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1 e and Necator americanus], and Strongyloides stercoralis).
2 nus), Trichuris trichiura, and Strongyloides stercoralis.
3 nd neutrophils to control infections with S. stercoralis.
4 e immune responses in mice against larval S. stercoralis.
5 tures consistent with B. dermatitidis and S. stercoralis.
6 e candidate against infection with larval S. stercoralis.
7 he Ss-riok-2 promoter in post free-living S. stercoralis.
8 se precursor from the nematode Strongyloides stercoralis.
9 encoding gene (Ss-riok-2) from Strongyloides stercoralis, a medically important parasitic nematode (O
11 nematode parasites, including Strongyloides stercoralis, Ancylostoma spp., and Necator americanus.
13 L3i) of the parasitic nematode Strongyloides stercoralis and compared the results to Caenorhabditis e
14 olecular and cellular biological study of S. stercoralis and of parasitic nematodes generally, and pr
16 Schistosoma mansoni, hookworm, Strongyloides stercoralis, and Mansonella perstans were the most preva
18 , including the human pathogen Strongyloides stercoralis, and their close relatives that are facultat
20 ophils were exposed to soluble Strongyloides stercoralis antigens, and the expression of various surf
22 s study provides a useful mouse model for S. stercoralis autoinfection and suggests a therapeutic str
25 signature.' Strikingly, in comparisons of S. stercoralis clusters to C. elegans homologs with RNAi kn
29 earance of infections, such as Strongyloides stercoralis, commonly found in the developing world but
31 solved the 3-dimensional structure of the S. stercoralis DAF-12 ligand-binding domain cocrystallized
32 nt in myeloperoxidase (MPO) infected with S. stercoralis had significantly decreased larval killing.
35 Protective immunity to larval Strongyloides stercoralis in mice has been shown to be dependent on an
43 reduced the pathogenic iL3 population in S. stercoralis, indicating the potential use of DAF-12 liga
44 gG antibody easily differentiated between S. stercoralis-infected and uninfected patients (P< .0001)
45 Tc9, Th17/Tc17, and Th22/Tc22 cells in 15 S. stercoralis-infected individuals and 10 uninfected indiv
47 umbers of adult and first-stage larvae in S. stercoralis-infected NSG mice, no hyperinfection was obs
48 ) than in uninfected (n=3) and Strongyloides stercoralis-infected patients (n=4), and greater in pati
49 ficant immunoreactivity was observed with S. stercoralis-infected sera, but a small number of patient
50 ranslocation is a feature of asymptomatic S. stercoralis infection and is associated with an inflamma
51 and with or without coexistent Strongyloides stercoralis infection before and after anthelmintic ther
52 that individuals with LTB and coexistent S. stercoralis infection have significantly lower levels of
59 native to ivermectin for the treatment of S. stercoralis infection, given that only slight difference
61 atory response in asymptomatic Strongyloides stercoralis infection, we measured the plasma levels of
68 subsets along with cytokine responses in S. stercoralis infections, which suggest the importance of
74 ed immune function, direct development of S. stercoralis is favored, whereas, in individuals with les
76 Here we show that the infective larva of S. stercoralis is strongly attracted to an extract of mamma
77 dauer or nondauer stages, matches between S. stercoralis L1 and C. elegans nondauer-expressed genes d
79 as determined that eosinophils killed the S. stercoralis larvae in naive mice, whereas these cells we
82 antigen-stimulated cytokine responses in S. stercoralis-LTB coinfection is reversible (for the most
83 om patients with parasitologically proven S. stercoralis or filarial infections and from healthy, uni
85 eated mice undergoing hyperinfection with S. stercoralis Overall, this study provides a useful mouse
86 as seen in those infected with Strongyloides stercoralis (P < 0.05) and when all helminth infections
87 ice, which are eosinophil deficient, with S. stercoralis resulted in development of primary and secon
90 bodies are both protective against larval S. stercoralis, they recognize different antigens and utili
92 s, and the intestinal parasite Strongyloides stercoralis was investigated in persons infected with on
93 ive third-stage larvae (L3) of Strongyloides stercoralis was shown to be dependent on immunoglobulin
95 tent mice are resistant to infection with S. stercoralis, we hypothesized that NSG mice, which have a
96 mical patterns of Ss-riok-2 expression in S. stercoralis, we observed expression patterns of a transg
98 ites Entamoeba histolytica and Strongyloides stercoralis were predictors of LBW despite their low pre
100 lopment of infective larvae of Strongyloides stercoralis, which may facilitate hyperinfection and, he
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