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1  years of cosmic history in a short, extreme starburst.
2 disk, rather than in a merger-driven nuclear starburst.
3 star-formation rates in protogalaxies and in starbursts.
4 that can be observed as luminous quasars and starbursts.
5 on of H(2) is not co-spatial with the buried starburst activity and originates outside the obscured r
6 ative corneal curvature is a risk factor for starbursts after laser-assisted in situ keratomileusis (
7 he retina, individual dendritic sectors of a starburst amacrine cell (SAC) are preferentially activat
8 ieving radially symmetric arborization of On starburst amacrine cell (SAC) dendrites and normal SAC s
9 ymmetric GABAergic inhibition exerted by the starburst amacrine cell (SAC), a cholinergic and GABAerg
10 inputs and neurotransmitter release sites on starburst amacrine cell dendrites: the excitatory input
11                             In contrast, the Starburst amacrine cell has GABA(B) receptors in a subpo
12    Aside from rare counterexamples (e.g. the starburst amacrine cell in retina), neurons are polarize
13                                          The starburst amacrine cell in the mouse retina presents an
14 ABA release from the presynaptic interneuron starburst amacrine cell in the mouse retina.
15                        These features of the starburst amacrine cell network suggest that starburst c
16 two different strains of mice that differ in starburst amacrine cell number.
17 ltransferase-IR) but in less than 50% of the starburst amacrine cell somata.
18 tute a novel cholinergic, non-GABAergic, non-starburst amacrine cell type described for the first tim
19                                 In fact, the starburst amacrine cell was the only amacrine cell type
20 etina, this study reports that the displaced starburst amacrine cell, a unique cholinergic interneuro
21 scribed of these pairs is the very elaborate starburst amacrine cell, A5, which stains regularly in t
22 n the dendrites of an interneuron, i.e., the starburst amacrine cell, and that these responses are hi
23 neurons, that a presynaptic interneuron, the starburst amacrine cell, delivers direct inhibition to d
24  for generating direction selectivity in the starburst amacrine cell.
25 rlying direction-selective GABA release from starburst amacrine cells
26 ; and dendrites of orthotopic- and displaced-starburst amacrine cells (identified by choline acetyltr
27 ccur first in the retina in the dendrites of starburst amacrine cells (interneurons presynaptic to th
28 earch of clues, here we reconstruct Off-type starburst amacrine cells (SACs) and bipolar cells (BCs)
29 rupts self-avoidance of dendrites in retinal starburst amacrine cells (SACs) and cerebellar Purkinje
30 BA release during null-direction motion from starburst amacrine cells (SACs) and DS acetylcholine and
31 he mouse retina to show connectivity between starburst amacrine cells (SACs) and their known synaptic
32                                              Starburst amacrine cells (SACs) are an essential compone
33                                              Starburst amacrine cells (SACs) are thought to mediate t
34                   Pharmacologically isolated starburst amacrine cells (SACs) in perinatal rabbit reti
35                             The dendrites of starburst amacrine cells (SACs) in the mammalian retina
36 y selective wiring of inhibitory inputs from starburst amacrine cells (SACs) onto four subtypes of ON
37                                              Starburst amacrine cells (SACs) process complex visual s
38 recordings revealed that distal processes of starburst amacrine cells (SACs) received largely excitat
39 that local inhibition arising from GABAergic starburst amacrine cells (SACs) strongly contributes to
40 try in the inhibitory neurotransmission from starburst amacrine cells (SACs) to direction selective g
41 mission of acetylcholine (ACh) and GABA from starburst amacrine cells (SACs) to direction-selective g
42 lective ganglion cells (DSGCs) and GABAergic starburst amacrine cells (SACs), and the SACs then provi
43               In this study, we used retinal starburst amacrine cells (SACs), critical components of
44 y connected cholinergic interneurons, called starburst amacrine cells (SACs), generates spontaneous r
45 ne receptors (beta2-nAChRs) selectively from starburst amacrine cells (SACs), we show that mutual exc
46 to be initiated by the spontaneous firing of Starburst Amacrine Cells (SACs), whose dense, recurrent
47 ll-side inhibition, which is provided by OFF starburst amacrine cells (SACs).
48 of the mature inner retina are the so-called starburst amacrine cells (SACs).
49 ecorded in retinal ganglion cells (RGCs) and starburst amacrine cells (SACs).
50                Directional GABA release from starburst amacrine cells (SBACs) is critical for generat
51  established that the inhibition arises from starburst amacrine cells (SBACs) located on the null sid
52 tric GABA release from the dendritic tips of starburst amacrine cells (SBACs).
53 at glycinergic cells inhibit the cholinergic Starburst amacrine cells and are in turn inhibited by GA
54 mosaics by two retinal interneuron subtypes, starburst amacrine cells and horizontal cells.
55 Cholinergic "retinal waves" are initiated in starburst amacrine cells and propagate to retinal gangli
56 ch costratify near the processes of both the starburst amacrine cells and the ON-OFF directionally se
57                                              Starburst amacrine cells and their synaptic partners, ON
58 sitive bipolar cell provides input to the ON-starburst amacrine cells and/or the ON-plexus of the ON-
59                       We also confirmed that starburst amacrine cells are presynaptic to ganglion cel
60 lls in the ganglion cell layer revealed that starburst amacrine cells are the most KA-responsive cell
61 allowed imaging spontaneous calcium waves in starburst amacrine cells during development, and light-e
62 the DAPI-3 cell, we have also shown that the starburst amacrine cells exhibit no immunoreactivity for
63                                        Thus, starburst amacrine cells form the first identified netwo
64                                              Starburst amacrine cells have a synaptic asymmetry that
65 57BL/6J (B6) strain, although the mosaics of starburst amacrine cells in both strains are comparably
66                                              Starburst amacrine cells in the macaque retina were stud
67 udy has compared the dendritic morphology of starburst amacrine cells in two different strains of mic
68  that acetylcholine release from presynaptic starburst amacrine cells is crucial for its generation.
69 blocked by TTX, indicating that release from starburst amacrine cells is independent of sodium action
70                        The light response of starburst amacrine cells is initiated by glutamate relea
71 ude that the predominant excitatory input to starburst amacrine cells is mediated by AMPA receptors.
72                          A prominent role of starburst amacrine cells is the generation of directiona
73 erned by direction-selective inhibition from starburst amacrine cells occurring during stimulus movem
74 tive release of gamma-aminobutyric acid from starburst amacrine cells onto direction-selective gangli
75                               The network of starburst amacrine cells plays a fundamental role in the
76                                              Starburst amacrine cells release two classical neurotran
77 olution localization of GABA(B) receptors on starburst amacrine cells shows that they are discretely
78  (A) strain contains about one-quarter fewer starburst amacrine cells than does the C57BL/6J (B6) str
79 sitize rapidly, enhancing the sensitivity of starburst amacrine cells to moving or other rapidly chan
80 idence is found for inhibitory synapses from starburst amacrine cells to the ON-OFF DS GC.
81                         In the INL, presumed starburst amacrine cells were homogenous in appearance a
82                    Labeling the processes of starburst amacrine cells with antibodies against choline
83 f a mutually inhibitory relationship between starburst amacrine cells with overlapping dendrites.
84 re dictated by recurrent connectivity within starburst amacrine cells, and retinal ganglion cells act
85                          SV2C was present in starburst amacrine cells, other conventional synapses in
86 e of the neurotransmitter acetylcholine from starburst amacrine cells, the effect of DCG-IV on ON-OFF
87                An important issue is how the starburst amacrine cells, which are known to provide a m
88                                 For example, starburst amacrine cells, which lack syt 2 in adult reti
89 ing RGC loss in the Ndufs4 KO is the loss of starburst amacrine cells, which may be an important targ
90  RGCs by inhibiting transmitter release from starburst amacrine cells.
91 te receptor (mGluR) subtype are expressed in starburst amacrine cells.
92 f GABA(B) receptors on cholinergic/GABAergic starburst amacrine cells.
93 nd SV2C being important for the functions of starburst amacrine cells.
94 rret retina through immunotoxin depletion of starburst amacrine cells.
95 ly belonged to several different cholinergic starburst amacrine cells.
96 tly more distally to the processes of the ON-starburst amacrine cells.
97 e of the two mirror-symmetric populations of starburst amacrine cells.
98 ic, nicotinic acetylcholine (ACh) input from starburst amacrine cells.
99  production (by 30%) of RGCs, and absence of starburst amacrine cells.
100 bpopulation of W3 ganglion cells, but not to starburst amacrine cells.
101 rd GABAergic/cholinergic signals mediated by starburst amacrine cells.
102 y be generated by a mechanism independent of starburst amacrine cells.
103 ic acid (GABA) release from the dendrites of starburst amacrine cells.
104    Regular spacing was also observed for the starburst amacrine cells.
105 dritic field, is dependent on the density of starburst amacrine cells.
106 natal stages and adult retinas, the presumed starburst amacrine ChAT-IR cells formed two mirror-like
107  In the retina, the beautifully symmetrical 'starburst' amacrine cells interact with each other in a
108    The membrane excitability of cholinergic (starburst) amacrine cells was studied in the rabbit reti
109 h-clamp recordings from pairs of neighboring starburst and ganglion cells show that the rhythmic acti
110 ay limit uncorrected visual acuity and cause starbursts and glare at night.
111 son) showed a correlation with postoperative starbursts and pupil size and a decrease in starbursts w
112 rative dysphotopsia introduces glare, halos, starbursts and shadows in a small number of patients.
113 tifacts manifest themselves as glare, halos, starbursts and shadows.
114 ved that they were formed in intense nuclear starbursts and that they ultimately grew into the most m
115                               This ancestral starburst appears similar to those being found by submil
116 eparation of 19 kiloparsecs, the two massive starbursts are about to merge and form a passive ellipti
117  ganglion cell that does not stratify in the starburst band, this suggests that its GABA-dependent di
118 ynapses from a type of amacrine cell termed 'starburst' because of its regularly spaced, evenly radia
119 , but so far there has been no evidence that starbursts can propel substantial quantities of cold mol
120 te to the electrical isolation of individual starburst cell dendrites, a property thought essential f
121 o make these receptors active comes from the Starburst cell itself, making them autoreceptors.
122 ohistochemistry revealed their expression in starburst cell somata and dendrites.
123 cell, which then feeds back and inhibits the Starburst cell.
124  We report that the directional responses of starburst-cell dendrites and DS ganglion cells are highl
125 distribution of the two cotransporters along starburst-cell dendrites mediates direction selectivity.
126 evidence that the cholinergic outputs of the starburst cells affect the responses of the ganglion cel
127 tivity and that the directional responses of starburst cells and DS ganglion cells are exquisitely se
128 stead of being direction discriminators, the starburst cells appear to potentiate generically the res
129                             The processes of starburst cells are connected asymmetrically to directio
130 ll recording, Zheng et al. now show that the starburst cells are mutually excitatory during early dev
131 h maturation, the nicotinic synapses between starburst cells dramatically diminished, whereas the GAB
132 lamp recordings were made from 110 displaced starburst cells in a thin retina] slice preparation of r
133                                  We labelled starburst cells in living retinas, then killed them by t
134 ase, we performed whole-cell recordings from starburst cells in mouse retina.
135 al nicotinic and GABAergic synapses between, starburst cells in the perinatal rabbit retina.
136                         The same was true of starburst cells injected intracellularly with Neurobioti
137 ion cells show that the rhythmic activity in starburst cells is closely correlated with that in gangl
138  the directional discrimination in which the starburst cells participate, namely, that their choliner
139                                              Starburst cells project inhibition laterally ahead of a
140 starburst amacrine cell network suggest that starburst cells regulate their dendritic overlap to ensu
141                         After vision begins, starburst cells release GABA in a prolonged and Ca2+-dep
142 cking the K-Cl cotransporter resulted in the starburst cells responding equally to light moving in op
143 erstand the intrinsic membrane properties of starburst cells responsible for direction-selective GABA
144 endrites and KCC2 on the distal dendrites of starburst cells results in a GABA-evoked depolarization
145                                     Ablating starburst cells revealed no asymmetric contribution to t
146 fter eye opening (P10), such that all of the starburst cells tested before eye opening had conspicuou
147      We show that this transition allows the starburst cells to use their neurotransmitters for two c
148                     We report that displaced starburst cells undergo a dramatic transition from spiki
149                     Our results suggest that starburst cells use action potentials transiently during
150 er, in contrast to ganglion cells, displaced starburst cells usually do not generate spontaneous soma
151                      The Kv3-like current in starburst cells was absent in Kv3.1-Kv3.2 knock-out mice
152 e bipolar cells follow the pattern of the ON-starburst cells' processes.
153 sistently hyperpolarized and depolarized the starburst cells, respectively, and greatly reduced or el
154 olinergic and the GABAergic synapses between starburst cells.
155  immunolabeling favoring proximal regions of starburst cells.
156 glion cells in a manner opposite that of the starburst cells.
157 he bundles formed by the processes of the ON-starburst cells.
158 er an alternative concept of the function of starburst cells.
159 at S15 and included not only the presumptive starburst cholinergic amacrine cells but also a populati
160 ed a high level of synaptic intricacy in the starburst circuit and suggested differential, yet synerg
161                                   A 'maximum starburst' converts the gas into stars at a rate more th
162 contrast sensitivity without glare, halos or starbursts, defocus curves, optical scatter, retinal poi
163 l (i.t.) injection of 131I-labeled boronated starburst dendrimer (BSD) or BSD-EGF.
164 boronated precision macromolecule [boronated starburst dendrimer (BSD)] was chemically linked to EGF
165  bacteria using an optical sensor based on a starburst dendrimer film containing a lipophilic fluorop
166                          A fourth generation starburst dendrimer was boronated and linked to EGF usin
167                    Our results indicate that Starburst dendrimers can be effective carriers for the i
168               These studies demonstrate that Starburst dendrimers can transfect a wide variety of cel
169 onal release of gamma-aminobutyric acid from starburst dendrites and that the asymmetric distribution
170 the NKCC2 and KCC2 cotransporters located on starburst dendrites consistently hyperpolarized and depo
171  of the GABA reversal potential in different starburst dendritic compartments indicate that the GABA
172                    Individual sectors of the starburst dendritic field are directionally selective by
173 robably approximately 3, indicating that the starburst-driven wind limits the star-formation activity
174 ymptoms (double images, glare, halos, and/or starbursts), dry eye symptoms, participant satisfaction
175 l symptoms (double images, glare, halos, and starbursts), dry eye symptoms, satisfaction with vision,
176 ure enough to form the most massive, intense starbursts existed at least as early as 880 million year
177 at resemble spherical baskets, cuboid cages, starbursts, flowers, scaffolds, fences, and frameworks,
178 cluster appears to be experiencing a massive starburst (formation of around 740 solar masses a year),
179 g of plate-like rectangles and fan-shaped or starburst forms.
180 g far (more than 10 kiloparsecs) outside the starburst galaxies (which have radii of less than 1 kilo
181                                Consequently, starburst galaxies are ideal for studying the interplay
182 z > 4, indicating that the fraction of dusty starburst galaxies at high redshifts is greater than pre
183                                              Starburst galaxies at low redshifts, however, generally
184 bsorption lines in the spectra of six lensed starburst galaxies at redshifts near 2.5.
185                                              Starburst galaxies at the peak of cosmic star formation
186       Recent observations have revealed that starburst galaxies can drive molecular gas outflows thro
187 tions indicate additional contributions from starburst galaxies or heavily filtered quasar radiation.
188 onized by the discovery that luminous, dusty starburst galaxies were 1,000 times more abundant in the
189 ntial masses and alter the evolution of post-starburst galaxies.
190 population of luminous, high-redshift, dusty starburst galaxies.
191 ocess, especially in the central regions of 'starburst' galaxies where star formation is vigorous.
192                           Young clusters in 'starbursting' galaxies in the local and distant Universe
193  we report observations of NGC 253, a nearby starburst galaxy (distance approximately 3.4 megaparsecs
194 mpact (effective radius 100 parsecs) massive starburst galaxy at redshift 0.7, which is known to driv
195 re we report the identification of a massive starburst galaxy at z = 6.34 through a submillimetre col
196         Here we report that the nearby dwarf starburst galaxy Henize 2-10 (refs 5 and 6) contains a c
197 outside the galactic plane of the archetypal starburst galaxy M82 (refs 4 and 5), but so far there ha
198 solution x-ray observations of the prototype starburst galaxy Messier 82 (M82) obtained with the adva
199  times more mass than the cold gas in a post-starburst galaxy wind.
200    NGC 4449 is a nearby Magellanic irregular starburst galaxy with a B-band absolute magnitude of -18
201  redshift (z) distribution of dusty, massive starbursts has long been suspected to be biased low in z
202 hile positive dysphotopsia (glare, halos and starbursts) has been largely attributed to edge effects
203                                 In addition, starburst inhibition is itself directionally selective:
204 , indicating a coordinated conversion of the starburst network excitability from an early hyperexcita
205                During early development, the starburst network mediates recurrent excitation and spon
206 gham model in which GABA(B) receptors are on starburst, not glycinergic amacrine cells.
207 g individual amacrine cell circuits like the starburst or A17 circuit have demonstrated that single t
208 f 138) for a pigmented lesion with a typical starburst pattern seen via dermatoscopy.
209 e total number of photons emitted during the starburst phase is sufficient to ionize intergalactic me
210 intly to turbulence and gravity, extends the starburst phase of a galaxy instead of quenching it.
211                                              Starburst polyamidoamine (PAMAM) dendrimers are a new ty
212                                              Starburst polyamidoamine dendrimers are a new class of s
213  unknown how soon after the Big Bang massive starburst progenitors exist.
214 ce of gaps in the neutral gas enveloping the starburst region.
215 to expanding molecular shells located in the starburst region.
216  screen was ejected from the galaxy during a starburst several 10(8) years earlier and has subsequent
217       Expanding molecular shells observed in starburst systems such as NGC 253 and M 82 may facilitat
218 vy elements through intense, dust-enshrouded starbursts--that is, increased rates of star formation--
219       If the emission is from dust heated by starbursts, then the majority of stars in spheroids were
220  of glutamate is processed differentially by starbursts via AMPA receptors and DSGCs via NMDA recepto
221 ense gas, an essential feature of an immense starburst, which contributes, together with the active g
222 t have formed at early epochs in spectacular starbursts, which should be luminous phenomena when obse
223  starbursts and pupil size and a decrease in starbursts with wavefront-guided treatments compared wit

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