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   5 nd sacral levels of the skeleton, and showed sternal and pelvic malformations not previously observed
  
     7 rax (n = 2), pericarditis (n = 2), dislodged sternal bar (n = 3), and mildly hypertrophic scar (n = 1
  
     9 ound infection (involving the sternal wires, sternal bone, and/or mediastinum), and (3) score for add
  
    11  B (mouth-to-mouth ventilation), and step C (sternal (cardiac) compressions) into basic life support.
  
    13  of a cyanogen bromide (CNBr) (CB) digest of sternal cartilage revealed an alpha1(II)CB11 peptide dou
    14 polymerase chain reaction in embryonic skin, sternal cartilage, and tendon, but is barely detectable 
  
  
    17 r cartilage and perichondrium; articular and sternal chondrocytes expressed p38 isoforms alpha, beta,
    18 that Ctgf mRNA was down-regulated in primary sternal chondrocytes from Sox9(flox/flox) mice infected 
    19 rom engineered cartilage, generated by chick sternal chondrocytes grown in a hollow fiber bioreactor,
    20 pase 3/7 activities were examined in primary sternal chondrocytes isolated from 3-day-old neonatal Co
  
  
    23 1 transcription following treatment of chick sternal chondrocytes with growth factors was accompanied
  
    25  the Qp/Qs (1/36 v/s 8/20; P=0.001), delayed sternal closure (6/36 v/s 7/20; P=0.001), and extracorpo
    26 ediate increase in ventilation after delayed sternal closure (n = 3) or removal of pericardial blood 
    27 sociated with increased incidence of delayed sternal closure (p = 0.003) and longer duration of mecha
    28 ry function may be compromised after delayed sternal closure and that ventilatory support should be i
  
  
    31 study was to establish the impact of delayed sternal closure on expired tidal volume, respiratory sys
  
  
    34 lied in monitoring blood gases after delayed sternal closure to assess clinical responses to sternal 
  
  
    37 ac surgery, chromosomal anomaly, and delayed sternal closure were independently associated with incre
    38 amics and respiratory variables occur during sternal closure, often requiring adjustment of inotropic
    39 rculatory arrest; and postoperative--delayed sternal closure, sepsis, renal failure, pulmonary hypert
  
    41 e leak increased by > or = 10% after delayed sternal closure, thereby invalidating recorded changes i
  
    43    Of the 128 patients, 14 (11%) died before sternal closure; delayed sternal closure was performed i
  
  
    46 ors began treating these wounds with radical sternal debridement followed by closure using muscle or 
    47 er of an omphalocele, ectopia cordis, distal sternal defect, pericardial defect, anterior diaphragmat
    48 netrance and the expressivity of the rib and sternal defects are increased, suggesting synergistic in
  
    50    While a majority of these mice had severe sternal defects that compromised their ability to breath
  
  
  
    54 ting room for bleeding, and mediastinitis or sternal dehiscence requiring surgery were also less in t
    55 ing injury to or embolism from prior grafts, sternal dehiscence, phrenic nerve injury, excessive hemo
  
  
    58 e of death is likely due to abnormal rib and sternal development, leading to an inability to breathe.
    59     Causes included puncture or erosion by a sternal edge in three and tearing at the myocardial-ster
  
  
    62 ubcutaneous tissue but not extending down to sternal fixation wires), (2) deep wound infection (invol
  
    64 2 or more rib fractures, ruptured diaphragm, sternal fracture, and pulmonary contusion or laceration 
    65 nal synostosis, ocular proptosis, precocious sternal fusion, and abnormalities in secondary branching
  
  
    68 ges (total gentamicin of 260 mg) between the sternal halves at surgical closure (n = 753) vs no inter
  
    70 ifferences among the groups in occurrence of sternal infection (1.3%, 2.6%, and 1.4%, respectively; P
  
    72 d ventilation, prolonged postoperative stay, sternal infection, and bleeding) after adjustment for co
    73 y and morbidities (stroke, reoperation, deep sternal infection, ventilation >48 hours, postoperative 
  
  
    76 ocardial infarction, neurologic events, deep sternal infections, and atrial fibrillation), the level 
    77 he primary outcome was a composite of death, sternal infections, prolonged ventilation, cardiac arrhy
  
  
  
    81 or maintenance of intervertebral, carpal and sternal joints, and the joint fusion process commences a
  
  
    84 er extremity (n = 4), intrathoracic (n = 3), sternal (n = 34), breast (n = 3), chest wall (n = 18), a
  
  
    87 e of asynchronous ossification of one of the sternal ossification centers 1-4 (P >.003) and of occurr
  
  
  
  
    92  bypass surgery that had been complicated by sternal osteomyelitis caused by the Staphylococcus aureu
  
    94 n either side; however, unlike the ribs, the sternal precursors do not originate from the somites.   
  
  
  
    98 ingle-graft group (P=0.005), and the rate of sternal reconstruction was 1.9% versus 0.6% (P=0.002).  
    99 heral vascular access may be compromised and sternal reentry is more likely to result in cardiac inju
  
  
  
  
  
   105 ossification, as compared with the remaining sternal segments, was demonstrated or if ossification wa
   106 6.9% vs. leg sutured = 32.6%; p = 0.001, and sternal stapled = 14.9% vs. sternal sutured = 3.7%; p = 
  
   108 = 9.3% vs. leg stapled = 8.9%; p = 0.99, and sternal sutured = 0.4% vs. sternal stapled = 2.5%; p = 0
  
   110 cting injury, (4) chest wall tenderness, (5) sternal tenderness, (6) thoracic spine tenderness, and (
   111 202 consecutive patients who underwent trans-sternal thymectomy for symptomatic myasthenia gravis fro
   112  improvement, compared to conventional trans-sternal thymectomy, neither the pathologic diagnosis (pr
   113 es), (2) deep wound infection (involving the sternal wires, sternal bone, and/or mediastinum), and (3
   114 tricular tachycardia (20%); leg wound (15%); sternal wound (5%); pneumonia (5%); gastrointestinal com
  
   116 nd have reduced the mean hospital stay after sternal wound closure of these critically ill patients. 
   117 elation to flap choices, hospital days after sternal wound closure, and incidence rates of morbidity 
  
  
  
  
  
   123 /753 [6.5%] vs 46/749 [6.1%]; P = .77), deep sternal wound infection (14/753 [1.9%] vs 19/749 [2.5%];
   124 .0 [7.2]; P = .67), or rehospitalization for sternal wound infection (23/753 [3.1%] vs 24/749 [3.2%];
   125  control group, respectively, in superficial sternal wound infection (49/753 [6.5%] vs 46/749 [6.1%];
   126 2 cardiac surgical patients at high risk for sternal wound infection (diabetes, body mass index >30, 
   127 evention of S. aureus bacteremia and/or deep sternal wound infection (including mediastinitis) throug
   128 esity was a risk factor only for superficial sternal wound infection (P < .001; odds ratio, 2.3), leg
  
  
   131 d a significant reduction in mortality after sternal wound infection and have reduced the mean hospit
   132 e the first reported case of a postoperative sternal wound infection and pneumonia caused by in a hea
  
   134  in-hospital stays as well as the absence of sternal wound infection are the main advantages of this 
   135 al infarction, repeat revascularization, and sternal wound infection between propensity score-matched
  
  
  
   139 rnotomy (p = 0.008) and patients treated for sternal wound infection from 1988 to 1992 (p = 0.024).  
  
  
  
   143 ysis, there was no significant difference in sternal wound infection in 63 of 753 patients randomized
  
  
  
  
  
  
   150 ne use of prophylactic systemic antibiotics, sternal wound infection still occurs in 5% or more of ca
   151 ks of cardiovascular events, but the risk of sternal wound infection was increased (risk difference, 
  
  
   154 ve CVA (adjusted OR, 1.06; P=.765), risks of sternal wound infection were substantially increased in 
   155 epsis/septicemia, 0.5% [n=99]; isolated deep sternal wound infection, 0.5% [n=96]; isolated harvest/c
   156 414 [2.4%] versus 13 of 414 [3.1%]; P=0.279; sternal wound infection, 7 of 414 [1.7%] versus 13 of 41
   157 y, deep sternal wound infection, superficial sternal wound infection, infection at the saphenous vein
  
   159 re was no difference in operative mortality, sternal wound infection, or total complications between 
   160  analyzed included operative mortality, deep sternal wound infection, superficial sternal wound infec
  
   162  accident (CVA), postoperative bleeding, and sternal wound infection, were defined prospectively.    
  
  
  
   166 ications with the possible exception of deep sternal wound infections (11 [1.3%] vs. 3 [0.4%], p = 0.
  
   168 ate modeling BITA increased the risk of deep sternal wound infections only in emergent cases and in o
  
  
  
  
   173 and surgical site infections, including deep sternal wound, thoracotomy, and harvest/cannulation site
  
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