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1 ith many beneficial actions arising from D1R stimulation.
2 on of its downstream ERK cascade without EGF stimulation.
3 n frequency, reaching steady state 1 d after stimulation.
4 oked potentials associated with visual field stimulation.
5 rove performance further during intermittent stimulation.
6 d responsiveness to continuous or repetitive stimulation.
7 subthreshold depolarization preceding mAChR stimulation.
8 r Smad3 was a more precise outcome of ligand stimulation.
9 ices which are capable of more precise point stimulation.
10 ipid mediators in response to subsequent TLR stimulation.
11 oad conditions and/or at higher frequency of stimulation.
12 nce at 24 h post-transcranial direct current stimulation.
13 xternalization during surgery for deep brain stimulation.
14 n innate response to isolation or mechanical stimulation.
15 conductances can be activated with stronger stimulation.
16 regulated genes in patient T cells upon IL-2 stimulation.
17 ve T cells unresponsive to further antigenic stimulation.
18 the ability to induce rapid gravity or light stimulation.
19 e duration of inhibition resulting from, LHb stimulation.
20 ion potentials in the absence of presynaptic stimulation.
21 lf the energy of conventional high frequency stimulation.
22 air of distinctive neuronal responses to the stimulation.
23 s, and HNECs within 15 minutes of local TLR7 stimulation.
24 he net stimulatory effect of beta-adrenergic stimulation.
25 tivation and sterol biosynthesis after M-CSF stimulation.
26 DA ratio is observed following in vivo light stimulation.
27 and produced pro-inflammatory cytokines upon stimulation.
28 secretion in response to lipopolysaccharide stimulation.
29 nhuman primates induced by paired electrical stimulation.
30 tralized rotors by optical S1-S2 cross-field stimulation.
31 r to 3 initial months of VIM-only or VO-only stimulation.
32 both 6 months and 12 months of ongoing brain stimulation.
33 HCV inhibited the recall reaction to antigen stimulation.
34 less interferon-gamma in response to T-cell stimulation.
35 the anti-inflammatory role of MCL after PGN stimulation.
36 equences of transcranial alternating current stimulation.
37 code distinct stimulation patterns as massed stimulation.
38 ectrode and several types of multi-electrode stimulation.
39 nflammatory cytokines in response to ex vivo stimulation.
40 iquitinated targets of LUBAC following TRAIL stimulation.
41 on cocaine locomotion produced by peripheral stimulation.
42 duals in response to M. tuberculosis antigen stimulation.
43 re dramatic glycolytic responses to hormonal stimulation.
44 pression after interleukin-1 beta (IL-1beta) stimulation.
45 straint stress or optogenetic C1 neuron (C1) stimulation (10 min) protected mice from ischemia-reperf
46 nces in LV mechanics with altered adrenergic stimulation achieved through post-handgrip-exercise isch
47 between eating performance and environmental stimulation, adjusting for resident characteristics (i.e
49 ing cell; those episomes that did respond to stimulation, aggregated within large domains that appear
50 trol of initiation and assembly of CCPs, EGF stimulation also elicited a Ca(2+)- and PKC-dependent re
52 mals were divided into three groups based on stimulation and body weight (i.e., lean nonstimulated, o
54 hex as a direct target of RUNX1 and FLT3-ITD stimulation and confirmed high HHEX expression in FLT3-I
56 e use a novel paradigm of repetitive whisker stimulation and in vivo calcium imaging to assess tactil
57 exhibit a similar suppressive effect on TLR-stimulation and inflammatory cytokine expression from ma
58 oinflammatory cytokine response to bacterial stimulation and less human leukocyte antigen - antigen D
59 (RMTg) regions were activated by peripheral stimulation and LHb lesions reversed the inhibitory effe
60 ts link to conscious experience, and reviews stimulation and patient studies of the cortical basis of
63 ipids in human fibroblasts upon inflammatory stimulation and subsequent treatment with dexamethasone,
65 fects dependent on the relative phase of the stimulation and the internal cognitive processing state.
66 istic firing accommodation during maintained stimulation, and reduced membrane channel density causes
67 gnaling blockade upon myostatin or activin A stimulation, and this leads to only a small increase in
70 r pressure-time-product were achieved during stimulation assisted breaths in all 22 paced subjects (r
71 inal prostheses), and for closed-loop neural stimulation at a much larger scale than currently possib
72 alhm1 knockout cultures following mechanical stimulation at a pressure of 55 mmHg for 50 milliseconds
74 erformance in animals impaired by continuous stimulation but did not improve performance further duri
76 a fusion clamp, making release dependent on stimulation by Ca(2+)SIGNIFICANCE STATEMENT Syntaxin 1A
78 esynaptic cholinergic synapses to respond to stimulation by elevating presynaptic choline uptake and
81 it remains unknown whether electrical brain stimulation can be used to create a sense of ownership o
83 gical batteries were administered under four stimulation conditions: VIM on, VO on, both on, and both
85 ir axon terminals in the EB, and optogenetic stimulation coupled with electrophysiological recordings
86 tudies of the efficacy of 'awake' deep brain stimulation (DBS) for Parkinson's disease (PD) under loc
88 cal lesions or the application of deep brain stimulation (DBS) within circuits that modulate motor fu
90 this study, we used gene signatures of CD40L stimulation derived from human immature dendritic cells
94 olution sensory prostheses based on tailored stimulation (e.g., retinal prostheses), and for closed-l
96 cts as a crucial sensor for electrical field stimulation (EFS)-enhanced osteogenic response in osteop
97 m and local field potentials from deep brain stimulation electrodes in 9 Parkinson's disease patients
98 s involved in beta-arrestin-dependent Erk1/2 stimulation elicited by other GPCRs such as beta2-adrene
100 otspots in cortex, where mu opioid or orexin stimulations enhance the hedonic impact of sucrose taste
103 t optogenetic activation of D1R-SPNs reduces stimulation-evoked dopamine release and that bath applic
104 port was obtained by a transcranial magnetic stimulation experiment, where subjects whose frontal con
107 e, we use 180 pairs of transcranial magnetic stimulation for approximately 30 min over the hand repre
109 reased in a saturating manner with increased stimulation frequency; average [Ca(2+) ]i was a linear f
111 ot AMPA receptor blockade, prevents synaptic stimulation from facilitating D2R-induced ADPs, suggesti
112 umors overexpress cell migration and stromal stimulation gene signatures compared with their SOX11(-)
113 is known, however, how transcranial current stimulation generates such effects, and the search for b
114 ormance, but the transcranial direct current stimulation group demonstrated significant improvement i
115 cation accuracy of the transcranial magnetic stimulation-guided regions was validated in a second sam
117 of the brain employing transcranial magnetic stimulation has convincingly demonstrated a presymptomat
118 ion of neural synchrony through phase-locked stimulation has the potential to both increase the effic
119 The beneficial effects of parasympathetic stimulation have been reported in left heart failure, bu
120 ew methods for precisely controlling retinal stimulation, here we show that covert attention flexibly
126 gle-synapse level after distinct patterns of stimulation in motoneurons of Drosophila We found that t
127 xamine this issue, we used single glomerular stimulation in mouse olfactory bulb slices to measure th
129 EL) display diminished responsiveness to HEL stimulation in presence of soluble anti-HEL IgM antibodi
130 eatures using SPN type-specific chemogenetic stimulation in rodent models of PD (PD mice) and L-DOPA-
137 t me signal) on cardiac MSCs after both TLR4 stimulation in vitro and transplantation into the infarc
139 requency relationship and in beta-adrenergic stimulation, including decreasing and increasing firing
140 ow that ownership, but not mere visuotactile stimulation, increases the dominance of the hand percept
142 Rapid optogenetic silencing and electrical stimulation indicated that short-latency pathways linkin
143 s a selective defect in POLA2-binding and PP stimulation, indicating that these activities are critic
145 O animals exhibit a deficit in low-frequency stimulation-induced NMDAR-dependent long-term depression
147 We revealed that altering T-cell receptor stimulation influenced recruitment of mRNAs to heavy pol
148 e is a lack of evidence on how environmental stimulation influences individuals' eating performance a
149 ion in human primary CD4(+) T cells upon TCR stimulation, inhibiting NF-kappaB signaling via its effe
151 imulation with short latencies regardless of stimulation intensity, MC latencies correlate negatively
153 looks at both nutrition and early childhood stimulation interventions as part of an integrated life
155 or receptor superfamily molecule GITR, whose stimulation is closely linked to thymic Treg cell develo
157 ffector cytotoxic T lymphocytes upon antigen stimulation is necessary for successful antiviral, and a
158 mporal dynamic using closed-loop optogenetic stimulation is sufficient to increase movement in the TS
159 We paired cVNS with kilohertz electrical stimulation (KES) nerve block to preferentially activate
160 ng to record responses to concurrent thermal stimulation (left forearm) and visual attention tasks of
161 , it is not yet known whether or how sensory stimulation might trigger abnormal sensory processing at
164 nce and long term stability under electrical stimulation of a new electrode material fabricated from
166 NM myosin II is regulated during contractile stimulation of airway SM tissues by RhoA-mediated NM myo
167 sodium-restricted conditions, physiological stimulation of aldosterone was blunted with older age (b
168 ranscriptome profile, while the antagonistic stimulation of alpha7nAChR will achieve the opposite.
170 mpanied by increased ROS generation, and the stimulation of autophagy by rapamycin (Rap) remarkably s
172 raumatic brain injury (TBI), suggesting that stimulation of BDNF signaling pathways may facilitate fu
174 13B5 target sequence resulted in the robust stimulation of binding antibodies and, in a subset of im
175 rylation sites in alpha1C in beta-adrenergic stimulation of CaV1.2, and show that phosphoregulatory s
179 ChR activation increased responses evoked by stimulation of excitatory corticothalamic and inhibitory
180 in-1 small interfering RNA prevented hnRNP F stimulation of Foxo3alpha and downregulation of acetylat
183 ates HER2 at p1248 in PDAC cells, leading to stimulation of HER2 signaling cascade, including ERK1/2,
184 This effect can be partially mimicked by stimulation of Hmox1(+/+) SCs with monocyte chemoattract
185 -6R) alone had no effect on VEGF production, stimulation of HPMCs with IL-6 in combination with sIL-6
190 rrets that were conditioned using electrical stimulation of mossy fiber and climbing fiber afferents
193 roteinases (MMPs) and selective chemogenetic stimulation of nNOS interneurons recapitulated MMP activ
195 ia, an anomalous blending of senses in which stimulation of one sensory modality produces sensation i
197 sal oxytocin administration and chemogenetic stimulation of oxytocin neurons render males sensitive t
200 stglacial increase in Ca , which matched the stimulation of photosynthesis, suggesting that increases
205 tivate astrocytes through ephrin-B1-mediated stimulation of signal transducer and activator of transc
206 In vascular smooth muscle cells (VSMCs), stimulation of SOCs composed of canonical transient rece
208 results indicate that selective optogenetic stimulation of TH(VTA) neurons enhanced cerebral blood v
209 for RecN function that includes presynaptic stimulation of the bacterial repair pathway perhaps by c
210 r mechanical conditioning or pharmacological stimulation of the cGMP pathway and by using direct modu
213 investigated this using transcranial current stimulation of the rat (all males) motor cortex consisti
214 e a new axis for thymic emigration involving stimulation of the thymic microenvironment via type 2 cy
216 herosclerosis in response to pharmacological stimulation of thermogenesis linked to increased HDL lev
222 iation protein treslin in vitro Furthermore, stimulation of treslin phosphorylation does not occur by
223 otein, SHPS-1, resulting in pathophysiologic stimulation of vascular smooth muscle cell (VSMC) migrat
226 tudy, we investigated the effect of synaptic stimulation on Tau pathology and synapses in in vivo and
227 ls in response to a novel form of mechanical stimulation, or a pulsed wave at the frequency of 1.5 MH
228 his we used transcranial alternating current stimulation over the left dorsolateral prefrontal cortex
236 nd nucleotide-binding oligomerization domain stimulation promote ATG16L1 stabilization via IKKalpha-d
237 clusion These results suggest LIP ultrasound stimulation protects against brain injury in the hippoca
239 +) /Ca(2+) exchanger current that followed a stimulation protocol was significantly prolonged in PMCA
240 ates such effects, and the search for better stimulation protocols proceeds largely by trial and erro
241 d to females during reductions to adrenergic stimulation, providing preliminary evidence that LV twis
244 trated that lumbosacral spinal cord epidural stimulation (scES) and activity-based training can progr
246 y provoke VF if, and only if, the mechanical stimulation site overlaps the repolarization wave edge.
247 The impaired response propagation from the stimulation site was associated with lower expression of
248 raded emergence of gamma oscillations at the stimulation site while retaining propagating waves of ga
250 nd C fibers, respectively, based on required stimulation strengths, and shows segmental differences i
251 -CML stem cells were MHCI+ without IFN-gamma stimulation, suggesting that IFN-gamma sensitizes these
255 processing with transcranial direct current stimulation targeting the FPC while 141 healthy particip
256 gery (MI-BCI) or transcranial direct current stimulation (tDCS) has been used in stroke rehabilitatio
257 d in humans with transcranial direct current stimulation (tDCS) over the right dorsolateral prefronta
258 hypothesis: in humans, transcranial electric stimulation (tES) with a time course that mimics the end
259 stemic immunity by providing tonic microbial stimulation that can functionally replace intestinal bac
262 mbining the FCM with a transcranial magnetic stimulation (TMS) intervention that transiently perturbe
264 ossibility by means of transcranial magnetic stimulation (TMS) over the hand area of the primary moto
265 ss by combining online transcranial magnetic stimulation (TMS) with computational modeling of behavio
267 inhibitory repetitive transcranial magnetic stimulation to a subject-specific frontal-cingulate rewa
268 crucial intermediary processes that link TLR stimulation to DC maturation and the subsequent developm
269 rimary cilia are sensors of electrical field stimulation to induce osteogenesis of human adipose-deri
271 se results provide evidence that noninvasive stimulation to MDC nodes can enhance learning rates, the
272 Applications of transcranial direct current stimulation to modulate human neuroplasticity have incre
273 ymptom suppression is achieved by delivering stimulation to the ventrolateral thalamus, timed accordi
274 rate that transcranial static magnetic field stimulation (tSMS) over the somatosensory parietal corte
276 mechanism for reduced plateau [Ca(2+)]i upon stimulation was due to increased sarco/endoplasmic retic
278 ticipants compared with control subjects and stimulation was provided approximately 90 ms prior to mo
279 and perfusion MRI, and transcranial magnetic stimulation were used to study structural connectivity,
281 sformed into a type II medium by optogenetic stimulation which predominantly targets inhibitory neuro
283 he production of inflammatory cytokines upon stimulation with aminobisphosphonate-treated cells.
284 er vector increased luciferase activity upon stimulation with an anti-TREM2 antibody, which induces t
285 cells proliferated vigorously in response to stimulation with anti-CD3/CD28 beads and gave rise to CD
290 same extent as crypts from Cd44(+/+) mice on stimulation with HGF, but had the same response to EGF.
293 [Ca(2+) ]i was increased by beta-adrenergic stimulation with isoprenaline and increased in a saturat
296 ith uric acid for 24 h and then subjected to stimulation with lipopolysaccharide (LPS) in the presenc
297 Cs respond to olfactory sensory neuron (OSN) stimulation with short latencies regardless of stimulati
298 Ps were immediately rescued by pairing mAChR stimulation with subthreshold depolarization ( approxima
300 to-toxicity and to allow for long-term photo-stimulation without causing cellular damage, we formulat
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