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1 ity sensitivity, a hallmark feature of adult stereopsis.
2 ey perform very poorly on clinical tests for stereopsis.
3 tical correction used for the development of stereopsis.
4 valence of reduced visual acuity and lack of stereopsis.
5 d be an indicator of binocular disparity and stereopsis.
6 development is binocular vision, also called stereopsis.
7 esults was evident for both local and global stereopsis.
8 r than retinal disparity has a role in human stereopsis.
9 9) in both groups and most patients had good stereopsis.
10 but had the highest prevalence of fusion and stereopsis.
11 ease in our knowledge of the neural basis of stereopsis.
12 D images show the same bias when tested with stereopsis.
13 nce implicating this pathway in fine-grained stereopsis.
14 nism that is important for foveal vision and stereopsis.
15 bly cerebral and not specifically related to stereopsis.
16 pital junction appears to be specialized for stereopsis.
17 ternative measure of sensory binocularity to stereopsis.
21 malities included strabismus (40%), abnormal stereopsis (44%), and limited ocular motility (40%).
22 nnot account for the conscious perception of stereopsis, although combining the outputs of many V1 ne
25 plex in the VEP could be a neural marker for stereopsis and fNIRS demonstrated differences in HbO.
27 between fusion and MVEP symmetry and between stereopsis and MVEP symmetry; the concordance between MV
28 eye assessment history, colour vision, gross stereopsis and non-cycloplegic autorefraction) were cond
29 wing the development of binocular vision and stereopsis and reducing the incidence of strabismus.
31 ositive family history, subnormal random-dot stereopsis, and hypermetropic anisometropia each pose a
32 nt a standard evaluation of ocular motility, stereopsis, and ocular alignment with alternate prism co
35 , the critical periods for susceptibility of stereopsis are described using four-parameter developmen
36 preprogrammed, and the mechanisms turning on stereopsis are extremely experience-dependent in humans.
37 This conclusion supports the clinical use of stereopsis as a screening test for bilateral monocular f
38 , breast-feeding is associated with enhanced stereopsis at age 3.5 y, as is a maternal DHA-rich anten
40 further evidence for qualitatively immature stereopsis based on relative disparity at 4-6 months of
41 a, the critical period for susceptibility of stereopsis begins at 10.8 months and peaks at 20 months.
42 s, the critical period for susceptibility of stereopsis begins at 2.4 months and peaks at 4.3 months.
43 s no statistically significant difference in stereopsis between the 2 treatment groups: Frisby (conta
44 gh AC/A accommodative esotropia; evidence of stereopsis, binocularity (on Worth 4-dot testing), or im
45 ereoanomalous showed substantial recovery of stereopsis, both on psychophysical tests with stimuli th
46 ity, 46% of children gained binocular fusion/stereopsis, but the quality of fusion gained was greater
47 ements are usually not conjugate, precluding stereopsis, but they maintain a wide region of binocular
49 the variables of visual acuity gain or loss, stereopsis, contrast sensitivity, high-order aberrations
50 ordination of eye movements is essential for stereopsis (depth perception) and to prevent double visi
51 ld of view, illumination, resolution, color, stereopsis, effect on head motion, and user interface.
53 et the critical period for susceptibility of stereopsis extends through late infancy and early childh
55 s, daily periods of binocular vision rescued stereopsis from the 10-fold reduction observed with cont
56 ive whose clinical tests indicated a lack of stereopsis) had measurable stereopsis of several thousan
58 , a lack of techniques to probe invertebrate stereopsis has prevented any further progress for three
59 computational, and psychophysical studies of stereopsis have assumed that the perceived surface struc
60 ith the view that before the abrupt onset of stereopsis, human infants may detect the differences bet
61 ly after rather than before the emergence of stereopsis (i.e., when the binocular connections are rel
62 anisms, both offer identical predictions for stereopsis in almost every viewing condition, making it
63 r, some of these patients even use this poor stereopsis in judging the size of stereoscopically prese
65 f periods of binocular vision could preserve stereopsis in monkeys reared with optical strabismus.
66 ar vision largely preserves local and global stereopsis in monkeys reared with optical strabismus.
69 his poor stereopsis, we investigated whether stereopsis in these subjects could support size constanc
74 eeks) of misalignment after the emergence of stereopsis is sufficient to drastically reduce the funct
77 that the critical immaturity limiting infant stereopsis is the well-known insensitivity of the infant
79 ise revealed the properties of the recovered stereopsis: It has reduced resolution and precision, alt
80 explanations, both based on the geometry of stereopsis, lead to two diametrically opposed hypotheses
81 ile esotropes before the known onset age for stereopsis may be important for maintaining better binoc
83 he balance toward the weak eye) and improves stereopsis more so than the push-only protocol, which so
87 omalous retinal correspondence and defective stereopsis of microstrabismus appear to be consequences
88 dicated a lack of stereopsis) had measurable stereopsis of several thousand are seconds or better.
90 4.5 years of age a masked examiner assessed stereopsis on these patients using 3 different tests: (1
92 4 mo were more likely to achieve high-grade stereopsis, or stereoscopic vision, than were children w
93 at 4-6 months of age.SIGNIFICANCE STATEMENT Stereopsis, our ability to sense depth from horizontal i
96 Daily periods of binocular vision preserved stereopsis over 16 weeks of optical strabismus for one o
103 were also more likely to achieve high-grade stereopsis than were children whose mothers did not eat
104 turational factors indicated by the onset of stereopsis (the ability to detect depth in an image on t
106 are thought to form the neural substrate for stereopsis, the mere existence of disparity-selective ne
110 Here we provide evidence for the recovery of stereopsis through perceptual learning, the repetitive p
113 udies evaluating the correlation of BiS with stereopsis, visual field expansion, and quality of life
114 The median visual acuity for patients with stereopsis was better than for those without stereopsis
118 The median age at surgery for patients with stereopsis was younger than for those without stereopsis
119 To identify neuronal mechanisms underlying stereopsis, we characterized interactions between inputs
120 ate the functional consequences of this poor stereopsis, we investigated whether stereopsis in these
122 ience during early infancy severely disrupts stereopsis, yet the critical period for susceptibility o
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