ライフサイエンスコーパス: stimulation with

1 ta show that, under physiologically relevant stimulation with 10 pm cholesystokinin, the luminal acid

2 2+)]i response to approximately half-maximal stimulation with 100 nm ACh ( approximately EC50), [Ca(2

3 ating mediators and with six cytokines after stimulation with 19 bacterial, fungal, viral, and non-mi

4 ured cells) and increased up to 0.9 Hz after stimulation with 30 mM KCl in cultured cells.

5 t controlled IFN-gamma levels in response to stimulation with 6-kDa early secretory antigen target, w

6 n in the immune system is up-regulated after stimulation with a bacterial endotoxin (lipopolysacchari

7 ependent protein kinase A was abolished, and stimulation with a cAMP analog mimicked the adjuvant eff

8 Stimulation with a combination of anti-CD3 and IL-4 indu

9 man tonsillar and blood ILC2s in response to stimulation with a combination of IL-25, IL-33, thymic s

10 els and transients increased upon mechanical stimulation with a hydrogel, and single cells altered pr

11 d a sustained phosphorylation of Ser220 upon stimulation with a ligand.

12 ly identified sweet receptors and respond to stimulation with a panel of sweet compounds.

13 ed responses than static models, during both stimulation with a series of natural sounds and epochs o

14 They respond to an olfactory nerve stimulation with a short barrage of excitatory inputs me

15 with c-Fos-based activity maps generated by stimulation with a sour tastant, 30 mM citric acid.

16 henotype and mainly produced IFN-gamma after stimulation with A. fumigatus antigens.

17 nstant but contingent on the strength of the stimulation, with a larger modulation for stimuli involv

18 Endothelial stimulation with A23187 or thrombin caused constriction

19 ll-like receptor 9 expression increased upon stimulation with acetaminophen-induced acute liver failu

20 to Young, the [Ca(2+) ]i response to maximal stimulation with acetylcholine (3 mum, 2 min) was approx

21 Here, stimulation with acetylcholine induced Plk1 phosphorylat

22 Here stimulation with acetylcholine promoted the recruitment

23 Stimulation with ACh caused NM myosin filament assembly,

24 w)Syk(low) phenotype in NK cells: continuous stimulation with activation beads and DNA damage.

25 ine expression by CD4 and CD8 T cells, after stimulation with Ag85A, Ag85B, and TB10.4 peptide pools,

26 ivation of the mu-opioid receptor (MOR).D1LR stimulation with agonist SKF 38393 concentration-depende

27 signaling following physiological levels of stimulation with agonists that engage the phospholipase

28 8 MCD displayed a proliferative defect after stimulation with alpha-galactosylceramide.

29 After stimulation with alpha2M*, CS-GRP78 signaling activates

30 he production of inflammatory cytokines upon stimulation with aminobisphosphonate-treated cells.

31 were induced in isolated guinea pig OHCs by stimulation with an 8 V/cm external alternating electric

32 After stimulation with an agonist, an additional 30% of recept

33 er vector increased luciferase activity upon stimulation with an anti-TREM2 antibody, which induces t

34 However, the dynamics of calcium flux after stimulation with an APC in vivo remain to be fully under

35 Stimulation with an EphB2 extracellular domain-Fc fusion

36 l receptor activation pathways after ex vivo stimulation with anti-CD3 and anti-CD28 crosslink in CD4

37 Stimulation with anti-CD3 resulted in phosphorylation an

38 In vitro stimulation with anti-CD3/CD28 antibodies or type I inte

39 cells proliferated vigorously in response to stimulation with anti-CD3/CD28 beads and gave rise to CD

40 hils after stimulation with peanut and after stimulation with anti-IgE (CD63 peanut/anti-IgE) was ind

41 Stimulation with anti-IgE or IL-3 resulted in strong upr

42 B cell responses induced by B cell receptor stimulation with antigen.

43 In vitro stimulations with ANXA1 peptide resulted in decreased in

44 dilation of the hP2X7R channel on sustained stimulation with ATP(4).

45 ce that macrophage secretion of IL1beta upon stimulation with ATP, crystals or LPS is mediated by a r

46 priming with lipopolysaccharide, followed by stimulation with ATP, led to an activation of caspase 1

47 duced chemotaxis and IL-1beta secretion upon stimulation with ATP, probably due to direct targeting o

48 ba Dictyostelium discoideum produce ETs upon stimulation with bacteria or lipopolysaccharide in a rea

49 larization, but develop resistance following stimulation with bacterial DNA or CpG oligodeoxynucleoti

50 acterium acnes and Prevotella intermedia for stimulation, with best growth on membranes over "helper"

51 ocytes failed to produce TNF-alpha following stimulation with C. tropicalis Ags.

52 ng microbial cells (bacteria and yeast) upon stimulation with carbon source.

53 te these microenvironments in vitro, such as stimulation with CD154 and IL4.

54 Stimulation with CD3/CD28, PMA/ionomycin, or latency rev

55 iate or primed state of activation following stimulation with certain agonists.

56 TRPA1 is sensitised by repeated stimulation with chemical agonists in a calcium-free env

57 ence its affinity and avidity following cell stimulation with chemokines and/or ligands.

58 Stimulation with cholangiocyte supernatants from BDL WT

59 However, upon in vitro stimulation with CMV, CBMCs undergo increased proliferat

60 hrough increased phosphorylated (p)-Akt upon stimulation with CMVpp65.

61 Stimulation with cochlear implants and hearing aids is b

62 to combine magnetic-field-induced mechanical stimulation with confocal fluorescence microscopy and pr

63 K promoter activity and gene expression upon stimulation with constitutively active Notch1 in presenc

64 depolarization, our results show that prior stimulation with CpG oligodeoxynucleotide or Escherichia

65 ssociated with AID in the same B cells after stimulation with CpG.

66 Co-stimulation with CRH and AVP results in complex patterns

67 We combined thermal grill stimulation with crossing the fingers to investigate whe

68 Co-stimulation with CSE, IL-17A and aeroallergens further i

69 We found that overexpression of CXCR4 or stimulation with CXCL12 supports neuroblastoma tumorigen

70 h mRNA and protein levels increase during NK stimulation with cytokines (IL-12, IL-18, and IL-15).

71 tion by IGCs, MCs responded to sensory input stimulation with decreased depolarization and spiking fo

72 naling and via direct cannabinoid receptor 1 stimulation with Delta(9)-tetrahydrocannabinol.

73 g (p190)BCR-ABL-specific T cells in vitro by stimulation with dendritic cells pulsed with (p190)BCR-A

74 jacchus) responded to rapid time-varying CI stimulation with discharges that were not synchronized t

75 B cells possess selective sensitivity to TLR stimulation, with distinctive phenotypic and genetic sig

76 Recipient CD154+TcM induced by stimulation with donor cells were expressed as a fractio

77 nteractions with architectural proteins upon stimulation with ecdysone.

78 Exosomes, derived from HLSECs after stimulation with either type I or type III IFNs, control

79 tronic device that integrates electrotactile stimulation with electromyography, temperature, and stra

80 tronic device that integrates electrotactile stimulation with electromyography, temperature, and stra

81 Stimulation with ephrinA1, a ligand for EphA2, induced f

82 DWV, which perpetuates a loop of reciprocal stimulation with escalating negative effects on honey be

83 sed high levels of MIF and released MIF upon stimulation with Escherichia coli and group B Streptococ

84 producing MAIT cells were more frequent upon stimulation with Escherichia coli compared with healthy

85 ut astrocytes responded robustly to cortical stimulation with evoked Ca(2+)signals.

86 ard an M1 phenotype in response to secondary stimulation with EVs from P. brasiliensis.

87 duction, and responded vigorously to ensuing stimulation with exogenous and endogenous IL-12.

88 eated with JQ1/SGCBD01 and I-BET762 prior to stimulation with FCS and TGF-beta.

89 o lower back exertion, and electrical muscle stimulation with feedback control.

90 dividual showed loss of kinase activity upon stimulation with fibroblast growth factor.

91 larial-uninfected (Uninf) subjects following stimulation with filarial Ag (BmA) or with the M. tuberc

92 ing sleep and wakefulness, and during visual stimulation with fixation.

93 e in solution and responds to electric field stimulation with flexible movement.

94 nd neutrophil/eosinophil responsiveness upon stimulation with formyl-methionyl-leucyl phenylalanine w

95 in complementation and cyclase activity upon stimulation with forskolin and Galphas.

96 1-6 min and a second peak at 7-12 min after stimulation with forskolin.

97 Mechanistically, stimulation with FPR2 ligands resulted in down-regulatio

98 nished their ability to secrete insulin upon stimulation with glucose, which was reversed in the pres

99 ant forms of CSF2RB had reduced pSTAT5 after stimulation with granulocyte-macrophage colony-stimulati

100 ated basophils (CD63(+)/CCR3(+) cells; after stimulation with grass pollen) was substantially lower f

101 of oxidative stress or acidity condition the stimulation with Grisu(R) alone caused an improvement of

102 KIgammai2, impaired PI3K/Akt activation upon stimulation with growth factors or extracellular matrix

103 mary tubular epithelial cells after in vitro stimulation with H2O2 and in whole kidneys after unilate

104 cessive inflammatory response elicitation by stimulation with H2O2.

105 same extent as crypts from Cd44(+/+) mice on stimulation with HGF, but had the same response to EGF.

106 ivation is influenced by the strength of TCR stimulation, with high-strength TCR stimulation being as

107 oscillation became more entrained by visual stimulation with higher frequencies (>10 Hz).

108 ells from vaccinated subjects after in vitro stimulation with homologous (NF54) or heterologous (7G8)

109 as quantifiable for the first time 3 h after stimulation with hTNF-alpha and 12 h after stimulation w

110 stimuli, by FcepsilonRI cross-linking or by stimulation with hymenoptera venom allergens, were signi

111 addition, intracellular delivery of dsDNA or stimulation with IFN did elicit a rapid and pronounced r

112 Furthermore, stimulation with IFN-gamma led to enhanced STAT1 phospho

113 ction of SOCS1 and refractoriness to further stimulation with IFN-lambda3.

114 f either FcgammaRIV or FcgammaRIIB following stimulation with IFNgamma or IL-4, respectively.

115 ic vascular endothelial growth factor-C upon stimulation with IL-10.

116 Stimulation with IL-12 and IL-23 results in activation o

117 Upon stimulation with IL-13, IL-13Ralpha2 increased the extra

118 Stimulation with IL-1alpha/beta induced a pro-inflammato

119 eceptors, was assessed at baseline and after stimulation with IL-1beta, PGE2, and specific EP recepto

120 staining and confocal microscopy) after cell stimulation with IL-1beta.

121 nse to IL-15 and reduced proliferation after stimulation with IL-2 or IL-15, suggesting that STAT5 si

122 n rate of Grb2-deficient CD4(+) T cells upon stimulation with IL-2 or upon activation by allogeneic d

123 MC or polyclonal NK cells was potentiated by stimulation with IL-2.

124 elevant consequences of prolonged eosinophil stimulation with IL-3.

125 aT/+) CD8(+) T cells was studied in vitro by stimulation with IL-4 and IL-2 cytokines, and changes in

126 In addition, upon stimulation with IL-4 or IL-13, HR(-) ETPs expressing vi

127 acrophages did not release NE in response to stimulation with IL-4, and conditioned media from IL-4-s

128 d proliferative ability, further enhanced by stimulation with IL-7.

129 On stimulation with immobilized immune complexes, Ptpn22(-/

130 Additionally, in vitro stimulation with immune complexes resulted in significan

131 bserved that podocytes respond to mechanical stimulation with increased intracellular calcium concent

132 Stimulation with inflammatory cytokines and endotoxin im

133 However, in response to in vitro stimulation with influenza A/California/7/2009 (H1N1) Ag

134 vein vascular smooth muscle cells following stimulation with interleukin-1alpha and platelet-derived

135 influx in the mitochondria was observed upon stimulation with ionophores, nigericin, or ionomycin.

136 [Ca(2+) ]i was increased by beta-adrenergic stimulation with isoprenaline and increased in a saturat

137 We found that beta-AR stimulation with isoproterenol (0.1 muM) decreased the m

138 We observed that chronic beta-AR stimulation with isoproterenol (ISO) for 48 h reduced It

139 where it exerts anti-inflammatory actions on stimulation with its natural ligand alpha-melanocyte-sti

140 Stimulation with jellyfish-derived allergen showed expre

141 We found that mitogenic stimulation with keratinocyte growth factor (KGF) marked

142 e response modulation, as upon CD1d-mediated stimulation with KRN7000, a synthetic alpha-galactosylce

143 Here, we combine noninvasive brain stimulation with large-scale electrophysiological record

144 ut the HIV-1 reactivation kinetics following stimulation with latency-reversing agents.

145 Ovarian stimulation with letrozole has been proposed to reduce m

146 women with unexplained infertility, ovarian stimulation with letrozole resulted in a significantly l

147 In vitro stimulation with leukotriene D4 increased iNOS mRNA leve

148 rom these mice overproduced type 1 IFNs upon stimulation with ligands that activate TLR7 or TLR9.

149 After 1 h of stimulation with ligands, 87, 138, 1013, and 22 genes we

150 significantly reduced cell damage, allowing stimulation with light.

151 ith uric acid for 24 h and then subjected to stimulation with lipopolysaccharide (LPS) in the presenc

152 y Mefv(V726A/V726A) monocytes in response to stimulation with lipopolysaccharide also is dependent on

153 GM-CSF cultures both undergo maturation upon stimulation with lipopolysaccharide but respond differen

154 re studied ex vivo at baseline and following stimulation with lipopolysaccharide, oligodeoxynucleotid

155 -12, CXCL9, and CXCL10 induced by subsequent stimulation with lipopolysaccharide.

156 and an exaggerated inflammatory response on stimulation with lipopolysaccharide.

157 e of peripheral blood mononuclear cells upon stimulation with lipopolysaccharide.

158 onocyte tumor necrosis factor receptors upon stimulation with lipopolysaccharide.

159 r alpha (TNF-alpha) secretion in response to stimulation with lipopolysaccharides (LPS) and/or nigeri

160 TLR4 stimulation with lipopolysaccharides or live bacteria en

161 trolling IFN-gamma production in response to stimulation with live bacillus Calmette-Guerin (BCG; LOD

162 on 8q controlling IFN-gamma production after stimulation with live BCG (Bacillus Calmette-Guerin), an

163 Moreover, acute stimulation with LN accelerates axon outgrowth over a ti

164 After stimulation with LPS (Lipopolysaccharide), the monocytes

165 zed in macrophages and bronchial cells after stimulation with LPS and incubation with SET-M33.

166 e receptors with AG simultaneously with TLR4 stimulation with LPS increased the expression of the E3

167 are used equally in unpolarized macrophages, stimulation with LPS or IFN-gamma leads to increased tra

168 ly kinase Fyn in the production of TNF after stimulation with LPS was evaluated using bone marrow-der

169 g CCR7 and high levels of MHC II, even after stimulation with LPS.

170 ant metabolite produced by macrophages after stimulation with LPS.

171 om the surface of ovarian cancer cells after stimulation with lysophosphatidic acid.

172 uated in murine atherosclerotic aortas after stimulation with M-CSF or GM-CSF by using quantitative a

173 els, which are critical for coupling glucose stimulation with membrane depolarization.

174 Upon stimulation with membrane-associated antigen, CD23 KO ca

175 antitative PCR, and cytokine secretion after stimulation with mitogenic, TLR, and T-cell stimuli by c

176 continuous theta-burst transcranial magnetic stimulation with model-based functional MRI, we show tha

177 Although stimulation with most TLR agonists resulted in strong cy

178 ngly and selectively activated by peripheral stimulation with mouse faecal extracts.

179 In mDC:DO11.10 CD4 TC cocultures, stimulation with MPLA, but not with LPS, resulted in enh

180 of normal human lung fibroblasts (NHLFs) to stimulation with mtDNA and determined whether the glycol

181 Moreover, after stimulation with mycobacterial antigens plus Der p 1 all

182 We observed that stimulation with NOD1 or NOD2 ligand had no effect on th

183 Local beta-adrenergic receptor stimulation with noradrenaline (norepinephrine; NA, 50 m

184 Stimulation with NT (10 nM) increases the gene expressio

185 entral nucleus of amygdala (CeA) optogenetic stimulation with one option for earning intravenous coca

186 Mothers who received responsive stimulation (with or without enhanced nutrition) had sig

187 Children who received responsive stimulation (with or without enhanced nutrition) had sig

188 cells were measured after anti-CD3/anti-CD28 stimulation with or without autologous B cells.

189 y recruited to the plasma membrane after BCR stimulation with or without FcgammaRIIB coligation in hu

190 nted IL-6 production following ex vivo blood stimulation with other TLR agonists.

191 tokine-producing capacities were analyzed by stimulation with overlapping peptides spanning the large

192 , TLR4-, and TLR9-expressing HEK293 cells to stimulation with Pam3CSK4, polyinosinic-polycytidylic ac

193 However, we show that olfactory stimulation with particular odorants results in modulati

194 Stimulation with pathologically high levels of Vegf, or

195 In response to stimulation with PDGF, CRMP2 was dephosphorylated on Thr

196 of the percentage of CD63(+) basophils after stimulation with peanut and after stimulation with anti-

197 reased expression of granzyme B and TNF upon stimulation with peptide Ag ex vivo.

198 ines in macrophages and dendritic cells upon stimulation with peptidoglycan (PGN, the main cell wall

199 ry and intracellular cytokine staining after stimulation with phorbol myristate acetate and ionomycin

200 FNgamma(+)] and Th2 [CD4(+)IL4(+)] cells) to stimulation with phytohemagglutinin, leptin, and dust mi

201 NET formation after stimulation with platelet activating factor, ionomycin,

202 able of producing HIV mRNA and protein after stimulation with PMA/ionomycin and latency-reversing age

203 ) TIL and cytokine production in response to stimulation with polyclonal antigens or TAA.

204 nd TNF-alpha by CXCL4-moDCs exclusively upon stimulation with polyinosinic-polycytidylic acid, R848,

205 rst afferent stimulation or pairing afferent stimulation with postsynaptic depolarization.

206 Furthermore, RSR was enhanced by chemical stimulation with potassium chloride.

207 uce exaggerated chemokine responses to acute stimulation with pro-inflammatory cytokines.

208 se (PAK), translocates to the membrane after stimulation with protease-activated receptor agonists in

209 lcium imaging, we recorded responses to oral stimulation with prototypic taste stimuli.

210 Neutrophil stimulation with Pseudomonas aeruginosa, LPS, or PAM(3)C

211 In conclusion, 5-HT2A/1A receptor stimulation with psilocybin seems to reduce social pain

212 ne expression from healthy neutrophils after stimulation with purified albumin from SAH patient plasm

213 We previously reported that cholinergic stimulation with pyridostigmine (PY) induces anti-inflam

214 y lack the ability to combine different cell stimulations with rapid sample processing and precise fl

215 m peripheral blood of patients revealed that stimulation with receptor activator of nuclear factor ka

216 BdMphi also failed to make NO after stimulation with recombinant badger interferon gamma (bd

217 Quantification of IFN-gamma used whole-blood stimulation with recombinant HEV-capsid protein in the Q

218 thermore, in Vero cells infected with PIV-3, stimulation with recombinant IL-29/-28A/-28B does not ca

219 Upon stimulation with relevant antigen, naturally tolerant pa

220 r stimulation with hTNF-alpha and 12 h after stimulation with RMD, while p24 Gag protein was detected

221 Following mGluR stimulation with (S)-3,5-dihydroxyphenylglycine, the rat

222 FR network at multiple time points following stimulation with several ligands, enabling a quantitativ

223 e with the production of IL-4 in response to stimulation with several of these different antigens.

224 Cs respond to olfactory sensory neuron (OSN) stimulation with short latencies regardless of stimulati

225 o-dialysis probe to couple selective optical stimulation with simultaneous in vivo microdialysis, we

226 Subsequently, stimulation with SP induced upregulation of TLR expressi

227 their discharge rate in response to acoustic stimulation with species-specific calls.

228 blood T cells elaborated TH2 cytokines after stimulation with Spls, as is typical for allergens.

229 d cells before and up to 2 hours after their stimulation with stem cell factor, Fms-like tyrosine kin

230 Ps were immediately rescued by pairing mAChR stimulation with subthreshold depolarization ( approxima

231 After stimulation with such agents, TAK1 becomes rapidly and t

232 gs, mitral cells responded to high frequency stimulation with sustained responses, whereas external t

233 Mitral cells responded to high frequency ORN stimulation with sustained transmission, whereas externa

234 ot IL-6) production, respectively, following stimulation with synthetic TLR agonists or RNA-containin

235 reduced expression of ABCA1 as compared with stimulation with T0901317 alone, indicating that Seco A-

236 Upon in vitro stimulation with target cells, Zap70(low)Syk(low) NK cel

237 ls of IL-8, IL-6, CCL3, CCL4, and CCL2 after stimulation with TDM, whereas DC responded more strongly

238 Noninvasive brain stimulation with temporally patterned waveforms such as

239 Upon stimulation with Th1 cytokines or bacterial lipopolysacc

240 produced IFN-gamma and IL-2 but not IL-5 on stimulation with the aforementioned peptides.

241 (Ser-346/7) of CXCR4 are phosphorylated upon stimulation with the agonist CXCL12 as well as a CXCR4 p

242 sequencing at steady state and after in vivo stimulation with the alarmin cytokines IL-25 and IL-33.

243 Stimulation with the CXCR4 ligand SDF1alpha activated PI

244 ses to Pneumocystis species are dependent on stimulation with the cyst life cycle stage.

245 eceptor, can activate various effectors upon stimulation with the endogenous ligand angiotensin-II (A

246 males deposit the pheromone 9-tricosene upon stimulation with the food-odor apple cider vinegar.

247 After stimulation with the gammac-ligand IL-15, gammac-deficie

248 paB activation in these cells in response to stimulation with the GPCR agonist lysophosphatidic acid.

249 Stimulation with the inotropic and lusitropic agent isop

250 en granules (ZGs) in response to physiologic stimulation with the intestinal hormone cholecystokinin

251 Embryo cryopreservation after ovarian stimulation with the letrozole and follicle-stimulating

252 cytes failed to produce a Ca(2+) signal upon stimulation with the M. tuberculosis protein.

253 s evaluated in macrophage immune cells after stimulation with the microbial ligand lipopolysaccharide

254 Shedding of IL-23R was induced by stimulation with the phorbol ester phorbol 12-myristate

255 he current study analyzed effects of chronic stimulation with the physiological agonist diacylglycero

256 immunomodulatory properties before and after stimulation with the pro-inflammatory cytokine interfero

257 iferate and produce IFN-gamma in response to stimulation with the proteins of molecular weight >30 kD

258 hese areas were not activated during passive stimulation with the same sensory stimuli.

259 d IFN-beta and TNF-alpha induction following stimulation with the STING-dependent ligand 5,6-dimethyl

260 of TLR2 compared with healthy controls, and stimulation with the synthetic lipopeptide Pam3Cys, an a

261 nd nonreplicating virus as well as following stimulation with the TLR ligands Poly(I:C) and CpG.

262 rofibrotic myofibroblast phenotype driven by stimulation with the TLR9 agonist, CpG-DNA.

263 cell line to undergo degranulation upon the stimulation with their respective antigens.

264 onent of fungal cell walls, since intestinal stimulation with this moiety alone overrides disease sus

265 The expressed rADAMTS13 is released on stimulation with thrombin and collagen, but less with 2M

266 Endothelial stimulation with thrombin rapidly increased the endothel

267 P-selectin or alphaIIbbeta3 activation upon stimulation with thrombin, ADP-mediated alphaIIbbeta3 ac

268 CXCL13 was produced by monocytes after stimulation with TLR 7 and 8 ligands or HIV-1-derived ss

269 ed macrophages and dendritic cells following stimulation with TLR agonists in vitro.

270 ing infection with Listeria monocytogenes or stimulation with TLR ligands (Pam3CSK4, LPS, polyinosini

271 TNFalpha and IL-6 production upon secondary stimulation with TLR ligands, as well as bacterial and f

272 easured, as well as the response of PBMCs to stimulation with TLR ligands.

273 ld (>/=65 y) individuals were analyzed after stimulation with TLR4, TLR7/8, and retinoic acid-inducib

274 Stimulation with TLR7 ligand enhances formation of IRF5-

275 In contrast, stimulation with TLR7/8 ligand protected MDCs but not MD

276 demonstrated to be markedly decreased under stimulation with TNF for 24 and 48 h, while JNK inhibito

277 Stimulation with TNF-alpha or LPS increased the expressi

278 cant increases in IL-10 release by AEC after stimulation with TNF-alpha/IL-1beta (P = 0.024) or HDM (

279 reased tumor necrosis factor secretion after stimulation with Toll-like receptor ligands and M. tuber

280 suggest against the use of peripheral nerve stimulation with train of four alone for monitoring the

281 5) We suggest that peripheral nerve stimulation with train-of-four monitoring may be a usefu

282 the dose should be based on peripheral nerve stimulation with train-of-four monitoring.

283 arge motor axons, we find that physiological stimulation with trains of action potentials transiently

284 increased alongside SLC4A2 expression under stimulation with transforming growth factor beta1 (TGFbe

285 aluate the dendritic cell response following stimulation with trophic forms alone, with a normal mixt

286 We demonstrate that stimulation with trophic forms downregulated the express

287 In vitro stimulation with TSLP primed basophil migration to eotax

288 om P2X7R-deficient mice were unresponsive to stimulation with tumor cells, and chemotaxis of P2X7R-le

289 ed ubiquitin from TRAF6, NEMO and RIP1 after stimulation with tumor necrosis factor (TNF).

290 resses CD45RA (termed TEMRA) after antigenic stimulation with unknown molecular characteristics and f

291 th relevant antigen stimulation and not upon stimulation with unrelated antigen.

292 increases in peak Ca(2+) responses, whereas stimulation with urocortin1 that binds both receptors wi

293 recursors from the hair follicles, following stimulation with UV light.

294 metry after polyclonal and pathogen-specific stimulation (with varicella-zoster virus [VZV] and cytom

295 mRNA was degraded posttranscriptionally upon stimulation with various TLR ligands.

296 Similar YAP responses were provoked by stimulation with vasopressin or serum.

297 Direct stimulation with virus was sufficient to activate M2 gen

298 m CD4 T cells in acute infection, even after stimulation with virus-encoded TLR7/8 ligand.

299 Pharmacologic stimulation with Wnt agonists led to intracellular accum

300 The FZD4-G protein complex dissociates upon stimulation with WNT-3A, WNT-5A, WNT-7A, and WNT-10B.