ライフサイエンスコーパス: strand

1 triggered, ejecting Pol delta on the leading strand.

2 uanosine or 2-deoxyadenosine in the opposite strand.

3 , where and when TLS occurs on each template strand.

4 to Pol delta and recruits it to the lagging strand.

5 opening and trapping of the DNA non-template strand.

6 lting in a shortening of the C-terminal beta-strand.

7 ation, even in the presence of excess staple strands.

8 tructure to a 3-D network of one-dimensional strands.

9 = benzene bis-1,4-isonitrile to form polymer strands.

10 milar elongation between leading and lagging strands.

11 nsible for concerted cleavage of the two DNA strands.

12 of each of the complementary and homologous strands.

13 of the ability of ZO-1 to stabilize claudin strands.

14 formation of significant gaps in the nascent strands.

15 hat electrostatic contacts with the excluded strand act as a regulator for unwinding activity.

16 he gaps between two rods, with different DNA strands allows one to synthesize nanostructure assemblie

17 stimate the spontaneous occurrence of single-strand and double-strand breaks (DSB).

18 lusion, where the helicase encircles one DNA strand and excludes the other, acting as a wedge with an

19 rive plant transcription from EBEs on either strand and in both directions.

20 uous runs of >/=4 RNA nucleotides within DNA strand and the only common substrate between the two bac

21 multaneously associated with the growing DNA strands and Mg2 PPi crystals during the rolling circle p

22 ucture are created by as few as four or five strands and so identify amyloid secondary structure even

23 he G/APs inhibited the formation of parallel strands and these adopted antiparallel topologies.

24 ds Pol epsilon and tethers it to the leading strand, and PCNA (proliferating cell nuclear antigen) bi

25 onal stress by nicking and resealing one DNA strand, and some Top1-dependent mutations are due to tra

26 show that loss of RER in B. subtilis causes strand- and sequence-context-dependent GC --> AT transit

27 rom annual bone growth layer rings from dead-stranded animals, and then combined the bone and regiona

28 Synthesis-dependent strand annealing (SDSA) is the preferred mode of homolog

29 ms, homologous recombination (HR) and single strand annealing (SSA), and in conferring resistance to

30 DNA-single strand annealing proteins (SSAPs) are recombinases frequ

31 le-base bulges 7T8, 5A6 and 4A5 on the guide strand are stacked-into the duplex, with conformational

32 sion of a DNA strand using the complementary strand as a template.

33 ow that purified PBP2a can cross-link glycan strands bearing penta- and triglycine, but not monoglyci

34 Although claudin strand behavior in fibroblasts may not fully recapitulat

35 brane space (IMS) domain and a C-terminal 16-stranded beta-barrel domain.

36 Using a standard 3-stranded beta-sheet model, the WW domain, it was found t

37 ther technology specific artifacts including strand bias and low base quality at read ends.

38 sit that extensive resection of a DNA double-strand break (DSB) by a multisubunit helicase-nuclease m

39 ability of the HR pathway to complete double-strand break (DSB) repair by about 50%.

40 P1 and RIF1 foci, suggesting that DNA double-strand break (DSB) repair by homologous recombination (H

41 ning (NHEJ) is the most prominent DNA double strand break (DSB) repair pathway in mammalian cells.

42 omologous recombination (HR) is a DNA double-strand break (DSB) repair pathway that protects the geno

43 end-joining (NHEJ) is the predominant double-strand break (DSB) repair pathway throughout the cell cy

44 enuated the expression of several DNA double-strand break (DSB) repair proteins and formation of repa

45 Improper DNA double-strand break (DSB) repair results in complex genomic rea

46 omologous recombination (HR)-mediated double-strand break (DSB) repair, which is mediated through its

47 localized to the progerin-induced DNA double-strand break (DSB) sites, blocking DSB repair, which led

48 h, the spatio-mechanical processes of double-strand break (DSB)-repair, especially the auxiliary fact

49 us chromosomal locus containing a DNA double-strand break (DSB).

50 ate that the criterion used to select direct strand break appears to have a very significant role on

51 higher order oligomers to generate a double strand break in DNA.

52 the CometChip and the staining of DNA double-strand break marker, gammaH2AX.

53 olved in DNA base excision repair and single-strand break repair (SSBR) and is important for genetic

54 ning pathway plays a critical role in double strand break repair and is uniquely responsible for cell

55 A1 is best known for its functions in double-strand break repair and resolution of DNA replication st

56 genes to irradiation and inefficient double-strand break repair correlated with severe late radiatio

57 proteins, SFPQ and NONO, promotes DNA double-strand break repair via the canonical nonhomologous end

58 romosome inactivation, imprinting and double-strand break repair, and mutations in SMCHD1 contribute

59 lti-protein complexes involved in DNA single-strand break repair.

60 DNA to investigate the quality of the double-strand break repairs in the class-switch recombination p

61 hromatin mobility induced by a single double-strand break requires active microtubule function.

62 ease in the stability of RAD51 at DNA double-strand break sites and in the overall efficiency of HR.

63 re, thereby hiding telomere ends from double-stranded break repair and ATM signaling, whereas POT1 re

64 d the effect of contrast agent on DNA double-strand-break (DSB) formation in patients undergoing magn

65 aks, and pronounced susceptibility to single-strand breakage.

66 1 ends and catalyzes excision through double strand breaks (DSB) and the joining of newly excised tra

67 ced clustered DNA lesions (OCDL), DNA double-strand breaks (DSB), apoptosis, and the local and system

68 neous occurrence of single-strand and double-strand breaks (DSB).

69 mbination, a subset of programmed DNA double-strand breaks (DSBs) are repaired as crossovers, with th

70 been implicated in the repair of DNA double-strand breaks (DSBs) by homologous recombination (HR), t

71 is the primary repair pathway for DNA double strand breaks (DSBs) in humans.

72 tones are modified in response to DNA double strand breaks (DSBs) in vivo by the ARTs Adprt1a and Adp

73 DNA double-strand breaks (DSBs) prevent cells from entering mitosis

74 Double strand breaks (DSBs) represent highly deleterious DNA da

75 ein 80 (RAP80) helps recruit BRCA1 to double-strand breaks (DSBs) through the scaffold protein CCDC98

76 formation of recombination-initiating double-strand breaks (DSBs) via a feedback loop triggered by cr

77 ), the activity of which leads to DNA double-strand breaks (DSBs) within IgH switch (S) regions.

78 promote the formation of meiotic DNA double-strand breaks (DSBs), the precursors of cross-overs.

79 11 (PHF11) in 5' end resection at DNA double-strand breaks (DSBs).

80 normal XRCC1 recruitment at oxidative single-strand breaks (SSBs) as indicated by the requirement for

81 n, Mlh1-Mlh3 can generate religatable double-strand breaks and form an active nucleoprotein complex t

82 Homologous recombination repairs DNA double-strand breaks and must function even on actively transcr

83 P3(-/-) cells leads to widespread DNA double-strand breaks and synthetic lethality.

84 (eg, ataxia-telangiectasia), and DNA double-strand breaks are crucial to the modulation of early gen

85 hese methods involve the induction of double-strand breaks at endogenous loci followed by the identif

86 cumulation of DNA damage, genome-wide double-strand breaks enriched at Ssb-binding regions and CpG is

87 The repair of DNA strand breaks improves, as do serum protein concentratio

88 es an antioxidant barrier against DNA double strand breaks induced by TGFbeta expressed in the tumor

89 ve end-joining (alt-EJ) repair of DNA double-strand breaks is associated with deletions, chromosome t

90 ZMYM3 is recruited to DNA double-strand breaks through bivalent interactions with both hi

91 3BP1 activities is its recruitment to double-strand breaks via the interaction of the tandem Tudor do

92 DNA double-strand breaks were determined in peripheral blood mononu

93 agile X syndrome, are also subject to double-strand breaks within the repetitive tract followed by DN

94 for oxidized DNA lesions, double-strand DNA strand breaks, and pronounced susceptibility to single-s

95 ibiting cep-1/p53, endogenous meiotic double strand breaks, or the expression of MIRAGE1 DNA transpos

96 a potent environmental source of DNA double-strand breaks, potential drivers of genome structure evo

97 in cells, which are converted to DNA double-strand breaks.

98 lved in H2O2 breakdown; and 4) result in DNA strand breaks.

99 role on the final number of simulated double strand breaks.

100 genome integrity with the potential to cause strand breaks.

101 l for the repair and signaling of DNA double strand breaks.

102 romote efficient RAD51 loading at DNA double-strand breaks.

103 ows replication elongation and causes double-strand breaks.

104 arkable in the examination of how DNA double-stranded breaks (DSBs) are repaired, with many component

105 ses to introduce changes (rather than double-stranded breaks and donor templates) and offers potentia

106 C DNA species with a covalently closed minus strand but an open plus strand (closed minus-strand RC D

107 cess, a small and constant set of unique DNA strands can be used to create DNA origami arrays of incr

108 LYL and YVEALL, which consist of 4-9 peptide strands, can contain a significant amount of beta-sheet.

109 e detection of noise-induced hearing loss in stranded cetaceans.

110 alently closed minus strand but an open plus strand (closed minus-strand RC DNA [cM-RC DNA]) was dete

111 ution capillary electrophoresis-based single-strand conformation polymorphism (CE-SSCP).

112 egment termed the stalk, which adopts a beta-strand conformation, instead of forming an alpha-helix a

113 To address this, we developed a new single-strand consensus sequencing assay for the determination

114 M pore loops touch both the Watson and Crick strands, constraining duplex DNA in a bent configuration

115 gger delivery and 5-phosphorylation of guide strands correlating with gene knockdown, we employed a p

116 cture or changing from alpha helical to beta strand depending on the solvents and molecules added to

117 nd from the broken DNA molecule pairs with a strand derived from an intact DNA molecule.

118 Guide strands detected in RISC by AGO2 immuno-isolation repres

119 gest that the following base on the template strand dictates the addition of the mutated base.

120 lymerase with both reverse-transcriptase and strand displacement activities to obtain sensitivities o

121 d synthesis may serve to regulate sequential strand displacement and flap cleavage at other genomic s

122 uccessfully design and construct complex DNA strand displacement circuits.

123 e formation of FEN1 cleavage products during strand displacement on a nontelomeric substrate, suggest

124 e resection of clean DSBs by cleaving the 5' strand DNA approximately 10-20 nucleotides away from the

125 ty of MRN apparently directly cleaves the 5' strand DNA at more distal sites.

126 It is suggested that for double-strand DNA breaks that have initially formed a complex w

127 ysis, we establish that leading- and lagging-strand DNA polymerases function independently within a s

128 as evidence for oxidized DNA lesions, double-strand DNA strand breaks, and pronounced susceptibility

129 or AdnAB) generates the requisite 3' single-strand DNA substrate for RecA-mediated strand invasion.

130 ion to abasic sites ahead of nascent lagging strand DNA synthesis and subsequent bypass by error-free

131 atin promotes the regular priming of lagging-strand DNA synthesis by facilitating DNA polymerase alph

132 rrelation between the release of host double-stranded DNA (dsDNA) following rhinovirus infection and

133 train encodes a 9.8- to 9.9-kb linear double-stranded DNA (dsDNA) genome with large inverted terminal

134 orm a nucleoprotein filament (NPF) on double-stranded DNA (dsDNA) that is capable of unwrapping the n

135 Human adenoviruses (Ad) are double-stranded DNA (dsDNA) viruses associated with infectious

136 The ability to directly detect double-stranded DNA (dsDNA) without sequence-preference continu

137 for pairing the ssDNA with homologous double-stranded DNA (dsDNA), which serves as the template to gu

138 died as models for viruses containing double-stranded DNA (polymer) and condensing proteins (particle

139 e, a protein that binds complementary single-stranded DNA (ssDNA) and facilitates its annealing to du

140 h the cell cytoplasm, and deliver the single-stranded DNA (ssDNA) genome to the nucleus, where viral

141 Single-stranded DNA (ssDNA) is notable for its interactions wit

142 and facilitates the immobilization of single stranded DNA (ssDNA) probe sequences on a wide variety o

143 by template-directed synthesis with a single-stranded DNA (ssDNA) topological structure.

144 omain) that binds efficiently to both double-stranded DNA and G-quadruplex (G4) DNA.

145 s insert into nuclear genomes through double-stranded DNA break repair.

146 h either event protects bacteria from double-stranded DNA breakage and TLD.

147 ired DNA lesions, such as single- and double-stranded DNA breaks (SSBs and DSBs), and single-stranded

148 Here we introduced double-stranded DNA breaks into the nuclear genome of tobacco t

149 that it does not require formation of double-stranded DNA breaks or provision of a donor DNA template

150 g homologous recombination, repair of double stranded DNA breaks, and integron recombination.

151 and Rad50 to coordinate the repair of double-stranded DNA breaks.

152 At these double-stranded DNA ends, RarA couples the energy of ATP bindin

153 separation and partitioning of large single-stranded DNA fragments of the homologous chromosome pair

154 (especially IgM, P < .0001), and anti-double-stranded DNA IgG (P < .05).

155 that utilize RNA to find and cut the double-stranded DNA molecules at specific locations.

156 at these contigs correspond to linear single-stranded DNA molecules that fold onto themselves to form

157 id (chloroplast) genomes are circular double-stranded DNA molecules, typically between 100 and 200 kb

158 ng, condensation, and pairing between double-stranded DNA molecules.

159 HCR is a hairpin-free system in which double-stranded DNA monomers could dendritically assemble into

160 Due to the linear double-stranded DNA nature of the adenovirus genome, the cellul

161 via HDR following co-delivery with a single-stranded DNA oligonucleotide.

162 Single-stranded DNA oligonucleotides have unique, and in some c

163 fusion constants of several different single-stranded DNA oligonucleotides trapped in an MspA nanopor

164 rvation leads to accumulation of both single-stranded DNA regions and intracellular ROS, and interfer

165 d MLPA products and subsequently to a single stranded DNA reporter probe bearing a HRP molecule, foll

166 to prime second strand synthesis of a single-stranded DNA template and generate millions of pair-wise

167 e-stranded RNA of the viral genome to double-stranded DNA that is then integrated into the DNA of the

168 of HIV-1 is reverse transcribed into double-stranded DNA that ultimately integrates into the host-ce

169 PORTANCE The papillomavirus (PV) is a double-stranded DNA tumor virus infecting cervix, mouth, and th

170 virus (ASFV) is a highly pathogenic, double-stranded DNA virus with a marked tropism for cells of th

171 Papillomaviruses are small, double-stranded DNA viruses that encode the E2 protein, which c

172 t the emergence of previously unknown double-stranded DNA viruses which delineate and extend the dive

173 is strongly conserved in the complex double-stranded DNA viruses, including the herpesviruses and ma

174 with specific sequences of unmodified single-stranded DNA, and we have identified five tags that reac

175 eferentially bind the minor groove of double-stranded DNA, inhibit vaccinia virus infection by blocki

176 teins, including the telomeric repeat single-stranded DNA-binding protein Teb1 and its heterotrimer p

177 ingle stranded DNA-ssDNA) and hybrid (double stranded DNA-dsDNA) both via 3-NT reduction and guanine

178 ze smFRET data of structurally rigid, double-stranded DNA-oligonucleotides in aqueous buffer and in b

179 ble to detect hybridization of probe (single stranded DNA-ssDNA) and hybrid (double stranded DNA-dsDN

180 s task, RAD51 must be loaded onto the single-stranded DNA.

181 s the translocation and processing of double-stranded DNA.

182 labeled with three distinct monolayer double-stranded (ds) DNA-gold nanoparticles (DNA-AuNPs).

183 and unbind from transiently exposed template strands during DNA synthesis.

184 capacity to govern DNA topology and resolve strand entanglements during fundamental molecular proces

185 dinally organized chromosome axes and stable strand exchange of crossover-designated DSBs.

186 ependent of RAD51, which encodes the central strand exchange protein in yeast required for conservati

187 s apparent, indicating telomerase-mediated G-strand extension.

188 olymerase I to successfully inhibit template strand extension.

189 s continuously, indicating a deficiency in C-strand fill-in synthesis.

190 The covalently connected strands folded into hyperstable collagen triple helices

191 Several different strand-folding topologies have been reported for Q-quadr

192 surfaces to allow the use of longer capture strands for detection of proteins.

193 diffuses in the cytoplasm to an active 4beta-stranded form bound to the membrane and MinD.

194 le conformational change from a latent 6beta-stranded form that diffuses in the cytoplasm to an activ

195 eta self-recognition sites spanning the beta strands found in cross-beta protofibril structures, lead

196 rmation of a heteroduplex, in which a single-strand from the broken DNA molecule pairs with a strand

197 nd an anti-parallel beta sheet consisting of strands from the top and bottom molecules, revealing the

198 sing nucleases, in order to close the single-stranded gap and terminate further resection.

199 anded DNA breaks (SSBs and DSBs), and single-stranded gaps can block progression of the DNA replicati

200 ter CTC1 disruption and at early times net G-strand growth is apparent, indicating telomerase-mediate

201 n of natural gas that is currently flared or stranded, has not yet been reported.

202 stream of the cleavage site as an extra beta-strand in a central beta-sheet.

203 xes formed by folding of GGG(TTAGGG)3 single strands in buffered solutions containing Na(+) cations (

204 n replication of the lagging and the leading strands in human cells, respectively.

205 resented 16% of total 5-phosphorylated guide strands in liver, correlating with a 2.7 log10 reduction

206 Second, it incorporates the strand information in the alignment step for more accura

207 Mismatch correction of strand invasion heteroduplex DNA is strongly polar, favo

208 ecA family of recombinases catalyzes the DNA strand invasion reaction that takes place during homolog

209 ingle-strand DNA substrate for RecA-mediated strand invasion.

210 late into the growing duplex as the template strand is copied into XNA.

211 of duplex DNA necessitates that the leading strand is replicated continuously whereas the lagging st

212 replicated continuously whereas the lagging strand is replicated in discrete segments known as Okaza

213 t, second conformation wherein the last beta-strand is retracted to extend the Ser65 loop and shorten

214 y preferentially occurs at telomeric lagging strands leading to heterogeneous telomere lengths observ

215 In contrast, C-strand length decreases continuously, indicating a defic

216 single-cell migration and the multicellular, strand-like invasion required for angiogenesis.

217 The lack of C-strand maintenance leads to gradual shortening of the te

218 T-strands may subsequently randomly integrate into plant c

219 We find that repair of genomic lagging strand mismatches occurs bi-directionally in E. coli and

220 NP concentration, PNA concentration, and DNA strand mismatches.

221 ctured electrode array and exposed to single-stranded MLPA products and subsequently to a single stra

222 U:G mismatches is translated into the single-strand nick required for error-prone synthesis is an ope

223 Genome-wide analysis of the transcribed strand/nontranscribed strand (TS/NTS) repair ratio demon

224 of flexible polyelectrolytes, such as single-stranded nucleic acids (ssNAs), is complicated by the in

225 The target strand of interest is biotinylated and detected by the G

226 yses a nucleophilic attack that connects one strand of the leader-proximal repeat to the prespacer 3'

227 NA and repair discontinuities on the lagging strand of the replication fork.

228 ATP binding and hydrolysis to separating the strands of duplex DNA, creating flaps.

229 of FOXO protein via CRISPR-assisted, single-stranded oligodeoxynucleotide-mediated homology-directed

230 ADAPT employs an enriched library of single-stranded oligodeoxynucleotides (ssODNs) to profile compl

231 nge of capturing structurally highly dynamic strands onto defined substrate positions.

232 ers to the effector units, e.g., an antifuel strand or 18-crown-6-ether, reconfiguration to the origi

233 The opposite strand polarity of duplex DNA necessitates that the lead

234 at DNA synthesis by the leading- and lagging-strand polymerases in the replisome must be coordinated

235 studies in mice indicated that ARC-520 guide strands predominantly accumulated in liver.

236 ization reaction is the alignment of the two strands prior to base pairing.

237 copied, and regenerated by release of single-stranded product DNA.

238 strand but an open plus strand (closed minus-strand RC DNA [cM-RC DNA]) was detected by this approach

239 unmethylated sequences generate local double stranded regions resulting to digestion of unmethylated

240 different aspects of RNA ranging from single stranded regions to discrete secondary or tertiary struc

241 ly sensitive detection of dsDNA based on the strand replacement of dsDNA by peptide nucleic acid (PNA

242 n some controversial features of the lagging-strand replication.

243 eotides downstream on the top and bottom DNA strands, respectively, in an NTP-hydrolysis dependent re

244 s reaching 21% and 11% in single- and double-strands, respectively.

245 dge base on the interaction between positive-strand RNA [(+)RNA] viruses and cellular membranes that

246 nal Herpesvirus Workshop (IHW), the Positive-Strand RNA Virus Symposium (PSR), and the Gordon Researc

247 is virus (WEEV) are arthropod-borne positive-strand RNA viruses that are capable of causing acute and

248 conserved protein fold found in several plus-strand RNA viruses that binds to the small molecule ADP-

249 Arenaviruses are enveloped negative-strand RNA viruses that cause significant human disease.

250 l host factor for many positive-sense single-stranded RNA (+RNA) viruses including human pathogens he

251 Long double-stranded RNA (dsRNA) can silence genes of matching seque

252 lly been used as a model to study the double-stranded RNA (dsRNA) Reoviridae family, the members of w

253 e an ExoN, which functions to degrade double-stranded RNA (dsRNA) replication intermediates.

254 In particular, NP associates with the double-stranded RNA (dsRNA)-activated protein kinase (PKR), a w

255 hat topological structures containing single-stranded RNA (ssRNA) free of strong base pairing interac

256 okine and type I IFN responses to the double-stranded RNA analogue poly(I:C) are reduced in mouse mac

257 tivity that act in concert to convert single-stranded RNA of the viral genome to double-stranded DNA

258 With Coxsackievirus B3 (CVB3) being a single-stranded RNA virus, and the recent evidence that the NOD

259 ependent RNA polymerases of diverse positive-stranded RNA viruses.

260 identified viruses are positive-sense single-stranded RNA viruses.

261 R-loop, a three-stranded RNA/DNA structure, has been linked to induced g

262 Double-stranded RNAs (dsRNA) produced during human cytomegalovi

263 It relies on plants stably expressing double-stranded RNAs (dsRNAs) that target essential genes in pe

264 -phosphate and 3'-hydroxyl termini of single-stranded RNAs.

265 es in the helices and on the surface of beta-strands S3, S9, and S10.

266 et site, and subsequent R-loop formation and strand scission are driven by complementary base pairing

267 Foremost, DNA strand separation by transcription or increased torsiona

268 average ratio of 29.2% by targeting both DNA strands simultaneously with an over 98.6% coverage.

269 high-confidence lncRNAs based on analysis of strand-specific RNA-seq data from cassava shoot apices a

270 c approach with gene expression based on the strand-specific RNA-seq data from seedling, floral bud,

271 E. coli single strand (ss) DNA binding protein (SSB) is an essential pr

272 RICE proteins specifically degraded single-strand (ss) RNAs in vitro; but neither miRNAs nor miRNA*

273 Guanine-rich DNAs can fold into four-stranded structures that contain stacks of G-quartets.

274 5hmU is enriched in strand switch regions, telomeric regions, and intergenic

275 the loss of telomeres replicated by lagging-strand synthesis by a yet to be defined mechanism.

276 erative interaction with FEN1 during lagging-strand synthesis may serve to regulate sequential strand

277 reviously-selected reagents, to prime second strand synthesis of a single-stranded DNA template and g

278 cific G-rich/G4 motif located on the lagging strand template for DNA replication and supports Pif1 fu

279 e-steering promotes an unanticipated lagging-strand template-switch mechanism during replication.

280 tially removes DNA lesions from the template strand that block translocation of RNA polymerase II (Po

281 ion allows helicases to bypass blocks on the strand that is excluded from the central channel.

282 d-coil dimers, which assemble into a helical strand that runs along the whole approximately 1 mum len

283 e exhibiting chiral symmetry, which form two strands that interleave along the lattice sites.

284 tices, with dye molecules coupled to the DNA strands that link the particles together, in the form of

285 to specific sites of the DNA particle-linker strands, thereby modulating dye-nanoparticle distance (t

286 capitulate that of epithelial tight junction strands, this is the first direct demonstration of the a

287 s local dipoles in each layer of the stacked strands to align in a parallel fashion.

288 ted histidine ligands, which bridge adjacent strands to form an infinite metal-ligand chain along the

289 uses the hybridization of complementary DNA strands to model the formation of the SNARE four-helix b

290 otegravir (GSK1265744) is an HIV-1 integrase strand transfer inhibitor with potent antiviral activity

291 ent treatment guidelines recommend integrase strand transfer inhibitors (INSTIs) as components of ini

292 sis of the transcribed strand/nontranscribed strand (TS/NTS) repair ratio demonstrated that deletion

293 Residual local motions at the edges of the strands, underscored by enhanced (15)N R1rho relaxation

294 ansferase reaction during extension of a DNA strand using the complementary strand as a template.

295 termine the presence of individual, labelled strands using gel electrophoresis.

296 we created site-specifically modified single-stranded vectors for replication in primate (COS7) or Es

297 plication of the leading and the lagging DNA strands were reported in yeast and in human cancers, but

298 The viruses, all positive sense single-stranded, were classified into diverse orders/families.

299 erential elongation of the telomeric lagging strands, whereas telomerase positive cells exhibited sim

300 long-noncoding (lnc)RNA in the complementary strand which has cis-regulatory transcriptional control