1 e requiring angioplasty of a progressive FMD stenotic lesion.

2 n of the total myocardium in jeopardy from a stenotic lesion.

3 IL-2 and IL-15, which are amply expressed in stenotic lesions.

4 une responses appear to predominate in human stenotic lesions.

5  significantly higher in nonstenotic than in stenotic lesions (1.3 +/- 0.2 vs. 1.0 +/- 0.2, p < 0.001

6 icantly different between nonstenotic versus stenotic lesions (20 +/- 8 mm(2), n = 23 vs. 22 +/- 8 mm

7 gration, a key feature in the development of stenotic lesions after balloon injury.

8 n are not necessarily severely stenotic, and stenotic lesions are not necessarily unstable.

9                                     Notably, stenotic lesions, but not AAAs, contained mature forms o

10        We have shown that cells derived from stenotic lesions in infrainguinal vein grafts were signi

11 tion (PCI) should be considered for severely stenotic lesions in proximal coronaries that subtend a l

12                                      In five stenotic lesions, "negative remodeling" (Remodeling Inde

13 resorbable scaffold implantation in a simple stenotic lesion resulted in stable lumen dimensions and

14 om a stroke), probably culprit (not the most stenotic lesion upstream from a stroke), or nonculprit (

15 assified as either culprit (the only or most stenotic lesion upstream from a stroke), probably culpri

16 ow often early arterial wall changes lead to stenotic lesions, use of these modalities in combination

17                 Human vein graft-threatening stenotic lesions were identified by duplex scanning with

18                 The location and severity of stenotic lesions were recorded.

19 n of the first balloon-expandable valves for stenotic lesions with implantation in the pulmonic posit