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1 ked by TA through the mifepristone-sensitive steroid receptor.
2 ansactivation to a greater extent than other steroid receptors.
3 res of this binding mode are more similar to steroid receptors.
4 spective crystal structures of the ancestral steroid receptors.
5 d -21 proteins as conserved PATs for the sex steroid receptors.
6 ting where conserved aspects exist for other steroid receptors.
7 erones that participate in the activation of steroid receptors.
8 ns that do not require activation of cognate steroid receptors.
9 ermine whether midbrain CART neurons contain steroid receptors.
10 nds and redefine the quaternary structure of steroid receptors.
11  altering the molecular pathways targeted by steroid receptors.
12  translocation and function of classical sex steroid receptors.
13 hway leading to the production of functional steroid receptors.
14 able chromatin residence times for FoxA1 and steroid receptors.
15 of transcription by ligand-activated nuclear steroid receptors.
16 ontal cortex, are densely populated with sex steroid receptors.
17 ct as agonist and/or antagonist of different steroids receptors.
18  discrete shift in ligand specificity in the steroid receptors, a family of biologically essential ho
19           Comparable regional differences in steroid receptor abundances likely regulate morph-specif
20 Yet, the distinct roles of these proteins in steroid receptor action are poorly understood.
21 rs that target other sites in the pathway of steroid receptor action.
22 trol nucleus HVC (used as a proper name) and steroid receptor activation within HVC; local coactivati
23 the mouth of the pocket play a major role in steroid receptor activation.
24 eviously unidentified level of regulation in steroid receptor activation.
25 uct of the bi-functional long non-coding RNA steroid receptor activator RNA 1 (SRA1) that is part of
26                                              Steroid receptor activator RNA protein (SRA1p) is the tr
27 -coding RNAs: tRNA, U2 spliceosomal RNA, and steroid receptor activator RNA.
28 s, like the group II intron, rRNA, or lncRNA steroid receptor activator.
29 a major role for FOXA1 in modulating nuclear steroid receptor activity in breast and prostate cancer,
30 y metabolize precursor compounds into potent steroid receptor agonists locally within bone.
31           Post-translational modification of steroid receptors allows fine-tuning different propertie
32                                              Steroid receptors also exist in discrete cytoplasmic org
33                            The Nur77 nuclear steroid receptor and Bim, a proapoptotic BH3-only member
34 ent with selectivity for the GR versus other steroid receptors and a differentiated gene expression p
35 ysis on the intranuclear dwell times of four steroid receptors and a number of known cofactors.
36 a now demonstrate the oncogenic potential of steroid receptors and implicate altered AR function and
37 nding domain of Ydj1 directly interacts with steroid receptors and is required for the activity of di
38 e folding of Hsp90-dependent clients such as steroid receptors and many kinases involved in cellular
39 e a mechanism through which ID domain of the steroid receptors and other similar transcription factor
40 sm through which ID activation domain of the steroid receptors and other similar transcription factor
41 eus (PMV) expresses dense collections of sex steroid receptors and receptors for metabolic cues, incl
42 uminal compartments complete with functional steroid receptors and stem/progenitor cells able to reco
43 cted on nNOS and Q78 ataxin-3 but not on the steroid receptors, and Mdm2 did not affect any of the co
44 as a chaperone for receptor protein kinases, steroid receptors, and other intracellular signaling mol
45 ion, we hypothesized that nNOS cells contain steroid receptors, and the testosterone-induced restorat
46 L2 is required for normal gene regulation by steroid receptors, and we show that estrogen receptor be
47 lthough evidence suggests that the classical steroid receptors are capable of mediating many of these
48 cribed, but the mechanisms relevant to other steroid receptors are entirely unknown.
49                                              Steroid receptors are found in discrete cellular locatio
50                                              Steroid receptors are members of a nuclear receptor tran
51                                              Steroid receptors are pleiotropic transcription factors
52 AR FXXLF motif region and indirectly through steroid receptor-associated p300 and p160 coactivators.
53 and AR signaling by disrupting the Pus1p-SRA-steroid receptor axis.
54                                 We then used steroid receptor-based inducible activation system to co
55                                      Using a steroid receptor-based inducible activation system, we s
56  of the H295R steroidogenesis assay, and sex steroid receptor binding activity using the yeast estrog
57 cently, a novel effect of phosphorylation on steroid receptor biology was described.
58                             We show that the steroid receptor BRI1 localizes to both plasma membrane
59    Basal-like "triple negative" cancers lack steroid receptors but are cytokeratin (CK) 5-positive an
60 ase the DNA-binding affinity of PR and other steroid receptors by mechanisms that are not well define
61 -containing type III J protein implicated in steroid receptor chaperoning.
62  besides Met, another member of this family, steroid receptor co-activator (SRC) is required for the
63                           TBP also increased steroid receptor co-activator 1 (SRC-1) interaction with
64 uch as p300/CBP-associated factor (PCAF) and steroid receptor co-activator 1 (SRC-1), the full length
65 calreticulin, and caspase 3 and decreases in steroid receptor co-activator 1, Grp78/BiP, and annexin
66                       These data suggest the steroid receptor co-activator plays key roles in both JH
67 d binding domain (LBD) interactions with the steroid receptor co-activator-1 (SRC-1) peptide, displac
68 n 2 (AF2) surfaces of both receptors driving steroid receptor co-activator-1 (SRC1) interaction.
69 enobiotic receptor PXR with the co-activator steroid receptor co-activator-1.
70                                              Steroid receptor co-activator-3 (SRC-3/AIB1) is an oncog
71 ne (JH) receptor, Methoprene-tolerant (Met), steroid receptor coactivator (SRC) and GATAa but not ecd
72 tivators, VDR-interacting protein (DRIP) and steroid receptor coactivator (SRC) at different stages o
73 invasion, and metastasis, including the p160 steroid receptor coactivator (SRC) family composed of SR
74  the bHLH transcription factors studied, the steroid receptor coactivator (SRC) showed the most sever
75 rene-tolerant transcription factor (Met) and steroid receptor coactivator (SRC), would be expressed c
76                                          The steroid receptor coactivator (SRC)-1 enhances the activi
77 lencing mediator for retinoid and thyroid or steroid receptor coactivator (SRC)-1 with RARalpha versu
78 OOH-terminal interaction and are enhanced by steroid receptor coactivator (SRC)-1, whereas the bicalu
79 gamma) regulate bone metabolism, and because steroid receptor coactivator (SRC)-2 (TIF-2) enhances ER
80                                              Steroid receptor coactivator (SRC)-3, also called amplif
81 ne receptors involves the recruitment of the steroid receptor coactivator (SRC)/p160 coactivator LXXL
82  further dissect the roles of members of the steroid receptor coactivator (SRC)/p160 family in PR-med
83 eam target of progesterone receptor (PR) and steroid receptor coactivator (SRC-1) action in the uteru
84                             Both PXR and the steroid receptor coactivator (SRC-1) were found to bind
85 receptor beta (TRbeta) gene that cannot bind steroid receptor coactivator 1 (SRC-1) and Src-1(-/-) mi
86 y less ability than dexamethasone to trigger steroid receptor coactivator 1 (SRC-1) recruitment and h
87 risk HPV type 16 (HPV16) E7 oncoprotein with steroid receptor coactivator 1 (SRC-1), an essential com
88 eciphered a previously unappreciated role of Steroid receptor coactivator 1 (SRC1) in defining the li
89 iquitination greatly enhanced recruitment of steroid receptor coactivator 1 (SRC1), a coactivator cri
90 ement binding protein binding protein (CBP), steroid receptor coactivator 1 (SRC1), and protein argin
91 TCO-mediated interaction between CAR and the steroid receptor coactivator 1 or the glucocorticoid rec
92 D bound to DAC and the fourth LXXLL motif of steroid receptor coactivator 1 reveals that the GR ligan
93 ated the recruitment of TTF-1, p65, CBP, and steroid receptor coactivator 1 to the TBE region of the
94 ta in vitro and supported the recruitment of steroid receptor coactivator 1.
95                  Herein, we demonstrate that Steroid Receptor Coactivator 2 (SRC-2) orchestrates a hi
96 of the oncogenic transcriptional coregulator steroid receptor coactivator 2 (SRC-2), also known as NC
97 rs that block the association of TRbeta with steroid receptor coactivator 2 (SRC2), we identified a n
98 ding between VDR and a fluorescently labeled steroid receptor coactivator 2 peptide was applied to di
99 corporated them into a sequence derived from steroid receptor coactivator 2, which interacts with est
100 ion between the vitamin D receptor (VDR) and steroid receptor coactivator 2.
101                                              Steroid receptor coactivator 3 (SRC-3) coactivator phosp
102                                              Steroid receptor coactivator 3 (SRC-3) is an oncogenic n
103                                          The steroid receptor coactivator 3 (SRC-3) is overexpressed
104 that ERK3 interacted with and phosphorylated steroid receptor coactivator 3 (SRC-3), an oncogenic pro
105 rect interaction of the liganded TRbeta with steroid receptor coactivator 3 (SRC-3), which recruits p
106                                              Steroid receptor coactivator 3 (SRC-3/AIB1) interacts wi
107                                              Steroid receptor coactivator 3 (SRC-3/AIB1/ACTR/NCoA-3)
108 e of an active complex of DNA-bound ERalpha, steroid receptor coactivator 3 (SRC-3/NCOA3), and a seco
109 as a model, we have investigated the role of steroid receptor coactivator 3 (SRC3) in gene activation
110      Here, we report that the ER coactivator steroid receptor coactivator 3 (SRC3) is also a coactiva
111                We have previously shown that steroid receptor coactivator 3 (SRC3) is expressed and u
112 enced by histone acetylation and require the steroid receptor coactivator 3 (SRC3), which mediates in
113 ctivation of gene expression was enhanced by steroid receptor coactivator 3 coactivator, and required
114                                          The steroid receptor coactivator 3 gene (SRC-3) (AIB1/ACTR/p
115 ied in breast cancer 1 (AIB1), also known as steroid receptor coactivator 3 or NCOA3, is a transcript
116 parate assays, both the recruitment of SRC3 (steroid receptor coactivator 3, a transcriptional coacti
117                   Overexpression of the p160 steroid receptor coactivator ACTR is associated with bre
118 or CBP and the activation domain of the p160 steroid receptor coactivator ACTR.
119 R function by selectively competing with the steroid receptor coactivator AIB1 but not GRIP1 or SRC1
120         Overexpression and activation of the steroid receptor coactivator amplified in breast cancer
121                                          The steroid receptor coactivator amplified in breast cancer
122 or complex disassembly by methylation of the steroid receptor coactivator family coactivators and p30
123  the most active members inhibit the ERalpha/steroid receptor coactivator interaction with K i's in t
124 s induced by GR signaling, and the important steroid receptor coactivator PELP1 was also found to be
125 (Pol II), estrogen receptor alpha (ERalpha), steroid receptor coactivator proteins (SRC), and acetyla
126 stant patients showed high expression of the steroid receptor coactivator SRC-1.
127 ctivator REGgamma directs degradation of the steroid receptor coactivator SRC-3 by the 20S proteasome
128 ctivator REGgamma directs degradation of the steroid receptor coactivator SRC-3 by the 20S proteasome
129 rence (RNAi) depletions of CYC, MET, and the steroid receptor coactivator SRC/FISC.
130                              SRC-3/AIB1 is a steroid receptor coactivator with potent growth-promotin
131 xes, DRIP (VDR-interacting protein) and SRC (steroid receptor coactivator), during keratinocyte diffe
132     Also, coactivator binding studies with a steroid receptor coactivator-1 (receptor interaction dom
133                                              Steroid receptor coactivator-1 (SRC-1 or NCOA1) is overe
134                            In breast cancer, steroid receptor coactivator-1 (SRC-1) expression positi
135                                              Steroid receptor coactivator-1 (SRC-1) is a coactivator
136 o part of the active receptor complex is the steroid receptor coactivator-1 (SRC-1) which interacts w
137                                              Steroid receptor coactivator-1 (SRC-1), a coregulatory p
138     During the decade since the discovery of steroid receptor coactivator-1 (SRC-1), the first authen
139 lpha), which requires co-activators, such as steroid receptor coactivator-1 (SRC-1), to facilitate th
140 rs recruit coactivator proteins, such as the steroid receptor coactivator-1 (SRC1).
141 corticosterone and the fourth LXXLL motif of steroid receptor coactivator-1 (SRC1-4).
142 r activator RNA 1 (SRA1) that is part of the steroid receptor coactivator-1 acetyltransferase complex
143 e interactions of hCAR with the coactivators steroid receptor coactivator-1 and glucocorticoid recept
144 rinsic activity of p160 coactivators such as steroid receptor coactivator-1 and promoted the interact
145 e nuclear receptor interacting domain of the steroid receptor coactivator-1 as a model for exploring
146  and promoted the interaction between PR and steroid receptor coactivator-1 in a mammalian two-hybrid
147 ene and also remarkably reduces basal MR and steroid receptor coactivator-1 recruitment, unraveling a
148 t insulin facilitated the recruitment of the steroid receptor coactivator-1 to the SREBP-1c promoter.
149  interaction with fatty acid metabolites and steroid receptor coactivator-1 while increasing PPARalph
150 d to spironolactone, finerenone inhibits MR, steroid receptor coactivator-1, and RNA polymerase II bi
151 glucose increased PPARalpha interaction with steroid receptor coactivator-1, DNA binding, and transac
152 to bind the IL-4 promoter in the presence of steroid receptor coactivator-1, indicating that PPARalph
153 gate binding and dissociation of full-length steroid receptor coactivator-1a (SRC1a) from full-length
154 reviously demonstrated the potential role of steroid receptor coactivator-2 (SRC-2) as a co-regulator
155 sly shown that the transcriptional regulator steroid receptor coactivator-2 (SRC-2) controls activati
156 es and in HepG2 cells reduced recruitment of steroid receptor coactivator-2 by RORalpha at an endogen
157 gh-throughput screening to identify SMIs for steroid receptor coactivator-3 (SRC-3 or AIB1), a large
158       Here we demonstrate that reprogramming steroid receptor coactivator-3 (SRC-3) function by chang
159  Molecular Cell, Yu et al. reported that the steroid receptor coactivator-3 (SRC-3) has a novel cytop
160 ctivator amplified in breast cancer 1 (AIB1)/steroid receptor coactivator-3 (SRC-3) have been shown t
161 ent study aimed to elucidate the role of the steroid receptor coactivator-3 (SRC-3) in thyroid carcin
162 oncogene amplified in breast cancer 1 (AIB1)/steroid receptor coactivator-3 (SRC-3) induces mammary t
163             Here, we show that the oncogenic steroid receptor coactivator-3 (SRC-3) is a critical reg
164                                              Steroid receptor coactivator-3 (SRC-3) is a histone acet
165                                              Steroid receptor coactivator-3 (SRC-3) is a transcriptio
166          Phosphorylation and activity of the Steroid Receptor Coactivator-3 (SRC-3) is reduced upon H
167              Estrogen receptor (ER) recruits steroid receptor coactivator-3 (SRC-3) primary coactivat
168 y also caused a failure in downregulation of steroid receptor coactivator-3 (SRC-3), a potent ERalpha
169                                              Steroid receptor coactivator-3 (SRC-3)/AIB1 is a member
170                                              Steroid receptor coactivator-3 (SRC-3/AIB1) is an oncoge
171 sphorylated alternate-spliced isoform of the steroid receptor coactivator-3 (SRC-3Delta4) bridges EGF
172 d crystallographic studies, we identify that steroid receptor coactivator-3 (SRC3) (also named as amp
173 exes did not readily recruit the coactivator steroid receptor coactivator-3 (SRC3) or ER to the PS2 p
174 ociated with the displacement of ERalpha and steroid receptor coactivator-3 from the target EBRs lead
175 shown that TR recruits the coactivator SRC3 (steroid receptor coactivator-3) and that coactivator rec
176 ceptor-associated coactivator 3 (RAC3)/AIB-1/steroid receptor coactivator-3, a nuclear coregulator an
177      The three members of the p160 family of steroid receptor coactivators (SRC-1, SRC-2, and SRC-3)
178  growth pathways on which cancer cells rely, steroid receptor coactivators (SRC-1, SRC-2, and SRC-3)
179  interplay and demonstrated that it requires steroid receptor coactivators (SRC-2, SRC-3) and the med
180             Thus far, a mechanistic role for steroid receptor coactivators (SRCs) in the progression
181                                              Steroid receptor coactivators (SRCs) regulate nuclear re
182                                     The p160 steroid receptor coactivators (SRCs) SRC-1, SRC-2 [nucle
183                                   AR and its steroid receptor coactivators (SRCs; SRC-1, -2 and -3) w
184 CI, Gao and colleagues present evidence that steroid receptor coactivators 1 and 2 (SRC-1 and SRC-2)
185       Here, we evaluated the contribution of steroid receptor coactivators 1 and 2 (SRC-1 and SRC-2),
186                                   Currently, steroid receptor coactivators have been proposed to medi
187  in the recruitment of RNA polymerase II and steroid receptor coactivators SRC-2 and SRC-3, and chang
188 plicing in a manner differing from the other steroid receptor coactivators.
189 lting in a more selective destabilization of steroid receptors compared with kinase clients.
190 160 coactivators to hormone response element-steroid receptor complexes has been difficult to investi
191 e tetratricopeptide repeat proteins found in steroid receptor complexes, and Fkbp51 is an androgen re
192      Originally discovered as a component of steroid receptor complexes, it is now known to regulate
193 also known as immunophilins) in hormone-free steroid receptor complexes.
194 70 and Hsp90 interactions during assembly of steroid receptor complexes.
195                                              Steroid receptors comprise a homologous family of ligand
196                                              Steroid receptors comprise an evolutionarily conserved f
197 plasm and nucleus that stabilizes unliganded steroid receptors, controls the catalytic activity of ce
198 GE-A11) is a low-abundance, primate-specific steroid receptor coregulator in normal tissues of the hu
199 oteins, one of which is the primate-specific steroid receptor coregulator melanoma antigen-A11 (MAGE-
200 (20-45 min), p300 is recruited to ERalpha by steroid receptor coregulators (SRCs) for enhancer matura
201 to injury and in methyl-binding proteins and steroid receptor coregulatory proteins are also reported
202  processes as well as in the therapeutics of steroid receptor-dependent diseases.
203                           We will review how steroid receptor-dependent genomic signaling is affected
204 ntradicts the traditional understanding that steroid receptors dimerize in the absence of DNA, it is
205 SL was correlated with the LXR target genes, steroid receptor element-binding protein 1c and ATP bind
206                                 Despite that steroid receptors engage nucleosome-remodeling complexes
207 s simplex viral vector to express a chimeric steroid receptor ("ERGR") which binds glucocorticoids ye
208                  It has also been shown that steroid receptors exist outside the nucleus in many orga
209           However, the organization, gonadal steroid receptor expression, and activity of nPGi affere
210  these effects are initiated through altered steroid receptor expression; however, the immediate down
211 y structure analysis of other members of the steroid receptor family (estrogen, androgen, and progest
212                ERK5 induces the Nur77 orphan steroid receptor family members.
213 cocorticoid receptor (GR) is a member of the steroid receptor family of ligand-activated transcriptio
214 ctionally known phosphorylation sites in the steroid receptor family of transcription factors are loc
215 iously found that the ancestor of the entire steroid receptor family was highly specific for estrogen
216      Androgen receptor (AR), a member of the steroid receptor family, is a transcription factor that
217 ain (DBD) not shared by other members of the steroid receptor family.
218 r, there is compelling evidence for membrane steroid receptors for estrogen in hypothalamic and other
219 itin ligase function that targets, e.g., the steroid receptors for proteasomal degradation.
220               The N-terminal domain (NTD) of steroid receptors harbors a transcriptional activation f
221                                              Steroid receptors have classically been described as lig
222 lmitoylation and membrane trafficking of the steroid receptor in all organs.
223 ene to study the biological function of this steroid receptor in the epithelial compartment at define
224 s masked the biological significance of this steroid receptor in the mammary epithelium.
225  set of 62 known NR coregulators and the six steroid receptors in 12 nonoverlapping mouse brain regio
226 or coactivator 3 (SRC-3/AIB1) interacts with steroid receptors in a ligand-dependent manner to activa
227 on to the well-characterized role of the sex steroid receptors in fertility and reproduction, organs
228 (A(max)) in gene induction and repression by steroid receptors in general and glucocorticoid receptor
229 and neuroendocrine secretions, expression of steroid receptors in GnIH-ir nuclei, and GnIH inhibition
230               TRPM3 channels form ionotropic steroid receptors in the plasma membrane of pancreatic b
231              The transcriptional activity of steroid receptors, including AR, is dependent on interac
232 s PIP(2) and that aldosterone stimulation of steroid receptors induces PIP(3) formation.
233 d PRMT6 interaction occurs through the PRMT6 steroid receptor interaction motif, LXXLL, and the AR ac
234 nsgenic mouse model in which growth factor - steroid receptor interactions were explored.
235 rring more efficient functioning of this sex steroid receptor is associated with "masculinization" of
236            Ligand-mediated gene induction by steroid receptors is a multistep process characterized b
237 to have actions via binding to their cognate steroid receptors, it is becoming clearer that steroids
238               Structure activity studies and steroid receptor knockdown suggest that flurandrenolide
239                                     However, steroid receptor ligand-binding domains (LBDs) are inher
240  targets to selectively inhibit membrane sex steroid receptor localization and function.
241                        In the past 40 years, steroid receptors localized to the nucleus have been rec
242 ving cells to develop a single-cell model of steroid-receptor mediated gene activation.
243           However, little is known about the steroid receptor-mediated molecular mechanisms of action
244 ctively, these data provide insight into sex steroid receptor-mediated regulation of androgen-inactiv
245  reported to be a coactivator that regulates steroid receptor-mediated transcription and alternative
246 a dual-functional coregulator that regulates steroid receptor-mediated transcription and alternative
247                 Recent studies indicate that steroid receptor-mediated transcriptional initiation is
248 ta suggest that CpdA is a unique dual-target steroid receptor modulator that has a high potential for
249 c factors that contribute to the function of steroid receptor modulators, providing valuable insight
250 r research on pregnancy hormones and risk of steroid receptor-negative cancers is needed to further c
251 that tumor growth in nude rats bearing human steroid receptor-negative MCF-7 breast tumors can be sig
252 viduals with a common genetic variant in the steroid receptor (NR3C1) gene.
253                                   The orphan steroid receptor, Nur77, is thought to be a central part
254                 However, the identity of the steroid receptor PAT(s) is unknown.
255 e normal mammary gland does not occur in the steroid-receptor positive cells but rather in adjacent c
256 e of three evolutionarily diverged ancestral steroid receptor proteins using the Zipping and Assembly
257 e divergence in the evolutionary path of the steroid receptor proteins.
258 c knockout of a long non-coding RNA (lncRNA) steroid receptor RNA activator (SRA) (SRAKO) are resista
259 smic RISC proteins PACT, TRBP, and Dicer are steroid receptor RNA activator (SRA) binding NR coregula
260                                              Steroid receptor RNA Activator (SRA) is a long, noncodin
261 d secondary structure of a human lncRNA, the steroid receptor RNA activator (SRA), 0.87 kB in size.
262                  Both SF-1 and Dax-1 bind to steroid receptor RNA activator (SRA), a coactivator that
263 p68 (DDX5) and its associated noncoding RNA, steroid receptor RNA activator (SRA), form a complex wit
264 have recently shown that the non-coding RNA, steroid receptor RNA activator (SRA), functions as a tra
265              In a widely accepted model, the steroid receptor RNA activator protein (SRA protein; SRA
266 acterized by exquisite potency and favorable steroid receptor selectivity and pharmacokinetic profile
267 he role of pioneer can be reversed, with the steroid receptors serving to enhance binding of FoxA1.
268 chaperone activity of SNCG on stimulation of steroid receptor signaling in mammary gland and, thus in
269 ires pseudouridylation by Pus1p to stimulate steroid receptor signaling.
270 ein-based multiprotein chaperone complex for steroid receptor signaling.
271 C-3 transcriptional coregulatory activity in steroid receptor signalings.
272 as emerged as a critical mediator of nuclear steroid receptor signalling, manifest at least in part t
273 ide receptor imaging for 3 receptor systems--steroid receptors, somatostatin receptors, and growth fa
274 In the future, we hope to fully characterize steroid receptor-specific functions in the brain.
275 ription factor FoxA1 is a global mediator of steroid receptor (SR) action in hormone-dependent cancer
276  FKBP52, FKBP51, Cyp40, and PP5 are found in steroid receptor (SR) complexes, but their receptor-spec
277                                     Multiple steroid receptors (SR) have been proposed to localize to
278                                              Steroid receptors (SRs) are the largest family of metazo
279 olecular requirements for the recognition of steroid receptors (SRs) by the lincRNA growth arrest-spe
280                                Classical sex steroid receptors (SRs) localize at the plasma membranes
281     Because of their interactions with other steroid receptors, steroid-metabolizing enzymes, or othe
282 ydrolysis and the activation of a kinase and steroid receptor substrate in yeast cells.
283 g extracellular signal-regulated kinases and steroid receptors, suggesting that MVP is likely to be i
284                  VitD interaction with other steroid receptor superfamily receptors in peripheral blo
285 thyroid hormone receptors are members of the steroid receptor superfamily that interact with their DN
286 well studied in specific regions, brain-wide steroid receptor targets and mediators remain largely un
287 orrelations of mRNA expression levels of six steroid receptors that we provide constitute a rich reso
288 al L/HX7LL motif, selectively present in sex steroid receptors, that causes recruitment of TAB2 as a
289 both the apo- and hormone-bound forms of the steroid receptor to modulate its affinity for ligand, wh
290 oadened the search for and identification of steroid receptors to include nonnuclear sites in synapse
291 ing immunophilins FKBP51 and FKBP52 modulate steroid receptor trafficking and hormone-dependent biolo
292 ts activity of the androgen receptor (AR), a steroid receptor transcription factor required for PCa g
293  methylation of histones and coactivation of steroid receptor transcription.
294 fic expression of MAGE-11 results in greater steroid receptor transcriptional activity through direct
295 ecedented Fkbp52 function, direct control of steroid receptor transcriptional activity.
296      Lysine acetyltransferases interact with steroid receptors upon binding of an agonist and are rec
297       KDACs have been shown to interact with steroid receptors upon binding to an antagonist.
298 on by androgens makes it distinct from other steroid receptors, which are typically ubiquitinated and
299 (H100)Trp, converts 1E9 into a high-affinity steroid receptor with a ligand recognition profile simil
300 be identified through spatial correlation of steroid receptors with genome-wide mRNA expression acros

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