ライフサイエンスコーパス: steroid treatment

1 as used when patients did not respond to the steroid treatment.

2 glucocorticoid-sensitive 6TG1.1 cells before steroid treatment.

3 ses in group B; none were reversible despite steroid treatment.

4 eficient states such as diabetes, and during steroid treatment.

5 chronic rejection and was down-regulated by steroid treatment.

6 n challenge and decrease in asthmatics after steroid treatment.

7 midine incorporation was observed 24 h after steroid treatment.

8 al iNOS levels may reflect responsiveness to steroid treatment.

9 asis of the need or not for systemic cortico-steroid treatment.

10 rtality among patients who do not respond to steroid treatment.

11 mber amplifications, AR splice-variants, and steroid treatment.

12 and AHR that were partially reversible with steroid treatment.

13 utochthonous hepatitis E that resolved under steroid treatment.

14 venting AECOPD requiring both antibiotic and steroid treatment.

15 tion language, or type, dose, or duration of steroid treatment.

16 30 rejection episodes (73.3%) resolved with steroid treatment.

17 cial effect is synergistically enhanced with steroid treatment.

18 d retinal ganglion cells during intravitreal steroid treatment.

19 completely unresponsive to IVIG or high-dose steroid treatment.

20 nt) but progressed in three patients without steroid treatment.

21 r steroid treatment but can progress without steroid treatment.

22 t, ERbeta mRNA levels were unaffected by any steroid treatment.

23 on with progressive symptoms despite ongoing steroid treatment.

24 and persisted despite the cessation of oral steroid treatment.

25 ecovery of thymic function after a course of steroid treatment.

26 esses, duration of Crohn disease, and use of steroid treatment.

27 cations for evaluating patients on high-dose steroid treatment.

28 assist efforts to reduce the side effects of steroid treatments.

29 eas NO concentrations were not influenced by steroid treatment (3.0 +/- 0.4 ppm and 2.9 +/- 0.2 ppm,

30 trations were also higher in patients not on steroid treatment (3.4 +/- 0.2 ppm) than in steroid-trea

31 Under sustained steroid treatment, acute liver injury improved and hepat

32 to standard treatment and may substitute for steroid treatments aimed at controlling disease activity

33 n graft survival was seen compared with both steroid treatment alone (P < 0.05) and steroid combined

34 ase in graft survival was seen compared with steroid treatment alone (P = 0.045).

35 Steroid treatment and anticonvulsant use were related to

36 utations in NPHS2 do not respond to standard steroid treatment and have a reduced risk for recurrence

37 In spite of steroid treatment and plasmapheresis, his clinical statu

38 te rejection, which were reversed with bolus steroid treatment, and four were donor-specific antibody

39 iseases involving bone marrow, no history of steroid treatment, and no other risk factors for osteone

40 riasis severity, Charlson comorbidity index, steroid treatment, and socioeconomic status.

41 nically recognized as asthma, was quelled by steroid treatment, and was unmasked following corticoste

42 id a post-hoc analysis of the 3 year inhaled Steroid Treatment As Regular Therapy (START) study, done

43 PLEX and steroids treatment as time-dependent covariates were ent

44 aging approach revealed that bevacizumab and steroid treatment blocked leukocyte infiltration into im

45 The lesions improve after steroid treatment but can progress without steroid treat

46 ons in 10 patients regressed (in nine, after steroid treatment) but progressed in three patients with

47 in uveitic patients, intensive perioperative steroid treatment can lessen these complications.

48 Compared with intraoperative steroid treatment, combined preoperative and intraoperat

49 immediately recovered following cessation of steroid treatment, concurrent with restoration of the th

50 tivation and found that both denervation and steroid treatment contribute to the shift in inactivatio

51 r, or Pseudomonas aeruginosa infection, oral steroid treatment decreased Glut1 and PiT2 levels in blo

52 oxide, blood eosinophil counts, and inhaled steroid treatment did not influence cough parameters.

53 In patients not on steroid treatment ethane was correlated to airway obstru

54 as associated with the delayed initiation of steroid treatment for GVHD (0.95 months vs 3.0 months; P

55 ne average at 2 months was 56.0 for the oral steroid treatment group and 57.6 dB for the intratympani

56 In post hoc analyses, steroid treatment had an impact on survival in patients

57 While steroid treatment has proved ineffective, preliminary st

58 that is chronic in nature or uncontrolled by steroid treatment have shown good response to immunosupp

59 clonal bands (OCB) and beneficial effects of steroid treatments have provoked the hypothesis that EL

60 89 demonstrated a survival disadvantage with steroid treatment (I2 = 14%; relative benefit, 0.89 [CI,

61 The initial rejection episode responded to steroid treatment in 93.4% (tacrolimus) and 63.8% (CsA-M

62 cial effect is synergistically enhanced with steroid treatment in HR PK.

63 several side effects limit the usefulness of steroid treatment in humans leading to the quest for dev

64 Conversely, weekly steroid treatment in mdx mice improved muscle function a

65 We compared weekly versus daily steroid treatment in mouse models of acute muscle injury

66 The positive effects of chronic steroid treatment in muscular dystrophy are paradoxical

67 ined, whereas IL-13 levels were abrogated by steroid treatment in neonatal HDM-exposed mice and in EB

68 ps and then tested the rats during week 2 of steroid treatment in the eight-arm radial-arm version of

69 ws for the avoidance of maintenance adjuvant-steroid treatment in the majority of our patients.

70 We examined the role of denervation and steroid treatment in the shift of the voltage dependence

71 vertigo, recent evidence suggests that early steroid treatment in vestibular neuritis may improve lon

72 well as topical and anterior subconjunctival steroid treatments in uveitis induced by the intravitrea

73 Steroid treatment increased Na(+) transport to a similar

74 osite effects on Lep transcript abundance to steroid treatments, indicating that these transcriptiona

75 d cell number after the critical period when steroid treatment induces male sexual behavior.

76 parameters were used to show that intensive steroid treatment induces thymic involution and a profou

77 gh RSV-induced exacerbation was resistant to steroid treatment, inhibition of TNF-alpha and MCP-1 fun

78 of a net change in cell activity with acute steroid treatment is consistent with the possibility tha

79 s from early B-lineage cells,and response to steroid treatment is critical to successful ALL therapy.

80 Steroid treatment is often effective for uveitis associa

81 Adjunctive steroid treatment is underutilized in pneumococcal menin

82 molecular and genetic effects of intraocular steroid treatments is of clinical relevance.

83 oretical analysis of the data suggested that steroid treatment leads to receptors with a greater stab

84 Sex steroid treatment led to numerous alterations in gene ac

85 y not only increase our understanding of the steroid treatment mechanism but also help us to better m

86 Steroid treatment might aggravate glucocorticoid resista

87 Steroid treatment might be an option to prevent progress

88 ational follow-up of the Multicenter Uveitis Steroid Treatment (MUST) randomized clinical trial compa

89 The Multicenter Uveitis Steroid Treatment (MUST) Trial and Follow-up Study was a

90 Multicenter Uveitis Steroid Treatment (MUST) Trial masked BCVA measurements

91 educing AECOPD requiring both antibiotic and steroid treatment (n = 1,113; cumulative incidence analy

92 Within 48 hours of steroid treatment, neutrophils displayed an AnxA1(hi)CD6

93 ed genetic predisposition to the response to steroid treatment of acute allograft rejection.

94 Cs showed the most prominent reduction after steroid treatment of acute GvHD.

95 After prenatal steroid treatment of pregnant dams, 12 premature baboons

96 admitted for rehabilitation and intravenous steroid treatment of relapses.

97 The influence of steroid treatment on corneal infection in a Chinese hams

98 r133, we also evaluated the effects of acute steroid treatment on levels of phosphorylated CREB (pCRE

99 mphysema, we examined the effects of chronic steroid treatment on lung structure in adult rats.

100 used to determine the effects of axotomy and steroid treatment on ribosomal transcription and process

101 The effect of steroid treatment on the thymic output of T cells was ex

102 enal function, and most did not need chronic steroid treatment or calcineurin inhibitors.

103 periods (4 weeks) and may perpetuate despite steroid treatment or the immediate use of fast-acting im

104 rse-effect profile than conventional topical steroid treatments or other medical or surgical therapie

105 sk factors include bisphosphonate treatment, steroid treatment, osteoporosis, and head/neck radiation

106 After initialization of steroid treatment, polymerase chain reaction analyses of

107 Steroid treatment prevents the accumulation of reaper tr

108 Steroid treatment provided good relief of symptoms and p

109 le disease and with the level of maintenance steroid treatment required to control inflammatory activ

110 V acute graft-versus-host disease related to steroid treatment shows a trend toward a protective effe

111 Steroid treatment significantly decreases outflow facili

112 Steroid treatment significantly increased motoneuron siz

113 The steroid treatment similarly increased total cellular and

114 raftment (T3), at GvHD onset (T4), and after steroid treatment (T5) in 40 patients (7 Hodgkin's Disea

115 d in 54% of biopsies) was less responsive to steroid treatment than rejection without endarteritis, a

116 After steroid treatment, the beneficial effects of deep inspir

117 However, like steroid treatment, these regimens induce global immunosu

118 nd after 2 weeks of systemic glucocorticoid (steroid) treatment to identify immunological differences

119 s, or panuveitis enrolled in the Multicenter Steroid Treatment Trial and Follow-up Study.

120 and biases of recent low-dose (physiologic) steroid treatment trials limit their ability to provide

121 Despite similar baseline clinical severity, steroid treatment was associated with decreased 28-day s

122 Steroid treatment was associated with earlier onset of s

123 Clinical resistance to oral steroid treatment was defined as a drop in serum bilirub

124 Steroid treatment was the only variable that predicted r

125 Steroid treatment, which had been prescribed for the und