ライフサイエンスコーパス: stimulation by

1 nocytic MDSC express TLR9 and respond to CpG stimulation by 1) losing their ability to suppress T cel

2 V pretreatment enhanced both acute locomotor stimulation by 15 mg/kg cocaine and development of locom

3 ted from Met e 1-sensitized Balb/c mice upon stimulation by 18 synthetic peptides that span the full-

4 retreatment with LEV reduced acute locomotor stimulation by 2.0 g/kg alcohol, attenuated the developm

5 vM1 optogenetic activation preceded vibrissa stimulation by 20 ms.

6 LL-37 induction was observed in response to stimulation by 25(OH)D(3).

7 a(2+) release in response to Galphaq/PLCbeta stimulation by 30 to 40%.

8 In response to directional stimulation by a chemoattractant, cells rapidly activate

9 TLR4 stimulation by a high fat diet or LPS were both associat

10 nd Lyn) were tested for activation following stimulation by A9/I-A(q) Unexpectedly we found they are

11 d headgroup dependence, and show significant stimulation by acetate.

12 erentially to and required for transcription stimulation by acetylated double-variant chromatin.

13 neutral amino acids (Ala, Cys) reduced ENaC stimulation by acidic pH, whereas reintroduction of a ne

14 lation and that the previously characterized stimulation by acidic phospholipids is dependent on the

15 and cDCs from bone marrow cells under flt3L stimulation, by acting on lineage(-) flt3(+) precursors.

16 sive phenotype by reducing the set point for stimulation by activated TGF-beta.

17 Injury induced by scratch wounds or stimulation by addition of UTP caused a brief internaliz

18 sion decreased whole-blood aggregation after stimulation by ADP, an effect negated by adenosine-5'-0-

19 However, on stimulation by agonists such as cocaine, Sig-1Rs translo

20 ade bodies (WPBs), and is released following stimulation by agonists that raise intracellular Ca(2+)

21 rize the co-activation of SOCC and LTCC upon stimulation by agonists, and to determine the possible c

22 ibitor of adenosine kinase, folate transport stimulation by AICAR was absent.

23 Mechanistically, Treg stimulation by alpha-(1,3)-glucan was dependent on the P

24 Whereas NKT stimulation by alpha-Galactosylceramide required CD1d ex

25 e sensitivity of cortical neurons to sensory stimulation by altering the balance between excitation a

26 A distinctive feature of EcoLigA is its stimulation by ammonium ion.

27 Optimal production requires stimulation by an NF-kappaB inducer, most commonly an in

28 est a novel pathogenic mechanism in the TLR4 stimulation by anti-beta(2)GPI peptide Abs that links ad

29 s CD4(+) T-cell proliferation in response to stimulation by anti-CD3 and anti-CD28 antibodies.

30 Exogenous ROS mimicked stimulation by anti-Siglec-8 and was sufficient to induc

31 s-presentation of associated peptides and co-stimulation by APCs that interact with alpha(2)M.

32 Pase triggers T cell proliferation upon IL-2 stimulation by associating with PYGM and modulating its

33 both their basal peptidase activity and the stimulation by ATPgammaS.

34 Aldosterone-independent stimulation by AVP shifts the role of ENaC in the ASDN f

35 isolated from T1D subjects that responded to stimulation by B:11-23 peptide and denatured insulin pro

36 ecular changes that occur in the animal upon stimulation by bacteria.

37 Upon stimulation by bacterial antigens, enteric alpha-defensi

38 The activated genes were consistent with stimulation by bacterial lipopolysaccharide; an influx o

39 th heat-killed PG (HKPG) and PG-LPS prior to stimulation by bacterial PAMPs.

40 Chronic stimulation by bacterial pathogen associated molecular p

41 odel immune response of mouse macrophages to stimulation by bacterial toxin, and a spatially-resolved

42 PI3KC3 recruitment and autophagy stimulation by Barkor/Atg14(L) require the BATS domain.

43 4,5)P2, which is produced locally upon Wnt3a stimulation by beta-arrestin- and Dishevelled-associated

44 and glucagon-like peptide-1) in response to stimulation by bitter-tasting compounds.

45 (TNF1), that controlled TNF production after stimulation by both BCG alone and BCG plus IFN-gamma.

46 K2 appears to be selectively involved in LTR stimulation by both KSHV ORF45 and HIV-1 Tat.

47 Notably, distinct phases of the stimulation by both mtSSBs are distinguishable, and they

48 a fusion clamp, making release dependent on stimulation by Ca(2+)SIGNIFICANCE STATEMENT Syntaxin 1A

49 We used direct stimulation by calcium uncaging at identified, single-si

50 , feedback from ATP hydrolysis products, and stimulation by calcium were characterized in isolated mi

51 ide-out Ca(2+) signaling in response to IL-8 stimulation by catalyzing an increased density of Talin-

52 Normally, leptin potentiates vagal afferent stimulation by CCK but this is lost in obesity.

53 IL-4 alone induces only CCL17 but enhances stimulation by CD40L of both CCL17 and CCL22.

54 Stimulation by chemokines of integrin alpha4beta1-depend

55 Because neural stimulation by CI electrodes accounts for up to 90% of p

56 pe plants are almost completely resistant to stimulation by CK supplied to the vasculature.

57 re, we show that beta(3)-adrenergic receptor stimulation by CL 316,243 promotes adipose tissue neutro

58 alcn regulates RTN neuronal excitability and stimulation by CO2, independent of direct pH sensing, po

59 ar distribution of TLR4 before and after LPS stimulation by confocal microscopy.

60 f SGs undergoing exocytosis during sustained stimulation by controlling vesicular mobilization and tr

61 The results show that dual stimulation by CpG and CD40L for 48 h is optimal for IL-

62 g fibroblasts to Toll-like receptor 9 (TLR9) stimulation by CpG-oligodeoxynucleotide (CpG-ODN) was in

63 on of immunosuppression as well as antigenic stimulation by cross-presentation of EBV antigen from de

64 Here we examine the mechanisms of PP stimulation by CST using purified complexes derived from

65 Moreover, co-stimulation by CXCL12 together with soluble VCAM-1 poten

66 T cells, which are recruited to tumors upon stimulation by CXCL9 and CXCL10, were largely excluded f

67 ses (JAK) phosphorylate STAT3 in response to stimulation by cytokines or growth factors.

68 this reduction was due to decreased maximal stimulation by DAMGO, with no difference in EC(50) value

69 Thus, the subunit composition-dependent stimulation by DHA demonstrates that BK channels are eff

70 Here, we demonstrate that direct stimulation by dietary amino acids regulates the homeost

71 hese findings, enforced NOTCH3 expression or stimulation by DLL4 increased levels of activated NOTCH1

72 that both ATPase sites are required for the stimulation by DNA.

73 In this study, stimulation by doxorubicin, hypoxia and ionizing radiati

74 tivation of different NF-kappaB subtypes via stimulation by EGF and UV stress.

75 Stimulation by EGF does not appear to induce a change in

76 aracterized by disturbed responses linked to stimulation by either integrin ligands or immobilized im

77 K phosphorylation per se but potentiated the stimulation by either muscarinic agonists or 2-methyl-th

78 of canopy leaves (Asat ) also showed similar stimulation by elevated CO2 at +60 ppm as at +150 ppm CO

79 esynaptic cholinergic synapses to respond to stimulation by elevating presynaptic choline uptake and

80 vation of glycolysis through beta-adrenergic stimulation by endogenous catecholamines plays an import

81 In this study, we found that stimulation by endothelial cells can render resting CD4(

82 The stimulation by endothelial cells does not involve interl

83 nit alpha-deficient ASMCs responded to CXCL1 stimulation by enhancing p38 MAPK activation and ASMC mi

84 These include pathways of innate stimulation by environmental or endogenous pathogen-asso

85 istant to transformation by mutant K-ras and stimulation by epidermal growth factor.

86 unbinding force to LAMA5, and insignificant stimulation by epinephrine as compared to SS-RBCs from u

87 beta(2)-adrenergic receptor stimulation by epinephrine can enhance ERK1/2 activity o

88 The contractile stimulation by epinephrine was linked to drug tissue lev

89 one secreted by erythroblasts in response to stimulation by erythropoietin (EPO).

90 VSE is also required for Vta1-mediated Vps4 stimulation by ESCRT-III subunits Vps60 and Did2.

91 4-hydroxypentanoate from levulinate and its stimulation by ethanol is a potential public health conc

92 positively respond to moderate intensity SMF stimulation by exhibiting enhanced differentiation, func

93 Naive T cells respond to antigen stimulation by exiting from quiescence and initiating cl

94 of nontolerant anti-self T cells from strong stimulation by exogenous tolerogen.

95 Upon stimulation by exogenous Wnts, Rab8a-deficient cells sho

96 s lead to calcium oscillations following the stimulation by external ATP.

97 In mechanoreceptors, mechanical stimulation by external forces leads to the rapid openin

98 te maximal levels of CCL20 at 6 h, following stimulation by F. nucleatum cell wall (FnCW).

99 or RBL-2H3 cells challenged with CE prior to stimulation by FcepsilonRI cross-linking.

100 Response of cells to stimulation by fetal calf serum could be reproduced by t

101 Organoid branching was dependent on stimulation by FGF2, and Ptprb knockdown in mammary epit

102 ed that NL dendrites respond to differential stimulation by first decreasing the size of their unstim

103 In human proximal tubular epithelial cells, stimulation by fluvoxamine or oxidative stress caused th

104 B cell development being extended to include stimulation by foreign Ag, and also further the known zo

105 af1 promoter upon T cell receptor (TCR)/CD28 stimulation by forming a complex with histone acetyltran

106 Na2S potentiated CFTR stimulation by forskolin, but not that by IBMX.

107 t a secondary or enhancer reaction like cAMP-stimulation by forskolin.

108 On stimulation by G(s), the activities of ACs can be furthe

109 the GAP activity of p115 is not required for stimulation by Galpha(13), two hydrophobic motifs in RH

110 ss-linking it to the EF hand domain inhibits stimulation by Gbetagamma without altering basal activit

111 Selective stimulation by glucose of tanycyte cell bodies evokes ro

112 tic nerve activity in the basal state and no stimulation by glucose.

113 is phosphorylated on tyrosine residues upon stimulation by growth factors and that this event is cri

114 We study the role of metabolism and stimulation by growth factors, and show that metabolism

115 ution of integrins from focal adhesions upon stimulation by growth factors.

116 aplotype displayed a significantly increased stimulation by guanosine triphosphate compared with the

117 r cells, blocks RetGC catalytic activity and stimulation by guanylyl cyclase-activating proteins (GCA

118 LXR stimulation by GW3965 up-regulated genes involved in cho

119 We found that epithelial stimulation by HDM selectively increased the proliferati

120 A and protein levels, than did controls upon stimulation by heat-killed wild-type Porphyromonas gingi

121 x by NS5BDelta21, resulting in RNA synthesis stimulation by helicase.

122 the JAK/STAT3 signaling pathway induced upon stimulation by Heme treatment, were assessed using real

123 HRPII inhibits AT activity by preventing its stimulation by heparin.

124 d to be released from pancreatic islets upon stimulation by high glucose.

125 NMDA receptor stimulation by homocysteine was determined by patch clam

126 ereas H2S is excitatory and mediates sensory stimulation by hypoxia.

127 Inhibition of phosphorylation by Na(+) and stimulation by ibogaine occurred at concentrations that

128 ts became refractory to in vivo and in vitro stimulation by IFN-alpha during the second-phase virolog

129 Stimulation by IFN-gamma in skeletal muscle cells induce

130 Neutrophil stimulation by IgA or IgG ANCA led to degranulation and

131 let-activating factor subsequent to FcgammaR stimulation by IgG/Ag complexes.

132 R1 with IL-1R antagonist suppressed platelet stimulation by IL-1, so IL-1beta stimulates its own synt

133 horylation of STAT4 and STAT1 in response to stimulation by IL-12 and type I interferon (IFN), respec

134 monocytogenes did not require CD8(+) T cell stimulation by IL-12 produced in response to infection,

135 omes phosphorylated in human cells following stimulation by IL-1R and Toll-like receptor agonists, wh

136 Acute or chronic stimulation by IL-4 modified expression of more than 100

137 e of IL-31, which is produced in response to stimulation by IL-4.

138 Clotting stimulation by immobilized tissue factor induced localiz

139 normal fibroblast (NF) after IL-6*IL-6Ralpha stimulation by immunoassays.

140 1 is not required to mediate transcriptional stimulation by improvement of the -10 element.

141 primary cilia and also responded to hormonal stimulation by increase of intracellular Ca(2+) .

142 artner (SHP) upon farnesoid X receptor (FXR) stimulation by increasing BA concentrations.

143 H receptor (PTH1R) and do not respond to PTH stimulation by increasing cAMP production or migrating t

144 cer cells with mutant p53 respond to insulin stimulation by increasing cell proliferation and invasiv

145 Kv11.1 channels responded to beta-adrenergic stimulation by increasing I(Kv11.1).

146 ed their ability to respond to growth factor stimulation by increasing pAKT levels proportionally to

147 p-regulated in arthritis, largely because of stimulation by inflammatory cytokines such as IL-1beta.

148 f vigorous stimuli, and respond to antigenic stimulation by initiating cell cycle progression and fun

149 lation/activation of IRS-1 and AKT following stimulation by insulin, insulin-like growth factor-1, or

150 showed enhanced proinflammatory responses on stimulation by interferon-gamma and oxidized low-density

151 Stimulation by interferon-gamma increased intracellular

152 fically respond to the frequency of neuronal stimulation by intracellular Ca2+ transients, with a cle

153 its association with the chromatin, and now, stimulation by ionizing radiation, we hypothesize that P

154 nterfering RNA knockdown of BTN3A1 abolished stimulation by IPP that could be restored by re-expressi

155 e that systemic nonselective beta-adrenergic stimulation by ISO at concentrations that increase energ

156 Adrenergic stimulation by isoproterenol (1 muM) or forskolin (5 muM

157 Following TLR7/8 stimulation by its agonist R848, chemical inhibition of

158 ivation even in the absence of growth factor stimulation by its endogenous ligand, hepatocyte growth

159 However, CeA stimulation by itself failed to support behavioral self-

160 Nor did CeA stimulation by itself induce any aversive state that mot

161 alphavbeta3 integrin, which responds to bFGF stimulation by JAM-A release to regulate mitogen-activat

162 he effects of EGTA, reserpine, and prolonged stimulation by K(+).

163 tal variables in podsol soil-with consistent stimulation by labile organic matter that did overrule t

164 mobilized in growing cells or upon hormonal stimulation by LD-associated lipases and steryl ester hy

165 es endogenous Galphaq and is unresponsive to stimulation by leukotriene.

166 nal clusters--often a critical step in their stimulation by ligand--are poorly understood.

167 electric focusing signature of PKCdelta upon stimulation by ligands of the phorbol ester and bryostat

168 oinflammatory monocytes and neutrophils upon stimulation by ligands of Toll-like receptors or proinfl

169 ral and spatial precision afforded by neural stimulation by light holds promise as a powerful alterna

170 feron (IFN)-responsive genes (ISG) following stimulation by lipopolysaccharide (LPS) of Toll-like rec

171 ta is also inducibly degraded; however, upon stimulation by lipopolysaccharide (LPS), it is degraded

172 examethasone (Dex) treatment before or after stimulation by lipopolysaccharide (LPS).

173 The magnitude of IFN-beta stimulation by liposome-encapsulated poly(I:C) is marked

174 We propose that this direct B cell stimulation by live RABV-based vaccines is a potential m

175 otemporal reorganization of actin after cell stimulation by local force application.

176 Of these, the most recently discovered is stimulation by loop formation.

177 esidue F660, which is known to determine the stimulation by low pH in human TRPV1, is also essential

178 ol-based chemiluminescence and combinatorial stimulation by low-dose artemisinin to photoactivate PPI

179 Platelets express functional TLR4, and stimulation by LPS induced rapid splicing, translation,

180 tic cells preincubated with IFN-gamma before stimulation by LPS, suppression of p40 and IL-12p70 prod

181 14-3-3gamma was rapid, peaking within 3 h of stimulation by LPSs, and sustained over the course of AI

182 GLP-1 stimulation by luminal glucose (20%) secretion was block

183 cant in NHE3 regulation, being necessary for stimulation by lysophosphatidic acid of activity and inc

184 oci were measured by real-time PCR after the stimulation by M. leprae antigens in the PBMC (periphera

185 type 1 (Th1), Th2, and Th17 cells following stimulation by M. tuberculosis antigen and enhanced freq

186 Na2S enhanced CFTR stimulation by membrane-permeable 8Br-cAMP under inhibit

187 absence of pDCs in response to CpG-Dotap and stimulation by microbial pathogens, such as Leishmania m

188 sed on physiology, it can respond to sensory stimulation by mimicking tadpole swimming behavior.

189 or avoidance behavior following noxious heat stimulation by modifying the forward-to-reversal behavio

190 innate immunity due to inadequate Wnt ligand stimulation by monocytes provides an additional mechanis

191 and synthesized ECM proteins in response to stimulation by mouse UII.

192 common mechanisms of signaling via TLRs link stimulation by multiple pathogens to atherosclerosis.

193 at GSK3beta activity is suppressed following stimulation by multiple signal transduction pathways, ou

194 Effects of combined stimulation by murine CXCL9 and CXCL12, ligands of CXCR3

195 malignancies and are a prevalent outcome of stimulation by native, monovalent EGF, or NRG.

196 Unexpectedly, direct stimulation by neither IL-12 nor type I IFNs on pathogen

197 at baseline and increased 15-fold during BAT stimulation by norepinephrine (1 microg.kg(-1).min(-1)).

198 ndritic cells after Toll-like receptor (TLR) stimulation, by not completely explained mechanisms.

199 nly approximately 3-fold reduction in ATPase stimulation by nucleosomes.

200 uated by multiparameter flow cytometry after stimulation by overlapping peptide pools of BK virus ant

201 can be explained by the overriding of Ca(2+) stimulation by paracrine inhibition, because somatostati

202 Monocytes and macrophages stimulation by pathogen antigens results in activation o

203 ke protein 3 (SSL3), which prevents receptor stimulation by pathogen-associated lipopeptides.

204 TCR stimulation by peptide-MHC complexes on APCs requires pr

205 nges in these cell lines in response to PDGF stimulation by performing phospho-site profiling.

206 sion of CD28(-)CD8(+) T cells during chronic stimulation by persistent Ag.

207 2 disrupts catalysis, allosteric activation, stimulation by phosphatidylserine, and pharmacological i

208 acrophages as controlled release agents upon stimulation by physical and/or mechanical cues provided

209 nor whether this mechanism occurs following stimulation by physiological agonists is known.

210 al for both its polyadenylation activity and stimulation by PI4,5P(2).

211 Stimulation by PIP(2) injection was mimicked by injectin

212 adhesion to extracellular matrix proteins or stimulation by platelet-derived growth factor (PDGF), th

213 Upon stimulation by PLY, mast cells produced cysLTs that acti

214 ort a model in which antigen-specific T cell stimulation by PND APCs triggers IFNgamma, followed by C

215 ly cross-presented a cell-associated Ag upon stimulation by polyinosinic-polycytidylic acid or R848,

216 The lack of stimulation by polymerase alpha suggests the existence o

217 Thus, BTN3A1 is required for stimulation by prenyl pyrophosphates but does not bind t

218 uch more sensitive to statin inhibition than stimulation by prenyl pyrophosphates; however, the conti

219 nd left ventricular dilation after long-term stimulation by pressure overload.

220 ells, respond to Toll-like receptor-mediated stimulation by producing interleukin-12 and process and

221 CD8(+) T cells respond to TCR stimulation by producing proinflammatory cytokines, and

222 V1) reaches full activation, with no further stimulation by protons.

223 YHB1 mRNA decay stimulation by Puf proteins is also responsive to cellul

224 ifies ERF as a novel regulator of osteogenic stimulation by RAS-ERK signaling, potentially by competi

225 Unexpectedly, chronic stimulation by repetitive sounds, whisker deflection or

226 wing BND cells to fully respond to antigenic stimulation by restoring signaling through the B-cell re

227 dose after stimulation by THW compared with stimulation by rhTSH injections was 3.9, 27, and 1.4 for

228 onal tissue cultivated on the gate area upon stimulation by rising the extracellular K(+) concentrati

229 ponded specifically to CMV-phosphoprotein 65 stimulation by secreting IFN-gamma and killing virus pep

230 eloped in vivo responded in vitro to peptide stimulation by secreting interleukin 2 and IFN-gamma.

231 Naive T cells also require stimulation by self-pMHCs.

232 ter the nuclear accumulation of MRTF-A after stimulation by serum addition.

233 mouse PA-SMCs, TERT was expressed on growth stimulation by serum.

234 l to the rhythmic stimulation outlasting the stimulation by several cycles.

235 ic suppressor function could be preserved by stimulation by specific donor alloantigen and cytokines

236 gnal transduction upon T-cell receptor (TCR) stimulation by specifically suppressing the activation o

237 cultures in three dimensions in response to stimulation by sphingosine 1-phosphate and growth factor

238 e investigate the specific mechanism of AMSH stimulation by STAM proteins and the role of the STAM Vp

239 We demonstrate the utility of TI stimulation by stimulating neurons in the hippocampus of

240 lar maltose) but not trans-allostery (uptake stimulation by subsaturating cytochalasin B).

241 r features, including small size and optimal stimulation by surface-associated ligands.

242 r findings collectively indicate that miR-21 stimulation by T(3) and subsequent TIAM1 suppression pro

243 mediates trialysin protection and metabolic stimulation by T. cruzi, indicating that extracellular c

244 to that of other coactivators, its 140-fold stimulation by TCPOBOP was striking and unique.

245 f spectrotemporal receptive fields following stimulation by temporally coherent and incoherent tone s

246 tion, Gdown1 specifically blocked elongation stimulation by TFIIF, inhibited the termination activity

247 NFAT activity is required for ECM stimulation by TGF-beta.

248 s has been lively and ever-growing since its stimulation by the advent of click chemistry.

249 the same rate in the presence or absence of stimulation by the agonist, melanocyte-stimulating hormo

250 helial cells (GECs) undergo apoptosis due to stimulation by the bacteria or inflammatory cytokines.

251 ls (7), suggested differential CD4(+) subset stimulation by the different parasite stimuli.

252 (IFN-beta) and/or IFN-lambda1 in response to stimulation by the dsRNA analogue polyinosinic:polycytid

253 kinase focal adhesion kinase (FAK) upon cell stimulation by the extracellular matrix initiates integr

254 in the folded structure decreases the ATPase stimulation by the folded ribozyme.

255 We further show that ATPase stimulation by the H4 tail does not require a specific s

256 ns downstream of Dectin-2 in response to the stimulation by the hyphal form of Candida albicans, an o

257 To determine whether stimulation by the inhibitory protein coevolved with res

258 may imply oncogenic activation of MET or its stimulation by the ligand hepatocyte growth factor.

259 rt that oxytocin (Oxt) receptors (Oxtrs), on stimulation by the ligand Oxt, translocate into the nucl

260 s, spontaneous whisker movements and passive stimulation by the littermates cooperate, with comparabl

261 s, spontaneous whisker movements and passive stimulation by the littermates cooperate, with comparabl

262 us (self-generated movements) and exogenous (stimulation by the littermates) mechanisms cooperate in

263 ng from the whisker movements with touch and stimulation by the littermates, support: (1) a twofold h

264 induced by LXR activation and maintained RCT stimulation by the LXR ligand.

265 smic to nuclear translocation in response to stimulation by the mononuclear cytokine, TNF.

266 Stimulation by the NOTCH ligand DLL4 was associated with

267 ht to be important for long-distance poly(A) stimulation by the NRS.

268 raction is markedly disrupted after receptor stimulation by the specific agonist UK14304, suggesting

269 ggested by partial inhibition of cholinergic stimulation by the specific PKA inhibitor Rp-cAMPS.

270 1G4 stimulation by the stronger stimulus emptied the ER of s

271 his unprecedented example of intein splicing stimulation by the substrate of the invaded host protein

272 assessed by flow cytometry, without and with stimulation by the thromboxane analog U46619 or ADP.

273 of NK cells with ex vivo or in vivo cytokine stimulation, by the use of antibodies to induce antibody

274 In this study, we further characterize stimulation by these compounds and define pathways used

275 tio of the average tumor absorbed dose after stimulation by THW compared with stimulation by rhTSH in

276 TNFRSF25 stimulation by TL1A-Ig in vivo induced expansion of Treg

277 y suppressed the proinflammatory response to stimulation by TLR ligands.

278 e ablation of MARCKS, which had no effect on stimulation by TLR2, TLR3, and TLR5 agonists.

279 ) both in control conditions and also during stimulation by TNF-alpha.

280 ponses of neutrophils, which face cumulative stimulations by TNF-alpha, beta2-integrin engagement, C5

281 increased DNA synthesis and robust activity stimulation by TPP1-POT1.

282 tional magnetic resonance imaging with brain stimulation by transcranial magnetic stimulation.

283 factor 2 (TTF2) as well as block elongation stimulation by transcription factor IIF (TFIIF).

284 urthermore, migratory DCs were refractory to stimulation by transient exposure to TLR agonists, as th

285 rane and responds to uniform chemoattractant stimulation by transiently localizing to the cytosol.

286 report, we assessed the effects of autophagy stimulation by trehalose in a transgenic mouse model of

287 of linear regression analyses comparing fold stimulation by TSH of IP1 vs. cAMP production were 0.044

288 dramatic enhancement in settlement following stimulation by turbulent shear typical of wave-swept sho

289 h we propose the designation 'Tcarbs') after stimulation by two glycoconjugate vaccines.

290 pes of cortical pyramidal cells to patterned stimulation by two-photon glutamate uncaging.

291 importer, providing insights into the ATPase stimulation by unliganded MalE.

292 nuated airway responsiveness to methacholine stimulation by up to 42%, concomitantly reduced tissue r

293 a reduction of the electrical threshold for stimulation by up to approximately 40%, or voluntary, re

294 rein, we developed a neat, green approach of stimulation by using CO2 gas as "molecular drill" to pie

295 n markers was assessed after anti-IgM or CpG stimulation by using flow cytometry on B cells from pati

296 originate from melanoma-competent MCSCs upon stimulation by UVB, which induces MCSC activation and tr

297 ation and nuclear translocation of IRF3 upon stimulation by various inducers.

298 nscriptionally induced in ECs in response to stimulation by vascular endothelial growth factor (VEGF)

299 cells, tacrolimus also inhibited Akt and p38 stimulation by vascular endothelial growth factor, a maj

300 Upon stimulation by Wnt ligands, the canonical Wnt/beta-caten