ライフサイエンスコーパス: stenosis

1 urred in women without obstructive CAD (<50% stenosis).

2 in patients with nonobstructive CAD (1%-69% stenosis).

3 esence or absence of obstructive CAD (>/=50% stenosis).

4 ificantly stenosed (>/=50%), or patent (<50% stenosis).

5 (10.4%) posterior tibial arteries had >/=50% stenosis.

6 ventricular biopsies of patients with aortic stenosis.

7 ation for human atherosclerotic renal artery stenosis.

8 -eluting stent vs CABG in patients with LMCA stenosis.

9 be made because of concomitant severe aortic stenosis.

10 ma, n = 15) or nonmalignant (CP, n = 15) CBD stenosis.

11 ne model of hemodialysis arteriovenous graft stenosis.

12 ccumulation at the IVC wall after 6 hours of stenosis.

13 arent epicardial coronary artery thrombus or stenosis.

14 tudy patients with severe symptomatic aortic stenosis.

15 ibrosis in mice with unilateral renal artery stenosis.

16 ed to assess the severity of coronary-artery stenosis.

17 D risk factors overlap with those for aortic stenosis.

18 ibrosis in mice with unilateral renal artery stenosis.

19 gliosis, ventricle enlargement, or ventricle stenosis.

20 mitral valve regurgitation and mitral valve stenosis.

21 RI and PET/CT data of 6 patients with aortic stenosis.

22 making, and survival in patients with aortic stenosis.

23 associated with ipsilateral atherosclerotic stenosis.

24 ars across 37 advanced economies have aortic stenosis.

25 dict the functional significance of coronary stenosis.

26 ciated with greater severity of angiographic stenosis.

27 on screening for asymptomatic carotid artery stenosis.

28 soregulatory ability and significant luminal stenosis.

29 ent of high-risk patients with severe aortic stenosis.

30 erative lumbar spondylolisthesis with spinal stenosis.

31 th aortic stenosis, and 193 with mild mitral stenosis.

32 reased risk in patients with moderate aortic stenosis.

33 h measures of plaque prevalence, extent, and stenosis.

34 nificantly enhanced due to the presence of a stenosis.

35 or defining high risk in asymptomatic aortic stenosis.

36 in patients with asymptomatic severe aortic stenosis.

37 se (CAVD) is the most common cause of aortic stenosis.

38 e prevention of stroke due to carotid artery stenosis.

39 or outcome prediction in asymptomatic aortic stenosis.

40 -resolution MRI protocols above the level of stenosis.

41 s attenuates aortic hypermuscularization and stenosis.

42 rial-enhanced volume scans were acquired per stenosis.

43 n, severe aortic regurgitation, or subaortic stenosis.

44 her 1,262 (0.02%) were diagnosed with mitral stenosis.

45 positive predictive value of 90% for >/=70% stenosis.

46 is independent of epicardial coronary artery stenosis.

47 score (CACS) for predicting coronary artery stenosis.

48 in patients with asymptomatic severe aortic stenosis.

49 ronary artery disease without left main LMCA stenosis.

50 e optic disc, and bilateral transverse sinus stenosis.

51 with different severities of coronary artery stenosis.

52 d demand in the management of carotid artery stenosis.

53 pendymal cells in aging-associated ventricle stenosis.

54 many intermediate-risk patients with aortic stenosis.

55 n was divided into normal, regurgitation, or stenosis.

56 the treatment of vertebral or basilar artery stenosis.

57 group), 58 (66.7%) had none or less than 50% stenosis, 29 (33.3%) had obstructive CAD (>/=50% stenosi

58 ea (8%), ascites (3%), fatigue (3%), gastric stenosis (3%), hepatic failure (3%), liver abscesses (3%

59 In 10 118 patients with no ipsilateral stenosis, 339 (6.7%) of 5047 patients in the ticagrelor

60 hereas women were more likely to have aortic stenosis (34.5% versus 44.1%, P<0.001).

61 dividuals with a diagnosis of carotid artery stenosis (4615 women and 5964 men; mean [SD] age, 65.6 [

62 ithin 1 year for moderate or severe valvular stenosis (64 [15.0%]), and routine surveillance of cardi

63 maly (4 patients, 6.5%), pulmonic atresia or stenosis (7 patients, 11.5%), truncus arteriosus (6 pati

64 osis, 29 (33.3%) had obstructive CAD (>/=50% stenosis), 7 (8%) with single-vessel disease, and 22 (25

65 [33.1%] vs 57 [24.1%], P < .001) and aortic stenosis (728 [38.7%] vs 51 [21.5%], P < .001).

66 e sclerosing cholangitis (75%) and malignant stenosis (9.5%).

67 l stenosis, and baseline percentage diameter stenosis, a strong trend was preserved in terms of targe

68 d point of the study was percentage diameter stenosis after 6 months measured by angiography.

69 Histopathology revealed lower area stenosis (AMA-BRS, 21.0+/-6.1% versus BVS 31.0+/-4.5%; P

70 ases, 32 cases were positive for significant stenosis and a total number of stenotic segments was 45.

71 derstanding of the pathophysiology of aortic stenosis and as a biomarker end point in clinical trials

72 anical intervention for patients with aortic stenosis and concomitant reduced ejection fraction.

73 Ventricle stenosis and fusion of the lateral ventricle (LV) walls

74 of patients with asymptomatic severe aortic stenosis and help to validate current guidelines for ser

75 ement (SAVR) for patients with severe aortic stenosis and high surgical risk.

76 Patients with severe, symptomatic aortic stenosis and high/intermediate surgical risk were enroll

77 203 patients with severe symptomatic aortic stenosis and increased surgical risk, as determined by t

78 to June 2016 using search terms intracranial stenosis and intracranial atherosclerosis.

79 in high-risk patients with coexisting aortic stenosis and LM disease.

80 show comparable safety in patients with LMCA stenosis and low to intermediate-complexity coronary art

81 of NAFLD is associated with coronary artery stenosis and need for coronary intervention, but not inc

82 g in these patients with asymptomatic aortic stenosis and no manifest atherosclerotic disease or diab

83 al of 1767 patients with asymptomatic aortic stenosis and no manifest atherosclerotic disease were an

84 For patients with asymptomatic severe aortic stenosis and normal left ventricular function, current p

85 The incidence of carotid artery stenosis and plaques, cardiac embolic source, TIA/stroke

86 ry of patients with medically managed aortic stenosis and preserved left ventricular function in an e

87 th a higher prevalence of significant aortic stenosis and regurgitation.

88 t available data on stress testing in aortic stenosis and subsequently summarizes its potential for g

89 ation and activation within the graft drives stenosis and that TGF-beta receptor 1 (TGF-betaR1) inhib

90 Depending of the severity of the aortic stenosis and the presence of concomitant heart disease a

91 Patients had severe aortic stenosis and were at increased surgical risk based on th

92 obstructive coronary artery disease (>/=70% stenosis) and 68 (5.5%) had ACS.

93 for discriminating between CAD (presence of stenosis) and the non-stenosis condition was 5.35 with 8

94 opathy (HOCM), 10 patients with aortic valve stenosis, and 14 healthy individuals using [(11)C]-aceta

95 17 had aortic regurgitation, 471 with aortic stenosis, and 193 with mild mitral stenosis.

96 sis, subjected to non-flow-limiting coronary stenosis, and after preadministration of caffeine.

97 on, diabetes mellitus, target vessel, serial stenosis, and baseline percentage diameter stenosis, a s

98 ricular hypertrophy, cardio-oncology, aortic stenosis, and ischemic heart disease.

99 T measures of coronary-artery calcium, total stenosis, and plaque did not differ significantly betwee

100 hypertension, iron deficiency anemia, aortic stenosis, and prior bariatric surgery.

101 Tonsillar herniation, foramen magnum stenosis, and severe osteomyelitis of the jaw were commo

102 of abnormal diffusion-weighted MRI, carotid stenosis, and transient ischaemic attack within 1 week o

103 ients with BAV undergoing surgery for aortic stenosis (aorta diameter </=45 mm: BAVnon-dil or >45 mm:

104 ine patients with asymptomatic severe aortic stenosis (aortic valve area <1 cm(2), peak jet velocity

105 Stenosis application increased plasma levels of soluble

106 Background: Atherosclerotic renal artery stenosis (ARAS) is associated with high blood pressure (

107 s like appendicitis and hypertrophic pyloric stenosis are all supported by good clinical evidence.

108 utaneous tracheostomy, particularly tracheal stenosis, are unclear.

109 dynamic cardiac consequences of aortic valve stenosis (AS) and aortic valve regurgitation (AR).

110 LV) systolic dysfunction and moderate aortic stenosis (AS) are more frequent with advancing age and o

111 In patients with severe aortic stenosis (AS) at intermediate surgical risk, treatment w

112 m in patients with severe symptomatic aortic stenosis (AS) in routine clinical practice.

113 RATIONALE: Calcific aortic stenosis (AS) is characterized by calcium deposition in

114 Aortic valvular stenosis (AS) is the most common cause of cardiac valvul

115 Severe aortic stenosis (AS) most often presents with reduced aortic va

116 n modify the natural history of aortic valve stenosis (AS).

117 cular risk factors and development of aortic stenosis (AS).

118 d flow as it occurs, for instance, in aortic stenosis (AS).

119 symptomatic patients with very severe aortic stenosis (AS); however, the definition of very severe AS

120 tissue was procured in patients with aortic stenosis (AS, n=9) and dilated cardiomyopathy (DCM, n=6)

121 imal models (eg, swine with chronic coronary stenosis); as well as human subjects.

122 predictions for the future of physiological stenosis assessment, outlining developments for both iFR

123 ting or DA in terms of angiographic diameter stenosis at 6 months and target lesion revascularization

124 Patients with severe aortic stenosis at increased risk for surgery had improved 3-ye

125 In patients with severe aortic stenosis at increased surgical risk, TAVR was associated

126 for patients with severe symptomatic aortic stenosis at intermediate surgical risk, TAVR volume is p

127 ve to surgery in patients with severe aortic stenosis at intermediate surgical risk, with a different

128 ted option for treating patients with aortic stenosis at intermediate to high or prohibitive surgical

129 l interest in patients with unstable carotid stenosis at risk of stroke.

130 mic stroke: 18 patients with culprit carotid stenosis awaiting carotid endarterectomy and 8 controls

131 l aortic valve replacement for severe aortic stenosis between 2012 and 2014 at our institution.

132 or individuals diagnosed with carotid artery stenosis between October 1, 2006, and September 30, 2010

133 lue for the determination of coronary artery stenosis but not for discriminating between different se

134 bgroups defined by sex and degree of carotid stenosis, but there was a nonsignificant trend suggestin

135 cits and detection of per-vessel obstructive stenosis by invasive coronary angiography were also comp

136 y stenoses (1104 patients), percent diameter stenosis by visual estimation (DSVE) and by quantitative

137 ular disease (CVD) and calcific aortic valve stenosis (CAVS) is substantial.

138 ented more often with moderate/severe aortic stenosis compared with men.

139 to undergo procedural management for carotid stenosis compared with those in the salary-based setting

140 tween CAD (presence of stenosis) and the non-stenosis condition was 5.35 with 88.6% sensitivity and 8

141 r importance for patients with severe aortic stenosis considering transcatheter aortic valve replacem

142 tients with left main coronary artery (LMCA) stenosis, coronary artery bypass grafting (CABG) has bee

143 Stenosis created by inflammation and anatomic variations

144 ses (69% versus 73%; P=0.3), considering 50% stenosis cut point.

145 e evaluated 1023 patients with severe aortic stenosis deemed high or extreme risk for surgery treated

146 urve for different levels of coronary artery stenosis did not have sufficient sensitivity and specifi

147 e respective values of angiographic diameter stenosis (DS) and fractional flow reserve (FFR) in predi

148 Stenosis due to intimal hyperplasia development often oc

149 wed all moderate- or greater-severity aortic stenosis echocardiography studies with concomitant cathe

150 was carotid endarterectomy for patients with stenosis exceeding 50% to 60%.

151 issue provides a clinical overview of aortic stenosis, focusing on screening, diagnosis, treatment, a

152 aphy results showed a lower percent diameter stenosis for FP-PES (38.78% [31.27-47.66]) compared with

153 Significant CAD was defined as stenosis greater than or equal to 50%; cardiovascular ev

154 >/=300 Agatston units (11.3%) and a luminal stenosis >/=50% (7.5%).

155 k of CAC >70th percentile for age or luminal stenosis >/=50% in male athletes (odds ratio, 1.08; 95%

156 infarction, including 3(42%) with a luminal stenosis >/=50% in the corresponding artery.

157 The best predictor of LMCA stenosis >/=50% was a PA diameter >/=40 mm.

158 LMCA stenosis >/=50% was detected in 48 of the 94 patients.

159 tments for patients with symptomatic carotid stenosis >/=70%.

160 istic curve of 0.87 (p < 0.001) for coronary stenosis >/=70%.

161 fractional flow reserve </=0.80 or diameter stenosis >/=80% on quantitative coronary angiography was

162 of 3-vessel disease and noninfarct diameter stenosis >/=90% and was particularly pronounced in patie

163 ffect was observed in patients with diameter stenosis >/=90% of noninfarct-related arteries (HR, 0.32

164 defined as cervical internal carotid artery stenosis (>50%) or occlusion.

165 The definition of severe aortic stenosis has classically and retrospectively been based

166 o association between SBP and risk of mitral stenosis (HR per 20 mmHg higher SBP 1.03; CI 0.93, 1.14;

167 Intracranial atherosclerotic stenosis (ICAS) is a common cause of ischemic stroke, an

168 ed EF, enabling reclassification to moderate stenosis in 25% of patients.

169 changed the indication to treat the coronary stenosis in 8 of 133 (6%) lesions.

170 isk patients with severe, symptomatic aortic stenosis in a randomized trial comparing TAVR (performed

171 t, elevated scores were predictive of >/=70% stenosis in all subjects (odds ratio [OR]: 9.74; p < 0.0

172 Hemodynamically significant stenosis in at least 1 coronary artery as indicated by a

173 cohort of 649 subjects, predictors of >/=70% stenosis in at least 1 major coronary vessel were identi

174 singly common for the management of vascular stenosis in congenital heart disease.

175 stic recoil (P < .001), as was cephalic arch stenosis in fistulas (P = .047) and autogenous fistulas

176 n the Treatment of Symptomatic Severe Aortic Stenosis in High Risk and Very High Risk Subjects Who Ne

177 n the Treatment of Symptomatic Severe Aortic Stenosis in High Risk and Very High Risk Subjects Who Ne

178 placement (TAVR) for the treatment of aortic stenosis in high- and intermediate-risk patients, but th

179 owed positive correlation to the grade of LV stenosis in nonaged mice (<10-month-old), and that the e

180 od and Drug Administration for severe aortic stenosis in patients who cannot undergo surgery and for

181 147 (9.6%) of 1539 patients with ipsilateral stenosis in the aspirin group had an occurrence of strok

182 103 (6.7%) of 1542 patients with ipsilateral stenosis in the ticagrelor group and 147 (9.6%) of 1539

183 bleeding events in patients with ipsilateral stenosis in the ticagrelor group compared with the aspir

184 osis) in 814 patients and severe CAD (>/=70% stenosis) in 708 patients.

185 126 patients, showed obstructive CAD (>/=50% stenosis) in 814 patients and severe CAD (>/=70% stenosi

186 and midterm outcomes for adults with aortic stenosis, including those at low to intermediate risk.

187 We found a treatment-by-atherosclerotic stenosis interaction (p=0.017).

188 r a treatment-by-ipsilateral atherosclerotic stenosis interaction in a subgroup analysis of patients

189 nsification, antihypertensive class, carotid stenosis intervention, and substance abuse referral for

190 Aortic stenosis is 1 of the most common heart valve diseases am

191 ntermediate-risk patients with severe aortic stenosis is associated with low mortality, strokes, and

192 eful to answer questions such as whether the stenosis is clinically relevant; the identification of t

193 c valve replacement (TAVR) for severe aortic stenosis is growing rapidly.

194 nobstructive coronary arteries (MINOCA [<50% stenosis]) is more common among younger patients and wom

195 For regional detection of obstructive stenosis, ischemic total perfusion deficit areas under t

196 s influence the management of carotid artery stenosis, it is not well understood whether a preference

197 Patients with obstructive (>/=50% stenosis) left main (LM) coronary artery disease (CAD) a

198 Low-gradient severe aortic stenosis (LGSAS) with preserved ejection fraction (EF) i

199 ostic significance of nonobstructive (1%-49% stenosis) LM CAD, including sex-specific differences, ha

200 artery disease (NOCAD; wall irregularities, stenosis <60%), and women with NOCAD in particular, rema

201 e goals in patients with a history of aortic stenosis may need to be redefined.

202 Under left-anterior descending coronary stenosis, MBF increased in response to hypercapnia and a

203 In the absence of stenosis, mean MBF under hypercapnia was 2.1 +/- 0.9 mL/

204 ces of functional significance of a coronary stenosis measured without hyperemia.

205 In contrast to patients with aortic valve stenosis, MEE was not improved in patients with HOCM aft

206 " ependymal cells in aging-related ventricle stenosis; moreover, they also contribute to the progress

207 ost common indication for surgery was aortic stenosis (n = 225 [72.6%]).

208 Subgroup analysis showed that stenosis occurring less than 3 months (10-year graft sur

209 terior descending (58%), with a mean percent stenosis of 58+/-12% and a mean FFR of 0.82+/-0.09.

210 Lesions had mean area stenosis of 84.4% (SD 10.2), fractional flow reserve of

211 scularisation or asymptomatic carotid artery stenosis of at least 50%), or coronary artery disease wi

212 hy at university hospitals in Europe for CBD stenosis of malignant (pancreatic cancer, n = 20 or chol

213 and specificity in the detection of coronary stenosis of more than 50% compared with detection of ICA

214 -pump via thrombosis of the outflow graft or stenosis of the anastomosis to the aorta (4 events; 0.00

215 model, DVT was induced by partial ligation (stenosis) of the inferior vena cava.

216 y artery disease (CAD) was defined as >/=50% stenosis on angiography by core laboratory.

217 (5) what degree of stenosis on CTA warrants CT-FFR?

218 The degree of carotid artery stenosis on histology correlated well with the ultrasoun

219 Patients with confirmed LMCA stenosis on selective coronary angiography had PCI.

220 tion, and severity of the noninfarct-related stenosis on the effect of fractional flow reserve-guided

221 Carotid artery stenosis on the involved side was worse in AF and OIS co

222 subjected to bilateral common carotid artery stenosis or a sham operation and fed normal or cilostazo

223 Patients with moderate to severe mitral stenosis or mechanical heart valves were excluded from t

224 xcluded patients with moderate/severe mitral stenosis or mechanical heart valves, but variably includ

225 moderate aortic/mitral regurgitation, aortic stenosis, or prior valve surgery (bioprosthesis replacem

226 between different levels of coronary artery stenosis (P<0.001) and there was a significant positive

227 ic disc [p=0.881], and bilateral dural sinus stenosis [p=0.837], Mann-Whitney U test).

228 peration to relieve severe prosthetic aortic stenosis (PAS) is increasing.

229 ents were performed in HOCM and aortic valve stenosis patients 4 months after surgery.

230 Studies of TAVR in low-flow severe aortic stenosis patients have demonstrated that TAVR has a sign

231 medically treated angina and severe coronary stenosis, PCI did not increase exercise time by more tha

232 andomized clinical trial, patients with LMCA stenosis, PCI vs CABG, exclusive use of drug-eluting ste

233 gating the role of TAVR in lower-risk aortic stenosis populations, in patients with aortic regurgitat

234 Stenosis, present in 27.5% of patients, was associated w

235 risk with severe asymptomatic carotid artery stenosis randomly assigned to carotid artery stenting or

236 Atherosclerotic renal artery stenosis reduces renal blood flow (RBF) and amplifies st

237 lence, valve morphology, dysfunction (aortic stenosis/regurgitation), aortopathy, and complications (

238 ment of patients with low-flow severe aortic stenosis remains challenging.

239 d revascularization for asymptomatic carotid stenosis, require reassessment given advances in both me

240 .13 to 0.36), proportional to mean hyperemic stenosis resistance (Spearman rho =0.61; P=0.001).

241 ry end point was the difference in hyperemic stenosis resistance index between measurements before an

242 Mean hyperemic stenosis resistance was 0.37+/-0.19 Hg/cm/s for wire-onl

243 ved increases in coronary pressure gradient, stenosis resistance, and flow velocity did not reach sta

244 nts with severe atherosclerotic renal artery stenosis scheduled for PTRA.

245 s with known bicuspid valve, moderate aortic stenosis, severe mitral regurgitation, severe aortic reg

246 tent with a significant increase in relative stenosis severity (k coefficient, P<0.0001), in keeping

247 In patients with stable coronary disease, stenosis severity as assessed by FFR is a major and inde

248 tatus, left ventricular function, and aortic stenosis severity between patients with (n = 202) and pa

249 g-state physiological assessment of coronary stenosis severity using the instantaneous wave-free rati

250 bjects matched for age, kidney function, and stenosis severity.

251 sults were documented for resting indices of stenosis severity.

252 urgery include giant cell arteritis, carotid stenosis, stroke, hypercoagulable state, and DM with oph

253 graphy as part of the TWIST (Tweesteden Mild Stenosis) study.

254 ine in 3 groups of canines: without coronary stenosis, subjected to non-flow-limiting coronary stenos

255 Renal artery stenosis surgery (n = 10) or sham surgery (n = 5) was pe

256 wn that among high-risk patients with aortic stenosis, survival rates are similar with transcatheter

257 inally, pooled estimates of trials with LMCA stenosis tended overall to differ significantly from tho

258 of valve obstruction in patients with aortic stenosis, there is an independent association between po

259 lanin in the IVC increased after 48 hours of stenosis to a substantially higher extent in mice with a

260 surgical mechanical interventions for aortic stenosis (transcatheter aortic valve replacement) may al

261 measured in 2141 patients with severe aortic stenosis treated with TAVR in the PARTNER I trial (Place

262 k and inoperable patients with severe aortic stenosis undergoing a transcatheter aortic valve replace

263 n patients with atherosclerotic renal artery stenosis undergoing PTRA.

264 nical trial of patients with calcific aortic stenosis undergoing SAVR at 18 North American centers be

265 HODS AND Thirty patients with severe carotid stenosis underwent (18)F-fluorodeoxyglucose-positron emi

266 Fifteen patients with aortic stenosis underwent repeated 18F-fluoride PET-CT.

267 ant recipients (KT) with severe aortic valve stenosis underwent transfemoral TAVI.

268 ary outcome of angiographic percent diameter stenosis was 33.6+/-17.7% for DA+DCB versus 36.4+/-17.6%

269 between different levels of coronary artery stenosis was determined using receiver operating charact

270 Tortuosity or stenosis was evaluated if localized Doppler velocity ele

271 lence of rectal and large intestinal atresia/stenosis was higher for ART births compared with non-ART

272 The degree of stenosis was measured by the North American Symptomatic

273 iabetes had 15,887 postoperative angiograms; stenosis was quantified for 7,903 internal thoracic arte

274 ally symptomatic ipsilateral atherosclerotic stenosis was reported in 3081 (23%) of 13 199 patients.

275 In the multivariate analysis, ostial stenosis was significantly associated with procedural su

276 that the extent of LV-wall adhesions and LV stenosis was significantly lower in mid-aged (>10-month-

277 t 6 months angiography, the percent diameter stenosis was significantly lower in patients treated by

278 The proportion of severe CAD (>/=70% stenosis) was lower in patients who had hypertensive res

279 murine DVT model of inferior vena cava (IVC) stenosis, we demonstrate that mice with general inducibl

280 th diabetes have more severe coronary artery stenosis, we hypothesized that graft patency is worse in

281 gically guided assessment of coronary-artery stenosis were randomly assigned to undergo revasculariza

282 III+ aortic regurgitation and severe aortic stenosis were seen in 37% and 10%, respectively.

283 vere aortic regurgitation, and severe aortic stenosis were seen in 7%, 18%, and 2%, whereas 91% and 5

284 inst CEA for the treatment of carotid artery stenosis were selected.

285 y in symptomatic patients with severe aortic stenosis, whereas the management of asymptomatic patient

286 ve to surgery in patients with severe aortic stenosis who are at high surgical risk, less is known ab

287 dard therapy for patients with severe aortic stenosis who are at high surgical risk.

288 parative outcomes among patients with aortic stenosis who are at intermediate surgical risk.

289 evolving treatment for patients with aortic stenosis who require valve replacement.

290 t study included patients with severe aortic stenosis who underwent TAVR in the Society of Thoracic S

291 ng asymptomatic patients with severe carotid stenosis who were not at high risk for surgical complica

292 300 patients with asymptomatic severe aortic stenosis who were seen in the ambulatory Minneapolis Hea

293 95% CI, 2.2-4.4) patients have severe aortic stenosis with 1.9 million (95% CI, 1.3-2.6) eligible for

294 patients with malignant vs nonmalignant CBD stenosis with 100% accuracy.

295 e epidemiology and pathophysiology of aortic stenosis with heart failure and reduced ejection fractio

296 who had intracranial large-vessel occlusion/stenosis with sparse collaterals showed hypoperfusion by

297 dysfunction is intricately linked to aortic stenosis, with over 25% patients presenting for transcat

298 y artery bypass grafting in intermediate LAD stenosis without functional evidence of ischemia.

299 R included more postdilatations, pure aortic stenosis without regurgitation, and possibly more pacing

300 approaches to revascularization for carotid stenosis, yet contemporary data on trends in rates and o