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1 gth of these contextual influences vary with stimulus intensity.
2 eurons were sensitive to changes in auditory stimulus intensity.
3 ies in a manner similar to the modulation of stimulus intensity.
4  and implicit time decreased with increasing stimulus intensity.
5 of alpha-synuclein is graded with respect to stimulus intensity.
6 of the b-wave were examined as a function of stimulus intensity.
7 cape from heat were appropriately related to stimulus intensity.
8 came more strongly nonlinear with increasing stimulus intensity.
9 nly between 8.3 and 35 ms-less with brighter stimulus intensity.
10 raded forces in a manner capable of encoding stimulus intensity.
11 g dynamic range that were independent of the stimulus intensity.
12 on the basis of their response to increasing stimulus intensity.
13 on could be graded in amplitude according to stimulus intensity.
14  apparent 'failures' at fixed suprathreshold stimulus intensity.
15 duration (10-50 s) increased with increasing stimulus intensity.
16 ition was systematically modified by varying stimulus intensity.
17 erage by up to 23%, depending on channel and stimulus intensity.
18 over, learning was facilitated by increasing stimulus intensity.
19 d as a function of increasing attenuation of stimulus intensity.
20 ich showed a steep sigmoidal relationship to stimulus intensity.
21 lity of perceived stimulus across a range of stimulus intensity.
22 ding model, whereas posterior insula encoded stimulus intensity.
23 .05) the area of the AP evoked at submaximal stimulus intensity.
24 rical signals whose amplitude is graded with stimulus intensity.
25 response, and that it is graded according to stimulus intensity.
26 5) the area of the AP evoked at a submaximal stimulus intensity.
27 ion of the trend of increasing or decreasing stimulus intensity.
28 gnitude and duration of decreases in noxious stimulus intensity.
29 h spikes, but their phase does not vary with stimulus intensity.
30 ad and increased in proportion to increasing stimulus intensity.
31 on appears to exist only in neurons tuned to stimulus intensity.
32 ave amplitude was evaluated as a function of stimulus intensity.
33  were related to the ambient light level and stimulus intensity.
34 of how field L response properties depend on stimulus intensity.
35 neural coding depends on the distribution of stimulus intensities.
36 cruiting more presynaptic fibers with higher stimulus intensities.
37 erence, but are exquisitely sensitive to low stimulus intensities.
38 e of protective inhibition at relatively low stimulus intensities.
39 onses to prolonged light, especially at high stimulus intensities.
40 nel subtypes that together detect a range of stimulus intensities.
41 gh (90% of maximum) and low (30% of maximum) stimulus intensities.
42 iable and remained distinct across different stimulus intensities.
43 or exhibit less tolerance than men for given stimulus intensities.
44 allows neurons to respond to a wide range of stimulus intensities.
45 from those in wild-type mice at low scotopic stimulus intensities.
46 ologically relevant frequencies, and at high stimulus intensities.
47 n is an all-or-nothing event over a range of stimulus intensities.
48 at constant phase throughout a wide range of stimulus intensities.
49 e (i.e., decreases in selectivity) at higher stimulus intensities.
50 ially for series of ascending and descending stimulus intensities.
51 d and dark-adapted conditions with different stimulus intensities.
52 he cortex and hippocampus was tested at four stimulus intensities: 0.0, 0.25, 0.5, and 1.0 mA.
53 carotid sinus and by electrical stimulation (stimulus intensity: 0.5 V, frequencies 5, 10, and 25 pul
54 ignaling because they occur at low levels of stimulus intensity [3].
55 s differentiators that transiently normalize stimulus intensity-a property potentially derived from a
56 a-threshold acceleration levels, we used two stimulus intensities, acceleration steps of 0.5 degrees
57                            At higher mesopic stimulus intensities activating both RBCs and cone bipol
58                                 Changing the stimulus intensity altered the amplitude but not the tim
59     Each neuron was tested across a range of stimulus intensities and multisensory responses evaluate
60  repertoire, albeit inducible only at higher stimulus intensities and with briefer expression.
61 he first AP was advanced >5 ms by increasing stimulus intensity and across multiple spikes during bur
62 s between ascending and descending trends in stimulus intensity and alters the magnitude of pain sens
63 sis showed that the NS neurones encoded both stimulus intensity and area (probe size) significantly b
64 s paper we describe the relationship between stimulus intensity and cochlear nerve discharge rate (th
65 e of TES phase-locked neurons increased with stimulus intensity and depended on the behavioral state
66 ntly modulated to reflect ongoing changes in stimulus intensity and dynamics that occur on a millisec
67 n bursts to the perforant path at a moderate stimulus intensity and I/O functions were reassessed 1 h
68 ly perceived as painful without changing the stimulus intensity and location.
69                              Irrespective of stimulus intensity and membrane potential, 78% (45/58) o
70  graded information covering a wide range of stimulus intensity and must be able to sustain this sign
71 fferent qualities and are also responsive to stimulus intensity and often to touch and temperature.
72 hat there is an inverse relationship between stimulus intensity and relative SERCA activity.
73 factors that can be considered (e.g., adding stimulus intensity and representational quality).
74  evolution of N1 amplitudes, increasing with stimulus intensity and showing largely significant diffe
75 sponses habituated more slowly at the higher stimulus intensity and slower repetition rate compared w
76 -discharge was significantly related to both stimulus intensity and stimulus area.
77 e normal functional dependency of the ERG on stimulus intensity and the normal response kinetics sugg
78                    Interaural differences in stimulus intensity and timing are major cues for sound l
79 is concept, the input-output relationship of stimulus intensity and TS-eEPSC amplitude shows an early
80 provement in muscle strength correlated with stimulus intensity and was identified in the absence of
81 ensory events; sensory responses scaled with stimulus intensity and were abolished by anesthesia.
82 ol, which in turn would equilibrate with the stimulus intensity (and therefore the number of open Ca(
83 n be compensated by extra attention or extra stimulus intensity) and that (b) a first threshold of th
84  and rise time, graded response to increased stimulus intensity, and no failures, suggesting a monosy
85 ith manipulations of interstimulus interval, stimulus intensity, and prepulse inhibition.
86 with presynaptic calcium influx, graded with stimulus intensity, and stable over a period of days.
87 g of action potentials typically varies with stimulus intensity, and the invariance of temporal repre
88 onal activity, and its effect interacts with stimulus intensity; and (4) noise generation--TMS adds r
89 eceptor neuron (ORN) output decreased; thus, stimulus intensity appeared to determine oscillation fre
90 tion in late paired-pulse depression at high stimulus intensities are unaffected by either NMDA or ki
91 he marginal shell provides information about stimulus intensity as a part of a reflex (or feedback ga
92  precipitous firing increase with increasing stimulus intensity, as compared to non-kindled GEPR-9s.
93 ters used clinically, i.e. 50 Hz, 0.2 ms and stimulus intensities at 30, 60 or 90% of MT.
94 b-wave responses (lambdamax = 506 nm) to all stimulus intensities at night but only to those intensit
95 Bullfrogs were more aggressive at the higher stimulus intensity at both repetition rates.
96 s have systematically mapped changes between stimulus intensity, attentional focus, neural activity,
97 esponses to light flashes at three different stimulus intensities before and after administration of
98 R-mediated EPSCs that were not evoked by low stimulus intensity before perfusion.
99    New stimuli were created in which overall stimulus intensity between short and long rise times was
100                        Thus, with increasing stimulus intensities, both the total number of SPR+ lami
101 y, similar to the effect seen with decreased stimulus intensity, but significantly different from the
102 n primary sensory circuits is matched to the stimulus intensity by the process of adaptation.
103                                              Stimulus intensities capable of disrupting the performan
104 e possible confound of rise time and overall stimulus intensity change (tones with shorter rise times
105    However, at high scotopic and low mesopic stimulus intensities, close to RBC saturation, a signifi
106 ustom system allowing full user control over stimulus intensity, color, and duration.
107        SICI was tested at seven conditioning stimulus intensities (CSIs; 40-100% of resting motor thr
108 atment context) with pain modulation through stimulus intensity cues (stimulus context) during functi
109 mulus-driven processing of expectations with stimulus intensity cues.
110  of the fly nonlinearly alters its tuning as stimulus intensity declines and oscillates spontaneously
111                     We found that increasing stimulus intensity decreased SLC latencies while increas
112 pecific, painful stimulus while increases in stimulus intensity do not produce increased activation.
113 5%) of the 471 patients required the maximum stimulus intensity during their index ECT course.
114                                      At high stimulus intensities, EPSPs caused by CSS and ISS became
115 mma nor high-gamma oscillations changed with stimulus intensity, even with a 10-fold increase.
116                  A 2- to 10-fold increase in stimulus intensity evoked a slow excitatory postsynaptic
117                                       At low stimulus intensities, firing rates are increased in Kv4.
118 vious finding in which individual TMS SI1mV (stimulus intensity for 1 mV MEP amplitude) sensitivity c
119 ized by the peak response are independent of stimulus intensity for a large portion of the dynamic ra
120 ally be confused with changes resulting from stimulus intensity, for example, the loudness of the utt
121 h circuitry allows the cortex to distinguish stimulus intensity from stimulus form.
122   MK-801 administration had no effect on the stimulus intensity function for early paired-pulse depre
123                                 Paired-pulse stimulus intensity functions were obtained from animals
124  deficit (difference in decibels between the stimulus intensity giving the patient's pupil response a
125                                              Stimulus intensity has no significant effect on DRRs.
126 loss of late paired-pulse depression at high stimulus intensities in naive and kindled rats.
127       PPR was sensitive to the extracellular stimulus intensity in all conditions.
128 0 microA) or high (approximately 500 microA) stimulus intensity in anaesthetized, vagotomized, neurom
129 the neural code underlying the perception of stimulus intensity in the somatosensory system.
130 ameter), the pupil response as a function of stimulus intensity in the treated, miotic eye did not di
131                       Sensory neurons encode stimulus intensity in their instantaneous spike rate and
132 ulus-evoked responses over the full range of stimulus intensities, including total darkness.
133                                   Increasing stimulus intensity increased the probability of evoking
134 ger than CSS-EPSPs and became shorter as the stimulus intensity increased while those of CSS-EPSPs re
135 ponses of the 39 units increased linearly as stimulus intensity increased, and the population stimulu
136 uffering glutamate-induced Ca2+ loads as the stimulus intensity increases.
137 essential for maintaining ITD selectivity as stimulus intensity increases.
138  a multivariate pattern signature-termed the stimulus intensity independent pain signature-1 (SIIPS1)
139 show that amygdala activity may be driven by stimulus intensity irrespective of valence, casting doub
140                             We conclude that stimulus intensity is best accounted for by the firing r
141 granule cell field potentials decreases when stimulus intensity is increased from moderate to high le
142 tion is proportional to the logarithm of the stimulus intensity is observed between the PL emission r
143  promoter is sufficiently strong, unless the stimulus intensity is very high.
144 in Kv4.2DN-expressing cells, whereas at high stimulus intensities, Kv4.2DN-expressing cells adapt str
145                                       At low stimulus intensities, licking resumed immediately on sti
146 andomized to ECT conditions that differed in stimulus intensity (low vs high dosage) and electrode pl
147           Symptoms increased with distending stimulus intensity (maximum pain, 2.1 +/- 0.4; nausea, 2
148 gating information about different ranges of stimulus intensity may be an important function of these
149                 Over a 4.0 log unit range of stimulus intensities measurements of A and V were seen t
150                                      At high stimulus intensities, neurons were sensitive to differen
151 conditioned stimulus modality, unconditioned stimulus intensity, number of training trials), conditio
152 electrical pulses to the hindpaw with varied stimulus intensity, number, and interstimulus interval.
153 ranial direct current stimulation (tDCS) use stimulus intensities of 2 mA despite the fact that blind
154  recordings were performed using four 470 nm stimulus intensities of 3, 30, 100 and 300 cd/m2.
155 icipant's visual sensitivity to recover to a stimulus intensity of 5x10(-3) cd/m(2) (a decrease of 3
156 r the primary motor cortex (M1) at different stimulus intensities on finger sequences of varying comp
157  The effects of stimulus repetition rate and stimulus intensity on bullfrog aggressive responses were
158 collectively encode bidirectional changes in stimulus intensity on multiple timescales, and how this
159 suggest that cortical regions either reflect stimulus intensity or additive effects of intensity and
160 on does not necessarily correspond to either stimulus intensity or cellular response.
161 onse patterns are not observed even when the stimulus intensity or identities were changed.
162 cular stimuli beyond the predictive value of stimulus intensity or self-reports of emotion.
163 m adjusts receptor sensitivity to background stimulus intensity over several orders of magnitude of a
164 thalamic EPSP had a graded relationship with stimulus intensity, owing to its slower-rising (2.9 +/-
165 ups in either protocol at any time or at any stimulus intensity (P > 0.05).
166 ociceptive reflex withdrawal are graded with stimulus intensity (p < 0.001), significantly correlated
167 d in spontaneous seizure-like behavior, high stimulus intensity population spikes in the absence of l
168 vations produced by the high and low thermal stimulus intensities presented with the high-intensity v
169                                Low levels of stimulus intensity released sufficient levels of leukotr
170 hanism for membrane internalisation at basal stimulus intensity remains largely unexplored.
171                                We determined stimulus intensity-response curves for anterior commissu
172                                              Stimulus intensity-response curves were shifted to the l
173  potentials more frequently and, at moderate stimulus intensities, showed irregular or stuttering fir
174 e of contrast is independent of the range of stimulus intensities signaled by the cells.
175       This relationship is unaffected by the stimulus intensity, size of the pupil, or age of the sub
176 ape of ITD tuning often depended strongly on stimulus intensity; some neurons had dynamic ranges of I
177 Weber's Law) used by sensory neurons to code stimulus intensity, suggesting how abstract cognitive op
178 threshold and steeper gradient while varying stimulus intensity, suggesting insufficiency of the homo
179 rea of the brain responds only to changes in stimulus intensity, suggesting that we directly detect o
180 in pain ratings evoked by small decreases in stimulus intensity -- suggests that dynamic activation o
181 be observed in the EMG activity, at stronger stimulus intensities than were required for resetting th
182 evoked by stimulation of cutaneous nerves at stimulus intensities that activated large mechanorecepti
183 d ascending and descending series of thermal stimulus intensities that maintained an average rating (
184       Physiological adaptation modulates the stimulus intensities that trigger such nocifensive behav
185 DeltaF/F(0)) demonstrated that at a constant stimulus intensity there was no change in the excitabili
186  of EPSPs were observed in DG cells, but the stimulus intensity threshold for EPSPs slightly increase
187 y decreased 0-3 h after reperfusion, and the stimulus intensity threshold for EPSPs transiently incre
188 eta-burst patterned stimulation at a maximal stimulus intensity through the perforant path electrode,
189 sed input/output (I/O) curves (EPSP slope vs stimulus intensity) to determine whether the sensitivity
190  reciprocally with hair cell depolarization (stimulus intensity) to produce constant synaptic phase.
191 ensory regions nor to rescaling of monotonic stimulus intensity tuning curves, but may rather represe
192 paired-pulse depression was observed at high stimulus intensities under all experimental conditions.
193 ower function with increasing attenuation of stimulus intensity up to the threshold.
194 ld light stimuli with a stepwise increase in stimulus intensity using a binocular infrared computeriz
195                                              Stimulus intensity, UVD, and starting vertical gaze dire
196                                   Decreasing stimulus intensity (velocity-acceleration) reduced the a
197 2+/high Mg2+ solution, but only when the low stimulus intensity was applied.
198 amount of pupil contraction as a function of stimulus intensity was compared between the brimonidine-
199                                              Stimulus intensity was correlated with the amount of evo
200                Response latency decreased as stimulus intensity was increased.
201  However, this difference was reduced as the stimulus intensity was increased.
202  were evoked from 59% of daPC neurons as the stimulus intensity was raised above a precise threshold.
203 bserved increase of behavioral response with stimulus intensity was the result of an increase of the
204 ons, reliably encoded f(0) changes even when stimulus intensity was varied randomly over a 20 dB rang
205 cit active Ca2+ release, even at the highest stimulus intensities we employed, although these same ce
206  motoneuron EPSP onset latencies varied with stimulus intensity, we proposed that the pathway from th
207  neurons as a function of cortical layer and stimulus intensity, we recorded intracellularly in vivo
208 n in EMG response over this time period when stimulus intensities were within the range of 1.2-1.5 ti
209       Synaptic potentials evoked by the same stimulus intensity were compared in Up/Down states.
210                               The effects of stimulus intensity were mediated by the neurologic pain
211 and the leech: summed spike counts represent stimulus intensity, whereas relative timing of first spi
212 hanoreceptor cells respond to a vast span of stimulus intensities, which they transduce into a limite
213  pupillary responses at multiple, controlled stimulus intensities while using varied stimulus pattern
214 namics of block allow the population to code stimulus intensity with flexibility and efficiency.
215 ease (caused by Cd(2+), baclofen, or reduced stimulus intensity) with whole-cell voltage clamp in CA1
216 y serves a primarily nonoverlapping range of stimulus intensities, with ganglion cells receiving eith
217 RFs expanded monotonically with increases in stimulus intensity, with some occupying essentially all
218  trains of action potentials (spikes) encode stimulus intensity within the onset time of the first ev

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