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1 hyperkinetic states such as those induced by stimulant drugs.
2                                 Increases in stimulant drug abuse may increase the rate of hospital a
3  underlying neurocognitive endophenotype for stimulant drug addiction.
4 that repeated treatment with the psychomotor stimulant drug amphetamine, which produces robust sensit
5 hown the safety and effectiveness of new non-stimulant drugs and long-acting formulations of methylph
6        Clandestine chemists synthesize novel stimulant drugs by exploiting structural templates known
7 lated on brain abnormalities associated with stimulant drug dependence.
8 the NAcc not only in the generation of acute stimulant drug effects but in sensitized responding to t
9     The research began with the finding that stimulant drugs enhanced memory in rats when administere
10 ectional designs, and without accounting for stimulant drug exposure.
11 ty and were not predicted by past or present stimulant drug exposure.
12 icated as mediating the rewarding effects of stimulant drugs; however, recent studies suggest that 5-
13 m may account for the therapeutic effects of stimulant drugs in clinical disorders such as ADHD.
14                                 The abuse of stimulant drugs is increasing in the western United Stat
15 reas transporter-mediated release induced by stimulant drugs is more restricted and unlikely to occur
16   The authors investigated the effect of the stimulant drug methylphenidate (given intravenously) on
17 itive to endogenous dopamine) induced by the stimulant drug methylphenidate in 20 controls and 20 det
18  how expectation affects the response to the stimulant drug methylphenidate in 25 cocaine abusers.
19 ility in the left ventral striatum, and that stimulant drugs modulate impulsivity and striatal D2/3 r
20 e administration of the first MPH dose to 40 stimulant drug-naive boys newly diagnosed with ADHD whil
21 l FH of substance use disorders (n = 16); 2) stimulant drug-naive healthy control subjects with no kn
22  (17 male, 7 female; age 23.0 +/- 6.2 years) stimulant drug-naive subjects who underwent PET [(11)C]r
23                                              Stimulant drugs of abuse have several effects on neural
24 city, promoting, for example, the actions of stimulant drugs of abuse such as cocaine.
25 eak link in the neurotoxicity seen following stimulant drugs of abuse.
26  shared by individuals who were dependent on stimulant drugs or used cocaine recreationally.
27                                              Stimulant drugs such as amphetamine are readily self-adm
28                                              Stimulant drugs such as cocaine and amphetamine have a h
29 exual desire could be one mechanism by which stimulant drugs such as cocaine and methamphetamine incr
30  seen in only a fraction of those exposed to stimulant drugs such as cocaine.
31  it so there is concern about treatment with stimulant drugs: this fear appears not to be substantiat
32 protected vaginal intercourse (p = 0.026) or stimulant drug use (p = 0.026), and more likely to repor
33 ey also reported significantly fewer days of stimulant drug use during the 3-month follow-up.
34 as associated with increased abstinence from stimulant drug use in stimulant-dependent patients with
35 ssue diseases, portal hypertension, diet and stimulant drug use, HIV infection, and congenital heart
36 vulnerability for dependence or with regular stimulant drug use.
37                                              Stimulant drug visits increased from 0.57 million to 2.8
38            Ephedrine and pseudoephedrine are stimulant drugs whose use is prohibited in athletic comp
39  function predict that the neural effects of stimulant drugs will vary according to task requirements

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