ライフサイエンスコーパス: streptomycin

1 veats associated with the common antibiotic, streptomycin.

2 t substantially affecting the sensitivity to streptomycin.

3 and high-level resistance to gentamicin and streptomycin.

4 red in a subset of mutants when treated with streptomycin.

5 ance to isoniazid, rifampicin, ofloxacin and streptomycin.

6 hich confers resistance to spectinomycin and streptomycin.

7 follicle growth in serum-free median without streptomycin.

8 ly, this mutation also confers resistance to streptomycin.

9 henicol, sulphamethoxazole, trimethoprim and streptomycin.

10 istance to the antibiotics spectinomycin and streptomycin.

11 retched cells that were exposed to 40 microM streptomycin.

12 tRNA genes (such as mutA) or by exposure to streptomycin.

13 monly used on plants are oxytetracycline and streptomycin.

14 se conformational changes in the presence of streptomycin.

15 istance to the antibiotics spectinomycin and streptomycin.

16 confers resistance to both spectinomycin and streptomycin.

17 thoxazole, trimethoprim, chloramphenicol and streptomycin.

18 (300 mg/day), and initial twice weekly (BIW) streptomycin.

19 owever, may be suboptimal for ethambutol and streptomycin.

20 00% for rifampin and ethambutol, and 95% for streptomycin.

21 TIW rifabutin (600 mg/dose), and initial BIW streptomycin.

22 increased to 93% for ethambutol and 100% for streptomycin.

23 uberculosis drugs, isoniazid, rifampicin and streptomycin.

24 ns of S. typhimurium after administration of streptomycin.

25 sensitivity of mycobacteria to rifampin and streptomycin.

26 Resistances to isoniazid (15.5%) and streptomycin (13.4%) were about twice as high as resista

27 atG S315T [isoniazid]; gidB 130 bp deletion [streptomycin]; 1957 [95% highest posterior density (HPD)

28 205 bp), pncA (pyrazinamide; 579 bp); rpsL (streptomycin; 196 bp), and embB (ethambutol; 185 bp).

29 ) were resistant to ampicillin, 649 (58%) to streptomycin, 402 (36%) to trimethoprim-sulfamethoxazole

30 stance most often to sulfamethoxazole (57%), streptomycin (56%), tetracycline (56%), ampicillin (52%)

31 Here we demonstrate that streptomycin, a bactericidal aminoglycoside that increas

32 Streptomycin, a blocker of SACs, had no effect upon the

33 In contrast, streptomycin, a nonspecific inhibitor of cationic MSCs,

34 mals received a 5 d course of treatment with streptomycin, a vestibulotoxic aminoglycoside.

35 ting hybrid RNA can then be immobilized on a streptomycin affinity matrix.

36 to the matrix, subsequent elution with free streptomycin allows efficient recovery of specific ribon

37 onses, as compared with animals treated with streptomycin alone.

38 Treatment with streptomycin altered the SCFAs in the cecum, significant

39 The aminoglycosides streptomycin, amikacin, and kanamycin and the cyclic pol

40 results for isoniazid, rifampin, ethambutol, streptomycin, amikacin, kanamycin, capreomycin, ofloxaci

41 Here, we show that exposure to streptomycin, an antibiotic known to promote mistranslat

42 SP204(1-1) was selected for resistance to streptomycin and 5-fluoro-2-deoxyuridine to distinguish

43 reas agreement was 93% for both rifampin and streptomycin and 90% for ethambutol.

44 se of Escherichia coli cells to penicillin G-streptomycin and cefazolin.

45 Other inhibitors of MSC such as streptomycin and GsTMx-4 also suppressed the expression

46 n products that increased resistance against streptomycin and kanamycin.

47 y, the rate of tRNA movement is increased by streptomycin and neomycin, decreased by tetracycline, an

48 eak strain acquired resistance to isoniazid, streptomycin and rifampicin by around 1973, indicating c

49 riplex, and differences in susceptibility to streptomycin and rifampin between this strain and M. tri

50 adenylation on 3''- and 9-hydroxyl groups of streptomycin and spectinomycin, respectively.

51 complexed with the antibiotics paromomycin, streptomycin and spectinomycin, which interfere with dec

52 analyses of an aggregated proteome caused by streptomycin and suggests that cellular defense against

53 f isolates that expressed resistance only to streptomycin and sulfamethoxazole (the StSu phenotype; 8

54 plasmid, encoding resistance to gentamicin, streptomycin and sulfonamides.

55 ptomycin, the Apt leaves the CS and binds to streptomycin and the Arch-shape structure is disassemble

56 antibiotics (erythromycin, tetracycline, and streptomycin) and whiB7 null mutants (Streptomyces and M

57 for strains resistant to both isoniazid and streptomycin, and 0.88 (95% CI, 0.53-1.47) for streptomy

58 rimethoprim-sulfamethoxazole, sulfisoxazole, streptomycin, and furazolidone increased from 7 (8%) of

59 h vancomycin or a combination of penicillin, streptomycin, and gentamicin (PSG) and then inoculated t

60 matched the purported source for ethambutol, streptomycin, and pyrazinamide.

61 ncing libraries, resistance to tetracycline, streptomycin, and sulfonamide/trimethoprim was assigned

62 resistance to the error-inducing antibiotic streptomycin, and the ribosomes from many such streptomy

63 ometry metabolomic features from a cohort of streptomycin- and rifampicin-resistant mutants grown in

64 ntly in white adipocytes (browning), whereas streptomycin antagonized TRPM8-mediated calcium entry, d

65 In contrast, the conversion of the streptomycin aptamer into functional riboswitches appear

66 In the presence of streptomycin, aptamer binds to its target and FAM-labele

67 In the absence of streptomycin, aptamer/FAM-labeled complementary strand d

68 by applying the approach to tetracycline and streptomycin aptamers.

69 In addition, pyranmycins, like streptomycin, are bacteriocidal while isoniazid (INH) is

70 c aptasensors were designed for detection of streptomycin based on aqueous gold nanoparticles (AuNPs)

71 ped for sensitive and selective detection of streptomycin, based on exonuclease I (Exo I), compliment

72 Mice treated with oral streptomycin before oral administration of MRSA develope

73 anged the electrochemical gradient such that streptomycin better accessed the cytoplasm.

74 The aminoglycosides paromomycin and streptomycin bind to the decoding center and induce rela

75 The streptomycin-binding aptamer, termed 'StreptoTag,' is em

76 of M. tuberculosis-infected A549 cells with streptomycin, but not cycloheximide, demonstrated a sign

77 In fura 2-loaded myocytes, HOE 642 and streptomycin, but not L-NAME, ablated the stretch-induce

78 mice treated perinatally with vancomycin or streptomycin by repeated intranasal administration of Sa

79 Our application to aptamers for streptomycin, chloramphenicol, neomycin B and ATP identi

80 A-site oligonucleotide, and paromomycin and streptomycin complexed to the 30S subunit was used as a

81 ntrast, mice whose water contained 5 g/liter streptomycin consistently became colonized at high level

82 A genetic system based on rpsL-streptomycin counter selection was developed to further

83 dentified mutations in 16S rRNA conferring a streptomycin dependence phenotype and from these derived

84 The structural basis for the streptomycin dependence phenotype of ribosomal protein S

85 Spontaneous suppressors of streptomycin dependence were also readily isolated.

86 ding ribosomal protein S4), which suppressed streptomycin dependence, were able to partially restore

87 isolated spontaneous streptomycin-resistant, streptomycin-dependent and streptomycin-pseudo-dependent

88 resistant, streptomycin-pseudo-dependent and streptomycin-dependent mutants described here is expecte

89 The streptomycin-dependent rpsL(P90K) mutant also showed sig

90 Streptomycin did not inhibit A-site cleavage in rpsL mut

91 Animals treated with streptomycin displayed vestibular system impairment as m

92 The aminoglycoside antibiotic streptomycin disrupts decoding by binding close to the s

93 These data reveal how streptomycin disrupts the recognition of cognate anticod

94 chanosensitive channel blocker gadolinium or streptomycin dramatically increased longitudinal conduct

95 %) accurate results for isoniazid, rifampin, streptomycin, ethambutol, amikacin, kanamycin, capreomyc

96 this study, we showed that 4- to 6-week-old streptomycin-fed C57BL/6 mice were susceptible to intest

97 cide vector carrying wild-type rpsL and used streptomycin for counterselection.

98 ary metabolites (penicillin, cephalosporins, streptomycin, fosfomycin, gramicidin S, rapamycin, indol

99 The discovery of Streptomyces-produced streptomycin founded the age of tuberculosis therapy.

100 channels with the aminoglycoside antibiotic streptomycin from onset of disease in the mdx mouse mode

101 s such as chloramphenicol, tetracycline, and streptomycin gather predominantly at the cell poles and

102 Prompt treatment with streptomycin, gentamicin, doxycycline, or ciprofloxacin

103 on Candida growth in optisol-gentamicin and streptomycin (GS) with and without antifungal supplement

104 current systemic administration of zVAD with streptomycin had both significantly greater hair cell su

105 Streptomycin has been demonstrated to inhibit mechanosen

106 that confers resistance to spectinomycin and streptomycin, has been considered critical for recovery

107 ette confers resistance to spectinomycin and streptomycin in both B. burgdorferi and Escherichia coli

108 response in the presence of the SAC blocker streptomycin in both muscle (80 microm) and myocytes (40

109 nce to the aminoglycosides spectinomycin and streptomycin in Escherchia coli, can be introduced into

110 l aptasensor was successfully used to detect streptomycin in milk and serum with LODs of 14.1 and 15.

111 aptasensors were successfully used to detect streptomycin in milk and serum.

112 be very effective for low-level detection of streptomycin in milk samples.

113 search Council (1948) study of the effect of streptomycin in the treatment of tuberculosis.

114 However, mice given streptomycin in their drinking water developed long-term

115 l distortion of 16S ribosomal RNA induced by streptomycin, including the crucial bases A1492 and A149

116 Using experimental evolution of streptomycin-independent pseudorevertants, we identified

117 d tobramycin, as compared to paromomycin and streptomycin, indicates differences in the efficacy of t

118 n the study of this antibiotic, a mysterious streptomycin-induced potassium efflux preceding any decr

119 only necessary for the previously described streptomycin-induced potassium efflux, but also directly

120 ate that the aminoglycosides paromomycin and streptomycin inhibit A-site cleavage of stop codons duri

121 These data suggest that streptomycin inhibits myogenic tone and K+-induced isome

122 In conclusion, streptomycin is not useful in the investigation of stret

123 The antibiotic streptomycin is widely used in the treatment of microbia

124 biotic susceptibility testing with rifampin, streptomycin, isoniazid, and ethambutol were compared to

125 nce or borderline resistance to at least one streptomycin-isoniazid-rifampin-ethambutol drug or PZA.

126 glycoside antibiotics, including gentamicin, streptomycin, kanamycin, amikacin, and paromomycin, have

127 eginning with sulfanilamide, penicillin, and streptomycin, led to additional strategies for managing

128 ver, substitutions that confer resistance to streptomycin likely represent a very distinct class of a

129 Transfer of the streptomycin marker was independent of the H. pylori com

130 um produced C. jejuni progeny containing the streptomycin marker.

131 Mice received 2 mg of streptomycin/ml of drinking water and 1,500 U of penicil

132 For kanamycin and streptomycin, molecular DST significantly outperformed p

133 ere, we used a toeprinting assay to how that streptomycin, neomycin, kanamycin, gentamycin, and hygro

134 etection for ofloxacin, chloramphenicol, and streptomycin of 0.3, 0.12, and 0.2 ng mL(-1), respective

135 ation, as transient selective suppression by streptomycin of bacterial growth in the intestine delaye

136 The action of streptomycin on rat cerebral arteries that exhibit press

137 -ray crystallography to define the impact of streptomycin on the decoding site of the Thermus thermop

138 hree antibiotics--neomycin, polymyxin B, and streptomycin--on diabetes development.

139 Early treatment and prophylaxis with streptomycin or gentamicin or the tetracycline or fluoro

140 contained aadA, which confers resistance to streptomycin, or aadA and dhfr, which confer resistance

141 ltured in media supplemented with penicillin-streptomycin (PenStrep) or vehicle control.

142 was similarly inhibited by 1.0 microgram of streptomycin per ml in fibroblast-conditioned tissue cul

143 methylthiolation is not a determinant of the streptomycin phenotype.

144 Experiments with streptomycin-pretreated caspase-1-deficient mice suggest

145 n acute inflammatory reaction in the ceca of streptomycin-pretreated mice that involves T-cell-depend

146 inflammation within 48 h after infection of streptomycin-pretreated mice, and the presence of the vi

147 n acute inflammatory reaction in the ceca of streptomycin-pretreated mice.

148 flammation caused by serotype Typhimurium in streptomycin-pretreated mice.

149 colonization of the ileum and caecum in the streptomycin-pretreated mouse model of colitis.

150 the bovine ligated ileal loop model and the streptomycin-pretreated mouse model, suggesting that thi

151 to elicit acute intestinal inflammation in a streptomycin-pretreated mouse model.

152 ating these inflammatory responses using the streptomycin-pretreated mouse model.

153 ot elicit marked inflammatory changes in the streptomycin-pretreated mouse model.

154 arly after serotype Typhimurium infection of streptomycin-pretreated wild-type mice regardless of fla

155 C57BL/6 mice were pretreated with streptomycin prior to receiving MET-1 or control, then g

156 -butyrolactone A-factor and consequently for streptomycin production in Streptomyces griseus).

157 to actII-ORF4 that is the final regulator of streptomycin production in Streptomyces griseus.

158 related, and suggests that the regulation of streptomycin production, which serves an important parad

159 at perinatal exposure to vancomycin, but not streptomycin, profoundly alters gut microbiota and enhan

160 is of ribosomes from streptomycin-resistant, streptomycin-pseudo-dependent and streptomycin-dependent

161 omycin-resistant, streptomycin-dependent and streptomycin-pseudo-dependent mutants of the thermophili

162 A good co-relation was observed between the streptomycin recoveries measured through the developed b

163 e, the miscoding antibiotics paromomycin and streptomycin rescue the defects in tRNA selection with t

164 an ultrasensitive biosensor for detection of streptomycin residues in milk samples using flow injecti

165 PCR results) were as follows: for high-level streptomycin resistance (HLSR), QA (12.0%) > PM-8 (5.2%)

166 neous mutants by the flanking, cotransformed streptomycin resistance and Pst I markers.

167 We propose a novel mechanism of streptomycin resistance by which A1408G influences confo

168 onitor the transfer, a chromosomally encoded streptomycin resistance cassette prearranged by a specif

169 rain 5051, was constructed by insertion of a streptomycin resistance gene cassette.

170 iazid, kanamycin, ofloxacin, rifampicin, and streptomycin resistance in Mycobacterium tuberculosis, a

171 ated that mutations conferring isoniazid and streptomycin resistance in this clone were acquired 50 y

172 allele of oppD interrupted by kanamycin and streptomycin resistance markers.

173 ance mutation is flanked on the 5' side by a streptomycin resistance mutation and on the 3' side by a

174 markers and heteroplasmic for the unselected streptomycin resistance trait.

175 he results for ethambutol, pyrazinamide, and streptomycin resistance were more variable.

176 cin resistance), strA and strB (which encode streptomycin resistance), class B tetA (which encodes te

177 ctor DNA and selection for spectinomycin and streptomycin resistance, respectively.

178 he frequency of nitrite-induced mutations to streptomycin resistance, which are known to occur in A x

179 sistance and 96% when testing for high-level streptomycin resistance.

180 and a 1-kb class I integron bearing aadA for streptomycin resistance.

181 However, the most common vehicles of streptomycin-resistance genes in human and plant pathoge

182 A fraction of streptomycin-resistance genes in plant-associated bacter

183 TB activity against rifampin, isoniazid, and streptomycin resistant Mtb strains.

184 osomes isolated from strains that are either streptomycin resistant or dependent for growth do not sh

185 e virG(icsA) deletion was constructed from a streptomycin-resistant (Str(r)) mutant of 1617 by a filt

186 mutations could be evolved from slow-growing streptomycin-resistant mutants.

187 A spontaneous streptomycin-resistant rpsL mutant of F. johnsoniae was

188 We propose that tmRNA.SmpB binds to streptomycin-resistant rpsL ribosomes less efficiently,

189 f rifampin-resistant S. gordonii DLl than of streptomycin-resistant S. gordonii SK12 from the hearts

190 , all animals were challenged with 5 x 10(6) streptomycin-resistant S. mutans strain SJ-r organisms.

191 reptomycin, and the ribosomes from many such streptomycin-resistant S12 mutants display decreased lev

192 A novel streptomycin-resistant strain of NTHi was developed, and

193 xytetracycline is rare, but the emergence of streptomycin-resistant strains of Erwinia amylovora, Pse

194 Whereas streptomycin-resistant strains were more commonly found

195 Testing additional ethambutol-resistant and streptomycin-resistant strains with two concentrations o

196 reptomycin, and 0.88 (95% CI, 0.53-1.47) for streptomycin-resistant strains.

197 ion along the entire intestinal tract by the streptomycin-resistant V. parahaemolyticus.

198 site cleavage in rpsL mutants, which express streptomycin-resistant variants of ribosomal protein S12

199 NA sequencing studies with low-fitness rpsL (streptomycin-resistant) mutants of E. coli with and with

200 We have isolated spontaneous streptomycin-resistant, streptomycin-dependent and strep

201 crystallographic analysis of ribosomes from streptomycin-resistant, streptomycin-pseudo-dependent an

202 had high-level resistance to gentamicin and streptomycin, respectively, including 10% that were resi

203 that treatment with the ototoxic antibiotic streptomycin results in a nearly complete elimination of

204 isolates yielding discrepant ethambutol and streptomycin results with a lower concentration of both

205 Streptomycin reversibly and concentration-dependently in

206 tion of wild-type rpsL on a plasmid restored streptomycin sensitivity, demonstrating that wild-type r

207 against isoniazid (INH), rifampin (RMP), and streptomycin (SM) resistant Mtb strains.

208 ultaneous, sensitive and visual detection of streptomycin (SM), tetracycline (TC), and penicillin G (

209 gs was associated with four-drug resistance (streptomycin, spectinomycin, sulfisoxazole, and tetracyc

210 Excellent recoveries were obtained from the streptomycin spiked milk samples in the range 98-99.33%,

211 onsistent with kinetic data, we observe that streptomycin stabilizes the near-cognate anticodon stem-

212 hat of rifampicin against the nonreplicating streptomycin-starved M. tuberculosis 18b-Lux strain, and

213 Ferrets treated with streptomycin (Stm) had higher counts of E. coli O157:H7

214 ned for selective and sensitive detection of streptomycin (STR) based on Exonuclease III (Exo III), S

215 rifampin, isoniazid (INH), pyrazinamide, or streptomycin (STR), 4 were resistant to STR and INH, and

216 n specific Asp-->Gly misreading, we asked if streptomycin (Str), an aminoglycoside antibiotic known t

217 Streptomycin (STR), isoniazid (INH), rifampin (RIF), eth

218 For the second-line drug streptomycin (STR), overall concordance between the agar

219 the activatable Stx2d are more virulent in a streptomycin (str)-treated mouse model of infection [let

220 dfrA12, and dfrA15], amoxicillin [bla(TEM)], streptomycin [strA-strB], chloramphenicol [cat-1], and e

221 Upon treatment with streptomycin, substantial changes in the microbial compo

222 ond was resistance to ampicillin, kanamycin, streptomycin, sulfamethoxazole, and tetracycline (the Am

223 s resistance to ampicillin, chloramphenicol, streptomycin, sulfamethoxazole, and tetracycline (the Am

224 istinct resistance to ampicillin, kanamycin, streptomycin, sulfamethoxazole, and tetracycline and to

225 acycline and to ampicillin, chloramphenicol, streptomycin, sulfamethoxazole, and tetracycline, respec

226 re resistant to ampicillin, chloramphenicol, streptomycin, sulfamethoxazole, and tetracycline.

227 g resistance to ampicillin, chloramphenicol, streptomycin, sulfamethoxazole-sulfisoxazole, and tetrac

228 uginosa as an agent conferring resistance to streptomycin, sulfanilamide, gentamicin, and carbenicill

229 re resistant to ampicillin, chloramphenicol, streptomycin, sulfisoxazole, and tetracycline.

230 ampicillin, chloramphenicol, nalidixic acid, streptomycin, sulfisoxazole, tetracycline, and trimethop

231 is resistant to ampicillin, chloramphenicol, streptomycin, sulfonamides, and tetracycline and has bec

232 nce to 5 drugs (ampicillin, chloramphenicol, streptomycin, sulfonamides, and tetracycline), has emerg

233 s resistance to ampicillin, chloramphenicol, streptomycin, sulfonamides, and tetracycline.

234 percent of the strains showed resistance to streptomycin, sulfonamides, or tetracycline only; all th

235 Levels of agreement for streptomycin tested at 2.0 and 6.0 micro g/ml were 77 an

236 complex antibiotics from nature: penicillin, streptomycin, tetracycline, and erythromycin, among othe

237 e critical concentrations for ethambutol and streptomycin that accurately detect susceptibility to th

238 n of ribosome inhibitors (chloramphenicol or streptomycin) that indirectly constrain transcription ra

239 In the absence of streptomycin the gate remains closed.

240 Upon addition of streptomycin, the Apt leaves the CS and binds to strepto

241 antimicrobials, including crystal violet and streptomycin (this phenotype could also be complemented)

242 ized model in which mice are pretreated with streptomycin to result in gut-restricted acute enteritis

243 which were also treated with an antibiotic (streptomycin) to knockdown the gut microbiota.

244 We demonstrated that streptomycin-treated adult C57BL/6 mice experienced prol

245 ns and in gastrointestinal colonization of a streptomycin-treated adult mouse.

246 detes dominated the intestinal microbiota of streptomycin-treated animals, while vancomycin promoted

247 an O157:H7 strain, is known to colonize the streptomycin-treated CD-1 mouse intestine by growing in

248 ds (SCFAs) present in the ileum and cecum of streptomycin-treated mice and untreated controls.

249 42 initially grew in the large intestines of streptomycin-treated mice but then failed to colonize (<

250 Neither conventional nor streptomycin-treated mice developed clinical signs or hi

251 Here we report that to colonize streptomycin-treated mice E. coli HS consumes six of the

252 ts exclusively to colonize the intestines of streptomycin-treated mice when it is the only E. coli st

253 Infection of streptomycin-treated mice with Salmonella was characteri

254 oth bacterial genetic backgrounds was fed to streptomycin-treated mice, together with the respective

255 Using streptomycin-treated mice, we were able to establish sub

256 creased spleen colonization by Salmonella in streptomycin-treated mice, with more of the pat mutant r

257 ve EAEC virulence factors to colonization of streptomycin-treated mice.

258 c conditions in cecal contents of saline- or streptomycin-treated mice.

259 In contrast, no colonization occurred in non-streptomycin-treated mice.

260 Previously we reported that the streptomycin-treated mouse intestine selected for two di

261 Previously, we reported that the streptomycin-treated mouse intestine selected nonmotile

262 rain E. coli EDL933 colonizes a niche in the streptomycin-treated mouse intestine that is distinct fr

263 strain MG1655, following passage through the streptomycin-treated mouse intestine, that has colonizat

264 t they appear to use different sugars in the streptomycin-treated mouse intestine.

265 bacterial biofilms in the mucus layer of the streptomycin-treated mouse intestine.

266 d as being important for colonization of the streptomycin-treated mouse large intestine by Escherichi

267 de that E. coli is the only bacterium in the streptomycin-treated mouse large intestine that respires

268 , there was no defect in colonization of the streptomycin-treated mouse model by the gadX mutant in c

269 Studies with the streptomycin-treated mouse model have produced important

270 te, and examined the effects in vivo using a streptomycin-treated mouse model of colonization.

271 pathogens, and murine models, including the streptomycin-treated mouse model of infection, are frequ

272 ne the role of respiratory metabolism in the streptomycin-treated mouse model of intestinal colonizat

273 Stx2d1-producing mutant was attenuated in a streptomycin-treated mouse model of STEC infection.

274 In the streptomycin-treated mouse model, more animals infected

275 In a streptomycin-treated mouse model, parenteral administrat

276 By using the streptomycin-treated mouse model, we found that cataboli

277 By using the streptomycin-treated mouse model, we found that Taconic

278 erobic respiration in colonization using the streptomycin-treated mouse model.

279 suppressed aggregated protein formation upon streptomycin treatment and increased aminoglycoside resi

280 Here we show that streptomycin treatment depleted commensal, butyrate-prod

281 However, streptomycin treatment did not show positive effects in

282 pansion were also observed in the absence of streptomycin treatment in genetically resistant mice but

283 Prenatal neomycin, polymyxin B, and streptomycin treatment protected NOD mice from diabetes

284 ock regulon was induced only by puromycin or streptomycin treatment, which lead to truncation or mist

285 in the mouse and the changes that occur upon streptomycin treatment.

286 ificantly increased hair cell survival after streptomycin treatment.

287 vancomycin, but increased dramatically after streptomycin treatment.

288 The streptomycin trial in the United Kingdom in the 1940s in

289 e demonstrations in clinical trials that (1) streptomycin was effective; (2) combination of drugs pre

290 ates to isoniazid, rifampin, ethambutol, and streptomycin was evaluated by comparing results to those

291 Monoclonal antibody specific to streptomycin was immobilized on to the thiol modified go

292 ptibility testing results for ethambutol and streptomycin was poor.

293 The binding of streptomycin was too weak to assess.

294 , conferring resistance to spectinomycin and streptomycin, was associated with an OXA-23 carbapenemas

295 nate, immune response because the effects of streptomycin were not observed during the early phases o

296 s to be highly specific to spectinomycin and streptomycin, while the acetyltransferase domain shows a

297 namic range (0.3-300 ng/mL) was obtained for streptomycin with a good linearity in the range 0.3-10 n

298 aptasensor exhibited high selectivity toward streptomycin with a limit of detection (LOD) as low as 1

299 y and concentration-dependently inhibited by streptomycin with an IC50 of 2.6 mM.

300 asensors showed excellent selectivity toward streptomycin with limit of detections as low as 73.1 and