ライフサイエンスコーパス: stool

1 the donor feces (~1011 per gram of human wet stool).

2 ool before and after FMT and also on donors' stool.

3 , weight loss, and waking at night to have a stool.

4 sts for direct detection of Campylobacter in stool.

5 l titers and increased viral shedding in the stool.

6 l cure at 24 hours and time to last unformed stool.

7 lowed by laxative and manual disimpaction of stool.

8 ted with Shigella who present without bloody stool.

9 epartment with jaundice, dark urine and pale stools.

10 ection of monthly surveillance and diarrheal stools.

11 al discomfort, rectal pain, and blood-tinged stools.

12 differed significantly from those in control stools.

13 alisation of virus and virus-specific IgA in stools.

14 by the demonstration of virus in children's stools.

15 illness, or both, and found BuV DNA in three stools (0.3%) and for the first time in a nasal swab (0.

16 umatosis (31.1% vs 47.2%; P = .01), blood in stool (11.8% vs 29.6%; P < .001), or mucus in stool (2.1

17 tool (11.8% vs 29.6%; P < .001), or mucus in stool (2.1% vs 5.6%; P = .048) but more likely to presen

18 liva (22 days), conjunctiva/tears (28 days), stool (29 days), vaginal fluid (33 days), sweat (44 days

19 e primary outcome was poliovirus shedding in stool 7 days after bivalent OPV challenge at 11 months.

20 tion (96%), methods used for conservation of stools (76%), the amount and type of stools used (for ex

21 Changes in stool acetate (P = 0.02) and total SCFAs (P = 0.05) were

22 Conclusion: Donor stool administered via colonoscopy seemed safe and was m

23 oliovirus type-specific IgA were measured in stool after a monovalent OPV type 2 challenge at 18 week

24 Only 14.6% of stool and 2.4% of urine samples yielded viral RNA.

25 amplification was used to detect Shigella in stool and blood matrixes at the single-digit CFU level.

26 sted by enzyme immunoassay for Campylobacter Stool and blood samples were assayed for markers of inte

27 of 21 CFU/mL and 18 CFU/mL were achieved in stool and blood, respectively, corresponding to 2-7 CFUs

28 uses have been studied exclusively by PCR in stool and detected only in patients with diarrhoea, alth

29 ype-2-specific antibodies can be measured in stool and develop in response to receipt of OPV type 2 e

30 Clinical data and infant stool and fasting HM samples were collected from 18 NW [

31 haw cycle-induced metabolic changes in crude stool and fecal water using a (1)H NMR spectroscopy-base

32 Furthermore, we analysed by BuV qPCR stool and nasal swab samples from 955 children with gast

33 Virus excretion in stool and nasopharynges was consistently observed, with

34 Virus excretion in stool and nasopharynges was consistently observed, with

35 cidence of 0.86 infections per child-year by stool and serology.

36 Paired stool and serum samples were collected from a subset of

37 factors for death were presence of blood in stool and severe dehydration.

38 cs for Salmonella isolated from blood versus stool and to determine resistance trends over time.

39 At these time points, stool and urine samples were collected.

40 her due to the detection of parasite eggs in stool and/or the presence of a concordant positive serol

41 donors (47%), materials used for collecting stools and the period of collection (96%), methods used

42 points to pneumatosis, 2 points to blood in stool, and 1 point each to abdominal tenderness and abdo

43 y also detected norovirus in directly boiled stool, and displayed better resistance to inhibitors tha

44 y tracts (LRT and URT, respectively), blood, stool, and urine.

45 was detected in 5.5% (408/7440) of diarrheal stools, and 344 (19.8%) children ever had rotavirus gast

46 mpylobacter selective media, four commercial stool antigen assays, and a commercial PCR assay.

47 estionable relevance of unconfirmed positive stool antigen CIDT results was supported by the finding

48 o question the use of commercially available stool antigen CIDTs as standalone tests for direct detec

49 icenter study to evaluate the performance of stool antigen CIDTs compared to culture and PCR for Camp

50 The stool antigen CIDTs ranged from 79.6% to 87.6% in sensit

51 sive performance data for four Campylobacter stool antigen CIDTs versus culture and molecular diagnos

52 true analytical and clinical performance of stool antigen CIDTs versus truly optimized culture condi

53 nts were tested for the presence of HP using stool antigen detection kit.

54 gen tests, and 73% were positive by just one stool antigen test.

55 n was confirmed using urease breath test and stool antigen test.

56 d positive predictive value of Campylobacter stool antigen tests were highly variable.

57 T, only one (0.48%) was positive by all four stool antigen tests, and 73% were positive by just one s

58 es include colonocytes (~107 per gram of wet stool), archaea (~108 per gram of wet stool), viruses (~

59 ility of using mRNA from exfoliated cells in stool as a means to surveil the distal small intestine i

60 ties (such as patient skin, nasopharynx, and stool) as well as environmental biofilms, in order to be

61 oms of 'straining' at 82.8%, 'lumpy and hard stool' at 74.2% and 'sensation of incomplete evacuation'

62 donor stool (heterologous) or patient's own stool (autologous) administered by colonoscopy.

63 ities of establishing such a service using a stool bank of prescreened donor stool including detail r

64 The development of a frozen stool bank of screened donor stool is an important step

65 robiota analyses were performed on patients' stool before and after FMT and also on donors' stool.

66 dpoints were adequate relief of symptoms and stool Bifidobacterium species abundance at 4 weeks.

67 tion tests identify BI/NAP1/027 rapidly from stool, but the emergence of closely related strains comp

68 6 months of life, whereas 74% had a positive stool by 2 years.

69 ternal breast milk, areolar skin, and infant stool by sequencing of the 16S ribosomal RNA gene.

70 Blinded specimen sets from human stool, chemostats, and artificial microbial communities

71 5 years of age presenting with AGE had their stools collected and tested for rotavirus by enzyme immu

72 ph nodes at days 1 and 3 p.i. and 5/6 pooled stools collected from day 1 to day 28 p.i.

73 r example, fresh or frozen), and duration of stool conservation (67%).

74 ecrease in abdominal pain and improvement in stool consistency on the same day for at least 50% of th

75 y since they regulate gut motor function and stool consistency, and targeted 5-HT4R selective drug in

76 vented weight loss, improved colitis scores (stool consistency, hematochezia, and mouse appearance),

77 There is substantial evidence that stool culture and parasitological examinations are of mi

78 The average length of time from stool culture collection to discharge was 3.4 days in th

79 Perirectal surveillance cultures and a stool culture grew Aeromonas species from three patients

80 ysis demonstrated 59% of the cost of routine stool culture to be attributable to the identification o

81 (CDST) to decrease the number of unnecessary stool cultures (STCUL), ova/parasite (O&P) examinations,

82 al reduction in the number of evaluations of stool cultures and the number of parasitological examina

83 Patients with stool cultures submitted were tested on the GI panel (n

84 clinical review and collection of blood and stool cultures.

85 ther measures included macronutrient intake, stool diaries, and fecal short-chain fatty acid concentr

86 The median time to last unformed stool did not differ between treatment arms (azithromyci

87 The microbial community structure in case stools differed significantly from those in control stoo

88 ies were distinct in milk, areolar skin, and stool, differing in both composition and diversity.

89 One study (n = 9989) found that FIT plus stool DNA test had better sensitivity in detecting CRC t

90 al immunochemical testing (FIT), multitarget stool DNA testing, flexible sigmoidoscopy with or withou

91 d the detection of toxigenic C. difficile in stool does not necessarily confirm the diagnosis of CDI.

92 efine whether FMT using a rationally derived stool donor is safe in recurrent HE compared to SOC alon

93 tool weight (76 +/- 12 g/d; P < 0.0001), and stool energy content (57 +/- 17 kcal/d; P = 0.003), but

94 were excluded, between-group differences in stool energy content and glucose tolerance increased, an

95 nt (57 +/- 17 kcal/d; P = 0.003), but not in stool energy density, were higher in the WG.

96 itive effects of whole grains on the RMR and stool energy excretion that favorably influence energy b

97 hat transfer of sterile filtrates from donor stool (FFT), rather than fecal microbiota, can be suffic

98 Using GII.4 huNoV positive stool filtrates, we demonstrated limited huNoV replicati

99 global collection, storage, and analysis of stool for microbiome studies.

100 ipation, pelvic pressure, and narrow-caliber stools for 2 months.

101 cross 8 countries contributed 7077 diarrheal stools for norovirus testing.

102 ke) (P < 0.0001), stool weight (P < 0.0001), stool frequency (P = 0.02), and short-chain fatty acid (

103 r 7 days before the study period; along with stool frequency and consistency.

104 No significant differences were observed in stool frequency or form or in short-chain fatty acid con

105 rable to controls by adulthood (P = NS), but stooling frequency remained higher in 44% of patients (P

106 rable to controls by adulthood (P = NS), but stooling frequency remained higher in 44% of patients (P

107 During faecal microbiota transplantation, stool from a healthy donor is transplanted to treat a va

108 re, we attempted to collect rectal swabs and stool from all participants.

109 rform a metagenome-wide association study on stools from 218 individuals with atherosclerotic cardiov

110 Analysis of 401 stools from 84 longitudinally sampled preterm infants de

111 rform a metagenome-wide association study on stools from individuals with atherosclerotic cardiovascu

112 resources to prevent C. difficile testing of stools from patients without clinically significant diar

113 of wet stool), viruses (~108 per gram of wet stool), fungi (~106 per gram of wet stool), protists, an

114 robiota, can be sufficient to restore normal stool habits and eliminate symptoms.

115 In all 5 patients, FFT restored normal stool habits and eliminated symptoms of CDI for a minimu

116 pert combined on nasopharyngeal aspirate and stool had intention-to-diagnose and per-protocol sensiti

117 , as a direct measure of human hemoglobin in stool has a number of advantages relative to conventiona

118 Fecal microbiota transplantation with donor stool (heterologous) or patient's own stool (autologous)

119 al CT scan revealed large amount of retained stool in the colon, bowel wall thickening and infiltrati

120 children had a Giardia positive surveillance stool in the first 6 months of life, whereas 74% had a p

121 illness <7 d duration comprising >/=3 loose stools in 24 h and >/=1 of the following: sunken eyes, s

122 d as the presence of diarrhea [>/=3 unformed stools in 24 h] and absence of laxative intake in the pr

123 vice using a stool bank of prescreened donor stool including detail regarding donor recruitment and s

124 nfected with hypervirulent ribotypes or with stool incontinence, to determine the rate of transmissio

125 ent of a frozen stool bank of screened donor stool is an important step in the standardization of the

126 81; 95% confidence interval, 1.56-2.11) than stool isolates.

127 (2) = 0.038), this was not true for neonatal stool (meconium; Mann-Whitney P > 0.05), and there was n

128 Disease activity and stool metagenomes at baseline, and weeks 14, 30, and 54

129 We applied MIDAS to stool metagenomes from 98 Swedish mothers and their infa

130 ol to evaluate best practices for sequencing stool metatranscriptomes.

131 On real supragingival plaque and stool MG datasets that were generated from healthy indiv

132 amined the functional potential of mucus and stool microbial communities in the mdr1a (-/-) mouse mod

133 on preceded colitis-induced inflammation and stool microbial differences only became apparent at coli

134 Stool microbiome composition was characterized using nex

135 The DNA Genotek OMNIgeneGut Stool Microbiome Kit was compared to the currently accep

136 Collection and stabilization of stool microbiome samples with the DNA Genotek collection

137 ic bacterial communities approximating human stool microbiomes to be used as a gold-standard for eval

138 ition and complexity of actual healthy human stool microbiomes.

139 inflammatory assessment, cognitive testing, stool microbiota analysis and brain MRI analysis.

140 The stool microbiota around the time of neutrophil recovery

141 sociated with low bacterial diversity in the stool microbiota early after transplant; however, the sp

142 of maternal pre-pregnancy body mass index on stool microbiota from 74 neonates, 18 born vaginally (5

143 associated with lower alpha diversity of the stool microbiota.

144 t diagnostic tests for S. mansoni infection (stool microscopy [samples prepared by sedimentation tech

145 parasites is traditionally accomplished via stool microscopy.

146 for eosinophils in the peripheral blood and stool mucus and allergen-specific lymphocyte stimulation

147 e inter-relatedness of tissue (invasive) and stool (noninvasive) datasets.

148 ion of the microbiota and the metabolites in stool of 183 subjects (82 UC, 50 CD, and 51 healthy cont

149 on pathway showed reduced persistence in the stool of infected mice, suggesting a role of 1,2-propane

150 Sequencing of viral nucleic acids from the stool of vaccinated, asymptomatic children and their clo

151 aseline and a decrease in frequency of loose stools of >/=50% from baseline, for 2 or more weeks duri

152 sitively detecting toxigenic C. difficile in stool, on-demand PCR may also be used to accurately pred

153 ally has been diagnosed by detecting eggs in stool or urine.

154 amples across multiple body sites, including stool, oral gingiva, nares, skin and vagina were collect

155 e observed on an inpatient research ward for stool output measurement and management of hydration.

156 f 44 adults with IBS and diarrhea or a mixed-stool pattern (based on Rome III criteria) and mild to m

157 rhoea (defined by WHO as three or more loose stools per day for less than 14 days).

158 l regarding donor recruitment and screening, stool preparation, and delivery of the FMT.

159 m of wet stool), fungi (~106 per gram of wet stool), protists, and metabolites.

160 unadjusted OR of 0.65 (95% CI 0.59-0.72) for stool relative to swab.

161 ining that no blood had been detected in the stool sample and were re-invited, if eligible, for scree

162 1606; 84.9%) having a Campylobacter-positive stool sample by 1 year of age.

163 The initial inoculum, a filtered clinical stool sample from the index gastroenteritis case cluster

164 dren aged <5 years hospitalized for AGE have stool sample tested for rotavirus antigen, was used to a

165 000-fold dilution from a norovirus positive stool sample.

166 nteropathogens on 878 acute watery diarrheal stools sampled from 14643 episodes captured by surveilla

167 Using stool samples (n = 298; aged 1-11 months) from a US birt

168 Consecutive stool samples (n = 312) positive for toxigenic C. diffic

169 Pre- and postintervention stool samples and rectal mucosal biopsies were collected

170 Metagenomics of patient stool samples at diagnosis revealed correlations between

171 t-chain fatty acids (SCFAs) were measured in stool samples by using gas chromatography.

172 the bacterial recognition patterns by IgA in stool samples collected at 1 and 12 months of age from c

173 d bacterial and eukaryotic gut microbiota in stool samples collected at 3 months of age using 16S and

174 V4 region of the bacterial 16S rRNA gene in stool samples collected before and 12 days after finishi

175 six IP taxa (11 subjects) were determined in stool samples collected before and after a challenge wit

176 The tests were performed on 479 stool samples collected from people admitted to the hosp

177 re, we examine the microbiome profile of 350 stool samples collected longitudinally for more than a y

178 We studied 1094 stool samples from 44 infants in the secondary cohorts.

179 We prospectively collected serial weekly stool samples from 66 patients who underwent HCT, starti

180 From August 2010 to July 2011, we collected stool samples from 723 children admitted with diarrhea (

181 ening for two specific RNA bacteriophages in stool samples from a longitudinal cohort of macaques sug

182 C. difficile was isolated from stool samples from a patient with two recurrent C. diffi

183 rray-based metatranscriptomics data from 233 stool samples from Europeans.

184 performed on fecal filtrate from donors and stool samples from FT recipients, as available.

185 Among the 111 patients enrolled, stool samples from nine were TC positive and four were E

186 315 stool samples from one series of 12 patients with CRC an

187 Mass spectrometry of stool samples identified novel candidate protein biomark

188 significantly enriched in CRC versus control stool samples in both series.

189 ant bacteria, bound to IgA or not, in infant stool samples in relation to allergy development.

190 ene in breast milk, areolar skin, and infant stool samples of 107 healthy mother-infant pairs.

191 and increased, respectively, in the 3-month stool samples of children who went on to have atopic whe

192 itative polymerase chain reaction testing of stool samples or 4-fold increase in serum antibody titer

193 nce of sequence reads from bacterial taxa in stool samples over time.

194 ommunity standard of freezing to store human stool samples prior to whole genome sequencing (WGS) for

195 Furthermore, metagenomic analysis of human stool samples reveals that the genetic capability of mic

196 n had 7601 diarrheal and 26 267 nondiarrheal stool samples tested for Campylobacter We describe a hig

197 udy was to evaluate the feasibility of using stool samples to detect the presence of H. pylori DNA wh

198 providing additional results in PCR-positive stool samples to guide therapy.

199 in commensal Escherichia coli isolated from stool samples was seen in children aged 3 or 6 months in

200 Stool samples were analysed for H. pylori antigen using

201 Stool samples were analysed for SCFA (acetic acid, propi

202 Stool samples were analyzed by mass spectrometry.

203 Diarrheal and nondiarrheal stool samples were collected and tested by enzyme immuno

204 Stool samples were collected every 2 weeks and during di

205 Stool samples were collected from donors (n = 3) and pat

206 In these individuals, stool samples were collected pretreatment (day 0), after

207 Maternal stool samples were examined for geohelminths by microsco

208 Stool samples were obtained from female IC patients and

209 Pre-colonoscopy stool samples were obtained from participants of screeni

210 ch markers and subsequent nutritional status.Stool samples were routinely collected between January 2

211 Stool samples were tested for norovirus by reverse-trans

212 During this surveillance period, 1379 stool samples were tested for the presence of norovirus.

213 The stool samples were thawed, homogenized, and used for 9 d

214 NA extracted from live parasites spiked into stool samples where the limits-of-detection were calcula

215 ues were not significantly different between stool samples with Bristol scores of 5, 6, and 7, betwee

216 nce full genomes from 507 norovirus-positive stool samples with reverse transcription-real-time PCR c

217 Of 294 total stool samples, 227 were deemed true positive.

218 of Health Human Microbiome Project (NIH HMP) stool samples, and they are transcribed under conditions

219 detection traditionally relies on diarrhoeal stool samples, but these are inconvenient to collect if

220 ure of blood, cerebrospinal fluid, urine, or stool samples, including bacteremia and bacterial mening

221 be used to accurately predict toxin-negative stool samples, thus providing additional results in PCR-

222 ium difficile from unformed (liquid or soft) stool samples.

223 articles and particles from patient sera and stool samples.

224 pisthorchiasis was based on PCR performed on stool samples.

225 ure-produced particles and patient blood and stool samples.

226 med 16S rRNA gene sequencing on 333 infants' stool samples.

227 cing Enterobacteriaceae (ESBL-E) in clinical stool samples.

228 d using 16S ribosomal RNA gene sequencing of stool samples.

229 tridium difficile identification in diarrhea stool samples.

230 ified and then pyrosequenced faecal DNA from stool samples.

231 osomal sequencing of prospectively collected stool samples.

232 Xpert performed on respiratory and stools samples enables rapid confirmation of tuberculosi

233 nd challenge with monovalent type 2 OPV, and stools samples were collected.

234 nfection in Eritrean refugees, compared with stool sedimentation microscopy.

235 Acute and convalescent stool, serum, and saliva samples from cases, exposed and

236 S. japonicum DNA present in human stool, serum, urine, and saliva was detected quantitativ

237 om the microbial communities in oral, nasal, stool, skin, and vagina, with Proteobacteria as the domi

238 ar diagnostic technology is available, and a stool specimen is not immediately available.

239 sing 1,084 prospectively collected preserved stool specimens across five clinical centers.

240 871 (76%) of 1147 stool specimens and 1024 (68%) of 1514 swabs were positi

241 le participants, 1147 (76%) of whom provided stool specimens and 1514 (>99%) provided swab specimens.

242 gen yield was 57% (871 of 1519 children) for stool specimens and 67% (1024 of 1519 children) for rect

243 tly from unpreserved or Cary-Blair-preserved stool specimens for the detection of Yersinia enterocoli

244 urpose, total DNA was extracted from 294 raw stool specimens from H. pylori-positive and -negative pa

245 Consecutive stool specimens from hospitalized patients with diarrhea

246 o, Y. enterocolitica, and P. shigelloides in stool specimens from patients suspected of acute gastroe

247 as 4800 system using prospectively collected stool specimens from patients suspected of having C. dif

248 tween July and October 2010, we tested 2,767 stool specimens from patients with gastrointestinal illn

249 C. difficile was isolated from stool specimens in 432 (10.9%) of all the patients who h

250 he enteropathogen yields of rectal swabs and stool specimens in children with diarrhoea or vomiting,

251 ted children, conducted surveillance, tested stool specimens in the laboratories, engaged with commun

252 arative yield adjusted odds ratios (ORs) for stool specimens relative to swabs were 1.24 (95% CI 1.11

253 Stool specimens were collected from 35 patients with cir

254 A total of 2,410 unformed, deidentified stool specimens were collected.

255 cobacter pylori DNA can be detected in human stool specimens with high sensitivity and can therefore

256 mmunoassay detection of rotavirus antigen in stool specimens.

257 for the identification of STEC in preserved stool specimens.

258 oducing Escherichia coli (STEC) in preserved stool specimens.

259 We studied the frequency of stool studies ordered before or on day 3 and after day 3

260 nts stated a surveillance preference for the stool test over colonoscopy (60.8 % vs 31.0 %; no prefer

261 Women were more likely to prefer the stool test than men (66.7 % vs. 53.6 %; p = .011).

262 The primary reason for preferring the stool test was that it would be done more frequently.

263 nts who met study criteria and had rotavirus stool testing performed and vaccine status confirmed, we

264 ting, flexible sigmoidoscopy with or without stool testing, computed tomographic colonography (CTC),

265 anel had an average of 0.58 other infectious stool tests compared with 3.02 in the control group (P =

266 olonoscopy, flexible sigmoidoscopy, CTC, and stool tests have differing levels of evidence to support

267 To identify novel protein biomarkers in stool that outperform or complement hemoglobin in detect

268 ls also remained detectable in the serum and stools throughout ribavirin monotherapy.

269 Paratyphi A in stool typically preceded onset of bacteremia.

270 At weeks 0 and 6, stool, urine and blood samples were collected, and funct

271 Helminth infections were ascertained by stool, urine and haemoparasitology.

272 tion of stools (76%), the amount and type of stools used (for example, fresh or frozen), and duration

273 of wet stool), archaea (~108 per gram of wet stool), viruses (~108 per gram of wet stool), fungi (~10

274 Standardizing the input stool volume did not improve CT toxin specificity.

275 Stool was collected from 5 donors selected by the patien

276 The time to virus negativity in stool was determined.

277 rotavirus antigen (cases) and children whose stool was negative (controls).

278 least 1 dose of Rotarix among children whose stool was positive for rotavirus antigen (cases) and chi

279 f oral Actinobacteria and oral Firmicutes in stool was positively correlated with subsequent GVHD; La

280 the phylum Firmicutes and Proteobacteria in stool was significantly decreased and Bacteroidetes and

281 Stool was sterile-filtered to remove small particles and

282 Stool was tested for C. difficile by toxin enzyme immuno

283 lic rate (RMR) (43 +/- 25 kcal/d; P = 0.04), stool weight (76 +/- 12 g/d; P < 0.0001), and stool ener

284 inols (a measure of WG intake) (P < 0.0001), stool weight (P < 0.0001), stool frequency (P = 0.02), a

285 f WGs in a weight-maintenance diet increases stool weight and frequency and has modest positive effec

286 Blood and stool were collected at baseline, 1, 3, 6 and 12 months.

287 Vomiting and bloody stool were frequently observed in both groups (approxima

288 andard samples); nasopharyngeal aspirate and stool were taken for all children, and a string test was

289 Stools were collected at enrollment and, for cases, afte

290 Stools were collected from 61 consecutive patients diagn

291 Secondary hyperparathyroidism and loose stools were more frequent after distal gastric bypass.

292 Enrollment and follow-up stools were tested by quantitative polymerase chain reac

293 Stools were tested for rotavirus and sera for antirotavi

294 ns rapid access to thoroughly screened donor stool when needed, without the ethical and logistical pr

295 icile toxin was subsequently detected in his stools, which is when he first raised the possibility of

296 n confirmed by the presence of free toxin in stool who were randomly assigned to receive one or more

297 ntery characterised by frequent scant bloody stools with fever, prostration, and abdominal cramps.

298 resence of Giardia in a monthly surveillance stool within the first 6 months of life decreased length

299 diarrhea case definition (>/=3 loose/liquid stools within 24 hours).

300 1-2 vomiting episodes plus 1-2 loose/liquid stools within 24 hours).