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1 esis of the hybrid polyketide/NRP antibiotic streptolydigin.
2 (AMPcPP), and with AMPcPP plus the inhibitor streptolydigin.
3 f bacterial RNA polymerase by the antibiotic streptolydigin.
4 ailed to show synergism with either aza-C or streptolydigin.
6 derivatives, rifabutin and rifapentine, and streptolydigin and sorangicin A, which are unrelated to
7 from that of elongation inhibitors, such as streptolydigin, and from the transcription initiation in
10 yses and molecular modeling, we identify the streptolydigin-binding region (Escherichia coli beta res
11 escribe the evolution of our approach to the streptolydigin class of antibiotics that target bacteria
12 bitors of protein synthesis was prevented by streptolydigin if added simultaneously, but was not reve
13 ive to either aza-C-induced DPC formation or streptolydigin, indicating that Mfd is not involved.
16 ructural model in which the NTP binds to the streptolydigin loop and upon pairing with the +1 DNA bas
18 ut not after inhibition of RNA elongation by streptolydigin or amino acid starvation of a stringent s
19 B (RNAP beta subunit) mutations that lead to streptolydigin resistance cause resistance to MccJ25.
22 n the synthesis and biological evaluation of streptolydigin, streptolydiginone, streptolic acid, and
24 rt, an ssrA mutant is also hypersensitive to streptolydigin, which blocks RNA polymerase elongation b
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