ライフサイエンスコーパス: stomach

1 ers (breast, lung, prostate, colorectal, and stomach).

2 IS expression (thyroid, salivary glands, and stomach).

3 of the brain, skin, prostate, pancreas, and stomach.

4 m cell function in adult small intestine and stomach.

5 for modulating vagal output activity to the stomach.

6 ng tubes to infuse alcohol directly into the stomach.

7 patients, 4 had identical Pa strains in the stomach.

8 ected NIS-mediated uptake in the thyroid and stomach.

9 likely related to the lower pH in the distal stomach.

10 f this oncobacterium to the acidic pH of the stomach.

11 pylori is a successful pathogen of the human stomach.

12 l surface and within the glands of the human stomach.

13 C model caused tumorigenesis of the proximal stomach.

14 inder terminate into the sling fibers of the stomach.

15 ight reverse preneoplastic metaplasia in the stomach.

16 e development of premalignant lesions in the stomach.

17 oids the liberation of protons from cells to stomach.

18 harshly acidic environment of the mammalian stomach.

19 he pancreas compared with liver, spleen, and stomach.

20 lumes of foam, liquid, and air layers in the stomach.

21 ract the acidic environment of the mammalian stomach.

22 tant for Helicobacter pylori to colonize the stomach.

23 ells) adenocarcinoma in the angularis of the stomach.

24 which also depends on residence time in the stomach.

25 vagal efferent motoneurones innervating the stomach.

26 of premalignant and malignant lesions in the stomach.

27 to study the fundus epithelium of the human stomach.

28 rergic relaxation in the proximal and distal stomach.

29 l barrier for using therapeutic drugs in the stomach.

30 and continue as sling/oblique muscle on the stomach.

31 specially under the acidic conditions of the stomach.

32 ion-related side effects in the absence of a stomach.

33 ical models of food degradation in the human stomach.

34 in aggregates under acidic conditions of the stomach.

35 ted through TCTP may contribute to IM in the stomach.

36 l for pathogenesis of the bacterium in human stomach.

37 were the thyroid (0.135 +/- 0.079 mSv/MBq), stomach (0.069 +/- 0.022 mSv/MBq), and salivary glands (

38 the kidneys, ranging from a maximum for the stomach (0.52 +/- 0.04 %ID/g) to almost negligible for t

39 FSPG SUVmean, 8.2; (18)F FDG SUVmean, 1.3), stomach ((18)F FSPG SUVmax, 3.6; (18)F FDG SUVmax, 1.6),

40 t a residual uptake in the kidney, lung, and stomach (9, 16, and 10 %IA/g, respectively) was also obs

41 lation of gastric secretions in the proximal stomach above the meal adjacent to the esophagogastric j

42 availability establishes a low percentage of stomach absorption capacity.

43 GAS was also able to survive stomach acid and decomposed in intestinal linings or aft

44 Half of hypermutated stomach adenocarcinomas are associated with NMD-elicit m

45 (FV/+) mice develop carcinoma in the thymus, stomach, adrenal medulla, and mammary gland but not in o

46 secretions that form a layer in the proximal stomach after ingestion of a liquid meal.

47 evaluated against two cancer cell lines from stomach (AGS, MKN-28) and one colon cancer cell (Caco-2)

48 The stomach, an organ derived from foregut endoderm, secrete

49 eptide synthesized by endocrine cells of the stomach and a key component of the gut-brain axis, is in

50 It is primarily secreted by the stomach and acts at its receptor, the growth hormone sec

51 oper placement of the tip of the tube in the stomach and all side ports inside it: [nose-to-ear-to-xi

52 Helicobacter pylori infects the human stomach and causes a spectrum of disease that includes g

53 GB) involves exclusion of major parts of the stomach and changes in admixture of gastro-pancreatic en

54 ration increased the proportion with a clear stomach and decreased the acidity of residual gastric li

55 volving the pancreas, but originating in the stomach and duodenum, acquired insertions with a similar

56 t hormones were significantly reduced in the stomach and duodenum, compared to lean, and returned to

57 (for example, RAD51C, PALB2 and MSH6 in AML, stomach and endometrial cancers, respectively).

58 ll as the United Kingdom's BE gene study and stomach and esophageal cancer study.

59 post surgery, mice were euthanized, and the stomach and esophagus were removed, fixed, and stained f

60 digestion requires fat processing within the stomach and fat sensing in the intestine.

61 tients with metastatic adenocarcinoma of the stomach and gastroesophageal junction.

62 SDH-deficient tumors occurred only in the stomach and had an indolent course.

63 e its niche for chronic infection inside the stomach and how its virulence factors interact with the

64 nsity of the static fluid present within the stomach and in the duodenal lumen is also described.

65 nated algae, microplastics were found in the stomach and in the gut.

66 Helicobacter pylori colonizes the human stomach and increases the risk for peptic ulcer disease

67 lity was determined in vitro under simulated stomach and intestinal digestion conditions.

68 In vitro release studies, through stomach and intestinal simulation, showed a sustained re

69 65 neurons) and mechanical distension of the stomach and intestine (GLP1R neurons).

70 nt major glycosidase under the conditions of stomach and intestine and degrade chitin substrates to p

71 nctions to compare and contrast Notch in the stomach and intestine.

72 ssively survive the harsh acid stress of the stomach and multiply into a mild acidic compartment with

73 lloproteinase-9 (MMP-9) was performed on the stomach and rectosigmoid areas.

74 pe 1a (GHS-R1a), is mainly secreted from the stomach and regulates food intake and energy homeostasis

75 issue expressed markers of 4 lineages of the stomach and self-organized into gland and pit domains.

76 em, simulating digestion in the upper tract (stomach and small intestine) of healthy adult humans.

77 s, had antioxidant capacities similar to the stomach and small intestine, containing parent compounds

78 lysis of 349 AYA patients with tumors of the stomach and small intestine, small-intestine location wa

79 ibacter and Streptococcus) were found in the stomach and small intestine, while anaerobic Lachnospira

80 imulates human digestion processes in mouth, stomach and small intestine.

81 monstrate that bitter taste receptors in the stomach and the oral cavity are involved in the regulati

82 were identified in myenteric ganglia of the stomach and varicose simple-type endings in the circular

83 Ha would be digested completely in the human stomach and would pose no risk in human food consumption

84 died primarily using methods that bypass the stomach and, therefore, fail to replicate the natural co

85 s postinjection, animals were euthanized and stomachs and small intestines were processed as whole mo

86 nerve ring, digestive system (e.g., cardiac stomach) and body wall-associated muscles (e.g., apical

87 neurons in the medial DMV projecting to the stomach, and (b) EGFP+ neurons in the lateral DMV projec

88 h they focused on sensations in their heart, stomach, and bladder.

89 ower from outside the body to the esophagus, stomach, and colon, respectively.

90 e the body endoscopically, at the esophagus, stomach, and colon.

91 matosis is the presence of air in esophagus, stomach, and duodenum simultaneously, which have never b

92 es the occurrence of colorectal, esophageal, stomach, and gastrointestinal cancers.

93 ontrast, in epithelial cells from the colon, stomach, and kidney, KCNE3 coassembles with KCNQ1 to for

94 In the colon, stomach, and kidney, KCNE3 coassembles with the alpha-su

95 uencies of vomiting, abdominal pain, swollen stomach, and loss of appetite, compared with people infe

96 ctive pulmonary disease, and cancers (liver, stomach, and lung), contributed much more to YLLs in 201

97 inal afferent endings were identified in the stomach, and most of them were CGRP immunoreactive.

98 bserved for lung, upper aerodigestive tract, stomach, and prostate cancer mortality [men and women co

99 In NIS KO mice, in the thyroid, stomach, and salivary gland, NIS is absent, and hence th

100 of the UGI system, including the esophagus, stomach, and small intestine.

101 et these specifications: caries lesions, the stomach, and the vagina.

102 e mechanisms that drive the curvature of the stomach are intrinsic to the gut tube itself.

103 ficient adsorption of mycotoxins in animals' stomachs are also carried out.

104 to hinder the delivery of FA at low pH (i.e. stomach) as well as able to deliver great amounts of the

105 phage polarization within H. pylori-infected stomachs, as assessed by Luminex technology and immunohi

106 n the proximal stomach to those in the total stomach) at 0, 1, 2, 3, and 4 h after ingestion of a mea

107 The stomach bacterium Helicobacter pylori is one of the most

108 irect signs involving the organs forming the stomach bed, like the spleen, pancreas and kidney.

109 approach to identify the microbiome from 27 stomach biopsies, and validated the presence of Helicoba

110 d using metatranscriptomic RNA sequencing of stomach biopsy specimens from individuals with different

111 and elastic in the acidic environment of the stomach but can be dissolved in the neutral-pH environme

112 ctly, either ending in the esophagus, in the stomach but too close to the esophagus, or too far into

113 EVS evoked relaxation of wild type stomachs, but the predominant response of W/W(V) stomach

114 Colonization of the human stomach by Helicobacter pylori and its role in causing g

115 o dissect the viable microbiota of the human stomach by metatranscriptomic analysis, and it shows tha

116 aining high levels of H. pylori loads in the stomach by modulating effector T cell responses at the g

117 5-year survival for stomach cancer in 2005-09 was high (54-58%) in Japan and

118 , the underlying mechanism of lncRNA GAS5 in stomach cancer is poorly understood.

119 Stomach cancer is the second most common cause of cancer

120 g into the 5'-UTR of four proto-oncogenes in stomach cancer sequencing data.

121 und that lncRNA GAS5 had lower expression in stomach cancer tissues than the normal counterparts.

122 ies for specific delivery of therapeutics to stomach cancer without damaging normal cells and tissues

123 produce major regressions of pancreatic and stomach cancer xenografts.

124 ratios were 5.76 (95% CI, 2.12 to 15.6) for stomach cancer, 3.61 (95% CI, 1.33 to 9.79) for kidney c

125 er), with a notably high frequency of 11% in stomach cancer.

126 n potentially be used for early diagnosis of stomach cancer.

127 ts for developing lncRNA-based therapies for stomach cancer.

128 lication for non-invasive early diagnosis of stomach cancer.

129 prevalent in survivorship of esophageal and stomach cancer.

130 health, and is associated with incidence of stomach cancer.

131 adder, esophagus, colon, lung, pancreas, and stomach cancers.

132 ovel mechanism of lncRNA GAS5 in suppressing stomach carcinogenesis, and the lncRNA GAS5/YBX1/p21 pat

133 in liver carcinoma, gastric lipase (LIPF) in stomach carcinoma, and thyroglobulin (TG) in thyroid car

134 Reprogramming of antral stomach cells assembled into bioengineered mini-organs i

135 ression at a variety of body sites including stomach, colon, liver, lung, and skin.

136 We looked at cancers of the stomach, colon, rectum, liver, lung, breast (women), cer

137 ivered to the heart, spleen, liver, kidneys, stomach, colon, small intestine, and pancreas, respectiv

138 ity; nasopharynx; other pharynx; esophageal; stomach; colon and rectum; liver; gallbladder and biliar

139 mutants are outcompeted by wild type during stomach colonisation, but no ligands had been mapped to

140 ow Notch regulates stem cell function in the stomach compared to intestine.

141 icant bacteria burden reduction in the mouse stomach compared with passive drug carriers, with no app

142 rotein hydrolysis was observed in the distal stomach (compared to the proximal stomach), likely relat

143 antial and significant correlations with the stomach-complaint scale of the GBB-24 (r = .71; p < .01)

144 The H. pylori population inside the stomach contains a subgroup of bacteria that are attache

145 o generalized trophic guilds using published stomach content analyses and quantified shape variation

146 Stomach content analysis (SCA) showed that small juvenil

147 Stomach content analysis confirmed predation on cnidaria

148 mice exhibited little circadian variation in stomach content or GVA mechanosensitivity.

149 rvals starting at zeitgeber time 0 (ZT0) and stomach content was measured.

150 SLD mice exhibited circadian fluctuation in stomach content, which peaked at ZT18 and reached a nadi

151 Over 4 h, stomach contents (foam, air, and liquid) were imaged usi

152 the only fish species in the study streams; stomach contents from a fish, highlighting the major rol

153 opes (delta(34)S) in yellow perch muscle and stomach contents showed that larger fish tended to feed

154 taxonomic resolution of prey collected from stomach-contents.

155 at significantly greater inflammation in the stomach, crypt distortion in the colon, and eosinophilia

156 The primary outcome was a clear stomach, defined as less than 40 mL of liquids and no so

157 The stomach-derived hormone ghrelin stimulates appetite thro

158 Ghrelin is a stomach-derived hormone that plays a key role in whole-b

159 eview highlights the molecular mechanisms of stomach development and discusses recent findings regard

160 Therefore, a better understanding of stomach development and stem cells can inform approaches

161 oduction, and eosinophil infiltration in the stomach-draining lymph nodes.

162 vitro digestion model, which includes mouth, stomach, duodenum, and colon phases.

163 Eosinophil densities were obtained from the stomach, duodenum, and rectosigmoid areas.

164 in the different compartments of the TIM-1 (stomach, duodenum, jejunum and ileum) and viscosity was

165 collected in each compartment of the TIM-1 (stomach, duodenum, jejunum and ileum) at different times

166 strointestinal tract examined, including the stomach, duodenum, jejunum, ileum, cecum, appendix, colo

167 The constant regeneration of stomach epithelium is driven by long-lived stem cells, b

168 In addition, the stomachs exhibit severe mucosal and muscular hypertrophy

169 The left wall of the primitive stomach expands more than the right wall, as the left ep

170 C digestion and remained active in the mouse stomach extract at pH 2.0.

171 ations of satiation on the varying degree of stomach filling during gastric emptying was compared bet

172 These conditions in the green stomach formed by the closed flytrap allow DmKT1 and DmH

173 Stomach fullness is a determinant of satiety.

174 g noticeable acute toxicity or affecting the stomach function, and the normal stomach pH is restored

175 adenocarcinoma and proximal polyposis of the stomach (GAPPS) is an autosomal-dominant cancer-predispo

176 ity occurs by derepression of characteristic stomach genes, some of which are also associated with pa

177 distinct bacterial microcolonies deep in the stomach glands and interact directly with gastric progen

178 ce factor CagA into the epithelium colonized stomach glands in mice, but did not activate stem cells

179 ns in the loads of ingested plastic in their stomachs, grouped as "no", "medium" (0.01-0.21 g; 1-14 p

180 The stomach has a significant capacity to distend (up to 2-4

181 and to include the complex interactions the stomach has with other organ systems.

182 Here, we show that cells of the antral stomach have a previously unappreciated propensity for c

183 Ghrelin is a stomach hormone normally associated with feeding behavio

184 Here we show that gastrin, a stomach hormone typically expressed in the pancreas only

185 of inflammation and malignant lesions in the stomach; however, preactivation of NOD1 before bacterial

186 ve biopsy in end organs, such as the lung or stomach; (iii) symptomatic cases with rising polymerase

187 DNA (vaccine type) was found in the resected stomach; immediate early (ORF63p) and late (gE) VZV prot

188 r it was linked with cancer of the liver and stomach in rodents.

189 enriched for genera from the oral cavity and stomach, including Fusobacterium, Megasphaera, Campyloba

190 ction was driven by group differences during stomach interoception (p=0.002, Bonferroni corrected), w

191 insula, and activation in this region during stomach interoception was correlated with measures of an

192 ied with an in vitro digestion model (mouth, stomach, intestine, but not colonic digestion).

193 egulation of expression observed pre-term in stomach, intestine, kidney and heart.

194 Biodistribution to lungs, stomach-intestine, liver, trachea-throat and blood at th

195 IMD(0) based on the division of the stomach into proximal and distal halves averaged 0.809 (

196 e liver, pancreas, gallbladder, endometrium, stomach, kidney, brain (benign), brain (malignant), colo

197 ], and one drink that was less stable in the stomach [less-stable foam (LSF)], and a nonaerated drink

198 plasma membrane, hyperacidifying the "green stomach"-like digestive organ, whereas subsequent ones c

199 Here we show that the stomach-like copper cell region (CCR) in the middle midg

200 the distal stomach (compared to the proximal stomach), likely related to the lower pH in the distal s

201 d dorsal pancreatic fates into intestinal or stomach lineages, respectively.

202 bumin in liver parenchyma, and lipase in the stomach lining.

203 adult primary tissues from small intestine, stomach, liver and pancreas into self-assembling 3D stru

204 absorbed dose were the gallbladder, spleen, stomach, liver, kidneys, and bladder.

205 velling through the harsh environment of the stomach lumen, H. pylori colonizes the mucosal surface a

206 hat, under conditions mimicking those of the stomach lumen, the most favoured reaction in tyramine is

207 der conditions similar to those in the human stomach lumen.

208 protrusion of the remaining tissue into the stomach lumen.

209 mucosa to avoid the microbicidal acid in the stomach lumen.

210 suggested increased risks of cancers of the stomach, lung, kidney, brain, and meninges; however, the

211 rt failure, or carcinomas of the colorectum, stomach, lung, prostate, or breast.

212 inal tract, the presence of fluid within the stomach may overlap with the pancreatic duct system and

213 alf the radiolabelled meal to empty from the stomach), measured at 5 weeks and 16 weeks in all patien

214 ations were performed at acupoints of either Stomach Meridian of Foot-Yangming (SMFY) or Gallbladder

215 e intestine meridian points LI4 and LI11 and stomach meridian points ST36 and ST44 were used.

216 tric organoids co-cultured with immortalized stomach mesenchymal cells (ISMCs).

217 s maintained in co-culture with immortalized stomach mesenchymal cells express robust numbers of surf

218 acteria respond in vivo to variations in the stomach microenvironment and at different stages of dise

219 cies of bacteria that can colonize the human stomach mucosa.

220 diseases without the deleterious effects on stomach mucosa.

221 anical advantage for penetrating the viscous stomach mucus layer.

222 Cs), whose function is to pump acid into the stomach, normally lack MIST1 and do not perform regulate

223 hotopic xenografts of MKN45/5FU cells in the stomach of nude mice revealed that these cells had a hig

224 Similarly, in the stomachs of H. pylori-infected patients, the human SLFN4

225 nd downstream MAPK signaling was observed in stomachs only when E-cadherin was absent.

226 close to the esophagus, or too far into the stomach or duodenum.

227 ellar endings (IGLEs) were identified in the stomach or esophagus.

228 ecurrent or metastatic adenocarcinoma of the stomach or gastro-oesophageal junction.

229 ecurrent or metastatic adenocarcinoma of the stomach or gastro-oesophageal junction.

230 r locally advanced carcinomas arising in the stomach or in the gastroesophageal junction in patients

231 a, less common causes being carcinoma of the stomach or pancreas, whereas diseases of the sternum pre

232 oembolization, nontarget embolization to the stomach or small bowel can result in gastrointestinal in

233 cant ulceration, bleeding, and oedema in the stomach or small intestine of wild-type (WT) mice; howev

234 ibitor that suppresses acid secretion in the stomach, or left untreated.

235 er, ovary, colon and/or rectum, pancreas, or stomach (P < .001).

236 of organoids from mouse tissues (intestine, stomach, pancreas, and liver) and human intestine and pa

237 The helical shape of the human stomach pathogen Helicobacter pylori has been suggested

238 with reductions in islet, hypothalamic, and stomach PC1 content.

239 563 +/- 140 and 32 +/- 9, respectively), and stomach (percentage injected dose/g at 90 min, 68 +/- 21

240 Erlotinib depends on stomach pH for its bioavailability.

241 urrently with a proton pump inhibitor (PPI), stomach pH increases, which results in a clinically rele

242 fecting the stomach function, and the normal stomach pH is restored within 24 h post motor administra

243 suggest that at least in rat, the glandular stomach plays a central role in the improvement of insul

244 The fundamental roles that the stomach plays in ingestion and digestion notwithstanding

245 tion of apical muscle, tube foot and cardiac stomach preparations from A. rubens.

246 , cause dose-dependent relaxation of cardiac stomach preparations, with greater potency/efficacy than

247 epithelial precursor cells of the glandular stomach, providing a new conditional model of gastric ca

248 euritis), brain (choriomeningoencephalitis), stomach, proximal duodenum, and pancreas.

249 e was an interaction between digestion time, stomach region, and almond type for gastric protein hydr

250 cobacter pylori into the glands of the mouse stomach relative to host mitotic progenitor cells, illus

251 ations of the stomach, specifically proximal stomach reservoir functions and antral emptying operatio

252 upport gastric function and to stimulate the stomach's proteolytic activities in FD.

253 of other common neoplasms, such as lung and stomach since mid-1980s, prostate cancer has become one

254 system including the oral cavity, esophagus, stomach, small and large intestines, pancreas, and liver

255 r there is a motility disorder affecting the stomach, small bowel, or colon.

256 cantly in all compartments, but first in the stomach, small intestine and ascending colon.

257 e artificial alimentary tract, including the stomach, small intestine and colon, was analyzed in norm

258 e digestive tract (mouth, throat, esophagus, stomach, small intestine, and colorectum) and digestive

259 tant mice develop remarkable dilation of the stomach, small intestine, bladder, and ureters attributa

260 d gastrointestinal tract (GIT) model: mouth; stomach; small intestine.

261 So far, stomach-specific biomarkers, gastric cancer(GC)-related

262 the unique biomechanical adaptations of the stomach, specifically proximal stomach reservoir functio

263 t, mesenchymal-epithelial interactions drive stomach specification, patterning, differentiation and g

264 to 490 mL), one drink that was stable in the stomach [stable foam (SF)], and one drink that was less

265 ment and discusses recent findings regarding stomach stem cells and organoid cultures, and their role

266 egulation include the motor functions of the stomach, such as the rate of emptying and accommodation,

267 k, including lung, colon, breast, liver, and stomach, suggest that studies of NTEs in other tissues a

268 in chemotaxis that are able to colonize the stomach surface but not the antral glands in mice do not

269 eeding sensor can help indicate blood in the stomach: the sensor is swallowed to detect active bleedi

270 pes (bladder, breast, liver, lung, prostate, stomach, thyroid, head and neck) and paired blood from 7

271 pectra datasets of serum, urine, cortex, and stomach tissue extracts from the rats were analysed by m

272 the potential for development of engineered stomach tissues as a renewable source of functional beta

273 o determine the localizations of H pylori in stomach tissues from humans and infected mice.

274 Stomach tissues from mice infected with PMSS1 had increa

275 le to the level of pepsinogen C in the mouse stomach tissues.

276 ommon food additive, in the acidic medium of stomach to form N- and C-nitroso compounds.

277 a need to survive the extreme acidity of the stomach to successfully colonize the human gut.

278 he origin of phase III contractions from the stomach to the duodenum (P = 0.001) and decreased hunger

279 IMD (ratio of gastric counts in the proximal stomach to those in the total stomach) at 0, 1, 2, 3, an

280 ulated gastric environment and fresh porcine stomach to validate the effectiveness and reliability of

281 ld-type (WT) and Gal3-deficient mice using a stomach tube.

282 ssure, and increasing spontaneous reports of stomach upset and headache, yet dropout rates were compa

283 Despite colonizing the murine stomach, ureB mutants failed to induce IL-1beta and IL-1

284 Devices resident in the stomach-used for a variety of clinical applications incl

285 of a graded thermal stimuli delivered to the stomach via fluid ingestion at 52, 37, 22 and 7 degrees

286 the buffet meal was inversely related to the stomach volume and area under the curve of hormone conce

287 ficant target expression in tumor, lung, and stomach was confirmed by immunohistochemistry.

288 e overall polyphenol bioaccessibility in the stomach was found to be 1.5% and 3.1% after F&V and PE c

289 -(11)C-methyl-taurolithocholic acid into the stomach was observed.

290 achs, but the predominant response of W/W(V) stomachs was contraction.

291 ability, while the ability to colonize mouse stomachs was significantly reduced in the DeltatrxA muta

292 survivability in the acid environment of the stomach, we created an acid-resistant strain, Ty21a-AR-S

293 administer lipid emulsions directly into the stomach, we show that inhibiting triacylglycerol digesti

294 preoperative hernias containing most of the stomach were more likely to recur after repair when comp

295 e revealed that SLFN4+ cells migrated to the stomach, where they exhibited myeloid-derived suppressor

296 ted in GAPPS and sporadic FGPs and in normal stomach, which suggests that 1B transcripts are more imp

297 nodose ganglia, and, after tracer transport, stomach whole mounts were collected.

298 w cellular plasticity in the adult liver and stomach will be examined, highlighting the diverse cell

299 r, in which a cast of hairs is formed in the stomach with its 'tail' extending up to varying lengths

300 Tumors occur throughout the stomach, with metastases documented in lymph nodes, lung