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1 not associated with any other rheumatologic stigmata.
2 atients with bleeding ulcers and higher risk stigmata.
3 gene in the development of additional Turner stigmata.
4 they have no other neurological or external stigmata.
5 he doses and timely management of endoscopic stigmata.
8 nts was localized in cauline leaves, pollen, stigmata, and floral primordia, and in the stems of youn
10 T); MEN 2B, by MTC, pheo, and characteristic stigmata; and familial MTC (FMTC), by the presence of MT
13 of 7.2 mm or greater as one of the high-risk stigmata, had a higher sensitivity (100%), negative pred
18 ferations, the KSHV-infected cells carry the stigmata of B lymphocytes, with plasmablastic features.
21 ology Group performance status (PS) of 1, no stigmata of chronic liver disease, and no ascites or enc
23 clinical CV risk, there may be a subset with stigmata of CV disease noted during the preoperative rad
24 icular bleeding because although they had no stigmata of diverticular hemorrhage, no other source of
26 All affected family members had no obvious stigmata of known genetic disorders associated with pneu
28 idity (0-3), diagnosis (0-2), and endoscopic stigmata of recent haemorrhage (0-2); the maximum possib
29 Two outcome variables were assessed: major stigmata of recent hemorrhage and need for endoscopic th
30 kall score (endoscopic triage) at predicting stigmata of recent hemorrhage and need for endoscopic th
39 ndromes may present without classic clinical stigmata or suspicious family history has led to increas
41 pies of the ATM gene that includes among its stigmata radiosensitivity and cancer susceptibility.
42 The arterial blood flow that underlies the stigmata rarely is monitored, but can be used to determi
46 rable improvement in the severity of chronic stigmata, such as syndesmyophytes and vertebral bridging
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