1 e monkeys were anisometropic, and three were
strabismic.
2 ach individual eye in 19 adults (7 esotropic
strabismics,
6 anisometropes and 6 controls).
3 ithin striate cortex (area V1) of normal and
strabismic,
adult macaque monkeys.
4 estionnaire (NEI VFQ-25) was performed in 42
strabismic adults over the age of 50 years at a single i
5 In nine
strabismic amblyopes (mean age, 32 years), the results c
6 in normal subjects, anisometropic amblyopes,
strabismic amblyopes and non-amblyopic esotropes.
7 Using the amblyopic eye,
strabismic amblyopes counted inaccurately, markedly unde
8 The results indicate that
strabismic amblyopes have mechanisms for binocular summa
9 Rather, counting deficits in
strabismic amblyopes reflected a higher-level limitation
10 gnification is normal in the foveal field of
strabismic amblyopes.
11 re newly diagnosed with anisometropic and/or
strabismic amblyopia and had not undergone previous trea
12 Abnormal visual development in
strabismic amblyopia drastically affects visual percepti
13 cular approach to treating anisometropic and
strabismic amblyopia has recently been advocated.
14 ld be a potential test to detect and monitor
strabismic amblyopia in young children.
15 Strabismic amblyopia is typically associated with severa
16 The treatment effect was greater for
strabismic amblyopia than for combined-mechanism amblyop
17 anisometropic amblyopes, in 6 patients with
strabismic amblyopia, and in 4 patients with combined an
18 part to distorted skull development causing
strabismic amblyopia, and OPG) were difficult to treat a
19 nown to be severely disrupted in adults with
strabismic amblyopia, could be a potential test to detec
20 and 71% positive predictive value (PPV) for
strabismic amblyopia.
21 rating acuity may underestimate the depth of
strabismic amblyopia.
22 n 4 patients with combined anisometropic and
strabismic amblyopia.
23 Sixty
strabismic and 80 control participants were prospectivel
24 ight strabismic, two anisometropic, and four
strabismic and anisometropic amblyopes (mean age, 8.5 +/
25 Optical treatment alone of
strabismic and combined-mechanism amblyopia results in c
26 Differences in gene expression between
strabismic and normal human EOMs point to a relevant con
27 mmetry in ODC width and area were similar in
strabismic and normal monkeys.
28 yes for the three less severe amblyopes (two
strabismic and one anisometropic).
29 he three more severely affected animals (one
strabismic and two anisometropic), the optimal spatial f
30 Comparison of SOA population activity in
strabismic animals and normal monkeys (described in the
31 and neural sensitivities are altered in the
strabismic animals compared with the normal animals.
32 The
strabismic animals had early-onset natural esotropia (th
33 minor effect on the vergence behavior of the
strabismic animals.
34 ours defined by Gabor patches is abnormal in
strabismic,
but not in anisometropic, amblyopia.
35 rally strabismic monkey and in a monkey made
strabismic by early postnatal alternating monocular occl
36 eural responses in nonamblyopic monkeys made
strabismic by surgery at the age of 10-60 d.
37 Although our data from
strabismic cats do not differ markedly from those report
38 of callosal neurons in the striate cortex of
strabismic cats to that in normally reared cats.
39 In
strabismic cats, we observed a dramatic shift in the ocu
40 Fifty-one
strabismic children and 130 normal controls ages 3 to 17
41 For
strabismic children, hyperacuity and grating acuity were
42 n may inform about causes and effects of the
strabismic condition in humans.
43 n of the peripheral oculomotor system to the
strabismic condition.
44 n among esotropic patients compared with non-
strabismic controls.
45 e deficiency of binocular connections in the
strabismic cortex was evident qualitatively as a "skip"
46 usion is a realistic goal in the majority of
strabismic CP children.
47 Strabismic (
cross-eyed) humans and animals show an imbal
48 always improves, if not permanently curing,
strabismic deviations.
49 FS) represent a subset of monogenic isolated
strabismic disorders that are characterized by restricti
50 s significantly different between normal and
strabismic EOM.
51 es that were significantly down-regulated in
strabismic EOMs; none was significantly upregulated.
52 Strabismic extraocular muscles (EOMs) differ from normal
53 Application of a prismatic correction to the
strabismic eye in order to achieve bifoveal stimulation
54 rmal age-matched infants, V1 neurons in both
strabismic groups exhibited reductions in sensitivity to
55 atomical markers in V1 of two experimentally
strabismic Macaca nemestrina monkeys.
56 The CO results in
strabismic macaque suggest that the nasal ODC bias promo
57 revealed by CO staining of V1 in a naturally
strabismic monkey and in a monkey made strabismic by ear
58 As do humans with large strabismus,
strabismic monkey display disconjugate saccadic eye move
59 One
strabismic monkey had infantile-onset, small-angle esotr
60 The other
strabismic monkey had naturally occurring, large-angle (
61 ctive error was also small in the normal and
strabismic monkeys (approximately 1 D to 2 D) as long as
62 ed Repair and 0.49 +/- 0.17 in the naturally
strabismic monkeys (P < 0.01).
63 Delayed Repair and naturally
strabismic monkeys also had motion VEP asymmetries of eq
64 e accommodation in awake-behaving normal and
strabismic monkeys and describe properties of photorefra
65 ors recorded binocular eye movements in four
strabismic monkeys and one unaffected monkey.
66 contrast, both Delayed Repair and naturally
strabismic monkeys had asymmetric motion VEP responses:
67 t an overabundance of neurons in V1 or V2 of
strabismic monkeys preferring the temporal-to-nasal dire
68 rsuit adaptation during monocular viewing in
strabismic monkeys with exotropia.
69 In layers 2/3 and 4B of the
strabismic monkeys, patchy projections and boutons termi
70 In both normal and
strabismic monkeys, small misalignment of the photorefra
71 Specific challenges posed by the
strabismic monkeys, such as possible misalignment of the
72 In all
strabismic monkeys, the sensitivity of V1 units to inter
73 accommodation in awake, behaving normal, and
strabismic monkeys.
74 ong orthotropic or naturally or artificially
strabismic monkeys.
75 onal bias was significantly reduced in these
strabismic monkeys.
76 strabismus were surprisingly small in infant
strabismic monkeys.
77 In both normal and
strabismic monkeys: (1) CO-blob compartment neurons show
78 ts were different across monkeys tested (two
strabismic,
one normal) but were similar for each monkey
79 dths of the ODCs having input from eyes with
strabismic or anisometropic amblyopia were reduced in pr
80 the sole initial treatment for children with
strabismic or combined-mechanism amblyopia before initia
81 .16] years; 11 boys and 9 girls) and 20 with
strabismic or mixed amblyopia (mean [SD] age, 4.90 [1.36
82 Among children treated for anisometropic,
strabismic,
or combined mechanism amblyopia, there is a
83 Strabismic owl monkeys also showed ocular dominance colu
84 visual deficits impacting quality of life in
strabismic patients and may represent a novel measure by
85 Strabismic patients demonstrate subnormal BiS and even b
86 For 1.25% LCA,
strabismic patients had a mean BiS score less than 1, in
87 Strabismic patients performed the same or worse on nearl
88 Additionally,
strabismic patients reported significantly worse ocular
89 Physicians treating
strabismic patients should recognize these quality-of-li
90 Mean BiS was significantly lower in the
strabismic patients than controls for LCA (2.5% and 1.25
91 A total of 130 postoperative
strabismic patients were studied.
92 This may explain why
strabismic patients who are not diplopic close 1 eye in
93 Pediatric
strabismic patients who underwent strabismus surgery bet
94 reoacuity and bifoveal fusion in a cohort of
strabismic patients with variable binocular sensory func
95 The dominant eye of
strabismic patients without anisometropia retained suppr
96 etely fixed in an infraducted (downward) and
strabismic position, and markedly limited and aberrant r
97 ctural basis for lack of binocular fusion in
strabismic primates, we investigated intrinsic horizonta
98 d differential expression between normal and
strabismic samples, with an adjusted P value of </= 0.05
99 ion for the amblyopic eye in one early-onset
strabismic subject with binocular suppression, indicatin
100 mpared in seven orthotropic subjects and six
strabismic subjects (four with primary microstrabismus a
101 All the
strabismic subjects demonstrated normal retinal correspo
102 suggest that the eye movement asymmetries in
strabismic subjects do not result from similar asymmetri
103 out anisometropia, consistent with classical
strabismic suppression of the nondominant eye.
104 Animals were rendered
strabismic surgically at 8-10 days of age and were allow
105 ely small proportion of cortical neurons; in
strabismics,
the influence of the two eyes was more near
106 Eight
strabismic,
two anisometropic, and four strabismic and a
107 normal monkeys revealed that the deficits in
strabismic V1 were due mainly to a loss of binocular con
108 ar ocular dominance columns if they are made
strabismic within a few weeks of birth.