ライフサイエンスコーパス: stenoses

1 ree of six shunts and moderate in two (focal stenoses).

2 asurements were completed in 28 patients (28 stenoses).

3 nts (38 portal vein and 12 hepatic vein-cava stenoses).

4 the deep femoral artery (one thrombosis, 13 stenoses).

5 and coronary CTA 1-6 d after PCS of culprit stenoses.

6 FFR was </=0.80 in 28 (45.9%) stenoses.

7 s, although the reverse held true for severe stenoses.

8 k between radiation and location of coronary stenoses.

9 identifying hemodynamically severe coronary stenoses.

10 scriminating between different severities of stenoses.

11 tenoses and identifying more unstable milder stenoses.

12 , Massachusetts) in treating coronary artery stenoses.

13 severe saphenous vein aorto-coronary bypass stenoses.

14 essel coronary artery disease, and left main stenoses.

15 xcellent vessel conspicuity and depiction of stenoses.

16 patients with severe (>/=70%) single-vessel stenoses.

17 There were 292 patients with 613 matched stenoses.

18 ngiography and DSA for detection of arterial stenoses.

19 erial image quality and presence of arterial stenoses.

20 ctive value for exclusion of coronary artery stenoses.

21 valuated vascular image quality and arterial stenoses.

22 mine the functional significance of coronary stenoses.

23 management of select benign tracheobronchial stenoses.

24 urate assessment of the presence of coronary stenoses.

25 ptomatic hemodynamically significant carotid stenoses.

26 with grafts free of significant (> or =75%) stenoses.

27 This applies to native and artificial stenoses.

28 5 treated lesions were 88 occlusions and 417 stenoses.

29 compared with post-procedural duplex-defined stenoses.

30 ) were correlated with the number of complex stenoses.

31 rs of the number of angiographically complex stenoses.

32 6% for the detection of significant coronary stenoses.

33 patients with focal, native coronary artery stenoses.

34 ion of moderate and severe epicardial artery stenoses.

35 ion scan were both abnormal, 70% had >or=50% stenoses.

36 que in patients without significant coronary stenoses.

37 coronary arteries with only mild or moderate stenoses.

38 had hemodynamically significant renal artery stenoses.

39 hemodynamically significant accessory artery stenoses.

40 ree patients had hemodynamically significant stenoses.

41 cation matched arteries with the most severe stenoses.

42 he physiologic assessment of coronary artery stenoses.

43 be present before the development of luminal stenoses.

44 , was the reference for defining significant stenoses.

45 PCI had revascularisation of all identified stenoses.

46 ate the hemodynamic significance of coronary stenoses.

47 discriminate malignant from nonmalignant CBD stenoses.

48 city for predicting functionally significant stenoses.

49 provided, presenting only focal intermediate stenoses.

50 is used to treat significant atherosclerotic stenoses.

51 detecting functionally significant coronary stenoses.

52 city for predicting functionally significant stenoses.

53 for the pressure-only assessment of coronary stenoses.

54 ermediate or borderline significant coronary stenoses: (1) pressure wire-derived coronary fractional

55 METHODS AND In 1382 coronary stenoses (1104 patients), percent diameter stenosis by v

56 D stenoses than controls or nonmalignant CBD stenoses (2.41 x 10(15) vs 1.60 x 10(14) nanoparticles/L

57 ks, 10 symptomatic marginal ulcers, 5 stomal stenoses, 3 bowel obstructions, 26 incisional hernias (n

58 At milder degrees of stenoses (50-69%), presence of collaterals was associate

59 n an additional 9 patients with intermediate stenoses (53+/-7%), 14 fractional flow reserve (FFR) mea

60 these two techniques of measuring CFR in 25 stenoses (6 vessels) artificially created by inflating s

61 stenoses (92%) and eight of 13 with coronary stenoses (62%) solely in the left anterior descending ar

62 Seventy-five stenoses (67 patients) underwent paired flow velocity as

63 have thrombus (42.5% versus 29.3%), tighter stenoses (72.0% versus 64.8%), and higher rates of TIMI

64 patients without significant coronary artery stenoses (76 +/- 37 ms vs. 38 +/- 23 ms, p < 0.001).

65 ivity for detection of all individual vessel stenoses (78% vs. 58%, p < 0.001) and patients overall (

66 In the 38 portal vein stenoses, 9 had prior perioperative portal vein and/or 5

67 eterization revealing 12 of 13 with coronary stenoses (92%) and eight of 13 with coronary stenoses (6

68 the per-stenosis analysis were 97% (29 of 30 stenoses), 96% (23 of 24 stenoses), 97% (29 of 30 stenos

69 ses), 96% (23 of 24 stenoses), 97% (29 of 30 stenoses), 96% (23 of 24 stenoses), and 96% (52 of 54 st

70 were 97% (29 of 30 stenoses), 96% (23 of 24 stenoses), 97% (29 of 30 stenoses), 96% (23 of 24 stenos

71 ding pressure by stenting of severe coronary stenoses, a proportional increase in vessel diameter is

72 was significantly smaller than DSQCA in mild stenoses, although the reverse held true for severe sten

73 condition with a common theme of multifocal stenoses and aneurysms in large arteries, accompanied by

74 Data for 50-69% stenoses and combinations of non-invasive tests were spa

75 in Eln(-)(/)(-) mice are also tortuous with stenoses and dilations.

76 between angiographic assessment of coronary stenoses and fractional flow reserve (FFR) is weak.

77 demonstrating a protective role with severe stenoses and identifying more unstable milder stenoses.

78 A total of 543 preoperative stenoses and occlusions were quantified and followed.

79 specificity were calculated for detection of stenoses and occlusions, as well as for confidence level

80 n images in the evaluation of central venous stenoses and occlusions.

81 on which coronary arteries have significant stenoses and on patient management.

82 In four arteries with severe stenoses and one occlusion (mean, 86%; range, 75%-100%),

83 uation of the ischemic potential of coronary stenoses and the expected benefit from revascularization

84 ment in multiple focal intracranial arterial stenoses and two demonstrated worsening.

85 ses), 97% (29 of 30 stenoses), 96% (23 of 24 stenoses), and 96% (52 of 54 stenoses), respectively.

86 nce of coronary calcification, morphology of stenoses, and anatomic characteristics, are under geneti

87 lates in vivo geometries, such as aneurysms, stenoses, and bifurcations, and supports endothelial cel

88 ved outcomes in small vessels, long coronary stenoses, and possibly saphenous vein graft intervention

89 uality of vessel definition, the severity of stenoses, and the presence of collateral vessels.

90 angiography for detection of coronary artery stenoses appears promising enough to warrant pursuit of

91 iagnosis of malignant common bile duct (CBD) stenoses are complex and lack accuracy.

92 ificant undergo PCI than when nonsignificant stenoses are treated.

93 identifying hemodynamically severe coronary stenoses as determined by fractional flow reserve (FFR).

94 at in patients with functionally significant stenoses, as determined by measurement of fractional flo

95 ween FFR and CFVR occurred in 31% and 37% of stenoses at the 0.75, and 0.80 FFR cut-off value, respec

96 val, 12 swine underwent surgical creation of stenoses at the left common carotid, right renal, and le

97 For detection of >/=70% stenoses based on angiographic criteria, a fully automat

98 mic flow velocity was observed when treating stenoses below physiological cut points; treating stenos

99 n apparent at sites with only modest luminal stenoses (but marked positive remodeling).

100 There were no significant PV stenoses, but adverse events included 1 episode of cardi

101 the functional significance of intermediate stenoses, but also have inherent limitations.

102 care for treatment of native coronary artery stenoses, but optimum treatment strategies for bare meta

103 om PCI was being considered, we assessed all stenoses by measuring FFR.

104 ion of hemodynamically significant (>or=50%) stenoses by using various image postprocessing methods,

105 th left main or ostial right coronary artery stenoses, bypass graft stenoses, chronic total occlusion

106 We hypothesized that these stenoses can be detected at rest without recourse to str

107 ion of plaques that may not produce critical stenoses causes many acute coronary syndromes (ACS).

108 nd the number and localization of functional stenoses changed in 32%.

109 right coronary artery stenoses, bypass graft stenoses, chronic total occlusions, planned two-stent bi

110 CI, hyperemic flow velocity is diminished in stenoses classed as physiologically significant compared

111 mic flow velocity increases 6-fold more when stenoses classed as physiologically significant undergo

112 higher in those with severe coronary artery stenoses compared to those with no vessel disease.

113 in patients with intermediate single-vessel stenoses, complex bifurcation and ostial branch stenoses

114 In 22 patients without significant coronary stenoses, contrast-enhanced MDCT (0.75-mm collimation, 4

115 ing and hyperemic flow velocity after PCI in stenoses defined physiologically by fractional flow rese

116 chemia may facilitate evaluation of moderate stenoses, designation of the culprit lesion, and predict

117 extracranial internal carotid artery (eICA) stenoses, detectable via submandibular Doppler sonograph

118 Long-term problems include coronary stenoses, distortion of the pulmonary arteries, dilatati

119 pigmentary retinopathy, and multiple tubular stenoses (e.g., bile ducts, ureters).

120 y enlargement) has expanded attention beyond stenoses evident by angiography to encompass the biology

121 Across all stenoses, extent of collaterals was a predictor for subs

122 w was higher than iFR flow in nonsignificant stenoses (FFR >0.75; mean FFR flow, 42.3+/-22.8 cm/s ver

123 artery disease and functionally significant stenoses, FFR-guided PCI plus the best available medical

124 In significant stenoses, flow velocity over the resting wave-free perio

125 e extent and complexity of residual coronary stenoses following percutaneous coronary intervention (P

126 ssure and flow velocity were measured in 216 stenoses from 186 patients with coronary disease.

127 iating fixed muscle hypertrophy and fibrotic stenoses from acute transmural inflammatory stenoses in

128 rams at 2 centers had each of their coronary stenoses graded serially by using 6 thresholds (grade 0

129 ced MR angiography for detection of arterial stenoses greater than 50% were 94% and 98% for reader 1

130 rator characteristic curve for prediction of stenoses > or = 70% by the MF method was 0.92 +/- 0.04 v

131 All coronary stenoses > or =30% diameter reduction (n =531 in 322 pat

132 m 96% survival for 1 stenosis > or =70% or 2 stenoses > or =50% (p = 0.013) to 85% survival for > or

133 with 3-vessel disease and noninfarct-related stenoses >/=90%, and in this subgroup, there was a nonsi

134 Patients with complex coronary stenoses had a significantly (P<0.001) higher PAPP-A/pro

135 Patients in whom all stenoses had an FFR of more than 0.80 received medical t

136 Patients in whom all stenoses had an FFR of more than 0.80 were entered into

137 nically dilate obstructive coronary arterial stenoses has vastly improved our approach to managing pa

138 g functionally significant coronary arterial stenoses; however, larger studies are required to determ

139 ngiography (one stenosis in 13 patients, two stenoses in 15 patients, and three stenoses in four pati

140 We studied 157 intermediate coronary stenoses in 157 patients, evaluated by FFR and CFVR betw

141 A total of 213 intermediate coronary stenoses in 184 patients were enrolled.

142 using the intracoronary pressure wire in 38 stenoses in 34 patients with significant coronary stenos

143 ents, two stenoses in 15 patients, and three stenoses in four patients).

144 onclusion Venous elastic recoil after PTA of stenoses in hemodialysis access circuits is common, but

145 Vessel diameters, frequency, and severity of stenoses in IVUS-imaged and nonimaged coronary arteries

146 ther PCI should be performed immediately for stenoses in nonculprit arteries is controversial.

147 ng can identify severe, unsuspected coronary stenoses in patients who had prior mediastinal irradiati

148 stenoses from acute transmural inflammatory stenoses in patients with Crohn's disease (CD) scheduled

149 R by CMR differentiates moderate from severe stenoses in patients with known or suspected CAD.

150 in therapy slows the progression of coronary stenoses in proportion to average low-density lipoprotei

151 mmed stenosis score (p = 0.002), integrating stenoses in series, was the best predictor of MFR(region

152 ns, tracheostomy-related deaths, or tracheal stenoses in survivors.

153 perform FFR in angiographically intermediate stenoses in the absence of stress testing or in the pres

154 vessel coronary artery disease that includes stenoses in the proximal left anterior descending artery

155 raphy as multifocal if there were at least 2 stenoses in the same arterial segment; otherwise, they w

156 heritabilities were identified for proximal stenoses, in particular, left main CAD (h2=0.49+/-0.12;

157 velocities, which were associated with worse stenoses (incidence risk ratio [IRR] = 5.1, P </= .0001

158 ate the treatment decisions for intermediate stenoses, indicative of a worrisome disconnect between r

159 were identified in 45.8%, including arterial stenoses, interruptions and ectasia in 7.6%.

160 sured distal to the stenosis; in part 2 (118 stenoses), intracoronary pressure alone was measured.

161 In part 1 (39 stenoses), intracoronary pressure and flow velocity were

162 mporary use in longer, more complex coronary stenoses is lacking.

163 e (FFR) measurement of intermediate coronary stenoses is recommended by guidelines when demonstration

164 The clinical significance of small-vessel stenoses is therefore questionable.

165 ies, and the prognostic significance of such stenoses is uncertain.

166 ndex of the hemodynamic severity of coronary stenoses, is derived from invasive measurements and requ

167 re no instances of graft vascular thromboses/stenoses/leaks (0%).

168 ortion of patients with significant coronary stenoses, left ventricular systolic dysfunction, and dea

169 nicity, fistula age, fistula type, number of stenoses, maximal angioplastic balloon diameter used, an

170 dentification of hemodynamically significant stenoses may be confounded by coronary remodeling.

171 Patients with severe bilateral carotid stenoses may be predisposed to ICH, particularly if ther

172 One hundred ninety-seven patients with 257 stenoses (mean diameter stenosis 48%) were studied.

173 For 70 to 99% stenoses, more extensive collaterals diminished risk of

174 noses, complex bifurcation and ostial branch stenoses, multivessel coronary artery disease, and left

175 ly occluded (n = 6) or compromised by ostial stenoses (n = 5).

176 the detection of CAA (n=11), coronary artery stenoses (n=2), and coronary occlusions (n=2).

177 In the case of bifurcation stenoses, new evidence suggests that opening a stenotic

178 DISCOVER-FLOW (Diagnosis of Ischemia-Causing Stenoses Obtained Via Noninvasive Fractional Flow Reserv

179 -related complications including thromboses, stenoses, occlusions, and aneurysms.

180 -related complications including thromboses, stenoses, occlusions, and aneurysms.

181 TIPS was more often associated with shunt stenoses/occlusions, recurrent hemorrhage, shunt revisio

182 dicated whenever hemodynamically significant stenoses occur.

183 Hepatic vein-cava stenoses occurred after a mean of 37.2 +/- 35.2 months,

184 RD, 3 RSS, and 3 FS, while hepatic vein-cava stenoses occurred in 2 LRD, 8 RSS, and 2 FS.

185 Portal vein stenoses occurred in 32 LRD, 3 RSS, and 3 FS, while hepa

186 Stenoses of 50% or greater were detected with accuracy,

187 e CT angiography for detecting or ruling out stenoses of 50% or more according to conventional angiog

188 Stenoses of 50% or more were considered obstructive.

189 d fashion on 287 of 569 baseline angiograms (stenoses of 50-99% and adequate collateral views) in the

190 ngiography and duplex US accurately depicted stenoses of 70% or more compared with those depicted at

191 s had coexistent hemodynamically significant stenoses of accessory and main renal arteries.

192 ts with hemodynamically significant isolated stenoses of accessory renal arteries were calculated.

193 women [mean age, 71 years]) with 55 coronary stenoses of at least 50% underwent coronary CT angiograp

194 re of generalized arteriopathy presenting as stenoses of elastic arteries and hypertension.

195 mputed tomography angiography, DSA) revealed stenoses of its main branches, indicating Takayasu arter

196 Stenoses of the coronary arteries were graded from 0 to

197 gly utilized for hemodynamically significant stenoses of the extracranial carotid artery.

198 The percent stenoses of the veins draining each independent lung wer

199 However, the impact of incidental stenoses on future cardiac events remains unknown.

200 detecting physiologically relevant coronary stenoses on MCE.

201 phy depicted 28 abnormalities in the CIA (27 stenoses, one dissection), 185 in the EIA (17 thromboses

202 section), 185 in the EIA (17 thromboses, 167 stenoses, one dissection), one in the common femoral art

203 ed by the presence of occlusive/subocclusive stenoses or FFR measurements </= 0.80 in vessels >2mm.

204 y and popliteal artery, and six patients had stenoses or occlusions below the popliteal artery.

205 lization techniques and angioplasty to treat stenoses or occlusions in 16 patients: 10 patients had h

206 s in 16 patients: 10 patients had high-grade stenoses or occlusions longer than 5 cm in the superfici

207 pecificity (0.56) in the detection of venous stenoses or occlusions.

208 entified with late portal vein or vena caval stenoses or thromboses from a cohort of 524 grafts with

209 were also analyzed for significant coronary stenoses (over 50% luminal narrowing) by segment, by art

210 o 0.78) to identify functionally significant stenoses (p = 0.04).

211 cluded were patients with stent occlusion or stenoses, peripheral arterial disease (ABI <1.0), sympto

212 CIA and EIA stenoses predominantly involved the distal and proximal

213 I in noninfarct coronary arteries with major stenoses (preventive PCI) is unknown.

214 th multiple arteries, localized renal artery stenoses produced focal elevations of R2*, suggesting ar

215 of the distribution of coronary vessels and stenoses provided a measure of myocardial jeopardy that

216 tive (r(2) = 0.52; p < 0.001) and artificial stenoses (r(2) = 0.54; p < 0.05), although the pressure-

217 neys downstream of high-grade renal arterial stenoses, R2* was elevated at baseline, but fell after f

218 f 48 patients were enrolled: 27 patients (40 stenoses) randomly assigned to the 1-minute group and 21

219 ve that the presence of endothelial cells in stenoses reduces platelet adhesion but increases sickle

220 Overall, the stenoses regressed from 79% at baseline (interquartile r

221 ently, the detection of noncritical coronary stenoses requires some form of stress.

222 Before randomisation, stenoses requiring PCI were identified on the angiogram.

223 , 96% (23 of 24 stenoses), and 96% (52 of 54 stenoses), respectively.

224 normal, luminal irregularities, and moderate stenoses, respectively, which were confirmed by catheter

225 Upon hip flexion, 23 CIA and 116 EIA stenoses showed kinking (mean amplitude, 76 degrees +/-

226 substantial number of coronary arteries with stenoses showing an FFR>0.80 present disturbed hemodynam

227 I in noninfarct coronary arteries with major stenoses significantly reduced the risk of adverse cardi

228 However, CTA assessment of coronary stenoses tends toward overestimation, and even among CTA

229 terminant of the natural history of coronary stenoses than anatomy (DS).

230 ile samples from patients with malignant CBD stenoses than controls or nonmalignant CBD stenoses (2.4

231 in 8 open-chest dogs with critical coronary stenoses that abolished flow reserve.

232 In the presence of coronary stenoses that abolished regional flow reserve, myocardia

233 rease in systolic VI was noted with coronary stenoses that resulted in progressive increases in the s

234 cal success was achieved in the treatment of stenoses that were resistant to high-pressure angioplast

235 y artery disease, we assessed the FFR in all stenoses that were visible on angiography.

236 evolve from mild-to-moderate coronary artery stenoses, that patients who experience a fatal coronary

237 amely a global coronary score and high-grade stenoses, the prevalence of atherosclerosis was analyzed

238 us coronary intervention of complex coronary stenoses, their use appears to be reasonably cost-effect

239 uminal angioplasty of dialysis access venous stenoses, there was no significant difference in postint

240 s of pressure wire drift are enough to cause stenoses to change classification.

241 re chronically instrumented with LAD and LCX stenoses to produce viable dysfunctional myocardium and

242 nature (anatomic or functional) of coronary stenoses to the perfused myocardium supplied by the targ

243 ly assigned to the 1-minute group and 21 (36 stenoses) to the 3-minute group.

244 Retrospective review of 175 cervical carotid stenoses treated with elective CAS from April 2001 to Fe

245 Three stenoses (two mild, one severe) were identified at SSFP

246 ses in 34 patients with significant coronary stenoses undergoing percutaneous intervention.

247 Seventeen patients with severe carotid stenoses underwent FDG-PET imaging 3 h after FDG adminis

248 Thirty-two subjects with carotid stenoses underwent noninvasive imaging with FDG positron

249 A total of 283 patients (310 coronary stenoses) underwent coronary angiography with FFR using

250 Mean MRA index of number and severity of stenoses was 0.84 +/- 0.68 (normal 0), % wall volume 74

251 sitivity for detecting more than 50% luminal stenoses was 89%; specificity, 65%; positive predictive

252 sitivity for detecting more than 70% luminal stenoses was 94%; specificity, 67%; positive predictive

253 angiography to depict substantial (>/= 50%) stenoses was assessed by using quantitative coronary ang

254 n defects was only 43+/-31%, and 68% of such stenoses were <50%.

255 The percentage stenoses were 21% +/- 22% versus 19% +/- 15% (not statis

256 MACE) at 2 years in 607 patients in whom all stenoses were assessed by FFR and who were treated with

257 Four hundred forty-seven stenoses were assessed with FFR, iFR, and whole-cycle Pd

258 A total of 157 stenoses were assessed.

259 These noncritical stenoses were classified as mild, moderate, or severe on

260 1 coronaries (78 patients) with intermediate stenoses were classified in 4 FFR and coronary flow rese

261 Post-procedural angiographic stenoses were compared with post-procedural duplex-defin

262 Relevant stenoses were correctly identified with SSFP MR angiogra

263 Variable stenoses were created in the left main coronary arteries

264 meter were assessed while graded, controlled stenoses were created in the stented segment by progress

265 When graded stenoses were created, a decrease in diameter of 18 +/-

266 Arterial stenoses were detected with contrast-enhanced MR angiogr

267 All deep femoral artery stenoses were diaphragm-like and involved the lateral ci

268 those with evidence of >10% coronary artery stenoses were divided into 2 groups, with either stable

269 The stenoses were divided into 4 groups according to FFR and

270 st 2001, 1058 patients with complex coronary stenoses were enrolled in the SIRIUS trial and randomize

271 More central venous stenoses were found at fistulography than at US.

272 Four to five intermediate-severity stenoses were generated in the left anterior descending

273 Portal vein stenoses were identified after bleeding (17/38), ascites

274 Stenoses were identified in 23 (68%) of 34 ankles, fibro

275 Hemodynamically significant (>50%) stenoses were identified in 88% (89 of 101) of patients;

276 Stenoses were labeled as severe if FFR </=0.80.

277 On subsequent angiograms, stenoses were observed in 16.2% (21/130) of nonimaged ar

278 tients with abnormal TCD and/or intracranial stenoses were placed on a transfusion program.

279 on-critical (n = 6) or flow-limiting (n = 4) stenoses were placed on coronary arteries of 10 open-che

280 Overall, 35 coronary artery stenoses were present.

281 rformed in three steps: (a)Coronary arterial stenoses were scored for severity and reader confidence

282 No coronary ostial stenoses were seen.

283 nformation required) and also recorded which stenoses were significant.

284 EIA stenoses were significantly longer in women (P < .003).

285 High-grade unilateral renal arterial stenoses were surgically created in eight pigs.

286 All ureteric stenoses were treated by surgical reconstruction.

287 ctive values for the presence of significant stenoses were: by segment (n = 935), 86%, 95%, 66%, and

288 R/CFVR discordance is mainly attributable to stenoses where CFVR is abnormal.

289 termine their accuracy, especially at 50-69% stenoses where the balance of risk and benefit for carot

290 ssessing functional significance of coronary stenoses, which is more accurate than resting indices an

291 MRA detected moderate to severe anastomotic stenoses, which were confirmed at catheter angiography a

292 lenge in patients with intracranial vascular stenoses who are potential candidates for bypass surgery

293 tenotic > or = 70% aorto-ostial renal artery stenoses, who underwent implantation of a balloon-expand

294 atherosclerosis, but only 39% had 1 or more stenoses with >or=50% narrowing.

295 iscriminated malignant from nonmalignant CBD stenoses with 100% accuracy.

296 malignant vs patients with nonmalignant CBD stenoses with 63.3% diagnostic accuracy.

297 evaluated the data sets for the presence of stenoses with diameter reduction of 50% or more, by usin

298 ses below physiological cut points; treating stenoses with fractional flow reserve </=0.80 gained Del

299 The model is used to compute shear rates in stenoses with growing boundaries.

300 Sixty-one stenoses with intermediate angiographic severity were st