
22 of activation of human plasminogen (HPg) by staphylokinase (Sak) required formation of an active com
24 of the staphylococcal plasminogen activator, staphylokinase (SAK), was analyzed for its ability to me
25 EA, SEG, and SEK and the fibrinolytic enzyme staphylokinase (Sak), was identified in the unannotated
27 nd in vitro and ex vivo assays, we show that staphylokinase, secreted by S. aureus, promoted the esta
28 In this manner, SKDelta59 behaves more like staphylokinase than like SK; it requires plasmin to form
29 ve evolved PAs [e.g., streptokinase (SK) and staphylokinase] that exploit the Pg system to infect ani
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