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1 delivering sterols to the mitochondrion for steroid synthesis.
2 and SF-1-dependent Cyp11a1 transcription for steroid synthesis.
3 at can further suppress de novo intratumoral steroid synthesis.
4 support region-specific regulation of brain steroid synthesis.
5 nd the 17,20 lyase activity required for sex steroid synthesis.
6 adrenocortical disease (PPNAD) and increased steroid synthesis.
7 ds 4-6, respectively, were prepared by total steroid synthesis.
8 stablished, other than as precursors for sex steroid synthesis.
9 ch drives P450scc expression and neuroactive steroid synthesis.
10 lowed by 17,20 lyase activity needed for sex steroid synthesis.
11 ct on adrenocorticotropic hormone-stimulated steroid synthesis.
13 of four sex steroids, and inhibitors of sex steroid synthesis (aminoglutethimide, ketoconazole, and
14 0c17-knockout mice would have disordered sex steroid synthesis and disordered brain DHEA production a
16 terol metabolism and bile-acid biosynthesis; steroid synthesis and metabolism; vitamin D(3) synthesis
17 F-1-induced leptin expression, modulates sex steroid synthesis by acting directly on steroidogenic ge
18 d in adrenal and gonadal tissues to initiate steroid synthesis by catalyzing the conversion of pregne
19 the availability of adrenal cholesterol for steroid synthesis by decreasing the expression of choles
21 nges in the expression of hormone receptors, steroid synthesis enzymes, and BMPs and their receptors.
23 eroids in the impeded pathways and excessive steroid synthesis in other adrenal biosynthetic pathways
26 he role of CYP17, a key enzyme mediating sex steroid synthesis, in Xenopus ovarian androgen productio
27 -2, animals received no further treatment; a steroid synthesis inhibitor, trilostane (TRL); TRL + R50
28 ats, the stress effect is blocked by adrenal steroid synthesis inhibitors, and mimicked by daily inje
29 genes associated with cancer, inflammation, steroid-synthesis, insulin-synthesis, neurotransmitter p
32 se endocrine disruption via perturbations in steroid synthesis (steroidogenesis) has become increasin
33 eceptor (LXR) ligands on StAR expression and steroid synthesis, suggesting HSL-mediated steroidogenes
35 t, all of the genes required for de novo sex steroid synthesis would be expressed in regions that wou
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